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Journal Article
Nature communications, ISSN 2041-1723, 2019, Volume 10, Issue 1, pp. 2961 - 14
Journal Article
The journal of clinical endocrinology and metabolism, ISSN 1945-7197, 2017, Volume 102, Issue 6, pp. 2029 - 2038
...). Serum leptin and C-reactive protein... 
PATHWAYS | WOMEN | IN-VITRO | MATERNAL OBESITY | ENDOCRINOLOGY & METABOLISM | FOLLICULAR-FLUID | CUMULUS CELLS | DEVELOPMENTAL COMPETENCE | OUTCOMES | MATURATION | ABUNDANCE | Body Composition | Ovulation Induction | Humans | Leptin - metabolism | Inhibitor of Differentiation Proteins - genetics | Gene Expression Profiling | Neoplasm Proteins - metabolism | Obesity - genetics | Oocyte Retrieval | Case-Control Studies | Young Adult | Overweight - metabolism | C-Reactive Protein - metabolism | Chemokine CXCL2 - genetics | Adult | Female | Neoplasm Proteins - genetics | Single-Cell Analysis | Chemokine CXCL2 - metabolism | Dual Specificity Phosphatase 1 - genetics | Oocytes - metabolism | Overweight - genetics | Lipid Metabolism | Inflammation | Inhibitor of Differentiation Proteins - metabolism | Nerve Tissue Proteins - genetics | Follicular Fluid - chemistry | Obesity - metabolism | Triglycerides - metabolism | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Carrier Proteins - metabolism | Sequence Analysis, RNA | Adolescent | Dual Specificity Phosphatase 1 - metabolism | Oxidative stress | Transcription factors | C-reactive protein | Fat metabolism | Body fat | Body weight | Lipids | Body composition | Oocytes | Gene sequencing | Body mass index | Embryogenesis | Body composition (biology) | Fertility | Lipid metabolism | Obesity | Follicular fluid | Metabolism | Gene expression | Ribonucleic acid--RNA | Insulin | Embryonic growth stage | Body mass | Leptin | Body size
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2007, Volume 293, Issue 4, pp. 2210 - 2218
...B and RhoA activation and upregulation of adhesion molecules ICAM-1 and VCAM-1, increased expression of monocyte chemoattractant protein, enhanced transendothelial migration of monocytes... 
Adhesion molecules | Inflammation | RhoA | Endothelial activation | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MOLECULE EXPRESSION | ATHEROSCLEROSIS | NECROSIS-FACTOR-ALPHA | KNOCKOUT MICE | endothelial activation | CANNABINOID RECEPTORS | inflammation | adhesion molecules | NITRIC-OXIDE | THERAPEUTIC TARGETS | PERIPHERAL VASCULAR DISEASE | POTENTIAL ROLE | CB2 RECEPTOR ACTIVATION | Inflammation - chemically induced | Leukocyte Rolling - drug effects | Tumor Necrosis Factor-alpha - metabolism | Receptor, Cannabinoid, CB2 - agonists | Humans | Male | Monocytes - metabolism | NF-kappa B - metabolism | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Coronary Vessels - metabolism | Lipopolysaccharides | Dose-Response Relationship, Drug | Inflammation - metabolism | Anti-Inflammatory Agents - therapeutic use | Chemokine CCL2 - metabolism | Disease Models, Animal | Coronary Vessels - drug effects | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Cannabinoids - pharmacology | Monocytes - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Cannabinoids - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Signal Transduction - drug effects | Inflammation - prevention & control | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects
Journal Article
Circulation, ISSN 0009-7322, 07/2014, Volume 130, Issue 1, pp. 35 - 50
Background-The relevance of the dissociation of circulating pentameric C-reactive protein (pCRP... 
Myocardial infarction | Microcirculation | Inflammation | C-reactive protein | Atherosclerosis | atherosclerosis | CRP | ANTIBODIES | CARDIAC & CARDIOVASCULAR SYSTEMS | COMPLEMENT ACTIVATION | myocardial infarction | PHOSPHOLIPASE A | RELEASE | MICROPARTICLES | microcirculation | REDUCTION | inflammation | LYSOPHOSPHATIDYLCHOLINE | APOPTOTIC CELLS | PERIPHERAL VASCULAR DISEASE | EXPRESSION | Chemotaxis, Leukocyte | Leukocyte Rolling - drug effects | Complement Activation | Rats, Wistar | Humans | Male | Muscle, Skeletal - metabolism | Myositis - chemically induced | Phosphorylcholine - analogs & derivatives | Biopolymers | Phospholipase A2 Inhibitors - pharmacology | Inflammation - metabolism | Hexanes - pharmacology | Inflammation - drug therapy | Myocardial Infarction - pathology | Phosphorylcholine - pharmacology | Protein Structure, Quaternary | Anti-Inflammatory Agents - therapeutic use | Carrier Proteins - chemistry | Cell Membrane - metabolism | Myositis - pathology | Reperfusion Injury - metabolism | Cell Membrane - drug effects | Muscle, Skeletal - blood supply | Carrier Proteins - physiology | Hexanes - therapeutic use | Phospholipase A2 Inhibitors - therapeutic use | Lipopolysaccharides - toxicity | Receptors, IgG - physiology | Reperfusion Injury - pathology | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Myositis - metabolism | Lysophosphatidylcholines - metabolism | Rats | Phospholipases A2 - metabolism | Myocardial Infarction - metabolism | Random Allocation | C-Reactive Protein - chemistry | C-Reactive Protein - physiology | Cell Adhesion - drug effects | Inflammation - etiology | Animals | Membrane Lipids - metabolism | Endothelial Cells - drug effects | Phosphorylcholine - therapeutic use | Care and treatment | Usage | Anti-inflammatory drugs | Physiological aspects | Development and progression | Research | Health aspects
Journal Article
The Journal of Physiology, ISSN 0022-3751, 2004, Volume 556, Issue 3, pp. 983 - 1000
Muscular adaptation to physical exercise has previously been described as a repair process following tissue damage. Recently, evidence has been published to... 
INDUCED INJURY | PHYSIOLOGY | IMMUNOREACTIVITY | ECCENTRIC EXERCISE | REGENERATION | LEUKEMIA INHIBITORY FACTOR | DENERVATED HUMAN MUSCLE | SATELLITE CELLS | STRENGTH | FACTOR-I | EXPRESSION | NEUROSCIENCES | Immunohistochemistry | Granulocytes - cytology | Cytokines - analysis | Humans | Middle Aged | DNA-Binding Proteins - analysis | Ki-67 Antigen - metabolism | Male | Muscle, Skeletal - metabolism | Proteins - analysis | Pain - metabolism | fas Receptor - metabolism | Fascia - chemistry | Oxygen Consumption - physiology | fas Receptor - analysis | Antigens, CD - metabolism | Antigens, CD - analysis | Running - physiology | C-Reactive Protein - metabolism | Aryl Hydrocarbon Receptor Nuclear Translocator | CD11b Antigen - analysis | Receptors, Cytokine - metabolism | Testosterone - blood | C-Reactive Protein - analysis | Hormones - blood | Leukocytes - chemistry | Interleukin-6 - metabolism | Lymphocytes - metabolism | CD56 Antigen - analysis | Lymphocytes - cytology | Insulin-Like Growth Factor I - analysis | Muscle, Skeletal - physiology | CD3 Complex - metabolism | Leukemia Inhibitory Factor Receptor alpha Subunit | Regression Analysis | Pain - physiopathology | Receptors, Cytokine - analysis | Adolescent | Antigens, Differentiation, Myelomonocytic - analysis | Creatine Kinase - metabolism | Transcription Factors - analysis | Leukocytes - metabolism | Insulin-Like Growth Factor I - metabolism | Receptors, OSM-LIF | Interleukin-6 - analysis | Monocytes - cytology | Receptors, Cell Surface - analysis | Monocytes - metabolism | Receptors, Aryl Hydrocarbon - analysis | DNA-Binding Proteins - metabolism | Testosterone - metabolism | Flow Cytometry | Interleukin-6 - blood | Exercise Test - methods | Muscle, Skeletal - chemistry | Fascia - metabolism | Heart Rate - physiology | Receptors, Aryl Hydrocarbon - metabolism | Adult | Female | Leukocyte Count | Leukemia Inhibitory Factor | Isometric Contraction - physiology | Cytokines - blood | Leukocytes - cytology | CD56 Antigen - metabolism | Creatine Kinase - blood | Cytokines - metabolism | Receptors, Cell Surface - metabolism | Transcription Factors - metabolism | CD3 Complex - analysis | Ki-67 Antigen - analysis | Proteins - metabolism | Hormones - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | CD11b Antigen - metabolism | Growth Substances - metabolism | Pain - diagnosis | Research Papers
Journal Article
Atherosclerosis, ISSN 0021-9150, 2012, Volume 221, Issue 2, pp. 375 - 382
Highlights ► Liraglutide can normalize TNF-α-induced pro-oxidant production in HUVEC probably through reduced expression of NADPH oxidase subunit gp91phox and... 
Cardiovascular | Oxidative stress | NF-κB | Endothelial cell | Inflammation | Apoptosis | CHRONIC HEART-FAILURE | TRANSLOCATION | PROTEIN-KINASE-C | PENTRAXIN 3 | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | IN-VITRO | NF-kappa B | ACUTE MYOCARDIAL-INFARCTION | DEGRADATION | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | VASCULAR NAD(P)H OXIDASE | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Reactive Oxygen Species - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Membrane Glycoproteins - metabolism | Protein Kinase C-alpha - metabolism | Apoptosis - drug effects | Humans | NADPH Oxidases - metabolism | Human Umbilical Vein Endothelial Cells - immunology | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Dose-Response Relationship, Drug | C-Reactive Protein - metabolism | Transfection | I-kappa B Kinase - metabolism | Time Factors | Inflammation Mediators - metabolism | Superoxide Dismutase - metabolism | Recombinant Proteins - metabolism | Glutathione Peroxidase - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Anti-Inflammatory Agents - pharmacology | Glucagon-Like Peptide 1 - analogs & derivatives | Cells, Cultured | Glucagon-Like Peptide 1 - pharmacology | Antioxidants - pharmacology | NADPH Oxidase 2 | Protein Transport | Catalase - metabolism | NF-kappa B - genetics | Signal Transduction - drug effects | Serum Amyloid P-Component - metabolism | Oxidative Stress - drug effects | Liraglutide
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 5/2013, Volume 110, Issue 21, pp. 8650 - 8655
.... This versatile recognition protein senses a wide variety of immune and nonimmune ligands, including pathogens and altered self components, and triggers the classical complement pathway... 
Proteins | Molecules | Cell lines | Materials | Cell aggregates | Ligands | Mass spectroscopy | Biochemistry | Electron microscopy | Molecular chains | C1 complex | Protein engineering | Innate immunity | B-CHAIN | ACTIVATION | 1ST COMPONENT | PROTEIN | MULTIDISCIPLINARY SCIENCES | LONG PENTRAXIN PTX3 | LIGAND-RECOGNITION | MANNAN-BINDING LECTIN | protein engineering | innate immunity | STRUCTURAL BASIS | PATHWAY | SUBCOMPONENT C1Q | Surface Plasmon Resonance | Complement C1r - genetics | Calcium - metabolism | Humans | Complement C1s - metabolism | Mutation, Missense | Complement C1q - metabolism | Complement C1r - metabolism | Complement C1s - genetics | Complement C1r - chemistry | Complement C1s - chemistry | C-Reactive Protein - metabolism | HEK293 Cells | Protein Structure, Quaternary | Binding Sites | Complement C1q - chemistry | Complement Activation - physiology | Protein Structure, Tertiary | Recombinant Proteins - metabolism | Gene Expression | Immunoglobulin G - chemistry | Serum Amyloid P-Component - chemistry | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Complement C1q - genetics | C-Reactive Protein - chemistry | Serum Amyloid P-Component - metabolism | Amino Acid Substitution | Immunoglobulin G - metabolism | Protein Binding - physiology | Bone morphogenetic proteins | Polypeptides | Research | Properties | Proteases | Protein binding | C-Reactive Protein | Calcium | Complement Activation | Immunoglobulin G | Biochemistry, Molecular Biology | Serum Amyloid P-Component | Recombinant Proteins | Complement C1q | Complement C1s | Complement C1r | Life Sciences | Protein Binding | Biomolecules | Biological Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2017, Volume 12, Issue 5, p. e0178525
...) from the harmful effects of lipopolysaccharide (LPS). We found that melatonin inhibited the LPS-binding protein-CD14-TLR4 signaling pathway in bMECs, which had opposing effects on pro-inflammatory and anti-inflammatory mediators... 
LIPOPOLYSACCHARIDE-INDUCED MASTITIS | DIAGNOSIS | OXIDATIVE STRESS | ACTIVATION | ACUTE-PHASE PROTEINS | PEROXYNITRITE | CHEMOKINES | MULTIDISCIPLINARY SCIENCES | AMNIOTIC-FLUID | EXPRESSION | NITRATION | Tumor Necrosis Factor-alpha - metabolism | Heme Oxygenase-1 - metabolism | Epithelial Cells - metabolism | Mammary Glands, Animal | Membrane Glycoproteins - metabolism | Epithelial Cells - drug effects | Colony-Stimulating Factors - metabolism | Lipopolysaccharide Receptors - metabolism | C-Reactive Protein - metabolism | Cattle Diseases - metabolism | Mastitis - metabolism | Mastitis - drug therapy | Cattle | Interleukin-1beta - metabolism | Interleukin 1 Receptor Antagonist Protein - metabolism | Female | Cattle Diseases - drug therapy | Interleukin-6 - metabolism | Acute-Phase Proteins - metabolism | Anti-Inflammatory Agents - pharmacology | Antioxidants - pharmacology | Toll-Like Receptor 4 - metabolism | Animals | Carrier Proteins - metabolism | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Melatonin - pharmacology | Mammary glands | Melatonin | Research | Health aspects | Lipopolysaccharides | Cell culture | Oxidative stress | C-reactive protein | Zoology | Glands | Cerebrospinal fluid | Cell surface | Antioxidants | Interleukin 6 | Proteins | Microorganisms | Metabolites | Amniotic fluid | Rodents | Interleukin 1 | Biosphere | Bacteria | Tumor necrosis factor-TNF | Amyloid | Gram-negative bacteria | Mammary gland | Astrocytoma | Public health | Mastitis | Advanced glycosylation end products | Antiinflammatory agents | Cytokines | Animal sciences | Inflammation | Epithelium | Gene expression | Pregnancy | Monocytes | Acute phase substances | Cell death | Ligands | Chemokines | Cancer | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0175824
Serum amyloid A (SAA) is an acute phase protein with cytokine-like and chemotactic properties, that is markedly up-regulated during various inflammatory conditions... 
C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | B TYPE-I | SCAVENGER RECEPTOR | POLYMORPHONUCLEAR LEUKOCYTES | ACUTE-PHASE REACTANT | PROTEIN-COUPLED RECEPTOR | MULTIDISCIPLINARY SCIENCES | SERUM-AMYLOID-A | CORONARY-ARTERY-DISEASE | HIGH-DENSITY-LIPOPROTEIN | Inflammation - chemically induced | Inflammation - pathology | Liver - pathology | Kidney - pathology | Serum Amyloid A Protein - pharmacology | Humans | Fluorescent Dyes - metabolism | Serum Amyloid A Protein - genetics | Inflammation - metabolism | Kidney - metabolism | MAP Kinase Kinase 4 - metabolism | Transfection | Biological Transport | Liver - drug effects | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | p38 Mitogen-Activated Protein Kinases - metabolism | Transgenes | Receptors, Scavenger - metabolism | Fluorobenzenes - metabolism | Kidney - drug effects | Lysosome-Associated Membrane Glycoproteins - metabolism | Serum Amyloid A Protein - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Liver - metabolism | Gene Expression Regulation | p38 Mitogen-Activated Protein Kinases - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Receptors, Scavenger - genetics | Inflammation - genetics | Lysosome-Associated Membrane Glycoproteins - genetics | MAP Kinase Kinase 4 - genetics | Mice | HeLa Cells | Mitogen-Activated Protein Kinase 1 - metabolism | Physiological aspects | Development and progression | Cellular signal transduction | Genetic aspects | Glycoproteins | Inflammation | Research | Heart | Plasma | Cytokines | Laboratories | Neutrophils | Lipids | Smooth muscle | JNK protein | Metabolism | Kinases | Proteins | Medicine | Studies | Immunology | Rheumatoid arthritis | Tumor necrosis factor-TNF | In vivo methods and tests | Diabetes | Kidney diseases | Localization | In vitro methods and tests
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, p. e0131295
Atherosclerosis is an inflammatory disease. As an inflammatory molecule, C-reactive protein (CRP... 
TRANSITION | KAPPA-B ACTIVATION | HYPERHOMOCYSTEINEMIA | RISK-FACTOR | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | DISEASE | ATHEROSCLEROSIS | INDUCED MIGRATION | PLASMA HOMOCYSTEINE | C-Reactive Protein - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Hyperhomocysteinemia - metabolism | Male | PPAR gamma - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | C-Reactive Protein - metabolism | Homocysteine - pharmacology | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Aorta, Thoracic - drug effects | Phosphorylation - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Muscle, Smooth, Vascular - drug effects | PPAR gamma - genetics | Homocysteine - antagonists & inhibitors | Hyperhomocysteinemia - pathology | Emodin - pharmacology | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Gene Expression Regulation | Aorta, Thoracic - metabolism | Rats | p38 Mitogen-Activated Protein Kinases - genetics | C-Reactive Protein - genetics | Muscle, Smooth, Vascular - cytology | Rats, Sprague-Dawley | Hyperhomocysteinemia - drug therapy | Aorta, Thoracic - cytology | Reactive Oxygen Species - antagonists & inhibitors | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | PPAR gamma - agonists | Hyperhomocysteinemia - genetics | Primary Cell Culture | Mitogen-Activated Protein Kinase 1 - metabolism | Atherogenesis | Phosphorylation | C-reactive protein | Smooth muscle | mRNA | Kinases | Experiments | Risk factors | Proteins | Rodents | Atherosclerosis | Tumor necrosis factor-TNF | Inhibition | Pretreatment | Immunoglobulins | Oxygen | Cytokines | Muscles | Extracellular signal-regulated kinase | Pharmacology | Inflammation | Gene expression | Emodin | Medicine | Molecular modelling | Arteriosclerosis | Pharmacy | Laxatives | Homocysteine
Journal Article
Journal Article