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Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 17763 - 11
We investigated the effect of a peroxisome proliferator-activated receptor alpha (PPAR?alpha) agonist ophthalmic solution in wound healing using a rat corneal... 
GAMMA | PPAR-ALPHA | CELLS | LIPID-METABOLISM | COLLAGEN | TIE2 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | FACTOR-KAPPA-B | EXPRESSION | BASEMENT-MEMBRANE | Eye Burns - drug therapy | Ophthalmic Solutions - pharmacology | Burns, Chemical - metabolism | Corneal Neovascularization - drug therapy | Collagen Type III - metabolism | Male | Angiopoietin-1 - metabolism | RNA, Messenger - metabolism | Angiopoietin-2 - metabolism | Corneal Injuries - metabolism | Myofibroblasts - metabolism | Cicatrix - metabolism | Fenofibrate - pharmacology | Cornea - drug effects | Alkalies - pharmacology | Neutrophils - metabolism | Wound Healing - drug effects | Disease Models, Animal | Endothelial Cells - metabolism | Neutrophils - drug effects | Rats | Myofibroblasts - drug effects | Up-Regulation - drug effects | Cornea - metabolism | Cicatrix - drug therapy | Macrophages - metabolism | Animals | Corneal Neovascularization - metabolism | Neovascularization, Pathologic - drug therapy | Burns, Chemical - drug therapy | Eye Burns - metabolism | Macrophages - drug effects | PPAR alpha - agonists | Vascular Endothelial Growth Factors - metabolism | Neovascularization, Pathologic - metabolism | Corneal Injuries - drug therapy | Cornea | Wound healing | Angiopoietin | Stroma | Collagen (type III) | Leukocytes (neutrophilic) | Inflammation | Gene expression | Macrophages | Endothelial cells | Vascularization | Fenofibrate | Rodents | Vascular endothelial growth factor
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 08/2016, Volume 311, Issue 2, pp. E436 - E448
Burn trauma results in prolonged hypermetabolism and skeletal muscle wasting. How hypermetabolism contributes to muscle wasting in burn patients remains... 
Hypermetabolism-induced oxidative stress | Mitochondria proteases | Cachexia | Burn injury | Mitochondrial unfolded protein response | Up-Regulation | ATP-Dependent Proteases - genetics | Oxidative Stress | TOR Serine-Threonine Kinases - metabolism | Humans | Middle Aged | Mitochondria, Muscle - metabolism | Male | Multiprotein Complexes - genetics | Muscle, Skeletal - metabolism | Mitochondrial Proteins - genetics | RNA, Messenger - metabolism | Case-Control Studies | Mechanistic Target of Rapamycin Complex 1 | Mitochondrial Membrane Transport Proteins - genetics | Young Adult | Multiprotein Complexes - metabolism | TOR Serine-Threonine Kinases - genetics | Mitochondrial Proteins - metabolism | Proteolysis | Cachexia - etiology | Muscle Proteins - metabolism | Adult | Burns - metabolism | Female | Body Surface Area | Burns - complications | Cachexia - metabolism | Real-Time Polymerase Chain Reaction | Mitochondrial Membrane Transport Proteins - metabolism | Cachexia - genetics | Oxygen Consumption | Endopeptidase Clp - genetics | Endopeptidase Clp - metabolism | Mitochondria - metabolism | Metabolism | Blotting, Western | Proteasome Endopeptidase Complex - genetics | Adolescent | Proteasome Endopeptidase Complex - metabolism | ATP-Dependent Proteases - metabolism | Burns - genetics | Physiological aspects | Muscles | Burns and scalds | Mitochondria | Research | Call for Papers | mitochondria proteases | hypermetabolism-induced oxidative stress | burn injury | mitochondrial unfolded protein response | cachexia
Journal Article
Burns, ISSN 0305-4179, 12/2018, Volume 44, Issue 8, pp. 1863 - 1869
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 08/2010, Volume 182, Issue 3, pp. 351 - 359
Rationale: Hyperglycemia and insulin resistance have been shown to increase morbidity and mortality in severely burned patients, and glycemic control appears... 
Burn | Sepsis | Insulin | Pediatric | Morbidity | burn | SENSITIVITY INDEXES | insulin | GLUCOSE CONTROL | SEVERE TRAUMA | pediatric | ENDOTOXEMIC RATS | TREATMENT IMPROVES | RESPIRATORY SYSTEM | LIVER-FUNCTION | SYSTEMIC INFLAMMATORY RESPONSE | ENDOPLASMIC-RETICULUM | morbidity | CRITICALLY-ILL PATIENTS | sepsis | MUSCLE CATABOLISM | CRITICAL CARE MEDICINE | Body Composition | Hypoglycemia - epidemiology | Sepsis - prevention & control | Bilirubin - metabolism | Prospective Studies | Blood Urea Nitrogen | Humans | Alkaline Phosphatase - metabolism | Male | Haptoglobins - metabolism | Hyperglycemia - drug therapy | Creatinine - metabolism | Retinol-Binding Proteins - metabolism | Interleukin-6 - blood | Female | C-Reactive Protein - analysis | Burns - complications | Child | Hypoglycemic Agents - therapeutic use | Burns - mortality | Bone Density | Complement C3 - metabolism | Insulin Resistance | Sepsis - epidemiology | Biomarkers - blood | Trauma Severity Indices | Wound Infection - epidemiology | Aspartate Aminotransferases - metabolism | alpha-Macroglobulins - metabolism | Multiple Organ Failure - prevention & control | Prealbumin - metabolism | Wound Infection - prevention & control | Hyperglycemia - etiology | Alanine Transaminase - metabolism | Blood Glucose - metabolism | Transferrin - metabolism | Insulin - therapeutic use | C. Critical Care
Journal Article
FASEB Journal, ISSN 0892-6638, 07/2011, Volume 25, Issue 7, pp. 2500 - 2508
We have recently shown that antibody-induced blockade of C5a, C5a receptors, or IL-17A greatly reduced the harmful outcomes of sepsis. In the current study,... 
Cytokine receptors | Cytokines | IL-17A | Cardiomyopathy | C5a | CONTRACTILE DYSFUNCTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HUMAN SEPTIC SHOCK | CARDIAC DYSFUNCTION | NECROSIS-FACTOR-ALPHA | cytokine receptors | CELL BIOLOGY | HEART | BURN TRAUMA | CALCIUM | BIOLOGY | MITOCHONDRIAL DYSFUNCTION | cytokines | TNF-ALPHA | cardiomyopathy | Receptor, Anaphylatoxin C5a - genetics | Tumor Necrosis Factor-alpha - metabolism | Chemokines - blood | Tumor Necrosis Factor-alpha - blood | Complement C5a - metabolism | Male | RNA, Messenger - metabolism | Interleukin-6 - blood | Sepsis - metabolism | Chemokine CCL3 - blood | Inflammation Mediators - metabolism | Chemokine CCL3 - metabolism | Receptors, Cytokine - metabolism | Receptors, Chemokine - genetics | Chemokine CXCL2 - metabolism | Interleukin-6 - metabolism | Receptors, Cytokine - genetics | Cytokines - blood | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Mice, Inbred C57BL | RNA, Messenger - genetics | Receptors, Chemokine - metabolism | Inflammation Mediators - blood | Rats | Reverse Transcriptase Polymerase Chain Reaction | Chemokine CXCL2 - blood | Rats, Sprague-Dawley | Sepsis - genetics | Mice, Knockout | Interleukin-17 - metabolism | Animals | Myocytes, Cardiac - metabolism | Chemokines - metabolism | Mice | Receptor, Anaphylatoxin C5a - metabolism | Research Communications
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 02/2001, Volume 88, Issue 2, pp. 167 - 174
ABSTRACT —Bone marrow (BM)–derived circulating endothelial precursor cells (CEPs) are thought to play a role in postnatal angiogenesis. Emerging evidence... 
Mobilization | AC133 | Vascular endothelial growth factor receptor 2 | Circulating endothelial precursor cells | Vascular endothelial growth factor | PROGENITOR CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | vascular endothelial growth factor receptor 2 AC133 | ISCHEMIA | circulating endothelial precursor cells mobilization | GROWTH-FACTOR VEGF | vascular endothelial growth factor | SURFACE | PERIPHERAL VASCULAR DISEASE | GENE-TRANSFER | NEOVASCULARIZATION | HEMATOLOGY | Endothelium, Vascular - cytology | Leukocytes, Mononuclear - metabolism | Receptors, Vascular Endothelial Growth Factor | Vascular Endothelial Growth Factor A | Cadherins - metabolism | Vascular Endothelial Growth Factors | Blood Vessels - metabolism | Cell Count | Humans | Lymphokines - blood | Glycoproteins - metabolism | Stem Cells - cytology | Lewis X Antigen - metabolism | RNA, Messenger - metabolism | Stem Cells - metabolism | Receptors, Growth Factor - genetics | Flow Cytometry | Peptides - metabolism | Macrophage-1 Antigen - metabolism | Colony-Forming Units Assay | Cadherins - genetics | von Willebrand Factor - metabolism | Burns - blood | Cells, Cultured | Receptor Protein-Tyrosine Kinases - metabolism | Reverse Transcriptase Polymerase Chain Reaction | AC133 Antigen | Endothelial Growth Factors - blood | Animals | Receptor Protein-Tyrosine Kinases - genetics | Endothelium, Vascular - metabolism | Coronary Artery Bypass | Leukocytes, Mononuclear - cytology | Mice | Antigens, CD | Receptors, Growth Factor - metabolism
Journal Article
Alcohol, ISSN 0741-8329, 2008, Volume 42, Issue 5, pp. 349 - 361
Abstract This report is a summary of the symposium on Alcohol, Intestinal Bacterial Growth, Intestinal Permeability to Endotoxin, and Medical Consequences,... 
Psychiatry | Bacterial growth | Alcohol | Endotoxin | Nitric oxide | Intestinal permeability | Acetaldehyde | alcohol | ACETALDEHYDE-INDUCED INCREASE | nitric oxide | EPITHELIAL TIGHT JUNCTIONS | CROHNS-DISEASE | NITRIC-OXIDE SYNTHASE | SUBSTANCE ABUSE | PARACELLULAR PERMEABILITY | bacterial growth | INDUCED LIVER-INJURY | CHAIN KINASE EXPRESSION | intestinal permeability | INFLAMMATORY-BOWEL-DISEASE | HEPATIC STELLATE CELLS | TOLL-LIKE RECEPTOR-4 | PHARMACOLOGY & PHARMACY | TOXICOLOGY | acetaldehyde | endotoxin | Endotoxins - blood | Intestines - drug effects | Acetaldehyde - metabolism | Zinc - metabolism | Humans | Glutamine - metabolism | Intestines - metabolism | Gram-Negative Bacteria - metabolism | Bacterial Translocation - drug effects | Intestinal Mucosa - drug effects | Liver Diseases, Alcoholic - etiology | Receptor, Epidermal Growth Factor - metabolism | Ethanol - adverse effects | Burns - metabolism | Alcohol Drinking - metabolism | Avena - metabolism | Endotoxins - metabolism | Permeability | Alcohol Drinking - adverse effects | Liver Diseases, Alcoholic - metabolism | Gram-Negative Bacteria - drug effects | Animals | Intestines - microbiology | Gram-Negative Bacteria - growth & development | Probiotics - therapeutic use | Nitric Oxide - metabolism | Studies | Respiratory distress syndrome | Small intestine | Alcoholism | Rodents | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2015, Volume 10, Issue 12, p. e0143730
Severe thermal injury induces a pathophysiological response that affects most of the organs within the body; liver, heart, lung, skeletal muscle among others,... 
POLY(ADP-RIBOSE) POLYMERASE | RESPONSES | ACTIVATION | PLASMA | INHIBITION | MULTIDISCIPLINARY SCIENCES | ACUTE LUNG INJURY | HYDROGEN-SULFIDE | DAMAGE | TRAUMA | Liver - pathology | Mitochondria, Heart - metabolism | Oxidative Stress | Mitochondria, Liver - metabolism | Male | Neutrophil Infiltration | Myocardium - metabolism | Burns - metabolism | Lung - metabolism | Disease Models, Animal | Lung - pathology | DNA, Mitochondrial - metabolism | Macrophages - pathology | Liver - metabolism | Myocardium - pathology | Mitochondria - metabolism | Mitochondria - pathology | Animals | Energy Metabolism | Burns - pathology | Mice | Mice, Inbred BALB C | DNA Damage | Lipid Peroxidation | Peroxidase - metabolism | Oxidative stress | Burns and scalds | Care and treatment | DNA damage | Patient outcomes | Mitochondrial DNA | Research | Immunohistochemistry | Heart | Pathogenesis | Liver | Poly(ADP-ribose) | ADP | Inflammatory diseases | Time dependence | Mitochondria | Alterations | Bioenergetics | Thermal injury | Ribose | Rodents | Surgery | Bacteria | Peroxidase | Polymers | Injuries | Deoxyribonucleic acid--DNA | Injury analysis | Cardiac muscle | Organs | Muscles | Fragments | Inflammation | Metabolism | Fatty acids | Trauma | Bacteremia | Malondialdehyde | Skeletal muscle | Musculoskeletal system | Damage detection | Blood circulation | Lungs | Infiltration | Anesthesiology | Electron transport | Burns | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 09/2016, Volume 6, Issue 1, p. 32514
Endothelial-to-mesenchymal transition (EndMT) has been implicated in a variety of aberrant wound healing conditions. However, unambiguous evidence of EndMT has... 
TENDONS | PROGENITORS | SCLERAXIS | MULTIDISCIPLINARY SCIENCES | Hindlimb | Leukocyte Common Antigens - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - genetics | Cadherins - metabolism | Tenotomy | Endothelial Cells - transplantation | Male | Epithelial-Mesenchymal Transition - genetics | Burns, Electric - pathology | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Burns, Electric - genetics | Receptor, TIE-2 - metabolism | Cell Differentiation | Cadherins - genetics | SOX9 Transcription Factor - metabolism | Gene Expression | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Burns, Electric - metabolism | Endothelial Cells - metabolism | Mice, Inbred C57BL | Mesenchymal Stromal Cells - metabolism | Mice, Transgenic | Aggrecans - metabolism | Aggrecans - genetics | Receptor, TIE-2 - genetics | Animals | Receptor, Platelet-Derived Growth Factor alpha - genetics | Sp7 Transcription Factor - genetics | Sp7 Transcription Factor - metabolism | Leukocyte Common Antigens - genetics | Luminescent Proteins - genetics | Mice | Mesenchymal Stromal Cells - pathology | Primary Cell Culture | Endothelial Cells - pathology | SOX9 Transcription Factor - genetics | Luminescent Proteins - metabolism | Cell culture | Animal models | Wound healing | Mesenchyme | Sox9 protein | Transplantation | CD45 antigen | Endothelial cells | Arteriosclerosis | Rodents | Fibrosis | Ligands | Aggrecan
Journal Article
Cardiovascular Research, ISSN 0008-6363, 07/2010, Volume 87, Issue 2, pp. 211 - 217
Journal Article