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1. Breastfeeding and human lactation
by Wambach, Karen, editor and Riordan, Jan, editor
2015, Fifth edition., ISBN 1449697283, xx, 966 pages
Infant Nutritional Physiological Phenomena | Lactation | Milk, Human | Breast Feeding
Book
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2. Journal of mammary gland biology and neoplasia
Journal of mammary gland biology and neoplasia, ISSN 0183-3021, 1996
Breast | Mammary glands | Physiology | Cancer | Tumors | Diseases
Journal
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3. Full Text Breastfeeding and maternal and infant health outcomes in developed countries
by Ip, Stanley and Tufts-New England Medical Center. Evidence-based Practice Center and United States. Agency for Healthcare Research and Quality
2007, Evidence report/technology assessment, Volume no. 153
Developed countries | Infants | Nutrition | Breastfeeding | Health aspects
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4. HIV transmission through breastfeeding: a review of available evidence
by Newell, Marie-Louise and World Health Organization (WHO) and United Nations . Joint Programme on HIV/AIDS (UNAIDS) and United Nations . Population Fund (UNFPA) and United Nations . Children's Fund (UNICEF)
2004, ISBN 9241562714, 25
AIDS (Disease) in infants -- Prevention | Infants -- Health and hygiene | Breastfeeding -- Health aspects | Infants -- Nutrition | AIDS (Disease) -- Transmission
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5. Full Text Estrogen regulates Hippo signaling via GPER in breast cancer
by Zhou, Xin and Wang, Shuyang and Wang, Zhen and Feng, Xu and Liu, Peng and Lv, Xian-Bo and Li, Fulong and Yu, Fa-Xing and Sun, Yiping and Yuan, Haixin and Zhu, Hongguang and Xiong, Yue and Lei, Qun-Ying and Guan, Kun-Liang
Journal of Clinical Investigation, ISSN 0021-9738, 05/2015, Volume 125, Issue 5, pp. 2123 - 2135
The G protein coupled estrogen receptor (GPER) mediates both the genomic and nongenomic effects of estrogen and has been implicated in breast cancer... 
MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | DRUG-THERAPY | ACTIVATION | PATHWAY | GROWTH | GPR30 | PROTEIN-COUPLED RECEPTORS | SIZE-CONTROL | TAZ | 17-BETA-ESTRADIOL | Carcinoma, Ductal, Breast - genetics | Neoplasms, Hormone-Dependent - metabolism | Protein-Serine-Threonine Kinases - analysis | Phosphorylation | Estrogens - pharmacology | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Neoplasms, Hormone-Dependent - physiopathology | Breast Neoplasms - physiopathology | Breast Neoplasms - metabolism | Neoplasms, Hormone-Dependent - genetics | RNA Interference | Phospholipase C beta - physiology | Cell Division | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Female | Transcription, Genetic | Phosphoproteins - physiology | Receptors, G-Protein-Coupled - drug effects | Carcinoma, Ductal, Breast - metabolism | Protein-Serine-Threonine Kinases - metabolism | Receptors, G-Protein-Coupled - physiology | Estrogens - physiology | GTP-Binding Protein alpha Subunits, Gq-G11 - genetics | rho-Associated Kinases - physiology | Transcription Factors - physiology | Protein Kinase C - physiology | Carcinoma, Ductal, Breast - physiopathology | Protein-Serine-Threonine Kinases - physiology | GTP-Binding Protein alpha Subunits, Gq-G11 - antagonists & inhibitors | GTP-Binding Protein alpha Subunits, Gq-G11 - physiology | Tumor Suppressor Proteins - physiology | Adaptor Proteins, Signal Transducing - physiology | Receptors, Estrogen - drug effects | Animals | Breast Neoplasms - genetics | Cell Transformation, Neoplastic | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | Protein Processing, Post-Translational | Cell Movement | Receptors, Estrogen - physiology | Tumor Suppressor Proteins - analysis | Cell receptors | Estrogen | Physiological aspects | Development and progression | Breast cancer | Cellular signal transduction | Research | Studies | Kinases | Rodents | Index Medicus | Abridged Index Medicus | Oncology
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6. Infant and child nutrition worldwide
: issues and perspectives
by Falkner, Frank T., 1918-2003
1991, ISBN 9780849388149, xiv, 297, iii
Infant formulas | Child Nutrition | Nutrition | Breast Feeding | Infant Food | Infant Nutrition | Infants | Children | Breastfeeding | Social aspects | Health aspects
Book
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7. The breast: morphology, physiology, and lactation
by Vorherr, Helmuth
1974, ISBN 9780127280509, x, 282
Lactation | Breast | Diseases
Book
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8. Physiology of mammary glands
by Yokoyama, Akira and Mizuno, Hideo and Nagasawa, Hiroshi
1978, ISBN 083911303X, x, 387
Mammary glands | Glandes mammaires
Book
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9. Lactation
: physiology, nutrition, and breast-feeding
by Neville, Margaret C and Neifert, Marianne R
1983, ISBN 9780306413117, xvi, 466
Lactation | Breastfeeding | Infants -- Nutrition
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10. Full Text Cooperation of breast cancer proteins PALB2 and piccolo BRCA2 in stimulating homologous recombination
by Masson, Jean-Yves and Cai, Hong and Coulombe, Yan and Stasiak, Andrzej and Dion-Côté, Anne-Marie and Launay, Hélène and Xia, Bing and Buisson, Rémi and Stasiak, Alicja Z
Nature Structural & Molecular Biology, ISSN 1545-9993, 10/2010, Volume 17, Issue 10, pp. 1247 - 1254
Inherited mutations in human PALB2 are associated with a predisposition to breast and pancreatic cancers. PALB2's tumor-suppressing effect is thought to be... 
POLY(ADP-RIBOSE) POLYMERASE | COMPLEX | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOUBLE-STRAND BREAKS | HISTONE H2AX | FANCONI-ANEMIA | CELL BIOLOGY | RAD51 | BIOPHYSICS | IN-VIVO | SUSCEPTIBILITY GENE | D-LOOP FORMATION | DNA-REPAIR | Recombination, Genetic - physiology | DNA, Neoplasm - metabolism | Humans | Neoplasm Proteins - physiology | DNA Repair - physiology | Molecular Sequence Data | Structure-Activity Relationship | DNA Breaks, Double-Stranded | BRCA2 Protein - physiology | Breast Neoplasms - metabolism | Base Sequence | Tumor Suppressor Proteins - chemistry | Tumor Suppressor Proteins - genetics | Female | Protein Interaction Domains and Motifs | Nuclear Proteins - genetics | Nucleic Acid Conformation | Fanconi Anemia Complementation Group N Protein | Peptide Fragments - metabolism | Neoplasm Proteins - chemistry | Nuclear Proteins - chemistry | Poly(ADP-ribose) Polymerase Inhibitors | Protein Interaction Mapping | Tumor Suppressor Proteins - physiology | Peptide Fragments - chemistry | Apoptosis Regulatory Proteins | Models, Biological | Rad51 Recombinase - chemistry | Rad51 Recombinase - physiology | BRCA2 Protein - chemistry | Nuclear Proteins - physiology | Poly (ADP-Ribose) Polymerase-1 | Breast cancer | Genetic aspects | Research | BRCA mutations | Ovarian cancer | Proteins | Mutation | Molecular biology | Prostate cancer | Index Medicus | homologous recombination | BRCA2 | PALB2
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11. Full Text A Nonapoptotic Cell Death Process, Entosis, that Occurs by Cell-in-Cell Invasion
by Overholtzer, Michael and Mailleux, Arnaud A and Mouneimne, Ghassan and Normand, Guillaume and Schnitt, Stuart J and King, Randall W and Cibas, Edmund S and Brugge, Joan S
Cell, ISSN 0092-8674, 2007, Volume 131, Issue 5, pp. 966 - 979
Epithelial cells require attachment to extracellular matrix (ECM) to suppress an apoptotic cell death program termed anoikis. Here we describe a nonapoptotic... 
CELLBIO | HUMDISEASE | APOPTOSIS | LUNG | ACTIVATION | DISRUPTION | CONTRACTION | CANNIBALISM | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | CARCINOMA | PHOSPHATIDYLSERINE | CELL BIOLOGY | rho-Associated Kinases - physiology | Myosin Type II - physiology | Humans | Extracellular Matrix - metabolism | Cell Communication - physiology | Autophagy - physiology | Cadherins - physiology | rho GTP-Binding Proteins - physiology | Lysosomes - metabolism | Adherens Junctions - physiology | Breast Neoplasms - pathology | Cell Adhesion - physiology | Models, Biological | Apoptosis - physiology | Tumor Cells, Cultured | Carcinoma - pathology | Actins - physiology | Cell death | Biochemistry | Index Medicus
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12. Full Text Serotonergic regulation of melanocyte conversion: A bioelectrically regulated network for stochastic all-or-none hyperpigmentation
by Lobikin, Maria and Lobo, Daniel and Blackiston, Douglas J and Martyniuk, Christopher J and Tkachenko, Elizabeth and Levin, Michael
Science Signaling, ISSN 1945-0877, 10/2015, Volume 8, Issue 397, pp. ra99 - ra99
Experimentally induced depolarization of resting membrane potential in "instructor cells" in Xenopus laevis embryos causes hyperpigmentation in an all-or-none... 
EPITHELIAL-MESENCHYMAL TRANSITION | CELL FATE DECISION | BREAST-CANCER | STEM-CELLS | DOUBLE-NEGATIVE FEEDBACK | POTASSIUM CHANNELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | POSITIVE-FEEDBACK | NEURAL CREST | MOLECULAR BIOELECTRICITY | TARGETING ION CHANNELS | CELL BIOLOGY | Serotonin - physiology | SOXE Transcription Factors - physiology | Machine Learning | Stochastic Processes | Gene Expression Regulation, Developmental | Melanocytes - cytology | Cell Shape | Computer Simulation | Skin Pigmentation - physiology | Transcription, Genetic | Cyclic AMP - metabolism | Membrane Potentials - drug effects | Transcription Factors - physiology | Xenopus laevis - embryology | Ivermectin - pharmacology | Melanocytes - physiology | CREB-Binding Protein - physiology | Cyclic AMP-Dependent Protein Kinases - physiology | Hyperpigmentation - physiopathology | Animals | Models, Biological | Xenopus Proteins - physiology | Signal Transduction - physiology | Larva - physiology | Xenopus laevis - physiology | Cell Movement | Pituitary Gland - physiology | Index Medicus
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13. Full Text Interacting with human physiology
by Pavlidis, I and Dowdall, J and Sun, N and Puri, C and Fei, J and Garbey, M
Computer Vision and Image Understanding, ISSN 1077-3142, 2007, Volume 108, Issue 1, pp. 150 - 170
We propose a novel system that incorporates physiological monitoring as part of the human–computer interface. The sensing element is a thermal camera that is... 
Facial tracking | Sleep apnea | Thermal imaging | Cardiac pulse | Stress | Human–computer interaction | Blood flow | Breath rate | Human-computer interaction | stress | DIAGNOSIS | facial tracking | USER | thermal imaging | COMPUTER SCIENCE, ARTIFICIAL INTELLIGENCE | ENGINEERING, ELECTRICAL & ELECTRONIC | BREAST-CANCER | human-computer interaction | TEMPERATURE | COMPUTER | breath rate | blood flow | sleep apnea | FACE | cardiac pulse
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14. Full Text miR‐124 inhibits cell proliferation in gastric cancer through down‐regulation of SPHK1
by Xia, Jintang and Wu, Zhiqiang and Yu, Chunping and He, Weiling and Zheng, Haiqing and He, Yulong and Jian, Weihua and Chen, Lianzhou and Zhang, Luanjing and Li, Wen
The Journal of Pathology, ISSN 0022-3417, 08/2012, Volume 227, Issue 4, pp. 470 - 480
SPHK1 expression is elevated in gastric cancer and is associated with shorter survival times for patients. However, the molecular mechanism of SPHK1... 
miR‐124 | gastric cancer | proliferation | SPHK1 | FOXO1 | miR-124 | MICRORNA GENES | SPHINGOSINE KINASE 1 | TUMOR SUPPRESSION | AKT | PATHOLOGY | FORKHEAD TRANSCRIPTION FACTORS | BREAST-CANCER | ONCOLOGY | PROGNOSTIC MARKER | DEGRADATION | UP-REGULATION | PROGRESSION | Phosphotransferases (Alcohol Group Acceptor) - physiology | Adenocarcinoma - pathology | Cell Proliferation | Humans | Cyclin-Dependent Kinase Inhibitor p27 - physiology | Stomach Neoplasms - pathology | Forkhead Transcription Factors - physiology | Proto-Oncogene Proteins c-akt - physiology | Stomach Neoplasms - physiopathology | Down-Regulation - physiology | Adenocarcinoma - physiopathology | Cell Transformation, Neoplastic | Cell Cycle - physiology | Cell Line, Tumor | Signal Transduction - physiology | Cyclin-Dependent Kinase Inhibitor p21 - physiology | Forkhead Box Protein O1 | MicroRNAs - physiology | In Vitro Techniques | Index Medicus
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15. Full Text Genome and Hormones: Gender Differences in Physiology: Invited Review: Cell localization, physiology, and nongenomic actions of estrogen receptors
by Levin, Ellis R
Journal of Applied Physiology, ISSN 8750-7587, 10/2001, Volume 91, Issue 4, pp. 1860 - 1867
Division of Endocrinology, Veterans Affairs Medical Center, Long Beach, Long Beach 90822; and Departments of Medicine and Pharmacology, University of... 
SPORT SCIENCES | PHYSIOLOGY | ACTIVATED PROTEIN-KINASE | LIGAND-BINDING | cell proliferation | HUMAN-BREAST-CANCER | ER-BETA | signal transduction | NITRIC-OXIDE SYNTHASE | apoptosis | steroid receptors | EPIDERMAL GROWTH-FACTOR | PLASMA-MEMBRANE | nuclear receptors | TARGETED DISRUPTION | TISSUE DISTRIBUTION | membrane receptors | GENE-TRANSCRIPTION
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16. Full Text HER2/Neu (ErbB2) signaling to Rac1-Pak1 is temporally and spatially modulated by transforming growth factor β
by Wang, Shizhen Emily and Shin, Incheol and Wu, Frederick Y and Friedman, David B and Arteaga, Carlos L
Cancer Research, ISSN 0008-5472, 10/2006, Volume 66, Issue 19, pp. 9591 - 9600
In HER2 (ErbB2)-overexpressing cells, transforming growth factor beta (TGF-beta), via activation of phosphoinositide-3 kinase (PI3K), recruits actin and... 
BREAST-CANCER CELLS | P21-ACTIVATED KINASE | TGF-BETA | CYTOSKELETAL REORGANIZATION | ONCOLOGY | PHOSPHATIDYLINOSITOL 3-KINASE | PROTEIN-KINASE | RHO-GTPASES | MESENCHYMAL TRANSITION | NF-KAPPA-B | MAMMARY EPITHELIAL-CELLS | p21-Activated Kinases | Adenocarcinoma - pathology | Pseudopodia - ultrastructure | Receptor, ErbB-2 - genetics | Humans | Recombinant Fusion Proteins - physiology | Neuropeptides - physiology | Proto-Oncogene Proteins c-vav - physiology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Receptor, ErbB-2 - physiology | Actinin - physiology | rac GTP-Binding Proteins - genetics | Mammary Neoplasms, Experimental - pathology | Female | Receptor, ErbB-2 - antagonists & inhibitors | Neuropeptides - genetics | rac GTP-Binding Proteins - physiology | Cell Line, Tumor - metabolism | Protein-Serine-Threonine Kinases - physiology | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Transforming Growth Factor beta1 - genetics | Transforming Growth Factor beta1 - physiology | Cell Line, Tumor - ultrastructure | Animals | rac1 GTP-Binding Protein - physiology | Breast Neoplasms - pathology | Neoplasm Metastasis - physiopathology | Phosphatidylinositol 3-Kinases - physiology | Signal Transduction - physiology | Mice | Pseudopodia - physiology | Actins - physiology | rac1 GTP-Binding Protein - genetics | Index Medicus
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17. Full Text Soluble E-cadherin: A critical oncogene modulating receptor tyrosine kinases, MAPK and PI3K/Akt/mTOR signaling
by Brouxhon, S.M and Kyrkanides, S and Teng, X and Athar, M and Ghazizadeh, S and Simon, M and O'banion, M.K and Ma, L
Oncogene, ISSN 0950-9232, 01/2014, Volume 33, Issue 2, pp. 225 - 235
E-cadherin, a cell-cell adhesion glycoprotein, is frequently downregulated with tumorigenic progression. The extracellular domain of E-cadherin is cleaved by... 
squamous cell carcinomas | soluble E-cadherin | HER/ErbB receptors | oncogenecity | UV-irradiation | IGF-1R | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | FACTOR-I | ULTRAVIOLET-IRRADIATION | CELL BIOLOGY | TUMOR PROMOTION | GROWTH-FACTOR RECEPTOR | BREAST-CANCER CELLS | ONCOLOGY | GASTRIC-CANCER | CELL CARCINOMA PROGRESSION | PROSTATE-CANCER | GENETICS & HEREDITY | TRANSGENIC MICE | MAP Kinase Signaling System - physiology | Carcinoma, Squamous Cell - metabolism | Humans | Receptor Protein-Tyrosine Kinases - physiology | Cadherins - physiology | Proto-Oncogene Proteins c-akt - physiology | Skin Neoplasms - metabolism | Receptor, ErbB-2 - physiology | Carcinoma, Squamous Cell - etiology | Animals | Receptor, IGF Type 1 - physiology | Cell Line, Tumor | Phosphatidylinositol 3-Kinases - physiology | TOR Serine-Threonine Kinases - physiology | Mice | Skin Neoplasms - etiology | Extracellular Signal-Regulated MAP Kinases - physiology | Receptor, Epidermal Growth Factor - physiology | Physiological aspects | Protein tyrosine kinase | Cadherins | Oncogenes | Mitogen-activated protein kinases | Signal transduction | Neurons | Insulin-like growth factors | Index Medicus | HER | proliferation | invasion | Squamous cell carcinomas | migration | ErbB receptors
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18. The psychology of health
: an introduction
by Phillips, Keith, 1951 and Pitts, Marian, 1948
1998, 2nd ed., ISBN 9780415150248, xviii, 410
The first edition of The Psychology of Health has become the standard recommended text for many courses. This completely revised and updated second edition... 
Clinical health psychology | Medicine and psychology | Medicine: General Issues | Psychological Science | Health Psychology | Medicine and pychology
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19. Full Text Cellular mechano-environment regulates the mammary circadian clock
by Yang, Nan and Williams, Jack and Pekovic-Vaughan, Vanja and Wang, Pengbo and Olabi, Safiah and McConnell, James and Gossan, Nicole and Hughes, Alun and Cheung, Julia and Streuli, Charles H and Meng, Qing-Jun
Nature Communications, ISSN 2041-1723, 01/2017, Volume 8, Issue 1, pp. 14287 - 14287
Circadian clocks drive similar to 24 h rhythms in tissue physiology. They rely on transcriptional/translational feedback loops driven by interacting networks... 
STEM-CELLS | HOMEOSTASIS | PATHWAY | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | BIOLOGY | GENE-EXPRESSION | ACTIN DYNAMICS | INDUCTION | Cell Self Renewal - physiology | Humans | CLOCK Proteins - genetics | Circadian Clocks - genetics | Vinculin - physiology |