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Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 2012, Volume 75, Issue 4, pp. 633 - 647
The radical response of peripheral nerves to injury (Wallerian degeneration) is the cornerstone of nerve repair. We show that activation of the transcription... 
SCIATIC-NERVE | MYELINATION | RAT | SIGNALING PATHWAY | PERIPHERAL NERVOUS-SYSTEM | WALLERIAN DEGENERATION | IN-VIVO | AXONAL REGENERATION | DORSAL-ROOT GANGLION | NEUROSCIENCES | NEGATIVE REGULATOR | Nerve Regeneration - physiology | Benzofurans | Cell Movement - genetics | Motor Neurons - pathology | Motor Neurons - ultrastructure | Spinal Cord - pathology | Sciatic Neuropathy - therapy | Adenoviridae - genetics | Disease Models, Animal | Microfluidic Analytical Techniques | Macrophages - ultrastructure | Microscopy, Electron, Transmission | Macrophages - pathology | Myelin Sheath - pathology | Gene Expression Regulation - genetics | Proto-Oncogene Proteins c-jun - genetics | Sciatic Neuropathy - pathology | Mice, Transgenic | Schwann Cells - pathology | Schwann Cells - metabolism | Motor Neurons - metabolism | Macrophages - metabolism | Animals | Schwann Cells - ultrastructure | Analysis of Variance | Proto-Oncogene Proteins c-jun - metabolism | Sciatic Neuropathy - physiopathology | Genetic Vectors - physiology | Mice | Myelin Sheath - ultrastructure | Sciatic Neuropathy - metabolism | Neurons | Neurosciences | Medical research | Genotype & phenotype | Transcription factors | Rodents | Leprosy | Grants | Mammals | Gene expression | Regeneration | Myelin | Neurodegeneration | Peripheral nerves | Schwann cells | Recovery of function | c-Jun protein | Trophic factors | Injuries | Radicals | Cell adhesion molecules
Journal Article
1994, 1, ISBN 0849345731, 310
This book introduces and analyzes the crucial role of AP-1 in cell growth, proliferation, differentiation, and apoptosis. AP-1 is the endpoint of several... 
Jun oncogenes | Fos oncogenes | Transcription factors | Biochemistry
Book
Nature medicine, ISSN 1546-170X, 2011, Volume 17, Issue 12, pp. 1668 - 1673
Tissue homeostasis requires an effective, limited wound-healing response to injury. In chronic disease, failure to regenerate parenchymal tissue leads to the... 
FIBROSIS | MEDICINE, RESEARCH & EXPERIMENTAL | JUND | ANTAGONIST | RAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | INJURY | PROLIFERATION | CELL BIOLOGY | HEPATIC STELLATE CELLS | BILE-DUCT LIGATION | GROWTH | Immunohistochemistry | Receptor, Serotonin, 5-HT2B - metabolism | Transforming Growth Factor beta1 - metabolism | Hepatic Stellate Cells - metabolism | Male | Wound Healing | Serotonin - pharmacology | Hepatocytes - cytology | Receptor, Serotonin, 5-HT2B - drug effects | Mitogen-Activated Protein Kinase 1 - genetics | Urea - analogs & derivatives | Indoles - pharmacology | Hepatocytes - drug effects | Hepatic Stellate Cells - drug effects | Signal Transduction | Proto-Oncogene Proteins c-jun - genetics | Electrophoresis, Polyacrylamide Gel | Mice, Inbred C57BL | Cells, Cultured | Rats | Liver Cirrhosis - therapy | Transforming Growth Factor beta1 - genetics | Serotonin 5-HT2 Receptor Antagonists - pharmacology | Rats, Sprague-Dawley | Mice, Knockout | Animals | Proto-Oncogene Proteins c-jun - metabolism | Liver Cirrhosis - pathology | Cell Proliferation - drug effects | Mice | Chronic Disease | Urea - pharmacology | Mitogen-Activated Protein Kinase 1 - metabolism | Proteins | Signal transduction | Cell growth | Liver diseases | Gene expression | Chronic illnesses | Cell Proliferation | Serotonin 5-HT2 Receptor Antagonists | Life Sciences | Indoles | Transforming Growth Factor beta1 | Serotonin | Biochemistry, Molecular Biology | Liver Cirrhosis | Receptor, Serotonin, 5-HT2B | Urea | Hepatocytes | Hepatic Stellate Cells | Mitogen-Activated Protein Kinase 1 | Proto-Oncogene Proteins c-jun
Journal Article
Journal Article
Blood, ISSN 0006-4971, 2016, Volume 127, Issue 14, pp. 1780 - 1789
Journal Article
STEM CELLS, ISSN 1066-5099, 05/2013, Volume 31, Issue 5, pp. 870 - 881
Accumulated evidence suggests that glioma stem cells (GSCs) may contribute to therapy resistance in high‐grade glioma (HGG). Although recent studies have shown... 
Cancer stem cell | Stress‐induced kinase | Glioma‐initiating cell | c‐Jun NH‐terminal kinases | Neural stem cell | Glioma-initiating cell | Stress-induced kinase | c-Jun NH-terminal kinases | INITIATING CELLS | PROTEIN | initiating cell | PROLIFERATION | CANCER | BRAIN-TUMORS | P53 | CELL & TISSUE ENGINEERING | CELL BIOLOGY | MELK | Glioma | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | HEMATOLOGY | GLIOBLASTOMA CELLS | SERINE/THREONINE KINASE | LEUCINE-ZIPPER KINASE | Humans | Brain Neoplasms - pathology | Cell Growth Processes - physiology | Brain Neoplasms - metabolism | Tumor Suppressor Protein p53 - genetics | Gene Knockdown Techniques | Glioma - metabolism | Glioma - genetics | Heterografts | Neoplastic Stem Cells - metabolism | Glioma - pathology | Neoplastic Stem Cells - pathology | Female | Protein-Serine-Threonine Kinases - metabolism | Signal Transduction | Proto-Oncogene Proteins c-jun - genetics | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | Brain Neoplasms - genetics | Protein-Serine-Threonine Kinases - biosynthesis | Animals | Mice, Nude | Proto-Oncogene Proteins c-jun - metabolism | Mice | Apoptosis - physiology | RNA | Growth | Chemotherapy | DNA microarrays | Gliomas | Nuclear radiation | Analysis | Stem cells | Genetic engineering | Tumor proteins | Intermediate filament proteins | Phosphotransferases | Cancer | Proteins | Tumors | glioma-initiating cell | neural stem cell | cancer stem cell | stress-induced kinase | JNK
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2010, Volume 285, Issue 10, pp. 7097 - 7110
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2015, Volume 290, Issue 30, pp. 18636 - 18649
Journal Article
Journal Article