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Journal of allergy and clinical immunology, ISSN 0091-6749, 09/2015, Volume 136, Issue 3, pp. 769 - 780
Background Inflammation and oxidative stress play critical roles in patients with chronic obstructive pulmonary disease (COPD). Mitochondrial oxidative stress... 
Allergy and Immunology | airway hyperresponsiveness | antioxidant | proliferation | MitoQ | inflammation | mitochondria | airway smooth muscle | Ozone | oxidative stress | chronic obstructive pulmonary disease | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Respiratory System - pathology | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Male | Pulmonary Disease, Chronic Obstructive - pathology | Bronchial Hyperreactivity - genetics | Ubiquinone - pharmacology | Muscle, Smooth - drug effects | Smoking - physiopathology | Organophosphorus Compounds - pharmacology | Myocytes, Smooth Muscle - drug effects | Pneumonia - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Bronchial Hyperreactivity - chemically induced | Signal Transduction | Mitochondria - pathology | Smoking - metabolism | Bronchial Hyperreactivity - pathology | Reactive Oxygen Species - antagonists & inhibitors | Electron Transport Chain Complex Proteins - metabolism | Pulmonary Disease, Chronic Obstructive - chemically induced | Respiratory System - drug effects | Airway Remodeling - genetics | Mice | Oxidative Stress - drug effects | Bronchial Hyperreactivity - drug therapy | Myocytes, Smooth Muscle - pathology | Pneumonia - pathology | Membrane Potential, Mitochondrial - drug effects | Pneumonia - chemically induced | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Myocytes, Smooth Muscle - metabolism | Ubiquinone - analogs & derivatives | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Electron Transport Chain Complex Proteins - genetics | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Muscle, Smooth - metabolism | Animals | Pneumonia - drug therapy | Respiratory System - metabolism | Aged | Muscle, Smooth - pathology | Oxidative stress | Care and treatment | Lung diseases, Obstructive | Inflammation | Pharmaceutical industry | Biomedical research | Disease | Laboratories | Mortality | Colleges & universities | Smooth muscle | Mitochondrial DNA | Metabolism | Defects | Antioxidants | Mitochondria | Hospitals | Biopsy | Chronic obstructive pulmonary disease | Apoptosis | Smoking | Index Medicus | Abridged Index Medicus | NO, Nitric oxide | ΔΨm, Mitochondrial membrane potential | ASM, Airway smooth muscle | NAC, N-acetylcysteine | RL, Lung resistance | BAL, Bronchoalveolar lavage | OCR, Oxygen consumption rate | dTPP, Decyltriphenylphosphonium bromide | AHR, Airway hyperresponsiveness | logPC100, Concentration of acetylcholine that increased lung resistance by 100 | GOLD, Global Initiative for Chronic Obstructive Lung Disease | ATP, Adenosine triphosphate | KC, Keratinocyte-derived cytokine | JC-1, 5,5′,6,6′-Tetrachloro-1,1′,3,3′-tetraethylbenzimidazolylcarbocyanine iodide | COPD, Chronic obstructive pulmonary disease | ROS, Reactive oxygen species | Mechanisms of Allergy and Clinical Immunology
Journal Article
PloS one, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, pp. e70351 - e70351
.... GSNOR activity has been shown to be elevated in human asthmatic lungs, resulting in diminished S-nitrosothiols and thus contributing to increased airway hyperreactivity... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Allergens | Bronchial Hyperreactivity - immunology | Chemokine CCL11 - biosynthesis | Eosinophils - drug effects | Interleukin-13 - antagonists & inhibitors | Glutathione Reductase - metabolism | Bronchial Hyperreactivity - drug therapy | Humans | Pneumonia - pathology | Male | Th2 Cells - immunology | Alcohol Dehydrogenase | Interleukin-5 - biosynthesis | Eosinophils - immunology | Th2 Cells - drug effects | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Interleukin-13 - biosynthesis | Chemokine CCL11 - antagonists & inhibitors | Interleukin-5 - antagonists & inhibitors | Pneumonia - chemically induced | Pneumonia - immunology | Female | Pyrimidinones - pharmacology | Eosinophils - pathology | Th2 Cells - pathology | Bronchial Hyperreactivity - chemically induced | Ovalbumin | Enzyme Inhibitors - pharmacology | Mice, Transgenic | Administration, Intranasal | Cell Movement - drug effects | Bronchial Hyperreactivity - pathology | Animals | Pneumonia - drug therapy | Benzoates - pharmacology | Mice | Mice, Inbred BALB C | Glutathione Reductase - antagonists & inhibitors | Inflammation | T cells | Analysis | Asthma | Health sciences | Smooth muscle | Mucus | Lymphocytes T | Alcohol dehydrogenase | Eotaxin | Alcohols | Respiratory tract | Interleukin 5 | Lymphocytes | Transgenic animals | Rodents | Respiratory tract diseases | Inhibition | Pharmaceutical sciences | Enzymes | Cytokines | Intracellular levels | Hypersensitivity | Environmental health | Intranasal administration | Interleukin 13 | Inhalation | CD4 antigen | Lungs | Regulation | Laboratory animals | Respiration | Binding sites | Reductase | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 02/2013, Volume 131, Issue 2, pp. 501 - 511.e1
Background Sphingosine-1-phosphate (S1P), which is produced by 2 sphingosine kinase (SphK) isoenzymes, SphK1 and SphK2, has been implicated in IgE-mediated... 
Allergy and Immunology | airway hyperresponsiveness | asthma | nuclear factor κB | mast cells | sphingosine kinase | Sphingosine-1-phosphate | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Lysophospholipids - metabolism | Asthma - metabolism | Bronchial Hyperreactivity - drug therapy | Humans | Goblet Cells - drug effects | NF-kappa B - metabolism | Amino Alcohols - pharmacology | Interferon-gamma - metabolism | Interleukins - metabolism | Inflammation - metabolism | Inflammation - drug therapy | Methacholine Chloride - pharmacology | Mast Cells - metabolism | Bronchial Hyperreactivity - metabolism | Female | Chemokine CCL2 - metabolism | Hyperplasia - drug therapy | Lung - metabolism | Sphingosine - metabolism | Hyperplasia - metabolism | Asthma - enzymology | Immunoglobulin E - metabolism | Mice, Inbred C57BL | Cells, Cultured | Ovalbumin - pharmacology | Asthma - drug therapy | Goblet Cells - metabolism | Mast Cells - drug effects | Asthma - chemically induced | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Bronchial Hyperreactivity - enzymology | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Bronchial Hyperreactivity - pathology | Sphingosine - analogs & derivatives | Animals | Lung - drug effects | Mice | Bronchoalveolar Lavage Fluid - chemistry | Phosphates | Medical colleges | Isoenzymes | Analysis | Sphingosine | Inflammation | Asthma | Studies | Cytokines | Lungs | Rodents | Kinases | Laboratory animals | Allergies | Index Medicus | Abridged Index Medicus
Journal Article
Respiratory research, ISSN 1465-9921, 02/2007, Volume 8, Issue 1, pp. 16 - 16
Mast cell-derived prostaglandin D2 (PGD2), may contribute to eosinophilic inflammation and mucus production in allergic asthma. Chemoattractant receptor... 
Inflammation - pathology | Bronchial Hyperreactivity - immunology | Receptors, Immunologic - antagonists & inhibitors | Inflammation - drug therapy | Bronchial Hyperreactivity - physiopathology | Sensitivity and Specificity | Asthma - immunology | Female | Prostaglandin Antagonists - pharmacology | Bronchoalveolar Lavage Fluid - cytology | Radioligand Assay | Receptors, Prostaglandin - antagonists & inhibitors | Disease Models, Animal | Pulmonary Eosinophilia - immunology | Immunization | Enzyme-Linked Immunosorbent Assay | Cells, Cultured | Sulfonamides - pharmacology | Asthma - drug therapy | Pulmonary Eosinophilia - pathology | Animals | Mice | Mice, Inbred BALB C | Carbazoles - pharmacology | In Vitro Techniques | Asthma - pathology | Allergens | Prostaglandins | Inflammation | Histochemistry | Asthma | Index Medicus | Research | Disease Models | Inflammation: pathology | Inbred BALB C | Medical and Health Sciences | Prostaglandin Antagonists: pharmacology | Medicin och hälsovetenskap | Pulmonary Eosinophilia: immunology | Klinisk medicin | Asthma: drug therapy | Receptors | Pulmonary Eosinophilia: pathology | Cultured | Lungmedicin och allergi | Asthma: immunology | Bronchoalveolar Lavage Fluid: cytology | Bronchial Hyperreactivity: immunology | Respiratory Medicine and Allergy | Immunologic: antagonists & inhibitors | Prostaglandin: antagonists & inhibitors | Sulfonamides: pharmacology | Cells | Inflammation: drug therapy | Clinical Medicine | Carbazoles: pharmacology | Bronchial Hyperreactivity: physiopathology | Animal | Asthma: pathology
Journal Article