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Journal of Clinical Investigation, ISSN 0021-9738, 05/2009, Volume 119, Issue 5, pp. 1109 - 1123
Imatinib mesylate (IM), a potent inhibitor of the BCR/ABL tyrosine kinase, has become standard first-line therapy for patients with chronic myeloid leukemia... 
CHRONIC MYELOGENOUS LEUKEMIA | MEDICINE, RESEARCH & EXPERIMENTAL | MALIGNANT GLIOMA-CELLS | BLAST CRISIS | CLINICAL RESISTANCE | BCR-ABL MUTATIONS | ENDOPLASMIC-RETICULUM | CYTOCHROME-C RELEASE | CASPASE ACTIVATION | IMATINIB RESISTANCE | CHRONIC MYELOID-LEUKEMIA | Transcription Factor CHOP - genetics | Neoplastic Stem Cells - cytology | Gene Expression - drug effects | Calcium - metabolism | Gene Expression - genetics | Microtubule-Associated Proteins - metabolism | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Endoplasmic Reticulum - metabolism | Antineoplastic Agents - therapeutic use | Autophagy - physiology | Thiazoles - therapeutic use | Autophagy - drug effects | Chloroquine - pharmacology | Neoplastic Stem Cells - metabolism | RNA Interference | Endoplasmic Reticulum - drug effects | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Macrolides - pharmacology | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Dasatinib | Chloroquine - therapeutic use | Piperazines - therapeutic use | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Mice, Inbred C3H | Xenograft Model Antitumor Assays | Fusion Proteins, bcr-abl - genetics | Animals | Cell Death - physiology | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Fusion Proteins, bcr-abl - antagonists & inhibitors | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | Benzamides | Macrolides - therapeutic use | Protein-Tyrosine Kinases - antagonists & inhibitors | Causes of | Physiological aspects | Genetic aspects | Chronic myeloid leukemia | Research | Drug therapy | Phagocytosis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2007, Volume 104, Issue 9, pp. 3378 - 3383
During pregnancy the uterine decidua is populated by large numbers of natural killer (NK) cells with a phenotype ${\rm CD}56^{\text{superbright}}{\rm... 
Cytometry | Phenotypes | Cytokines | Stromal cells | Stem cells | Cultured cells | Natural killer cells | Progenitor cells | Blood | Endometrium | Pregnancy | RECRUITMENT | NATURAL-KILLER-CELLS | GROWTH-FACTOR-BETA | INTERLEUKIN-15 | MULTIDISCIPLINARY SCIENCES | pregnancy | TRAFFICKING | C-KIT | RECEPTOR | HUMAN ENDOMETRIUM | EXPRESSION | SUBSET | Biological Sciences
Journal Article
European Journal of Immunology, ISSN 0014-2980, 02/2015, Volume 45, Issue 2, pp. 480 - 491
Anergic T cells can survive for long time periods passively in a hyporesponsive state without obvious active functions. Thus, the immunological reason for... 
CD28 | CTLA‐4 | Dendritic cell | Treg cell | T‐cell anergy | CTLA-4 | T-cell anergy | ENTEROTOXIN-B | BONE-MARROW | CLONAL ANERGY | MONOCLONAL-ANTIBODY | INDUCTION | IMMUNOLOGY | IN-VIVO | NUCLEAR FACTOR | TRANSCRIPTION FACTOR | TRANSGENIC MICE | SELF-TOLERANCE | Forkhead Transcription Factors - immunology | T-Lymphocytes, Regulatory - metabolism | Coculture Techniques | Dendritic Cells - immunology | Early Growth Response Protein 2 - genetics | Clonal Anergy | T-Lymphocytes, Regulatory - immunology | Interleukin-2 - immunology | Interleukin-2 Receptor alpha Subunit - genetics | NFATC Transcription Factors - immunology | CD28 Antigens - genetics | T-Lymphocytes, Regulatory - cytology | Dendritic Cells - drug effects | Cell Differentiation | Early Growth Response Protein 2 - immunology | Dendritic Cells - metabolism | Interleukin-2 Receptor alpha Subunit - immunology | Signal Transduction | Gene Expression Regulation - immunology | Lymphocyte Activation | Mice, Inbred C57BL | Cell Communication | CTLA-4 Antigen - genetics | CD28 Antigens - immunology | CTLA-4 Antigen - immunology | Forkhead Transcription Factors - genetics | Interleukin-2 - genetics | Transforming Growth Factor beta - pharmacology | T-Lymphocytes, Regulatory - drug effects | Animals | Interleukin-10 - genetics | Dendritic Cells - cytology | Mice | Mice, Inbred BALB C | Interleukin-10 - immunology | NFATC Transcription Factors - genetics | Bone morphogenetic proteins | Peptides | Dendritic cells | T cells | Transforming growth factors
Journal Article
European Journal of Immunology, ISSN 0014-2980, 10/2011, Volume 41, Issue 10, pp. 2840 - 2851
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2010, Volume 107, Issue 5, pp. 2159 - 2164
The beneficial effects of probiotics have been described in many diseases, but the mechanism by which they modulate the immune system is poorly understood. In... 
T lymphocytes | Probiotics | Inflammatory bowel diseases | Immune disorders | Population growth | Cytokines | Dendritic cells | B lymphocytes | Drug regulation | Mice | Inflammatory bowel disease | Inflammation | Atopic dermatitis | Rheumatoid arthritis | Regulatory T cell | MIGRATION | BACTERIA | MULTIDISCIPLINARY SCIENCES | RETINOIC-ACID | atopic dermatitis | regulatory T cell | INDUCTION | rheumatoid arthritis | FOXP3 | inflammatory bowel disease | EXPERIMENTAL ARTHRITIS | INFLAMMATORY-BOWEL-DISEASE | inflammation | ORAL TOLERANCE | MICE | EXPRESSION | Probiotics - administration & dosage | T-Lymphocytes, Regulatory - metabolism | Arthritis, Experimental - therapy | Dendritic Cells - immunology | Inflammatory Bowel Diseases - immunology | Dendritic Cells - pathology | Adoptive Transfer | Dermatitis, Atopic - therapy | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Arthritis, Experimental - pathology | CD11c Antigen - metabolism | Forkhead Transcription Factors - metabolism | Inflammatory Bowel Diseases - pathology | Dermatitis, Atopic - immunology | Immune System Diseases - pathology | Disease Models, Animal | Immune System Diseases - immunology | Mice, Transgenic | Arthritis, Experimental - immunology | Dermatitis, Atopic - pathology | Immunosuppression | Animals | Immune System Diseases - therapy | Mice, Inbred BALB C | Inflammatory Bowel Diseases - therapy | Biological Sciences
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2014, Volume 193, Issue 4, pp. 1622 - 1635
Journal Article
Immunity, ISSN 1074-7613, 10/2013, Volume 39, Issue 4, pp. 733 - 743
Journal Article