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American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 02/2014, Volume 189, Issue 4, pp. 401 - 407
Journal Article
American journal of respiratory and critical care medicine, ISSN 1073-449X, 2013, Volume 187, Issue 5, pp. 486 - 493
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2009, Volume 106, Issue 31, pp. 13088 - 13093
Journal Article
Annals of the American Thoracic Society, ISSN 2325-6621, 01/2016, Volume 13, Issue 1, pp. 17 - 24
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2008, Volume 122, Issue 2, pp. 225 - 235
Exercise-induced bronchoconstriction (EIB) is a consequence of evaporative water loss in conditioning the inspired air. The water loss causes cooling and... 
Allergy and Immunology | Epithelial injury | airway smooth muscle | eicosanoids | mast cells | microvascular permeability | β 2-agonists | β | agonists | INDUCED ASTHMA | MAST-CELL ACTIVATION | BRONCHIAL HYPERRESPONSIVENESS | INDUCED PLASMA EXUDATION | IMMUNOLOGY | CROSS-COUNTRY SKIERS | INDUCED BRONCHOSPASM | FISH-OIL SUPPLEMENTATION | SMOOTH-MUSCLE | beta-agonists | ALLERGY | PASSIVE SENSITIZATION | epithelial injury | CHLORINATED SWIMMING POOLS | Asthma, Exercise-Induced - etiology | Capillary Permeability | Bronchial Hyperreactivity - immunology | Bronchial Hyperreactivity - drug therapy | Humans | Bronchi - physiopathology | Epithelial Cells - physiology | Exercise | Asthma, Exercise-Induced - drug therapy | Bronchoconstriction - drug effects | Bronchi - blood supply | Bronchoconstriction - immunology | Muscle, Smooth - physiology | Adrenergic beta-Agonists - therapeutic use | Bronchi - pathology | Methacholine Chloride | Sports | Bronchial Hyperreactivity - etiology | Mast Cells - immunology | Mast Cells - physiology | Inflammation | Bronchial Hyperreactivity - pathology | Eicosanoids - metabolism | Asthma, Exercise-Induced - immunology | Asthma, Exercise-Induced - pathology | Bronchoconstrictor Agents | Athletes | Sports injuries | Cooling | Cold | Rodents | Smooth muscle | Accreditation | Olympic games | Asthma | Steroids
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 07/2003, Volume 168, Issue 1, pp. 54 - 62
New York City Fire Department rescue workers experienced massive exposure to airborne particulates at the World Trade Center site. Aims of this longitudinal... 
Firefighters | Reactive airways dysfunction syndrome | Inorganic particulate matter | Nonspecific bronchial hyper-reactivity | World Trade Center collapse | OCCUPATIONAL ASTHMA | BRONCHIAL RESPONSIVENESS | FOLLOW-UP | SYMPTOMS | HYPERRESPONSIVENESS | firefighters | DOSE-RESPONSE CURVES | SYNDROME RADS | nonspecific bronchial hyperreactivity | RESPIRATORY SYSTEM | INFLAMMATION | METHACHOLINE | inorganic particulate matter | reactive airways dysfunction syndrome | EXPOSURE | CRITICAL CARE MEDICINE | Air Pollutants - analysis | Predictive Value of Tests | Prospective Studies | Humans | Middle Aged | Occupational Diseases - etiology | Bronchial Provocation Tests | Bronchial Hyperreactivity - diagnosis | Cough - diagnosis | Male | Inhalation Exposure - analysis | Case-Control Studies | Occupational Diseases - diagnosis | Forced Expiratory Volume | Time Factors | Fires | Cough - epidemiology | Inhalation Exposure - adverse effects | Adult | Female | Surveys and Questionnaires | Asthma - etiology | Asthma - epidemiology | Asthma - diagnosis | Bronchial Hyperreactivity - etiology | Terrorism | Occupational Exposure - adverse effects | Explosions | Cough - etiology | Vital Capacity | Occupational Exposure - analysis | Air Pollutants - adverse effects | Bronchial Hyperreactivity - epidemiology | New York City - epidemiology | Occupational Diseases - epidemiology
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 134, Issue 1, pp. 63 - 72.e7
Background Most asthma begins in the first years of life. This early onset cannot be attributed merely to genetic factors because the prevalence of asthma is... 
Allergy and Immunology | natural killer cells | mouse model | diesel exhaust particles | IL-17 | IL-5 | asthma | IL-13 | Prenatal exposure | aryl hydrocarbon receptor | ENVIRONMENTAL-POLLUTANT | NATURAL-KILLER-CELLS | POLYCYCLIC AROMATIC-HYDROCARBONS | LYMPH-NODE CELLS | ALLERGIC AIRWAY INFLAMMATION | IMMUNOLOGY | CYTOKINE PRODUCTION | NK CELLS | INTRANASAL INSTILLATION | ALLERGY | ARYL-HYDROCARBON RECEPTOR | TH2 DIFFERENTIATION | Vehicle Emissions - toxicity | Bronchial Hyperreactivity - immunology | Humans | Interleukin-17 - immunology | Gene Expression Profiling | Killer Cells, Natural - pathology | Bronchial Hyperreactivity - genetics | Immunoglobulin E - immunology | Immunoglobulin E - genetics | Asthma - immunology | Killer Cells, Natural - immunology | Maternal Exposure - adverse effects | Female | Transcription, Genetic | Asthma - etiology | Disease Models, Animal | Bronchial Hyperreactivity - etiology | Disease Susceptibility | Mice, Inbred C57BL | Gene Expression Regulation | Receptors, Aryl Hydrocarbon - genetics | Interleukin-17 - genetics | Asthma - genetics | Pregnancy | Prenatal Exposure Delayed Effects - genetics | Bronchial Hyperreactivity - pathology | Animals | Ovalbumin - administration & dosage | Prenatal Exposure Delayed Effects - immunology | Mice | Air Pollutants - toxicity | Asthma - pathology | Receptors, Aryl Hydrocarbon - immunology | Prenatal Exposure Delayed Effects - pathology | Diesel motor exhaust gas | Pregnant women | Analysis | Prevalence studies (Epidemiology) | Asthma | Hypotheses | Immunization | Polycyclic aromatic hydrocarbons | Rodents | prenatal exposure | interleukin 5 | interleukin 13 | interleukin 17
Journal Article