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Nature Medicine, ISSN 1078-8956, 11/2010, Volume 16, Issue 11, pp. 1299 - 1304
Bitter taste receptors (TAS2Rs) on the tongue probably evolved to evoke signals for avoiding ingestion of plant toxins. We found expression of TAS2Rs on human... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | CONTRACTILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | RELAXATION | CELL BIOLOGY | HETEROGENEITY | LACTONES | ASTHMA | CHANNELS | MAMMALIAN TASTE | EXPRESSION | SPARKS | Asthma - metabolism | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Bronchi - physiopathology | Myocytes, Smooth Muscle - pathology | RNA, Messenger - metabolism | Airway Obstruction - metabolism | Bronchi - drug effects | Dose-Response Relationship, Drug | Muscle, Smooth - drug effects | Muscle Relaxation - physiology | Bronchoconstriction - drug effects | Bronchi - metabolism | Bronchi - pathology | Myocytes, Smooth Muscle - metabolism | Membrane Potentials - drug effects | Bronchoconstriction - physiology | Asthma - physiopathology | Cell Separation | RNA, Messenger - genetics | Airway Obstruction - physiopathology | Muscle, Smooth - metabolism | Membrane Potentials - physiology | Taste - physiology | Gene Expression Regulation - drug effects | Animals | Taste - drug effects | Asthma - complications | Calcium Signaling - drug effects | Airway Obstruction - pathology | Muscle, Smooth - physiopathology | Mice | Receptors, G-Protein-Coupled - genetics | Muscle Relaxation - drug effects | Muscle, Smooth - pathology | Saccharin - pharmacology | Taste buds | Care and treatment | Usage | Animal models in research | Physiological aspects | Smooth muscle | Genetic aspects | Airway obstruction (Medicine) | Research | Risk factors | Signal transduction | Taste | Calcium | Cellular biology | Respiratory diseases
Journal Article
Pharmacology and Therapeutics, ISSN 0163-7258, 09/2016, Volume 165, pp. 14 - 25
Smooth muscle cells provide crucial contractile functions in visceral, vascular, and lung tissues. The contractile state of smooth muscle is largely determined... 
Kv7 channel | Visceral smooth muscle | KCNQ | Vascular smooth muscle | Voltage-gated potassium channel | Airway smooth muscle | DRUG-THERAPY | KCNE-BETA-SUBUNITS | CHROMANOL 293B | DOWN-REGULATION | K(V)7 CHANNELS | FUNCTIONAL-CHARACTERIZATION | I-KS | RETIGABINE EZOGABINE | RECTIFIER K+ CURRENT | PHARMACOLOGY & PHARMACY | CEREBRAL VASOSPASM | Bronchodilator Agents - therapeutic use | Muscle, Smooth, Vascular - metabolism | Humans | Potassium Channel Blockers - therapeutic use | Molecular Targeted Therapy | Bronchoconstrictor Agents - therapeutic use | Muscle, Smooth, Vascular - physiopathology | Potassium Channel Blockers - adverse effects | KCNQ Potassium Channels - antagonists & inhibitors | Muscle, Smooth - drug effects | Bronchoconstriction - drug effects | Drug Design | Viscera - physiopathology | Muscle, Smooth, Vascular - drug effects | Vasoconstrictor Agents - pharmacology | Viscera - metabolism | Vasodilator Agents - pharmacology | KCNQ Potassium Channels - agonists | Viscera - drug effects | Muscle, Smooth - metabolism | Vasoconstriction - drug effects | Animals | Signal Transduction - drug effects | KCNQ Potassium Channels - metabolism | Respiratory System - metabolism | Muscle, Smooth - physiopathology | Respiratory System - drug effects | Respiratory System - physiopathology | Vasodilation - drug effects | Smooth muscle | Potassium channels
Journal Article
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 10/2013, Volume 170, Issue 4, pp. 835 - 846
Background and Purpose AVE 0991 (AVE) is a non‐peptide compound, mimic of the angiotensin (Ang)‐(1–7) actions in many tissues and pathophysiological states.... 
airway remodelling | asthma | cytokines | angiotensin‐(1–7) | Mas receptor | AVE 0991 | allergic lung inflammation | angiotensin-(1-7) | ARRIVE GUIDELINES | AVE-0991 | SENSITIZED RATS | AIRWAY | INFLAMMATION | EOSINOPHIL | MOUSE MODEL | PHARMACOLOGY & PHARMACY | RECEPTOR MAS | ACCUMULATION | EXPRESSION | Asthma - metabolism | Pulmonary Veins - drug effects | Receptors, G-Protein-Coupled - metabolism | Bronchoalveolar Lavage Fluid - immunology | Male | Peptide Fragments - pharmacology | Pulmonary Veins - metabolism | Molecular Mimicry | Pulmonary Artery - metabolism | Time Factors | Bronchoconstriction - drug effects | Asthma - immunology | Pulmonary Veins - immunology | Angiotensin I - pharmacology | Lung - metabolism | Receptors, G-Protein-Coupled - drug effects | Hypertrophy, Right Ventricular - metabolism | Disease Models, Animal | Proto-Oncogene Proteins - metabolism | Anti-Asthmatic Agents - pharmacology | Ovalbumin | Proto-Oncogene Proteins - drug effects | Angiotensin II - metabolism | Asthma - physiopathology | Cytokines - metabolism | Pulmonary Veins - physiopathology | Imidazoles - pharmacology | Lung - physiopathology | Pulmonary Artery - physiopathology | Pulmonary Artery - drug effects | Asthma - drug therapy | Asthma - chemically induced | Animals | Lung - drug effects | Hypertrophy, Right Ventricular - prevention & control | Mice | Mice, Inbred BALB C | Airway Remodeling - drug effects | Lung - blood supply | Chronic Disease | Lung - immunology | Pulmonary Artery - immunology | Analysis | Angiotensin | Asthma | Research Papers | angiotensin-(1–7)
Journal Article
Respiratory Research, ISSN 1465-9921, 03/2015, Volume 16, Issue 1, p. 42
Background and purpose: The persistent influx of neutrophils into the lung and subsequent tissue damage are characteristics of COPD, cystic fibrosis and acute... 
Adhesion molecules | VAP-1/SSAO | Lung inflammation | Neutrophils | COPD | INDUCED ASTHMA | RECRUITMENT | VASCULAR ADHESION PROTEIN-1 | AIRWAY INFLAMMATION | SENSITIVE AMINE OXIDASE | CXCR2 ANTAGONIST | INDUCED LUNG INFLAMMATION | RESPIRATORY SYSTEM | IN-VIVO | INFECTION | CYSTIC-FIBROSIS | Respiratory Tract Infections - enzymology | Cell Adhesion Molecules - antagonists & inhibitors | Rhinovirus - pathogenicity | Cecum - microbiology | Picornaviridae Infections - virology | Lipopolysaccharides | Lung - enzymology | Cecum - surgery | Bronchoconstriction - drug effects | Asthma - immunology | Pneumonia - immunology | Respiratory Tract Infections - etiology | Asthma - virology | Neutrophil Infiltration - drug effects | Disease Models, Animal | Klebsiella Infections - enzymology | Asthma - physiopathology | Anti-Inflammatory Agents - pharmacology | Pneumonia - enzymology | Asthma - enzymology | Allylamine - analogs & derivatives | Enzyme Inhibitors - pharmacology | Lung - physiopathology | Cell Adhesion Molecules - metabolism | Picornaviridae Infections - enzymology | Lung - drug effects | Respiratory Tract Infections - immunology | Picornaviridae Infections - physiopathology | Mice, Inbred BALB C | Endothelial Cells - enzymology | Leukocyte Rolling - drug effects | Rats, Wistar | Benzamides - pharmacokinetics | Picornaviridae Infections - drug therapy | Dose-Response Relationship, Drug | Ligation | Amine Oxidase (Copper-Containing) - antagonists & inhibitors | Enzyme Inhibitors - pharmacokinetics | Allylamine - pharmacology | Anti-Inflammatory Agents - pharmacokinetics | Benzamides - pharmacology | Klebsiella Infections - drug therapy | Picornaviridae Infections - immunology | Pneumonia - etiology | Mice, Inbred C57BL | Punctures | Respiratory Tract Infections - drug therapy | Klebsiella Infections - microbiology | Asthma - drug therapy | Amine Oxidase (Copper-Containing) - metabolism | Endothelial Cells - immunology | Animals | Pneumonia - drug therapy | Klebsiella Infections - immunology | Klebsiella pneumoniae - pathogenicity | Allylamine - pharmacokinetics | Lung - immunology | Endothelial Cells - drug effects | Health aspects | Analysis | Endothelium | VAP-1 | SSAO | Research
Journal Article
Journal Article
Journal Article
Journal Article
Immunological Reviews, ISSN 0105-2896, 07/2011, Volume 242, Issue 1, pp. 205 - 219
The adoption of the concept that asthma is primarily a disease most frequently associated with elaboration of T‐helper 2 (Th2)‐type inflammation has led to the... 
remodeling | environmental injury | phenotypes | inflammation | origins | progression | asthma | epithelium | Progression | Origins | Phenotypes | Environmental injury | Inflammation | Epithelium | Remodeling | Asthma | RETICULAR BASEMENT-MEMBRANE | CHEMOKINE RECEPTOR 4 | DENDRITIC CELLS | INCREASED EXPRESSION | IMMUNOLOGY | CHILDHOOD ASTHMA | THYMIC STROMAL LYMPHOPOIETIN | TIGHT JUNCTIONS | EPIDERMAL-GROWTH-FACTOR | GOBLET CELL HYPERPLASIA | BRONCHIAL-ASTHMA | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Hypersensitivity - immunology | Infant | Th2 Cells - immunology | Rhinovirus - physiology | Hypersensitivity - drug therapy | Airway Remodeling - immunology | Picornaviridae Infections - virology | Epithelium - immunology | Signal Transduction - immunology | Allergens - immunology | Asthma - immunology | Anti-Inflammatory Agents - therapeutic use | Adult | Respiratory Tract Infections - virology | Rhinovirus - immunology | Child | Respiratory System - immunology | Cytokines - immunology | Adaptive Immunity | Epithelium - drug effects | Picornaviridae Infections - immunology | Asthma - physiopathology | Epithelium - metabolism | Inflammation - immunology | Immunity, Innate | Asthma - drug therapy | Th2 Cells - metabolism | Mice, Knockout | Animals | Hypersensitivity - physiopathology | Epithelial Cells - immunology | Respiratory System - drug effects | Respiratory Tract Infections - immunology | Respiratory System - physiopathology | Mice | Mucus - immunology | Allergens | Allergy | Dendritic cells | Asthma in children | Development and progression | Disease susceptibility | Health aspects | Allergic reaction | Hypersensitivity | Helper cells | Lymphocytes T | Atopy | Respiratory tract | Bronchoconstriction | Respiratory tract diseases | Microenvironments | Children | Pollutants | Growth factors | Adoption
Journal Article