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1. Full Text Genetic Susceptibility and Predictors of Paradoxical Reactions in Buruli Ulcer
by Barogui, Yves Thierry and Klis, Sandor-Aian and Johnson, Roch Christian and Phillips, Richard O and van der Veer, Eveline and van Diemen, Cleo and van der Werf, Tjip S and Stienstra, Ymkje
PLoS Neglected Tropical Diseases, ISSN 1935-2735, 04/2016, Volume 10, Issue 4, p. e0004594
INTRODUCTION: Buruli ulcer (BU) is the third most frequent mycobacterial disease in immunocompetent persons after tuberculosis and leprosy. During the last... 
MANAGEMENT | TUBERCULOSIS | MYCOBACTERIUM-ULCERANS | CLINICAL-EFFICACY | DISEASE | ANTIMICROBIAL TREATMENT | INFECTION | ANTIBIOTIC-TREATMENT | MYCOLACTONE | VITAMIN-D DEFICIENCY | Genetic Predisposition to Disease | Humans | Genotype | Male | Randomized Controlled Trials as Topic | Vitamin D - blood | Anti-Infective Agents - administration & dosage | Radioimmunoassay | Treatment Failure | Buruli Ulcer - genetics | Cation Transport Proteins - genetics | Female | Vitamin D - analogs & derivatives | Buruli Ulcer - pathology | Polymorphism, Single Nucleotide | Buruli Ulcer - drug therapy | Care and treatment | Vitamin D | Buruli ulcer | Genetic variation | Calcifediol | Dosage and administration | Genetic aspects | Alfacalcidol | Research | Genetic polymorphisms | Studies | Genotype & phenotype | Tuberculosis | Disease | Antibiotics | Ulcers | Leprosy | Vitamin deficiency | Health risk assessment | Localization | Tropical diseases
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2. Full Text Genetic Variation in Autophagy-Related Genes Influences the Risk and Phenotype of Buruli Ulcer
by Capela, Carlos and Dossou, Ange Dodji and Silva-Gomes, Rita and Sopoh, Ghislain Emmanuel and Makoutode, Michel and Menino, João Filipe and Fraga, Alexandra Gabriel and Cunha, Cristina and Carvalho, Agostinho and Rodrigues, Fernando and Pedrosa, Jorge
PLoS Neglected Tropical Diseases, ISSN 1935-2727, 04/2016, Volume 10, Issue 4, p. e0004671
Buruli ulcer (BU) is a severe necrotizing human skin disease caused by Mycobacterium ulcerans. Clinically, presentation is a sum of these diverse pathogenic... 
Autophagy-Related Proteins | Genetic Predisposition to Disease | Risk Assessment | Humans | Male | Nod2 Signaling Adaptor Protein - genetics | Buruli Ulcer - epidemiology | Genotyping Techniques | Mycobacterium ulcerans - immunology | Autophagy | Host-Pathogen Interactions | Young Adult | Carrier Proteins - genetics | Adolescent | Buruli Ulcer - genetics | Adult | Female | Buruli Ulcer - pathology | Polymorphism, Single Nucleotide | Ubiquitin-Protein Ligases - genetics | Child | Autophagy (Cytology) | Development and progression | Buruli ulcer | Genetic variation | Analysis | Risk factors | Haplotypes | Health care | Pathogens | Disease | Funding | Genes | Genomics | Infections | Gender | Ulcers | Skin diseases | Cytoskeleton | Influence
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3. Full Text Human genetics of mycobacterial disease
by Dallmann-Sauer, Monica and Correa-Macedo, Wilian and Schurr, Erwin
Mammalian Genome, ISSN 0938-8990, 8/2018, Volume 29, Issue 7, pp. 523 - 538
Mycobacterial diseases are caused by members of the genus Mycobacterium, acid-fast bacteria characterized by the presence of mycolic acids within their cell... 
Life Sciences | Human Genetics | Cell Biology | Animal Genetics and Genomics | COMMON VARIANTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | LEPROSY SUSCEPTIBILITY | BURULI ULCER | LOCUS | INFECTIOUS-DISEASES | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | PULMONARY TUBERCULOSIS | LINKAGE ANALYSIS | SINGLE-NUCLEOTIDE POLYMORPHISMS | RNA EXPRESSION | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease | Genome-Wide Association Study | Genetic Association Studies | Leprosy - microbiology | Humans | Mycobacterium - physiology | Tuberculosis - microbiology | Leprosy - immunology | Mycobacterium Infections - immunology | Host-Pathogen Interactions - immunology | Tuberculosis - immunology | Buruli Ulcer - immunology | Animals | Buruli Ulcer - microbiology | Host-Pathogen Interactions - genetics | Tuberculosis - genetics | Mycobacterium Infections - genetics | Buruli Ulcer - genetics | Mycobacterium Infections - microbiology | Quantitative Trait Loci | Leprosy - genetics | Genetic Linkage | Tuberculosis | Disease | Cell walls | Complications | Buruli ulcer | Leprosy | Skin diseases | Mycolic acids | Genomes | Genetic factors | Gene expression
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4. Full Text Microdeletion on chromosome 8p23.1 in a familial form of severe Buruli ulcer
by Vincent, Quentin B and Belkadi, Aziz and Fayard, Cindy and Marion, Estelle and Adeye, Ambroise and Ardant, Marie-Françoise and Johnson, Christian R and Agossadou, Didier and Lorenzo, Lazaro and Guergnon, Julien and Bole-Feysot, Christine and Manry, Jeremy and Nitschké, Patrick and Theodorou, Ioannis and Casanova, Jean-Laurent and Marsollier, Laurent and Chauty, Annick and Abel, Laurent and Alcaïs, Alexandre and Franco-Beninese Buruli Res Grp and Franco-Beninese Buruli Research Group
PLoS Neglected Tropical Diseases, ISSN 1935-2727, 04/2018, Volume 12, Issue 4, p. e0006429
Buruli ulcer (BU), the third most frequent mycobacteriosis worldwide, is a neglected tropical disease caused by Mycobacterium ulcerans. We report the clinical... 
SEQUENCING-BASED DISCOVERY | IMMUNITY | RECEPTOR BETA-1 DEFICIENCY | HUMAN BETA-DEFENSIN-2 | TUBERCULOSIS | MYCOBACTERIUM-ULCERANS | DISEASE | INFECTION | ANTIMICROBIAL PEPTIDES | EXPRESSION | PARASITOLOGY | TROPICAL MEDICINE | Benin | Chromosomes, Human, Pair 8 - genetics | Sequence Deletion | Genome-Wide Association Study | Mycobacterium ulcerans - physiology | Humans | Child, Preschool | Genotype | Male | Whole Exome Sequencing | Phenotype | Buruli Ulcer - microbiology | Pedigree | Adolescent | Buruli Ulcer - genetics | Female | Consanguinity | Genetic Linkage | Buruli ulcer | Chromosomes | Research | Laboratories | Funding | Infections | Genomes | Gene deletion | Immunology | Clonal deletion | Genetic analysis | Deletion | Genetics | Supervision | Predictive control | Linkage analysis | Cytokines | Gene expression | Ribonucleic acid--RNA | Patients | Hospitals | Tuberculosis | Infectious diseases | Genotyping | Ulcers | Chromosome 8 | Skin | Mycobacteriosis | Non-coding RNA | Life Sciences | Cancer | RNA | Ribonucleic acid
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5. Full Text Multiple Introductions and Recent Spread of the Emerging Human Pathogen Mycobacterium ulcerans across Africa
by Vandelannoote, Koen and Meehan, Conor J and Eddyani, Miriam and Affolabi, Dissou and Phanzu, Delphin Mavinga and Eyangoh, Sara and Jordaens, Kurt and Portaels, Françoise and Mangas, Kirstie and Seemann, Torsten and Marsollier, Laurent and Marion, Estelle and Chauty, Annick and Landier, Jordi and Fontanet, Arnaud and Leirs, Herwig and Stinear, Timothy P and de Jong, Bouke C
Genome biology and evolution, ISSN 1759-6653, 03/2017, Volume 9, Issue 3, pp. 414 - 426
Buruli ulcer (BU) is an insidious neglected tropical disease. Cases are reported around the world but the rural regions of West and Central Africa are most... 
bacterial pathogen transmission | microbial population genomics | molecular evolution | phylogeography | RECOMBINATION | READ ALIGNMENT | PHYLOGENETIC ANALYSIS | CAUSATIVE AGENT | GENOME | EVOLUTIONARY BIOLOGY | TRANSMISSION | DISEASE BURULI ULCER | REDUCTIVE EVOLUTION | GENETICS & HEREDITY | POPULATION-STRUCTURE | INFECTION | Genetics, Population | Genome, Bacterial | Humans | Africa | Phylogeny | Sequence Analysis, DNA | Genetic Variation | Buruli Ulcer - transmission | Buruli Ulcer - microbiology | Mycobacterium ulcerans - genetics | Mycobacterium ulcerans - pathogenicity | Buruli Ulcer - genetics | Polymorphism, Single Nucleotide | Evolution, Molecular
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6. Full Text Susceptibility to Buruli ulcer is associated with the SLC11A1 (NRAMP1) D543N polymorphism
by Stienstra, Y and Van der Werf, TS and Oosterom, E and Nolte, IM and Van der Graaf, WTA and Etuaful, S and Raghunathan, PL and Whitney, EAS and Ampadu, EO and Asamoa, K and Klutse, EY and Meerman, GJT and Tappero, JW and Ashford, DA and van der Steege, G
GENES AND IMMUNITY, ISSN 1466-4879, 04/2006, Volume 7, Issue 3, pp. 185 - 189
Similar to other mycobacterial diseases, susceptibility to Buruli ulcer (Mycobacterium ulcerans infection) may be determined by host genetic factors. We... 
GENE | Buruli ulcer | SLC11A1 | WEST AFRICANS | genetic susceptibility | NRAMP1 | UMCG Approved | LEPROSY | Genetic susceptibility | GENETICS & HEREDITY | IMMUNOLOGY | Genetic Predisposition to Disease | Gene Frequency | Humans | Skin Ulcer - genetics | Male | Skin Ulcer - microbiology | Mycobacterium Infections, Nontuberculous - complications | Polymorphism, Genetic | Aspartic Acid - genetics | Mycobacterium ulcerans | Adolescent | Adult | Cation Transport Proteins - genetics | Female | Aspartic Acid - chemistry | Mycobacterium Infections, Nontuberculous - genetics | Child | Amino Acid Substitution | Asparagine - chemistry | Asparagine - genetics | Mycobacterium | Care and treatment | Usage | Mycobacteria | Genetic aspects | Research | Health aspects | Risk factors | Genetic polymorphisms
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7. Full Text Genetic Diversity of Staphylococcus aureus in Buruli Ulcer
by Amissah, Nana Ama and Glasner, Corinna and Ablordey, Anthony and Tetteh, Caitlin S and Kotey, Nana Konama and Prah, Isaac and van der Werf, Tjip and Rossen, John W and van Dijl, Jan Maarten and Stienstra, Ymkje
PLoS Neglected Tropical Diseases, ISSN 1935-2735, 02/2015, Volume 9, Issue 2, p. e0003421
Background Buruli ulcer (BU) is a necrotizing skin disease caused by Mycobacterium ulcerans. Previous studies have shown that wounds of BU patients are... 
SKIN INFECTIONS | STRAINS | RESISTANT | TOXIN | MYCOBACTERIUM-ULCERANS | DISEASE | COLONIZATION | EPIDERMOLYSIS-BULLOSA | NASAL CARRIAGE | FUNCTIONAL LIMITATIONS | MECHANISMS | INFECTION | IDENTIFICATION | CARE | PARASITOLOGY | TROPICAL MEDICINE | Mycobacterium ulcerans - isolation & purification | Humans | Male | Ghana | Staphylococcal Infections - complications | Buruli Ulcer - complications | Microbial Sensitivity Tests | Methicillin-Resistant Staphylococcus aureus - pathogenicity | Coinfection - microbiology | Buruli Ulcer - microbiology | Mycobacterium ulcerans - genetics | Bacterial Typing Techniques | Mycobacterium ulcerans - pathogenicity | Minisatellite Repeats - genetics | Adult | Female | Genetic Variation - genetics | Staphylococcal Infections - microbiology | Methicillin-Resistant Staphylococcus aureus - isolation & purification | Nose - microbiology | Methicillin-Resistant Staphylococcus aureus - genetics | Care and treatment | Genetic aspects | Buruli ulcer | Identification and classification | Health aspects | Staphylococcus aureus | Studies | Microorganisms | Wound healing | Hospitals | Antibiotics | Skin diseases | Staphylococcus infections | Genetic diversity | Patients | Bacteriology | Hygiene
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8. Full Text Genetic diversity of PCR-Positive, culture-negative and culture-positive Mycobacterium ulcerans isolated from Buruli ulcer patients in Ghana
by Williamson, Heather and Phillips, Richard and Sarfo, Stephen and Wansbrough-Jones, Mark and Small, Pamela
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e88007
Culture of Mycobacterium ulcerans from Buruli ulcer patients has very low sensitivity. Thus confirmation of M. ulcerans infection is primarily based on PCR... 
FISH | DIAGNOSIS | EVOLUTION | CAUSATIVE AGENT | MULTIDISCIPLINARY SCIENCES | GENOME | Genetic Variation | Mycobacterium ulcerans - isolation & purification | Buruli Ulcer - microbiology | Biopsy | Mycobacterium ulcerans - genetics | Humans | Minisatellite Repeats - genetics | Buruli Ulcer - pathology | Ghana | Polymerase Chain Reaction - methods | Multiculturalism | Medicine, Experimental | Medical research | Genetic aspects | Analysis | Biological diversity | Laboratories | Tissue culture | Cultures | Infections | Histology | Genomes | Genetic diversity | Epidemiology | Patients | Polymerase chain reaction | Fish diseases | Sensitivity | Microscopy | Buruli ulcer | Ulcers | Bacteria | Genotypes | Deoxyribonucleic acid--DNA | Teaching hospitals | Deoxyribonucleic acid | DNA
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9. Full Text The genome, evolution and diversity of Mycobacterium ulcerans
by Röltgen, Katharina and Stinear, Timothy P and Pluschke, Gerd
Infection, Genetics and Evolution, ISSN 1567-1348, 04/2012, Volume 12, Issue 3, pp. 522 - 529
â–º We review genome, evolution and genetic diversity of â–º Evolution is dominated by acquisition of a virulence plasmid. â–º Extensive genome downsizing and... 
Plasmid pMUM001 | Clonal population structure | Mycobacterium ulcerans | Mycolactone | Genetic typing | Reductive evolution | INFECTIOUS DISEASES | POLYKETIDE TOXIN | NUCLEOTIDE POLYMORPHISMS | BURULI ULCER | DRUG-RESISTANCE | PROFILE ANALYSIS | FRAGMENT-LENGTH-POLYMORPHISM | GENETIC DIVERSITY | MYCOLACTONE-PRODUCING MYCOBACTERIA | POPULATION-STRUCTURE | CLINICAL ISOLATE | Genome, Bacterial | Chromosomes, Bacterial - genetics | Virulence Factors - genetics | Mycobacterium ulcerans - classification | Humans | Macrolides - metabolism | Phylogeny | Plasmids - metabolism | DNA Transposable Elements | Genetic Variation | Mycobacterium ulcerans - metabolism | Chromosomes, Bacterial - metabolism | Animals | Buruli Ulcer - microbiology | Mycobacterium ulcerans - genetics | Mycobacterium ulcerans - pathogenicity | Plasmids - genetics | Mycobacterium marinum - genetics | Virulence Factors - metabolism | Mycobacterium marinum - metabolism | Evolution, Molecular | Genomics | Universities and colleges | Biological diversity
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10. Full Text Amoebae as Potential Environmental Hosts for Mycobacterium ulcerans and Other Mycobacteria, but Doubtful Actors in Buruli Ulcer Epidemiology
by Gryseels, Sophie and Amissah, Diana and Durnez, Lies and Vandelannoote, Koen and Leirs, Herwig and de Jonckheere, Johan and Silva, Manuel T and Portaels, Françoise and Ablordey, Anthony and Eddyani, Miriam
PLoS Neglected Tropical Diseases, ISSN 1935-2727, 08/2012, Volume 6, Issue 8, p. e1764
Background: The reservoir and mode of transmission of Mycobacterium ulcerans, the causative agent of Buruli ulcer, remain unknown. Ecological, genetic and... 
REAL-TIME PCR | ACANTHAMOEBA-CASTELLANII | AVIUM | CAUSATIVE AGENT | GROWTH | SP NOV | COCULTURE | IDENTIFICATION | VIRULENCE | PARASITOLOGY | TROPICAL MEDICINE | FREE-LIVING AMEBAS | Humans | Molecular Sequence Data | DNA, Bacterial - chemistry | Ghana | Buruli Ulcer - epidemiology | Microbial Viability | Sequence Analysis, DNA | DNA Transposable Elements | Mycobacterium - isolation & purification | Buruli Ulcer - transmission | DNA, Bacterial - genetics | Buruli Ulcer - microbiology | Environmental Microbiology | Disease Reservoirs | Amoeba - microbiology | Phagocytosis | Mycobacterium | Care and treatment | Buruli ulcer | Mycobacteria | Development and progression | Research | Health aspects | Epidemiology | Amoeba
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11. Full Text On the origin of Mycobacterium ulcerans, the causative agent of Buruli ulcer
by Doig, Kenneth D and Holt, Kathryn E and Fyfe, Janet A.M and Lavender, Caroline J and Eddyani, Miriam and Portaels, Françoise and Yeboah-Manu, Dorothy and Pluschke, Gerd and Seemann, Torsten and Stinear, Timothy P
BMC Genomics, ISSN 1471-2164, 06/2012, Volume 13, Issue 1, pp. 258 - 258
Background: Mycobacterium ulcerans is an unusual bacterial pathogen with elusive origins. While closely related to the aquatic dwelling M. marinum, M. ulcerans... 
GENOME SEQUENCE | PROTEIN | MYCOLACTONE-PRODUCING MYCOBACTERIA | REDUCTIVE EVOLUTION | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | TUBERCULOSIS | GENETICS & HEREDITY | PERIPHERAL-BLOOD | DIVERSITY | MARINUM | GENE DECAY | VIRULENCE | Geography | Mycobacterium ulcerans - isolation & purification | Africa | Genetic Loci - genetics | Selection, Genetic | Macrolides - metabolism | Phylogeny | Open Reading Frames - genetics | Sequence Analysis, DNA | Sequence Homology, Amino Acid | Genome, Bacterial - genetics | Pseudogenes - genetics | DNA, Bacterial - genetics | Buruli Ulcer - microbiology | Time Factors | Mycobacterium ulcerans - genetics | Plasmids - genetics | Polymorphism, Single Nucleotide - genetics | Evolution, Molecular | Mycobacterium | Genetic aspects | Research | Bacterial genetics | Mycobacteria | Analysis | Genes | Genomics | Genomes | Single nucleotide polymorphisms | Hypotheses | Ulcers | Colleges & universities | Bacteria | Bayesian analysis | Manuscripts | Pathogens | Cell survival | Cell walls | Data processing | Single-nucleotide polymorphism | Suppressor cells | polyketides | Buruli ulcer | Plasmids | Stem cells | Evolution | Toxins
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12. Full Text Exposure risk for infection and lack of human-to-human transmission of Mycobacterium ulcerans disease, Australia
by O’Brien, Daniel P and Wynne, James W and Buultjens, Andrew H and Michalski, Wojtek P and Stinear, Timothy P and Friedman, N. Deborah and Hughes, Andrew and Athan, Eugene
Emerging Infectious Diseases, ISSN 1080-6040, 05/2017, Volume 23, Issue 5, pp. 837 - 840
We conducted epidemiologic and genetic analyses of family clusters of Mycobacterium ulcerans (Buruli ulcer) disease in southeastern Australia. We found that... 
INFECTIOUS DISEASES | MOSQUITOS | ECOLOGY | INSECTS | SOUTHEASTERN AUSTRALIA | BURULI ULCER | IMMUNOLOGY | Genome, Viral | Mycobacterium ulcerans - isolation & purification | Mycobacterium ulcerans - classification | Humans | Middle Aged | Child, Preschool | Male | Risk | Buruli Ulcer - epidemiology | Environmental Exposure | Phylogeny | Australia - epidemiology | Incidence | Young Adult | Buruli Ulcer - transmission | Buruli Ulcer - microbiology | Mycobacterium ulcerans - genetics | Adolescent | Aged, 80 and over | Adult | Female | Aged | Polymorphism, Single Nucleotide | Child | infection | bacteria | transmission family clusters | Disease, Australia | Dispatch | epidemiology | Mycobacterium ulcerans | exposure risk | Buruli ulcer | tuberculosis and other mycobacteria | genome sequencing | Mycobacterium ulcerans disease | Australia | human-to-human transmission | Exposure Risk for Infection and Lack of Human-to-Human Transmission of
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13. Full Text Virulence potential of Staphylococcus aureus isolates from Buruli ulcer patients
by Amissah, Nana Ama and Chlebowicz, Monika A and Ablordey, Anthony and Tetteh, Caitlin S and Prah, Isaac and van der werf, Tjip S and Friedrich, Alex W and van Dijl, Jan Maarten and Stienstra, Ymkje and Rossen, John W
International Journal of Medical Microbiology, ISSN 1438-4221, 2017, Volume 307, Issue 4-5, pp. 223 - 232
Abstract Buruli ulcer (BU) is a necrotizing infection of the skin and subcutaneous tissue caused by Mycobacterium ulcerans. BU wounds may also be colonized... 
Infectious Disease | Medical Education | Virulence genes | Buruli ulcer | Enterotoxins | Staphylococcus aureus | Mobile genetic elements | TOXINS | SEPSIS | DIABETIC FOOT ULCERS | SHOCK | SUPERANTIGENS | GENES | MRSA | GHANA | MYCOLACTONE | MICROBIOLOGY | VIROLOGY | Leukocidins - genetics | Staphylococcus aureus - genetics | Genes, Bacterial | Virulence Factors - genetics | Staphylococcus aureus - isolation & purification | Hemolysin Proteins - genetics | Humans | Bacterial Proteins - genetics | Genomic Islands | Wound Healing | Sequence Analysis, DNA | Staphylococcus aureus - pathogenicity | Prophages - genetics | Staphylococcal Infections - diagnosis | DNA, Bacterial - genetics | Buruli Ulcer - diagnosis | Buruli Ulcer - microbiology | Superantigens - genetics | Exotoxins - genetics | Bacterial Toxins - genetics | Staphylococcal Infections - microbiology | Medical research | Transposons | Genes | Genomics | Staphylococcus aureus infections | Medicine, Experimental | Virulence (Microbiology) | Drug resistance in microorganisms | Anopheles | Septic shock | Skin
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