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1. Full Text c-Rel is crucial for the induction of Foxp3+ regulatory CD4 + T cells but not TH17 cells
by Visekruna, Alexander and Huber, Magdalena and Hellhund, Anne and Bothur, Evita and Reinhard, Katharina and Bollig, Nadine and Schmidt, Nicole and Joeris, Thorsten and Lohoff, Michael and Steinhoff, Ulrich
European Journal of Immunology, ISSN 0014-2980, 03/2010, Volume 40, Issue 3, pp. 671 - 676
C-Rel | IL-2 | Treg
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2. Full Text REL, encoding a member of the NF-kappa B family of transcription factors, is a newly defined risk locus for rheumatoid arthritis
by Gregersen, PK and Amos, CI and Lee, AT and Lu, Y and Remmers, EF and Kastner, DL and Seldin, MF and Criswell, LA and Plenge, RM and Holers, VM and Mikuls, TR and Sokka, T and Moreland, LW and Bridges, SL and Xie, G and Begovich, AB and Siminovitch, KA
NATURE GENETICS, ISSN 1061-4036, 07/2009, Volume 41, Issue 7, pp. 820 - U77
We conducted a genome-wide association study of rheumatoid arthritis in 2,418 cases and 4,504 controls from North America and identified an association at the... 
CELLS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | ITGAM | VARIANTS | INCEPTION COHORT | GENETICS & HEREDITY | C-REL | ASSOCIATION | EXPRESSION
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3. Full Text IRF5 promotes inflammatory macrophage polarization and T(H)1-T(H)17 responses
by Krausgruber, T and Blazek, K and Smallie, T and Alzabin, S and Lockstone, H and Sahgal, N and Hussell, T and Feldmann, M and Udalova, IA
NATURE IMMUNOLOGY, ISSN 1529-2908, 03/2011, Volume 12, Issue 3, pp. 231 - U66
Polymorphisms in the gene encoding the transcription factor IRF5 that lead to higher mRNA expression are associated with many autoimmune diseases. Here we show... 
COLLAGEN-INDUCED ARTHRITIS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | IFN REGULATORY FACTOR-1 | GM-CSF | GENE-EXPRESSION | C-REL | KAPPA-B | IMMUNOLOGY | IL-23 | IL-12 | T-CELLS
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4. Full Text IKKα limits macrophage NF-κB activation and contributes to the resolution of inflammation
by Lawrence, Toby and Bebien, Magali and Liu, George Y and Nizet, Victor and Karin, Michael
Nature, ISSN 0028-0836, 04/2005, Volume 434, Issue 7037, pp. 1138 - 1143
Inflammation and innate immunity involve signalling pathways leading to the production of inflammatory mediators. Usually such responses are self-limiting, but... 
UBIQUITIN-MEDIATED PROTEOLYSIS | RECOGNITION | SIGNALING PATHWAY | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | KINASE | TRANSCRIPTION | GENE-EXPRESSION | C-REL | TRANSACTIVATION DOMAIN | MICE
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5. Full Text Circuitry of nuclear factor κB signaling
by Hoffmann, Alexander and Baltimore, David
Immunological Reviews, ISSN 0105-2896, 04/2006, Volume 210, Issue 1, pp. 171 - 186
Over the past few years, the transcription factor nuclear factor (NF)-kappa B and the proteins that regulate it have emerged as a signaling system of... 
TRANSCRIPTION FACTORS | TARGET GENES | INDUCED APOPTOSIS | TEMPORAL CONTROL | C-REL | IKK-ALPHA | TNF-ALPHA | IMMUNOLOGY | BINDING-SITES | TRANSDUCTION PATHWAY | SELECTIVE GENE ACTIVATION
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6. Full Text Introduction to NF-κB: Players, pathways, perspectives
by Gilmore, T.D
Oncogene, ISSN 0950-9232, 10/2006, Volume 25, Issue 51, pp. 6680 - 6684
This article serves as an introduction to the collection of reviews on nuclear factor-kappaB (NF-kappa B). It provides an overview of the discovery and current... 
IkappaB | IKK | Rel | Signal transduction | NF-kappaB | Transcription factor | transcription factor | KBF1 | PROTEIN | signal transduction | BIOCHEMISTRY & MOLECULAR BIOLOGY | C-REL | DNA-BINDING SUBUNIT | RETICULOENDOTHELIOSIS VIRUS | DROSOPHILA | CELL BIOLOGY | ONCOGENE | V-REL | ONCOLOGY | PRODUCT | GENETICS & HEREDITY | DORSAL
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7. Full Text I kappa B-NS Protein Mediates Regulatory T Cell Development via Induction of the Foxp3 Transcription Factor
by Schuster, M and Glauben, R and Plaza-Sirvent, C and Schreiber, L and Annemann, M and Floess, S and Kuhl, AA and Clayton, LK and Sparwasser, T and Schulze-Osthoff, K and Pfeffer, K and Huehn, J and Siegmund, B and Schmitz, I
IMMUNITY, ISSN 1074-7613, 12/2012, Volume 37, Issue 6, pp. 998 - 1008
Forkhead box P3 positive (Foxp3(+)) regulatory T (Treg) cells suppress immune responses and regulate peripheral tolerance. Here we show that the atypical... 
ACTIVATION | DISRUPTION | GENE | CARMA1 | INFLAMMATION | THYMIC DEVELOPMENT | RECEPTOR | C-REL | SELF | IMMUNOLOGY | EXPRESSION
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8. Full Text IκBβ acts to inhibit and activate gene expression during the inflammatory response
by Rao, Ping and Hayden, Mathew S and Long, Meixiao and Scott, Martin L and West, A. Philip and Zhang, Dekai and Oeckinghaus, Andrea and Lynch, Candace and Hoffmann, Alexander and Baltimore, David and Ghosh, Sankar
Nature, ISSN 0028-0836, 08/2010, Volume 466, Issue 7310, pp. 1115 - 1119
The activation of pro-inflammatory gene programs by nuclear factor-kappa B (NF-kappa B) is primarily regulated through cytoplasmic sequestration of NF-kappa B... 
RHEUMATOID-ARTHRITIS | DNA-BINDING | SIMILARITIES | SEQUENCES | TUMOR NECROSIS FACTOR | PHOSPHORYLATION | BIOSYNTHESIS | MULTIDISCIPLINARY SCIENCES | ALPHA | C-REL | DEFICIENT MICE
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9. Full Text Shaping the nuclear action of NF-κB
by Greene, Warner C and Chen, Lin-Feng
Nature Reviews Molecular Cell Biology, ISSN 1471-0072, 05/2004, Volume 5, Issue 5, pp. 392 - 401
The NF-kappaB/REL family of transcription factors pivotally control the inflammatory and immune responses, as well as other genetic programmes that are central... 
TATA-BINDING PROTEIN | TUMOR-NECROSIS-FACTOR | P65 SUBUNIT | GENE-EXPRESSION | CASEIN KINASE-II | C-REL | IKK-ALPHA | HISTONE H3 PHOSPHORYLATION | TRANSCRIPTION FACTOR-IIB | FACTOR-ALPHA | CELL BIOLOGY
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10. Full Text FOXP3 and scurfy: How it all began
by Ramsdell, Fred and Ziegler, Steven F
Nature Reviews Immunology, ISSN 1474-1733, 2014, Volume 14, Issue 5, pp. 343 - 349
It has been 65 years since the scurfy mutation arose spontaneously in mice at the Oak Ridge National Laboratory in the United States, and it is 13 years since... 
X-LINKED SYNDROME | ROR-GAMMA-T | TRANSCRIPTION FACTOR FOXP3 | REGULATORY T-CELLS | GENE-EXPRESSION | LYMPHORETICULAR DISEASE | C-REL | SF MOUSE | LINEAGE SPECIFICATION | IMMUNOLOGY | AUTOIMMUNE-DISEASE | Forkhead Transcription Factors - immunology | Immune System - immunology | Animals | Humans | T-Lymphocytes, Regulatory - immunology
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11. Full Text Malt1 Protease Is Critical in Maintaining Function of Regulatory T Cells and May Be a Therapeutic Target for Antitumor Immunity
by Cheng, LQ and Deng, N and Yang, NX and Zhao, XQ and Lin, X
JOURNAL OF IMMUNOLOGY, ISSN 0022-1767, 05/2019, Volume 202, Issue 10, pp. 3008 - 3019
The paracaspase Malt1 is a key molecule in mediating Ag receptor-induced NF-kappa B activation in lymphocytes, but the role of Malt1 in the function of... 
ACTIVATION | DIFFERENTIAL REQUIREMENT | GENE | LYMPHOMA | C-REL | IMMUNOLOGY | CLEAVAGE | T(11/18)(Q21,Q21) | IDENTIFICATION | NF-KAPPA-B | FOXP3
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12. Full Text NF-kappaB signaling pathways in neurological inflammation: A mini review
by Shih, Ruey-Horng and Wang, Chen-Yu and Yang, Chuen-Mao
Frontiers in Molecular Neuroscience, ISSN 1662-5099, 12/2015, Volume 8, p. 77
The NF-kappa B (nuclear factor K-light-chain-enhancer of activated B cells) transcription factor family is a pleiotropic regulator of many cellular signaling... 
Neuroprotection | Adhesion molecules | Neuroinflammation | NF-kappaB | Proinflammatory transcription factors | BETA-PEPTIDE TOXICITY | ALZHEIMERS-DISEASE | NEURONAL CELL-DEATH | NEUROSCIENCES | factors | FACTOR C-REL | B KINASE-ALPHA | proinflammatory transcription | TUMOR-NECROSIS-FACTOR | neuroinflammation | adhesion molecules | neuroprotection | TOLL-LIKE RECEPTORS | GENE-EXPRESSION | SPINAL-CORD-INJURY | TRANSCRIPTION FACTOR | Physiological aspects | Development and progression | Transcription factors | Inflammation | Neurons | Health aspects | proinflammatory transcription factors | Signaling Pathways | neuroninflammation | neuronprotection | NF-KappaB
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13. Full Text The IκB Kinase Regulates Chromatin Structure during Reconsolidation of Conditioned Fear Memories
by Lubin, Farah D and Sweatt, J. David
Neuron, ISSN 0896-6273, 2007, Volume 55, Issue 6, pp. 942 - 957
Previously formed memories are susceptible to disruption immediately after recall due to a necessity to be reconsolidated after retrieval. Protein translation... 
SYSNEURO | DNA | MOLNEURO | INDUCED GENE-EXPRESSION | ACTIVATION | LONG-TERM-MEMORY | ACETYLATION | HISTONE ACETYLTRANSFERASE ACTIVITY | IKK-ALPHA | C-REL | TRANSCRIPTION FACTOR | PROTEIN-SYNTHESIS | NEUROSCIENCES | RETRIEVAL
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14. Full Text Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1
by Barbie, David A and Tamayo, Pablo and Boehm, Jesse S and Kim, So Young and Moody, Susan E and Dunn, Ian F and Schinzel, Anna C and Sandy, Peter and Meylan, Etienne and Scholl, Claudia and Fröhling, Stefan and Chan, Edmond M and Sos, Martin L and Michel, Kathrin and Mermel, Craig and Silver, Serena J and Weir, Barbara A and Reiling, Jan H and Sheng, Qing and Gupta, Piyush B and Wadlow, Raymond C and Le, Hanh and Hoersch, Sebastian and Wittner, Ben S and Ramaswamy, Sridhar and Livingston, David M and Sabatini, David M and Meyerson, Matthew and Thomas, Roman K and Lander, Eric S and Mesirov, Jill P and Root, David E and Gilliland, D. Gary and Jacks, Tyler and Hahn, William C
Nature, ISSN 0028-0836, 11/2009, Volume 462, Issue 7269, pp. 108 - 112
The proto-oncogene KRAS is mutated in a wide array of human cancers, most of which are aggressive and respond poorly to standard therapies. Although the... 
APOPTOSIS | ESSENTIAL GENES | ACTIVATION | K-RAS | SCREEN | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | LUNG ADENOCARCINOMA | CLASSIFICATION | NF-KAPPA-B | TUMOR-CELL SURVIVAL | Lung Neoplasms - genetics | Neoplasms - metabolism | Signal Transduction | Cell Survival | Humans | Lung Neoplasms - metabolism | Proto-Oncogene Proteins c-rel - metabolism | Lung Neoplasms - pathology | Gene Expression Profiling | Oncogene Protein p21(ras) - metabolism | Genes, Lethal | Neoplasms - genetics | RNA Interference | Alleles | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Line, Tumor | Neoplasms - pathology | bcl-X Protein - metabolism | Protein-Serine-Threonine Kinases - metabolism | Apoptosis | Genes, ras - genetics | Oncogene Protein p21(ras) - genetics | Cancer patients | Care and treatment | Genetic aspects | Research | Cancer | Proteins | Genes | Kinases | Cells | Methods | Tumors
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