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1. Full Text The genetic landscape of mutations in Burkitt lymphoma
by Love, Cassandra and Sun, Zhen and Jima, Dereje and Li, Guojie and Zhang, Jenny and Miles, Rodney and Richards, Kristy L and Dunphy, Cherie H and Choi, William L and Srivastava, Gopesh and Lugar, Patricia L and Rizzieri, David A and Lagoo, Anand S and Bernal-Mizrachi, Leon and Mann, Karen P and Flowers, Christopher R and Naresh, Kikkeri N and Evens, Andrew M and Chadburn, Amy and Gordon, Leo I and Czader, Magdalena B and Gill, Javed I and Hsi, Eric D and Greenough, Adrienne and Moffitt, Andrea B and McKinney, Matthew and Banerjee, Anjishnu and Grubor, Vladimir and Levy, Shawn and Dunson, David B and Dave, Sandeep S
Nature Genetics, ISSN 1061-4036, 12/2012, Volume 44, Issue 12, pp. 1321 - 1325
Burkitt lymphoma is characterized by deregulation of MYC, but the contribution of other genetic mutations to the disease is largely unknown. Here, we describe... 
B-CELL LYMPHOMA | FREQUENT | AMPLIFICATION | GENETICS & HEREDITY | HUMAN EXOMES | EXPRESSION | CANCER | C-MYC ONCOGENE | SOMATIC MUTATIONS | GENOME | REVEALS | Translocation, Genetic | Glutamate Formimidoyltransferase - genetics | Ammonia-Lyases - genetics | Membrane Proteins - genetics | Humans | GTP-Binding Protein alpha Subunits, G12-G13 - genetics | Inhibitor of Differentiation Proteins - genetics | Molecular Sequence Data | Transcription Factors - genetics | Sequence Analysis, DNA | Homeodomain Proteins - genetics | Chaperonin Containing TCP-1 - genetics | Base Sequence | Genes, myc - genetics | Burkitt Lymphoma - genetics | Cell Line, Tumor | Mutation | Genome, Human | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | DNA Helicases - genetics | Lymphoma, Large B-Cell, Diffuse - genetics | Proto-Oncogene Proteins c-ret - genetics | Usage | Gene mutations | Exome sequencing | Development and progression | Diagnosis | Research | Health aspects | Risk factors | Burkitt's lymphoma | Genetics | Lymphomas | Cancer | Index Medicus
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2. Full Text B-cell Lymphomas with Concurrent IGH-BCL2 and MYC Rearrangements Are Aggressive Neoplasms with Clinical and Pathologic Features Distinct from Burkitt Lymphoma and Diffuse Large B-cell Lymphoma
by Snuderl, Matija and Kolman, Olga K and Chen, Yi-Bin and Hsu, Jessie J and Ackerman, Adam M and Cin, Paola Dal and Ferry, Judith A and Harris, Nancy Lee and Hasserjian, Robert P and Zukerberg, Lawrence R and Abramson, Jeremy S and Hochberg, Ephraim P and Lee, Hang and Lee, Alfred I and Toomey, Christiana E and Sohani, Aliyah R
American Journal of Surgical Pathology, ISSN 0147-5185, 03/2010, Volume 34, Issue 3, pp. 327 - 340
B-cell lymphomas with concurrent IGH-BCL2 and MYC rearrangements, also known as "double-hit'' lymphomas (DHL), are rare neoplasms characterized by highly... 
BCL2 | Cytogenetics | High-grade B-cell lymphoma | MYC | Burkitt lymphoma | Diffuse large B-cell lymphoma | high-grade B-cell lymphoma | SURGERY | MALIGNANT-LYMPHOMAS | TRANSLOCATIONS | T(14/18) | C-MYC | FOLLICULAR LYMPHOMA | diffuse large B-cell lymphoma | PATHOLOGY | cytogenetics | POLYMERASE-CHAIN-REACTION | POOR-PROGNOSIS | ACUTE LYMPHOBLASTIC-LEUKEMIA | NON-HODGKINS-LYMPHOMA | OF-THE-LITERATURE | Predictive Value of Tests | Humans | Lymphoma, B-Cell - therapy | Middle Aged | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | Male | Lymphoma, B-Cell - genetics | Burkitt Lymphoma - mortality | Burkitt Lymphoma - pathology | Genes, Immunoglobulin Heavy Chain | Young Adult | Lymphoma, Large B-Cell, Diffuse - therapy | Time Factors | Karyotyping | Polymerase Chain Reaction | Aged, 80 and over | Burkitt Lymphoma - genetics | Adult | Female | Retrospective Studies | Child | Burkitt Lymphoma - therapy | Lymphoma, B-Cell - mortality | World Health Organization | Burkitt Lymphoma - classification | Lymphoma, Large B-Cell, Diffuse - pathology | Risk Assessment | Kaplan-Meier Estimate | Proportional Hazards Models | Antineoplastic Combined Chemotherapy Protocols | Terminology as Topic | Immunophenotyping | In Situ Hybridization, Fluorescence | Treatment Outcome | Lymphoma, B-Cell - classification | Lymphoma, Large B-Cell, Diffuse - mortality | Gene Rearrangement, B-Lymphocyte, Heavy Chain | Lymphoma, Large B-Cell, Diffuse - classification | Adolescent | Lymphoma, B-Cell - pathology | Aged | Proto-Oncogene Proteins c-myc - genetics | Neoplasm Staging | Lymphoma, Large B-Cell, Diffuse - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Index Medicus
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3. Full Text Lymphomas with concurrent BCL2 and MYC translocations: The critical factors associated with survival
by Johnson, Nathalie A and Savage, Kerry J and Ludkovski, Olga and Ben-Neriah, Susana and Woods, Ryan and Steidl, Christian and Dyer, Martin J. S and Siebert, Reiner and Kuruvilla, John and Klasa, Richard and Connors, Joseph M and Gascoyne, Randy D and Horsman, Douglas E
Blood, ISSN 0006-4971, 2009, Volume 114, Issue 11, pp. 2273 - 2279
BCL2 and MYC are oncogenes commonly deregulated in lymphomas. Concurrent BCL2 and MYC translocations (BCL2(+)/MYC+) were identified in 54 samples by karyotype... 
CHROMOSOMAL ALTERATIONS | B-CELL LYMPHOMA | BURKITT-LYMPHOMA | SOUTHWEST-ONCOLOGY-GROUP | CLINICAL PRESENTATION | MALIGNANT-LYMPHOMAS | NON-HODGKINS-LYMPHOMAS | C-MYC | FOLLICULAR LYMPHOMA | HEMATOLOGY | ELDERLY-PATIENTS | Translocation, Genetic | Follow-Up Studies | Humans | Middle Aged | Male | Antineoplastic Agents - administration & dosage | Lymphoma, Large B-Cell, Diffuse - metabolism | Burkitt Lymphoma - mortality | Proto-Oncogene Proteins c-bcl-2 - metabolism | Burkitt Lymphoma - genetics | Adult | Female | Retrospective Studies | Gene Expression Regulation, Neoplastic - genetics | Burkitt Lymphoma - drug therapy | Databases, Factual | Chromosomes, Human, Pair 18 - metabolism | Antibodies, Monoclonal, Murine-Derived | Lymphoma, Large B-Cell, Diffuse - drug therapy | Rituximab | Burkitt Lymphoma - metabolism | Survival Rate | Chromosomes, Human, Pair 14 - metabolism | Proto-Oncogene Proteins c-myc - metabolism | Lymphoma, Large B-Cell, Diffuse - mortality | Disease-Free Survival | Antibodies, Monoclonal - administration & dosage | Aged | Proto-Oncogene Proteins c-myc - genetics | Chromosomes, Human, Pair 18 - genetics | Lymphoma, Large B-Cell, Diffuse - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Chromosomes, Human, Pair 14 - genetics | Index Medicus | Abridged Index Medicus | Lymphoid Neoplasia
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4. Full Text Oncogenically active MYD88 mutations in human lymphoma
by Ngo, Vu N and Young, Ryan M and Schmitz, Roland and Jhavar, Sameer and Xiao, Wenming and Lim, Kian-Huat and Kohlhammer, Holger and Xu, Weihong and Yang, Yandan and Zhao, Hong and Shaffer, Arthur L and Romesser, Paul and Wright, George and Powell, John and Rosenwald, Andreas and Muller-Hermelink, Hans Konrad and Ott, German and Gascoyne, Randy D and Connors, Joseph M and Rimsza, Lisa M and Campo, Elias and Jaffe, Elaine S and Delabie, Jan and Smeland, Erlend B and Fisher, Richard I and Braziel, Rita M and Tubbs, Raymond R and Cook, J.R and Weisenburger, Denny D and Chan, Wing C and Staudt, Louis M
Nature, ISSN 0028-0836, 02/2011, Volume 470, Issue 7332, pp. 115 - 121
The activated B-cell-like (ABC) subtype of diffuse large B-cell lymphoma (DLBCL) remains the least curable form of this malignancy despite recent advances in... 
TNFAIP3 A20 | PATHWAYS | SURVIVAL | B-CELL LYMPHOMA | GENE | SIGNATURES | SUBTYPES | MULTIDISCIPLINARY SCIENCES | KINASE | INHIBITORS | NF-KAPPA-B | Interleukin-1 Receptor-Associated Kinases - metabolism | Phosphorylation | Interleukin-1 Receptor-Associated Kinases - genetics | Interleukin-1 Receptor-Associated Kinases - biosynthesis | Humans | Molecular Sequence Data | NF-kappa B - metabolism | Janus Kinases - metabolism | Myeloid Differentiation Factor 88 - chemistry | RNA Interference | Burkitt Lymphoma - genetics | Toll-Like Receptors - metabolism | STAT3 Transcription Factor - metabolism | Protein Structure, Tertiary | Amino Acid Sequence | Oncogenes - genetics | Cytokines - metabolism | Lymphoma, Large B-Cell, Diffuse - pathology | Signal Transduction | Cell Survival | Cytokines - secretion | Mutant Proteins - genetics | Myeloid Differentiation Factor 88 - genetics | Mutant Proteins - metabolism | Mutation - genetics | Lymphoma, B-Cell, Marginal Zone - genetics | Receptors, Interleukin-1 - metabolism | Sequence Analysis, RNA | Lymphoma, Large B-Cell, Diffuse - classification | Mutant Proteins - chemistry | Cell Line, Tumor | Hydrophobic and Hydrophilic Interactions | High-Throughput Nucleotide Sequencing | Lymphoma, Large B-Cell, Diffuse - genetics | Myeloid Differentiation Factor 88 - metabolism | Amino Acid Substitution | Lymphomas | Genetic aspects | Models | Gene mutations | Observations | Abnormalities | Mutation | Kinases | Toxicity | Apoptosis | Tumors | Index Medicus
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5. Full Text Recurrent mutation of the ID3 gene in Burkitt lymphoma identified by integrated genome, exome and transcriptome sequencing
by Richter, Julia and Schlesner, Matthias and Hoffmann, Steve and Kreuz, Markus and Leich, Ellen and Burkhardt, Birgit and Rosolowski, MacIej and Ammerpohl, Ole and Wagener, Rabea and Bernhart, Stephan H and Lenze, Dido and Szczepanowski, Monika and Paulsen, Maren and Lipinski, Simone and Russell, Robert B and Adam-Klages, Sabine and Apic, Gordana and Claviez, Alexander and Hasenclever, Dirk and Hovestadt, Volker and Hornig, Nadine and Korbel, Jan O and Kube, Dieter and Langenberger, David and Lawerenz, Chris and Lisfeld, Jasmin and Meyer, Katharina and Picelli, Simone and Pischimarov, Jordan and Radlwimmer, Bernhard and Rausch, Tobias and Rohde, Marius and Schilhabel, Markus and Scholtysik, René and Spang, Rainer and Trautmann, Heiko and Zenz, Thorsten and Borkhardt, Arndt and Drexler, Hans G and Möller, Peter and MacLeod, Roderick A. F and Pott, Christiane and Schreiber, Stefan and Trümper, Lorenz and Loeffler, Markus and Stadler, Peter F and Lichter, Peter and Eils, Roland and Küppers, Ralf and Hummel, Michael and Klapper, Wolfram and Rosenstiel, Philip and Rosenwald, Andreas and Brors, Benedikt and Siebert, Reiner and ICGC MMML-Seq Project and the ICGC MMML-Seq Project
Nature Genetics, ISSN 1061-4036, 12/2012, Volume 44, Issue 12, pp. 1316 - 1320
Burkitt lymphoma is a mature aggressive B-cell lymphoma derived from germinal center B cells(1). Its cytogenetic hallmark is the Burkitt translocation... 
INACTIVATION | DIAGNOSIS | B-CELL LYMPHOMA | MYC | GENETICS & HEREDITY | CHRONIC LYMPHOCYTIC-LEUKEMIA | MICE | PROTEINS | CANCER | Chromosomes, Human, Pair 8 - genetics | Somatic Hypermutation, Immunoglobulin | Humans | Inhibitor of Differentiation Proteins - genetics | Molecular Sequence Data | Male | Chromosome Mapping | Transcriptome - genetics | Sequence Analysis, DNA | Genes, Immunoglobulin | Base Sequence | Genes, myc - genetics | Burkitt Lymphoma - genetics | Female | Mutation | Genome, Human | Neoplasm Proteins - genetics | Chromosomes, Human, Pair 14 - genetics | Cohort Studies | Translocation, Genetic - genetics | Usage | Prognosis | Gene mutations | Exome sequencing | Development and progression | Diagnosis | Research | Health aspects | Risk factors | Burkitt's lymphoma | Proteins | Awards & honors | DNA methylation | Lymphomas | Genomes | Index Medicus
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6. Full Text Impact of oncogene rearrangement patterns on outcomes in patients with double‐hit non‐Hodgkin lymphoma
by Landsburg, Daniel J and Petrich, Adam M and Abramson, Jeremy S and Sohani, Aliyah R and Press, Oliver and Cassaday, Ryan and Chavez, Julio C and Song, Kevin and Zelenetz, Andrew D and Gandhi, Mitul and Shah, Namrata and Fenske, Timothy S and Jaso, Jesse and Medeiros, L. Jeffrey and Yang, David T and Nabhan, Chadi
Cancer, ISSN 0008-543X, 02/2016, Volume 122, Issue 4, pp. 559 - 564
To the authors' knowledge, the current study is the first large comparative analysis of double‐hit lymphomas by patterns of oncogene rearrangement. Patterns of... 
BCL6 (B‐cell lymphoma 6) | non‐Hodgkin lymphoma | gene rearrangement | BCL2 (B‐cell lymphoma 2) | MYC | BCL6 (B-cell lymphoma 6) | non-Hodgkin lymphoma | BCL2 (B-cell lymphoma 2) | RITUXIMAB PLUS CYCLOPHOSPHAMIDE | TRANSLOCATIONS | IN-VIVO EFFICACY | DOXORUBICIN | B-CELL LYMPHOMA | PREDNISONE | BCL6 | ONCOLOGY | VINCRISTINE | EXPRESSION | Doxorubicin - therapeutic use | Prognosis | Cytarabine - therapeutic use | Humans | Middle Aged | Male | Methotrexate - therapeutic use | Bone Marrow Neoplasms - genetics | Lymphoma, B-Cell - genetics | Cyclophosphamide - therapeutic use | Lymphoma, Non-Hodgkin - pathology | Burkitt Lymphoma - pathology | Burkitt Lymphoma - genetics | Female | Burkitt Lymphoma - drug therapy | Databases, Factual | Lymphoma, B-Cell - drug therapy | Lymphoma, Non-Hodgkin - drug therapy | Ifosfamide - therapeutic use | Lymphoma, Large B-Cell, Diffuse - drug therapy | Lymphoma, Large B-Cell, Diffuse - pathology | Central Nervous System Neoplasms - genetics | Etoposide - therapeutic use | Survival Rate | Germinal Center - pathology | Lymphoma, Non-Hodgkin - genetics | DNA-Binding Proteins - genetics | Genes, bcl-2 - genetics | Rituximab - administration & dosage | Central Nervous System Neoplasms - pathology | Dexamethasone - therapeutic use | Bone Marrow Neoplasms - pathology | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Gene Rearrangement | Genes, myc - genetics | Lymphoma, B-Cell - pathology | Vincristine - therapeutic use | Bone Marrow Neoplasms - drug therapy | Central Nervous System Neoplasms - drug therapy | Lymphoma, Large B-Cell, Diffuse - genetics | Prednisone - therapeutic use | Cohort Studies | Proto-Oncogene Proteins c-bcl-6 | Care and treatment | Chemotherapy | Usage | Non-Hodgkin's lymphomas | Diagnosis | Research | Oncogenes | Cancer | Index Medicus | Abridged Index Medicus
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7. Full Text The genomic landscape of mantle cell lymphoma is related to the epigenetically determined chromatin state of normal B cells
by Zhang, Jenny and Jima, Dereje and Moffitt, Andrea B and Liu, Qingquan and Czader, Magdalena and Hsi, Eric D and Fedoriw, Yuri and Dunphy, Cherie H and Richards, Kristy L and Gill, Javed I and Sun, Zhen and Love, Cassandra and Scotland, Paula and Lock, Eric and Levy, Shawn and Hsu, David S and Dunson, David and Dave, Sandeep S
Blood, ISSN 0006-4971, 05/2014, Volume 123, Issue 19, pp. 2988 - 2996
In this study, we define the genetic landscape of mantle cell lymphoma (MCL) through exome sequencing of 56 cases of MCL. We identified recurrent mutations in... 
BURKITT-LYMPHOMA | GERMINAL-CENTER FORMATION | TRANSCRIPTOME | VARIANTS | SIGNATURES | DNA | GENE-EXPRESSION | SEQUENCING DATA | HEMATOLOGY | SOMATIC MUTATIONS | HUMAN CANCER | Chromatin - metabolism | Epigenesis, Genetic | Genomics | Humans | Lymphoma, Mantle-Cell - pathology | Gene Regulatory Networks | Tumor Suppressor Protein p53 - genetics | Burkitt Lymphoma - pathology | Telomere-Binding Proteins - genetics | Burkitt Lymphoma - genetics | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | DNA Helicases - genetics | Lymphoma, Mantle-Cell - genetics | B-Lymphocytes - metabolism | Histone-Lysine N-Methyltransferase - genetics | Repressor Proteins - genetics | Germinal Center - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Sequence Analysis, DNA | Exome - genetics | Germinal Center - metabolism | Histones - genetics | Cyclin D1 - genetics | Retinoblastoma Protein - genetics | Histones - metabolism | Mutation | Ataxia Telangiectasia Mutated Proteins - genetics | Methylation | Chromatin - genetics | Index Medicus | Abridged Index Medicus | Lymphoid Neoplasia
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8. Full Text A Biologic Definition of Burkitt's Lymphoma from Transcriptional and Genomic Profiling
by Hummel, Michael and Bentink, Stefan and Berger, Hilmar and Klapper, Wolfram and Wessendorf, Swen and Barth, Thomas F.E and Bernd, Heinz-Wolfram and Cogliatti, Sergio B and Dierlamm, Judith and Feller, Alfred C and Hansmann, Martin-Leo and Haralambieva, Eugenia and Harder, Lana and Hasenclever, Dirk and Kühn, Michael and Lenze, Dido and Lichter, Peter and Martin-Subero, Jose Ignacio and Möller, Peter and Müller-Hermelink, Hans-Konrad and Ott, German and Parwaresch, Reza M and Pott, Christiane and Rosenwald, Andreas and Rosolowski, Maciej and Schwaenen, Carsten and Stürzenhofecker, Benjamin and Szczepanowski, Monika and Trautmann, Heiko and Wacker, Hans-Heinrich and Spang, Rainer and Loeffler, Markus and Trümper, Lorenz and Stein, Harald and Siebert, Reiner and Molecular Mechanisms Malignant Lym and Molecular Mechanisms in Malignant Lymphomas Network Project of the Deutsche Krebshilfe
The New England Journal of Medicine, ISSN 0028-4793, 06/2006, Volume 354, Issue 23, pp. 2419 - 2430
Analysis of gene-expression patterns led to the development of a molecular definition of Burkitt's lymphoma that distinguishes it from other types of mature... 
SURVIVAL | MEDICINE, GENERAL & INTERNAL | B-CELL LYMPHOMA | TRANSLOCATIONS | C-MYC | NON-HODGKINS-LYMPHOMA | CLASSIFICATION | LEUKEMIA | SUBGROUPS | CYTOGENETICS | CHEMOTHERAPY | Translocation, Genetic | Prognosis | Follow-Up Studies | Oligonucleotide Array Sequence Analysis | Humans | Lymphoma, B-Cell - diagnosis | Middle Aged | Male | Gene Expression Profiling | Genes, myc | Lymphoma, B-Cell - genetics | Burkitt Lymphoma - pathology | RNA, Neoplasm - analysis | Burkitt Lymphoma - genetics | Female | Transcription, Genetic | DNA, Neoplasm - analysis | Diagnosis, Differential | Gene Expression | Lymphoma, B-Cell - mortality | Survival Rate | Burkitt Lymphoma - diagnosis | Genes, bcl-2 | Lymphoma, B-Cell - classification | Genes, Immunoglobulin | Algorithms | Oncogene Proteins, Fusion - genetics | Genetic aspects | Genetic transcription | Research | Burkitt's lymphoma | Medical research | Chemotherapy | Lymphomas | Gene expression | Index Medicus | Abridged Index Medicus
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9. Full Text Burkitt lymphoma pathogenesis and therapeutic targets from structural and functional genomics
by Schmitz, Roland and Young, Ryan M and Ceribelli, Michele and Jhavar, Sameer and Xiao, Wenming and Zhang, Meili and Wright, George and Shaffer, Arthur L and Hodson, Daniel J and Buras, Eric and Liu, Xuelu and Powell, John and Yang, Yandan and Xu, Weihong and Zhao, Hong and Kohlhammer, Holger and Rosenwald, Aneas and Kluin, Philip and Mueller-Hermelink, Hans Konrad and Ott, German and Gascoyne, Randy D and Connors, Joseph M and Rimsza, Lisa M and Campo, Elias and Jaffe, Elaine S and Delabie, Jan and Smeland, Erlend B and Ogwang, Martin D and Reynolds, Steven J and Fisher, Richard I and Braziel, Rita M and Tubbs, Raymond R and Cook, James R and Weisenburger, Dennis D and Chan, Wing C and Pittaluga, Stefania and Wilson, Wyndham and Waldmann, Thomas A and Rowe, Martin and Mbulaiteye, Sam M and Rickinson, Alan B and Staudt, Louis M
Nature, ISSN 0028-0836, 10/2012, Volume 490, Issue 7418, pp. 116 - 120
Burkitt's lymphoma (BL) can often be cured by intensive chemotherapy, but the toxicity of such therapy precludes its use in the elderly and in patients with... 
IMMUNOGLOBULIN | B-CELL LYMPHOMA | PROTEINS | MULTIDISCIPLINARY SCIENCES | REGION | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Genomics | Humans | Inhibitor of Differentiation Proteins - genetics | Burkitt Lymphoma - metabolism | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Cyclin-Dependent Kinase 6 - metabolism | Inhibitor of Differentiation Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Receptors, Antigen, B-Cell - metabolism | Molecular Targeted Therapy | Neoplasm Proteins - metabolism | Burkitt Lymphoma - pathology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Cell Cycle | RNA Interference | Cyclin D3 - genetics | Genes, myc - genetics | Burkitt Lymphoma - genetics | High-Throughput Nucleotide Sequencing | Neoplasm Proteins - genetics | Burkitt Lymphoma - drug therapy | Cyclin D3 - metabolism | RNA sequencing | Usage | Care and treatment | Gene mutations | Observations | Identification and classification | Burkitt's lymphoma | Chemotherapy | Pathogenesis | Toxicity | Mutation | Kinases | Crystal structure | Index Medicus
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10. Full Text B-cell lymphoma patient-derived xenograft models enable drug discovery and are a platform for personalized therapy
by Zhang, Leo and Nomie, Krystle and Zhang, Hui and Bell, Taylor and Pham, Lan and Kadri, Sabah and Segal, Jeremy and Li, Shaoying and Zhou, Shouhao and Santos, David and Richard, Shawana and Sharma, Shruti and Chen, Wendy and Oriabure, Onyekachukwu and Liu, Yang and Huang, Shengjian and Guo, Hui and Chen, Zhihong and Tao, Wenjing and Li, Carrie and Wang, Jack and Fang, Bingliang and Wang, Jacqueline and Li, Lei and Badillo, Maria and Ahmed, Makhdum and Thirumurthi, Selvi and Huang, Steven Y and Shao, Yiping and Lam, Laura and Yi, Qing and Wang, Y. Lynn and Wang, Michael
Clinical Cancer Research, ISSN 1078-0432, 08/2017, Volume 23, Issue 15, pp. 4212 - 4223
Purpose: Patients with B-cell lymphomas often relapse after frontline therapy, and novel therapies are urgently needed to provide long-term remission. We... 
ONCOLOGY | RITUXIMAB | BONE-MARROW | INVOLVEMENT | IBRUTINIB | RESISTANCE | NON-HODGKIN-LYMPHOMA | CANCER | CHEMOTHERAPY | TRANSPLANTATION | FEATURES | Lymphoma, Follicular - drug therapy | Humans | Lymphoma, Mantle-Cell - pathology | Protein Kinase Inhibitors - adverse effects | Burkitt Lymphoma - pathology | Protein-Tyrosine Kinases - genetics | Lymphoma, Follicular - genetics | Burkitt Lymphoma - genetics | Lymphoma, B-Cell, Marginal Zone - pathology | Gene Expression Regulation, Neoplastic - drug effects | Lymphoma, Mantle-Cell - genetics |