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Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 3, pp. 769 - 780
Background Inflammation and oxidative stress play critical roles in patients with chronic obstructive pulmonary disease (COPD). Mitochondrial oxidative stress... 
Allergy and Immunology | airway hyperresponsiveness | antioxidant | proliferation | MitoQ | inflammation | mitochondria | airway smooth muscle | Ozone | oxidative stress | chronic obstructive pulmonary disease | ASTHMA FEATURES | MECHANISMS | IMMUNOLOGY | FRAGMENTATION | HYPERRESPONSIVENESS | BETA | RESPONSES | ALLERGY | ENERGY-METABOLISM | EXPRESSION | COPD | Respiratory System - pathology | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Male | Pulmonary Disease, Chronic Obstructive - pathology | Bronchial Hyperreactivity - genetics | Ubiquinone - pharmacology | Muscle, Smooth - drug effects | Smoking - physiopathology | Organophosphorus Compounds - pharmacology | Myocytes, Smooth Muscle - drug effects | Pneumonia - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Bronchial Hyperreactivity - chemically induced | Signal Transduction | Mitochondria - pathology | Smoking - metabolism | Bronchial Hyperreactivity - pathology | Reactive Oxygen Species - antagonists & inhibitors | Electron Transport Chain Complex Proteins - metabolism | Pulmonary Disease, Chronic Obstructive - chemically induced | Respiratory System - drug effects | Airway Remodeling - genetics | Mice | Oxidative Stress - drug effects | Bronchial Hyperreactivity - drug therapy | Myocytes, Smooth Muscle - pathology | Pneumonia - pathology | Membrane Potential, Mitochondrial - drug effects | Pneumonia - chemically induced | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Myocytes, Smooth Muscle - metabolism | Ubiquinone - analogs & derivatives | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Electron Transport Chain Complex Proteins - genetics | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Muscle, Smooth - metabolism | Animals | Pneumonia - drug therapy | Respiratory System - metabolism | Aged | Muscle, Smooth - pathology | Oxidative stress | Care and treatment | Lung diseases, Obstructive | Inflammation | Pharmaceutical industry | Biomedical research | Disease | Laboratories | Mortality | Colleges & universities | Smooth muscle | Mitochondrial DNA | Metabolism | Defects | Antioxidants | Mitochondria | Hospitals | Biopsy | Chronic obstructive pulmonary disease | Apoptosis | Smoking | NO, Nitric oxide | ΔΨm, Mitochondrial membrane potential | ASM, Airway smooth muscle | NAC, N-acetylcysteine | RL, Lung resistance | BAL, Bronchoalveolar lavage | OCR, Oxygen consumption rate | dTPP, Decyltriphenylphosphonium bromide | AHR, Airway hyperresponsiveness | logPC100, Concentration of acetylcholine that increased lung resistance by 100 | GOLD, Global Initiative for Chronic Obstructive Lung Disease | ATP, Adenosine triphosphate | KC, Keratinocyte-derived cytokine | JC-1, 5,5′,6,6′-Tetrachloro-1,1′,3,3′-tetraethylbenzimidazolylcarbocyanine iodide | COPD, Chronic obstructive pulmonary disease | ROS, Reactive oxygen species | Mechanisms of Allergy and Clinical Immunology
Journal Article
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 2, pp. 441 - 453
Background Traffic-related particulate matter (PM) has been linked to a heightened incidence of asthma and allergic diseases. However, the molecular mechanisms... 
Allergy and Immunology | airway hyperresponsiveness | Traffic-related particulate matter | diesel exhaust particles | ultrafine particles | Jagged 1 | Notch | asthma | allergic airway inflammation | aryl hydrocarbon receptor | AMBIENT PARTICULATE MATTER | OXIDATIVE STRESS | DENDRITIC CELLS | IMMUNOLOGY | POLLUTION | NRF2 | ALLERGY | CHEMICALS | airway hyper-responsiveness | HEALTH | TRANSCRIPTION FACTOR | Allergens - adverse effects | Bronchial Hyperreactivity - immunology | Dendritic Cells - immunology | Humans | Dendritic Cells - pathology | Gene Expression Profiling | Monocytes - immunology | Bronchial Hyperreactivity - genetics | CD11c Antigen - immunology | Respiratory Hypersensitivity - pathology | CD11c Antigen - genetics | Immunoglobulin E - genetics | Calcium-Binding Proteins - immunology | Serrate-Jagged Proteins | T-Lymphocytes, Helper-Inducer - immunology | Monocytes - pathology | Vehicle Emissions | T-Lymphocytes, Helper-Inducer - pathology | Disease Models, Animal | Jagged-1 Protein | Bronchial Hyperreactivity - chemically induced | Signal Transduction | Membrane Proteins - genetics | Particulate Matter - adverse effects | Respiratory Hypersensitivity - chemically induced | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Receptors, Aryl Hydrocarbon - genetics | Receptor, Notch1 - immunology | Membrane Proteins - immunology | Mice, Transgenic | Respiratory Hypersensitivity - immunology | Receptors, Cell Surface - immunology | Respiratory Hypersensitivity - genetics | Bronchial Hyperreactivity - pathology | Animals | Alleles | Mice | Primary Cell Culture | Receptor, Notch1 - genetics | Receptors, Aryl Hydrocarbon - immunology | Intercellular Signaling Peptides and Proteins - immunology | Calcium-Binding Proteins - genetics | Receptors, Cell Surface - genetics | Medical colleges | Air pollution | Asthma | Antigens | Aqueous solutions | Cytokines | Dendritic cells | Lymphocytes | Atherosclerosis | Traffic | Atoms & subatomic particles | Hydrocarbons | Inflammation | Pollutants
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2002, Volume 169, Issue 9, pp. 5315 - 5321
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 2, pp. 462 - 473
Background Steroid-resistant asthma is a major clinical problem that is linked to activation of innate immune cells. Levels of IFN-γ and LPS are often... 
Allergy and Immunology | pulmonary macrophages | MicroRNA | innate immunity | steroid-resistant airway hyperresponsiveness | protein phosphatase 2A | EMERGING ROLES | N-TERMINAL KINASE | INFLAMMATORY SUBTYPES | IMMUNOLOGY | CANCER-THERAPY | NEUTROPHILIC ASTHMA | INTERFERON-GAMMA | ALLERGY | DISEASE | GENE-EXPRESSION | GLUCOCORTICOID-RECEPTOR PHOSPHORYLATION | Oligonucleotides - genetics | Egg Hypersensitivity - drug therapy | Macrophages, Alveolar - pathology | MicroRNAs - antagonists & inhibitors | Bronchial Hyperreactivity - immunology | Eosinophils - drug effects | Bronchial Hyperreactivity - drug therapy | Humans | Protein Phosphatase 2 - immunology | Bronchial Hyperreactivity - genetics | Eosinophils - immunology | Luciferases - genetics | Egg Hypersensitivity - immunology | Dexamethasone - pharmacology | Egg Hypersensitivity - etiology | Luciferases - immunology | Asthma - immunology | Eosinophils - pathology | Genes, Reporter | Macrophages, Alveolar - immunology | Disease Models, Animal | Egg Hypersensitivity - genetics | Neutrophils - pathology | Bronchial Hyperreactivity - chemically induced | Ovalbumin | Signal Transduction | Neutrophils - drug effects | Gene Expression Regulation | Neutrophils - immunology | Protein Phosphatase 2 - genetics | Receptors, Glucocorticoid - immunology | MicroRNAs - immunology | Asthma - drug therapy | Asthma - chemically induced | Asthma - genetics | Oligonucleotides - metabolism | Animals | Macrophages, Alveolar - drug effects | Lipopolysaccharides - pharmacology | Receptors, Glucocorticoid - genetics | Glucocorticoids - pharmacology | Mice | Mice, Inbred BALB C | MicroRNAs - genetics | Primary Cell Culture | Interferon-gamma - pharmacology | Medical colleges | Phosphatases | Corticosteroids | Analysis | Pharmacy | Drugstores | Genetic translation | Asthma | Proteins | Phosphorylation | Disease | Pathogenesis | Rodents | MicroRNAs | Inflammation | Gene expression | Phosphatase
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 3, pp. 521 - 529
The Dutch hypothesis was first articulated in 1961, when many novel and advanced scientific techniques were not available, such as genomics techniques for... 
Allergy and Immunology | allergy | hyperresponsiveness | Dutch hypothesis | Asthma | chronic obstructive pulmonary disease | BRONCHIAL HYPERRESPONSIVENESS | INHALED CORTICOSTEROID TREATMENT | LUNG-FUNCTION | RANDOMIZED CONTROLLED-TRIAL | IMMUNOLOGY | OBSTRUCTIVE PULMONARY-DISEASE | ADENOSINE 5'-MONOPHOSPHATE | RISK-FACTOR | RESPIRATORY SYMPTOMS | LONG-TERM TREATMENT | AIR-FLOW OBSTRUCTION | Pulmonary Emphysema - diagnosis | Age Factors | Bronchial Hyperreactivity - immunology | Humans | Matrix Metalloproteinase 9 - immunology | Bronchial Hyperreactivity - diagnosis | Pulmonary Disease, Chronic Obstructive - pathology | Bronchial Hyperreactivity - genetics | Gene-Environment Interaction | Transforming Growth Factor beta1 - immunology | Immunoglobulin E - genetics | Matrix Metalloproteinase 9 - genetics | Asthma - immunology | Pulmonary Emphysema - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Asthma - diagnosis | Pulmonary Disease, Chronic Obstructive - diagnosis | Gene Expression | Pulmonary Emphysema - pathology | Genotype | Transforming Growth Factor beta1 - genetics | Asthma - genetics | Pulmonary Emphysema - immunology | Bronchial Hyperreactivity - pathology | Phenotype | Sex Factors | Polymorphism, Single Nucleotide | Pulmonary Disease, Chronic Obstructive - immunology | Asthma - pathology | Medical colleges | Gene expression | Analysis | Genomics | Bacterial infections | Physicians | Infections | Research | Disclosure | Patients | Allergies | Copyright | Genotype & phenotype | Hypotheses | Researchers | Air pollution | Chronic obstructive pulmonary disease | Accreditation | Clinical medicine
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2013, Volume 304, Issue 8, pp. L519 - L529
Proskocil BJ, Bruun DA, Jacoby DB, van Rooijen N, Lein PJ, Fryer AD. Macrophage TNF-alpha mediates parathion-induced airway hyperreactivity in guinea pigs. Am... 
Parathion | Organophosphorus pesticides | Tumor necrosis factor-a | Alveolar macrophages | Airway Hyperreactivity | PULMONARY PARASYMPATHETIC NERVES | airway hyperreactivity | PHYSIOLOGY | BRONCHIAL HYPERRESPONSIVENESS | alveolar macrophages | MUSCARINIC RECEPTOR DYSFUNCTION | DEPLETION | MECHANISMS | MAST-CELL | tumor necrosis factor-alpha | RESPIRATORY SYSTEM | parathion | organophosphorus pesticides | ASTHMA | EXPOSURE | Receptor, Muscarinic M2 - agonists | Macrophages, Alveolar - pathology | Bronchial Hyperreactivity - immunology | Clodronic Acid - pharmacology | Apoptosis - drug effects | Parathion - toxicity | Humans | Tumor Necrosis Factor-alpha - genetics | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Bronchial Hyperreactivity - genetics | Etanercept | Interleukin-1beta - metabolism | Tumor Necrosis Factor-alpha - immunology | Immunoglobulin G - pharmacology | Female | Receptor, Muscarinic M2 - antagonists & inhibitors | Interleukin 1 Receptor Antagonist Protein - pharmacology | Macrophages, Alveolar - immunology | Interleukin-1beta - antagonists & inhibitors | Bronchial Hyperreactivity - chemically induced | Guinea Pigs | RNA, Messenger - genetics | Bronchial Hyperreactivity - pathology | Animals | Receptors, Tumor Necrosis Factor | Macrophages, Alveolar - drug effects | Macrophage Activation - drug effects | Insecticides - toxicity | Tumor Necrosis Factor-alpha - antagonists & inhibitors | tumor necrosis factor-α
Journal Article