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FRONTIERS IN IMMUNOLOGY, ISSN 1664-3224, 06/2014, Volume 5
The ability of circulating blood monocytes to express C1q receptors (cC1qR and gC1qR) as well as to synthesize and secrete the classical pathway proteins C1q,... 
c1q | SYSTEMIC-LUPUS-ERYTHEMATOSUS | C1q in SLE | IMMUNOLOGY | C1q in autoimmunity | IMMATURE DENDRITIC CELLS | monocyte C1 | IN-VITRO | CYTOKINE PRODUCTION | COMPLEMENT SUBCOMPONENT-C1Q | DC-SIGN | APOPTOTIC CELLS | C1q and C1q receptors | TOLL-LIKE RECEPTORS | DC and C1 | BINDING | T-CELLS
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 2014, Volume 5, p. 278
The ability of circulating blood monocytes to express C1q receptors (cC1qR and gC1qR) as well as to synthesize and secrete the classical pathway proteins C1q,... 
C1q and C1q receptors | C1q | Monocyte C1 | DC and C1 | C1q in SLE | C1q in autoimmunity | c1q
Journal Article
Developmental and Comparative Immunology, ISSN 0145-305X, 2010, Volume 34, Issue 8, pp. 837 - 846
The globular C1q-domain-containing (C1qDC) proteins are a family of versatile pattern recognition receptors via their globular C1q (gC1q) domain to bind... 
Innate immunity | Complement system | Microbial agglutinating activity | Argopecten irradians | C1qDC protein | Pattern recognition receptor | C-REACTIVE PROTEIN | B-CHAIN | CRYSTAL-STRUCTURE | BINDING LECTIN | C1Q-DOMAIN-CONTAINING PROTEINS | IMMUNOLOGY | CHLAMYS-FARRERI | GLOBULAR HEAD | TUMOR-NECROSIS-FACTOR | ZOOLOGY | COMPLEMENT COMPONENT C1Q | SUBCOMPONENT C1Q | Receptors, Pattern Recognition - immunology | Actinomycetales Infections - genetics | Complement C1q - immunology | Hemocytes - immunology | Humans | Gram-Negative Bacterial Infections - immunology | Molecular Sequence Data | Phylogeny | Complement C1q - metabolism | Receptors, Pattern Recognition - metabolism | Pectinidae - genetics | Micrococcus luteus - immunology | Actinomycetales Infections - immunology | Mycoses - metabolism | Hepatopancreas - metabolism | Agglutination | Hemocytes - microbiology | Pichia - pathogenicity | Complement C1q - chemistry | Amino Acid Sequence | Micrococcus luteus - pathogenicity | Pectinidae - microbiology | Receptors, Pattern Recognition - chemistry | Gram-Negative Bacterial Infections - genetics | Mycoses - genetics | Protein Structure, Tertiary - genetics | Complement C1q - genetics | Listonella - pathogenicity | Immunity, Innate | Gram-Negative Bacterial Infections - metabolism | Hemocytes - metabolism | Protein Folding | Animals | Hepatopancreas - microbiology | Receptors, Pattern Recognition - genetics | Listonella - immunology | Mycoses - immunology | Actinomycetales Infections - metabolism | Hepatopancreas - immunology | Pichia - immunology
Journal Article
Nature Immunology, ISSN 1529-2908, 09/2013, Volume 14, Issue 9, pp. 917 - 926
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2013, Volume 110, Issue 21, pp. 8650 - 8655
Complement C1q is a hexameric molecule assembled from 18 polypeptide chains of three different types encoded by three genes. This versatile recognition protein... 
Proteins | Molecules | Cell lines | Materials | Cell aggregates | Ligands | Mass spectroscopy | Biochemistry | Electron microscopy | Molecular chains | C1 complex | Protein engineering | Innate immunity | B-CHAIN | ACTIVATION | 1ST COMPONENT | PROTEIN | MULTIDISCIPLINARY SCIENCES | LONG PENTRAXIN PTX3 | LIGAND-RECOGNITION | MANNAN-BINDING LECTIN | protein engineering | innate immunity | STRUCTURAL BASIS | PATHWAY | SUBCOMPONENT C1Q | Surface Plasmon Resonance | Complement C1r - genetics | Calcium - metabolism | Humans | Complement C1s - metabolism | Mutation, Missense | Complement C1q - metabolism | Complement C1r - metabolism | Complement C1s - genetics | Complement C1r - chemistry | Complement C1s - chemistry | C-Reactive Protein - metabolism | HEK293 Cells | Protein Structure, Quaternary | Binding Sites | Complement C1q - chemistry | Complement Activation - physiology | Protein Structure, Tertiary | Recombinant Proteins - metabolism | Gene Expression | Immunoglobulin G - chemistry | Serum Amyloid P-Component - chemistry | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Complement C1q - genetics | C-Reactive Protein - chemistry | Serum Amyloid P-Component - metabolism | Amino Acid Substitution | Immunoglobulin G - metabolism | Protein Binding - physiology | Bone morphogenetic proteins | Polypeptides | Research | Properties | Proteases | Protein binding | C-Reactive Protein | Calcium | Complement Activation | Immunoglobulin G | Biochemistry, Molecular Biology | Serum Amyloid P-Component | Recombinant Proteins | Complement C1q | Complement C1s | Complement C1r | Life Sciences | Protein Binding | Biomolecules | Biological Sciences
Journal Article
Cell, ISSN 0092-8674, 05/2016, Volume 165, Issue 4, pp. 921 - 935
Journal Article
Arkhiv Patologii, ISSN 0004-1955, 01/2018, Volume 80, Issue 1, pp. 46 - 51
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 03/2017, Volume 312, Issue 3, pp. F516 - F532
We have examined the pathogenic role of increased complement expression and activation during kidney fibrosis. Here, we show that PDGFR beta-positive pericytes... 
Pericytes | C1q | SYSTEM | C1Q | REPAIR | PHYSIOLOGY | PATHWAY | INJURY | UROLOGY & NEPHROLOGY | CKD PROGRESSION | RENAL ISCHEMIA-REPERFUSION | DISTINCT ROLES | CHRONIC KIDNEY-DISEASE | EXPRESSION | Wnt3A Protein - metabolism | Ureteral Obstruction - complications | Leukocyte Common Antigens - metabolism | Complement Activation | Complement C1q - immunology | Male | Complement C1q - metabolism | Renal Insufficiency, Chronic - metabolism | Complement C3 - deficiency | Time Factors | Pericytes - pathology | Inflammation Mediators - metabolism | Renal Insufficiency, Chronic - genetics | Kidney Tubules - pathology | Kidney Tubules - metabolism | Complement C3 - genetics | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Wnt Signaling Pathway | Disease Models, Animal | Receptor, Platelet-Derived Growth Factor beta - metabolism | Folic Acid | Kidney Tubules - immunology | Renal Insufficiency, Chronic - immunology | Cytokines - metabolism | Macrophages - pathology | Complement C3 - metabolism | Mice, Inbred C57BL | Pericytes - metabolism | Cell Communication | Genotype | Complement C1q - genetics | Disease Progression | Complement C3 - immunology | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Macrophages - metabolism | Pericytes - immunology | Phenotype | Animals | Fibrosis | Complement C1q - deficiency | Physiological aspects | Cell interaction | Immune response | Observations | Health aspects | pericytes
Journal Article