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animals (195) 195
transcription factor brn-3 (146) 146
brn-3 protein (126) 126
mice (117) 117
retina (109) 109
transcription factors - genetics (100) 100
dna-binding proteins - genetics (99) 99
retinal ganglion cells (96) 96
neurosciences (91) 91
expression (87) 87
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immunohistochemistry (42) 42
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retinal ganglion-cells (26) 26
cell survival (25) 25
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amacrine cells (19) 19
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photoreceptors (19) 19
polymerase chain reaction (19) 19
rats, sprague-dawley (19) 19
retinal ganglion cells - drug effects (19) 19
retinal ganglion cells - pathology (19) 19
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PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 7, p. e0177962
.... We performed comparative analyses in terms of gene and protein expression as well as functional characterizations... 
PROGENITOR CELLS | MESENCHYMAL STROMAL CELLS | STEM-CELLS | TRUNK | MULTIDISCIPLINARY SCIENCES | PERIODONTAL-LIGAMENT | ONTOGENY | GENERATION | SKIN-DERIVED PRECURSORS | DIFFERENTIATION | MELANOCYTES | RNA-Binding Proteins - genetics | Humans | Adipose Tissue - cytology | Melanocytes - metabolism | Neurons - cytology | Receptors, Nerve Growth Factor - metabolism | Neural Stem Cells - cytology | Schwann Cells - cytology | Neural Crest - metabolism | Adipose Tissue - metabolism | Mesenchymal Stromal Cells - cytology | Snail Family Transcription Factors - genetics | Melanocytes - cytology | Nestin - genetics | Adult | Female | Cell Differentiation | Neurons - metabolism | Dermis - cytology | Nuclear Proteins - genetics | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Gene Expression | Neural Crest - cytology | Dermis - metabolism | Snail Family Transcription Factors - metabolism | Microinjections | Bone Marrow Cells - cytology | Neural Crest - growth & development | Mesenchymal Stromal Cells - metabolism | Nuclear Proteins - metabolism | Schwann Cells - metabolism | Transcription Factor Brn-3A - metabolism | Chick Embryo | Nerve Tissue Proteins - genetics | Receptors, Nerve Growth Factor - genetics | Nerve Tissue Proteins - metabolism | Nestin - metabolism | Animals | Transcription Factor Brn-3A - genetics | Twist-Related Protein 1 - genetics | Twist-Related Protein 1 - metabolism | Neural Stem Cells - metabolism | SOX9 Transcription Factor - genetics | Bone Marrow Cells - metabolism | Mesenchymal Stem Cell Transplantation | RNA-Binding Proteins - metabolism | Adipose tissues | Comparative analysis | Skin | Neurosciences | Nestin | Adipose tissue | Leukocyte migration | Laboratories | Sox9 protein | Dermis | Melanocytes | Nervous system | Human tissues | Regeneration (physiology) | Cell adhesion & migration | Msi1 protein | Plasticity (neural) | Ethics | Immunology | Pathways | Surgery | Bone marrow | Hair | Hematology | Neurons | Tissue engineering | Schwann cells | Brn-3 protein | Gene expression | Embryos | Neural crest | Medicine | Regeneration | Human performance | Stromal cells | Stem cells | Cell migration | Dental pulp
Journal Article
PloS one, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e0186748
Our previous studies showed positive correlation between accumulation of proNGF, activation of RhoA and neuronal death in diabetic models. Here, we examined... 
APOPTOSIS | FAMILY GTPASES | INHIBITION | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | GANGLION-CELLS | RAT RETINA | SPINAL-CORD-INJURY | DEATH | NERVE GROWTH-FACTOR | P75 | Poly(ADP-ribose) Polymerases | Intravitreal Injections | Apoptosis - drug effects | Streptozocin | Caspase 3 - metabolism | Diabetes Mellitus, Experimental - genetics | Male | rhoA GTP-Binding Protein - metabolism | Receptors, Nerve Growth Factor - metabolism | rhoA GTP-Binding Protein - genetics | Retinal Ganglion Cells - metabolism | Retinal Degeneration - metabolism | Retinal Ganglion Cells - pathology | Proteolysis - drug effects | Caspase 3 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Diabetes Mellitus, Experimental - metabolism | Amides - pharmacology | Nerve Growth Factor - metabolism | Protein Precursors - genetics | Retinal Degeneration - genetics | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Nerve Growth Factor - genetics | Rats, Sprague-Dawley | Receptors, Nerve Growth Factor - genetics | Protein Precursors - metabolism | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Diabetes Mellitus, Experimental - pathology | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Primary Cell Culture | Retinal Degeneration - pathology | Retinal Ganglion Cells - drug effects | Complications and side effects | Retinal degeneration | Development and progression | Genetic aspects | Diabetes | Gene expression | Growth factors | Health aspects | Neuroprotection | Animal models | Retina | Activation | Kinases | Caspase-3 | Norwood, Charlie | Neurodegeneration | Rodents | Cleavage | Inhibition | Diabetes mellitus | Mortality | Caspase | Poly(ADP-ribose) polymerase | RhoA protein | JNK protein | Brn-3 protein | Tumor necrosis factor-α | Retinal ganglion cells | Cell death | Correlation analysis | Dolphins & porpoises | Apoptosis
Journal Article
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e97222
.... Endoplasmic reticulum stress was assessed using immunoglobulin binding protein (BiP), protein disulfide isomerase (PDI... 
Immunohistochemistry | Retina - metabolism | Age Factors | Protein Disulfide-Isomerases - metabolism | DNA Primers - genetics | Retinal Ganglion Cells - metabolism | Membrane Proteins - deficiency | Evoked Potentials, Visual - physiology | Polymerase Chain Reaction | Visual Acuity - physiology | Protein-Serine-Threonine Kinases - metabolism | Electroretinography | Microscopy, Electron, Transmission | Retinal Ganglion Cells - physiology | Contrast Sensitivity - physiology | Endoribonucleases - metabolism | Heat-Shock Proteins - metabolism | Retina - physiopathology | Visual Acuity - genetics | Blotting, Western | Mice, Knockout | Animals | Contrast Sensitivity - genetics | Mice | Unfolded Protein Response - physiology | Endoplasmic Reticulum Stress - physiology | Neurosciences | Amplitudes | Optic neuropathy | Visual pathways | Homeostasis | Retina | Visual evoked potentials | Action potential | Neuropathy | Kinases | Autophagy | Visual perception | Proteins | Atrophy | Histopathology | Protein folding | Rodents | Cell cycle | Stress response | Pancreas | Stresses | Inositol | Optic nerve | Diabetes mellitus | Brn-3 protein | Gene expression | Electron microscopy | Insulin | Stress | Latency | Electroretinograms | Beta cells | Retinal ganglion cells | Signaling | Brain research | Visual impairment | Protein disulfide-isomerase | Diabetes | Mutation | Endoplasmic reticulum | Apoptosis
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23194
Background: Neuron loss, glial activation and vascular degeneration are common sequelae of ischemia-reperfusion (I/R) injury in ocular diseases. The present... 
DIABETIC-RETINOPATHY | REGENERATION | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | GANGLION-CELLS | STAT3 | NF-KAPPA-B | EXPRESSION | DAMAGE | Retina - drug effects | Neurons - pathology | Retina - metabolism | Vascular Diseases - prevention & control | Vascular Diseases - pathology | Rats, Wistar | Apoptosis - drug effects | Male | NF-kappa B - metabolism | Glial Fibrillary Acidic Protein - metabolism | Retinal Ganglion Cells - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Retinal Ganglion Cells - pathology | Dose-Response Relationship, Drug | Retinal Vessels - pathology | Tubulin - metabolism | Time Factors | Neurons - metabolism | Neurons - drug effects | Reperfusion Injury - metabolism | STAT3 Transcription Factor - metabolism | Retinal Vessels - metabolism | Curcumin - pharmacology | Rats | Retinal Vessels - drug effects | Blotting, Western | Animals | Reperfusion Injury - prevention & control | Signal Transduction - drug effects | Chemokine CCL2 | Vascular Diseases - metabolism | Retina - pathology | Retinal Ganglion Cells - drug effects | Medical research | Ischemia | Neurons | Physiological aspects | Medicine, Experimental | Eye diseases | Tubulins | Apoptosis | Diabetic retinopathy | Phosphorylation | Multiple myeloma | Retina | Activation | Glial cells | Kinases | Neuronal-glial interactions | Signal transduction | Tubulin | Reperfusion | Epidermal growth factor | Rodents | Life sciences | Degeneration | Curcumin | Inhibition | Pretreatment | Trends | Capillaries | Stat1 protein | Injuries | NF-κB protein | Cytokines | Complications | Glial fibrillary acidic protein | Pancreatitis | Stat3 protein | Extracellular signal-regulated kinase | MAP kinase | Brn-3 protein | Studies | Signaling | Overexpression | Cell death | Pharmacy | Spinal cord injuries | Diabetes | Respiration | Laboratory animals | Alzheimers disease | Chemokines | Monocyte chemoattractant protein 1 | Intraocular pressure
Journal Article
PloS one, ISSN 1932-6203, 04/2016, Volume 11, Issue 4, p. e0153608
.... Retina tissue was used to determine retinal ganglion cell (RGC) number, while optic nerve tissue was examined for cellularity, myelin content, protein and lipid changes... 
RETINAL GANGLION-CELLS | HEAT-SHOCK PROTEINS | MICROTUBULE-ASSOCIATED PROTEINS | OPTIC-NERVE LESION | MULTIDISCIPLINARY SCIENCES | AXONAL-TRANSPORT | BASIC-PROTEIN | ALTERED EXPRESSION | CATHEPSIN-D | MYELIN-ASSOCIATED GLYCOPROTEIN | APOLIPOPROTEIN-E | Biomarkers - metabolism | Optic Nerve - pathology | Humans | Mice, Inbred C57BL | Brain Injuries - complications | Male | Proteome - analysis | Vision Disorders - pathology | Brain Injuries - physiopathology | Retinal Ganglion Cells - metabolism | Retinal Ganglion Cells - pathology | Animals | Nervous System Diseases - etiology | Nervous System Diseases - metabolism | Mice | Lipids - analysis | Chronic Disease | Vision Disorders - metabolism | Nervous System Diseases - pathology | Optic Nerve - metabolism | Vision Disorders - etiology | Brain | Complications and side effects | Prognosis | Physiological aspects | Research | Injuries | Visual perception | Immunohistochemistry | Multiple sclerosis | Traumatic brain injury | Target recognition | Lecithin | Neuropathology | Change detection | Retina | Lipids | Phospholipids | Biochemistry | Lysophosphatidylcholine | Proteins | Depolymerization | Visual system | Head injuries | Filaments | Demyelination | Apolipoprotein E | Microtubule-associated proteins | Microtubule-associated protein 1 | Nerves | Anesthesia | Neurofilaments | Cathepsin D | Species | Injury analysis | Phosphatidylethanolamine | Biochemical analysis | Optic nerve | Myelin | Heat shock proteins | Histology | Brn-3 protein | Metabolism | Retinal ganglion cells | Brain research | Concussion | Correlation analysis | Collagen | Laboratory animals | Dismantling | Heat shock
Journal Article
Journal of Comparative Neurology, ISSN 0021-9967, 06/2013, Volume 521, Issue 9, pp. 2165 - 2180
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 8, p. e0182407
Retinal ischemia is common in eye disorders, like diabetic retinopathy or retinal vascular occlusion. The goal of this study was to evaluate the potential... 
OPTIC-NERVE | AUTOIMMUNE GLAUCOMA MODEL | PROLIFERATIVE DIABETIC-RETINOPATHY | PRESSURE-INDUCED ISCHEMIA | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | RAT RETINA | GANGLION-CELLS | MACULAR DEGENERATION | GLIAL-CELLS | ENDOTHELIAL GROWTH-FACTOR | Amacrine Cells - drug effects | Microglia - metabolism | Cell Count | Humans | Photoreceptor Cells, Vertebrate - drug effects | Amacrine Cells - metabolism | Retina | Ranibizumab - therapeutic use | Vascular Endothelial Growth Factor A - metabolism | Protective Agents - therapeutic use | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Retinal Ganglion Cells - metabolism | Rhodopsin - metabolism | Aqueous Humor - metabolism | Synapses - metabolism | Protective Agents - pharmacology | Protein Binding - drug effects | Microfilament Proteins - metabolism | Ischemia - pathology | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Synapses - drug effects | Microglia - drug effects | Reperfusion Injury - pathology | Cholinergic Neurons - metabolism | RNA, Messenger - genetics | Rats | Cholinergic Neurons - drug effects | Ranibizumab - pharmacology | Animals | Ischemia - drug therapy | Mice | Photoreceptor Cells, Vertebrate - metabolism | Retinal Ganglion Cells - drug effects | Retinal diseases | Care and treatment | Occlusion | Immunohistochemistry | Intervention | Diabetic retinopathy | Neurosciences | Retinopathy | Disorders | Neurobiology | Activation | Eye | Macular degeneration | Proteins | Cell growth | Ischemia | Rodents | Eye (anatomy) | Vascular endothelial growth factor | Retinal cells | Glial fibrillary acidic protein | Diabetes mellitus | Brn-3 protein | Microglia | Vascular endothelial growth factor receptors | Studies | Polymerase chain reaction | Retinal ganglion cells | Pathology | Hospitals | Cell number | Morphology | Eye disorders | Amacrine cells | Photoreceptors | Diabetes | Glia | Apoptosis
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 10, p. e76347
Background: Visual information is conveyed from the retina to the brain via 15-20 Retinal Ganglion Cell (RGC) types. The developmental mechanisms by which RGC... 
SOMATOSENSORY NEURONS | NERVOUS-SYSTEM | AMACRINE CELLS | PRIMATE RETINA | MULTIDISCIPLINARY SCIENCES | VISUAL-SYSTEM | MOUSE RETINA | DIFFERENTIATION | DOMAIN FACTOR BRN-3B | MAMMALIAN RETINA | EXPRESSION | Retina - metabolism | Retina - growth & development | Cell Count | Homeodomain Proteins - metabolism | Transcription Factor Brn-3B - genetics | Cell Survival - genetics | Male | Mice, 129 Strain | Retinal Ganglion Cells - metabolism | Retinal Ganglion Cells - cytology | Retina - cytology | Gene Expression Regulation, Developmental | Rod Opsins - metabolism | Transcription Factor Brn-3C - genetics | Female | Mice, Inbred C57BL | Transcription Factor Brn-3C - metabolism | Transcription Factor Brn-3A - metabolism | Homeodomain Proteins - genetics | Transcription Factor Brn-3B - metabolism | Mice, Knockout | Animals | Transcription Factor Brn-3A - genetics | Epistasis, Genetic | Mice | Neurofilament Proteins - metabolism | Microscopy, Fluorescence | Ganglion | Genetic aspects | DNA binding proteins | Brain | Transcription factors | Gene regulation | Retina | Melanopsin | Defects | Proteins | Developmental stages | Specifications | Rodents | Physiology | Dendrites | Cell survival | Neurons | Axonogenesis | Brn-3 protein | Gene expression | Survival | Molecular chains | Retinal ganglion cells | Brn3 gene | Lamination | Insects | Islet-1 protein | Neural networks | Morphology | Alleles | Protein expression | Adults | Combinatorial analysis
Journal Article