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FRONTIERS IN IMMUNOLOGY, ISSN 1664-3224, 06/2014, Volume 5
The ability of circulating blood monocytes to express C1q receptors (cC1qR and gC1qR) as well as to synthesize and secrete the classical pathway proteins C1q,... 
c1q | SYSTEMIC-LUPUS-ERYTHEMATOSUS | C1q in SLE | IMMUNOLOGY | C1q in autoimmunity | IMMATURE DENDRITIC CELLS | monocyte C1 | IN-VITRO | CYTOKINE PRODUCTION | COMPLEMENT SUBCOMPONENT-C1Q | DC-SIGN | APOPTOTIC CELLS | C1q and C1q receptors | TOLL-LIKE RECEPTORS | DC and C1 | BINDING | T-CELLS
Journal Article
Developmental and Comparative Immunology, ISSN 0145-305X, 2010, Volume 34, Issue 8, pp. 837 - 846
The globular C1q-domain-containing (C1qDC) proteins are a family of versatile pattern recognition receptors via their globular C1q (gC1q) domain to bind... 
Innate immunity | Complement system | Microbial agglutinating activity | Argopecten irradians | C1qDC protein | Pattern recognition receptor | C-REACTIVE PROTEIN | B-CHAIN | CRYSTAL-STRUCTURE | BINDING LECTIN | C1Q-DOMAIN-CONTAINING PROTEINS | IMMUNOLOGY | CHLAMYS-FARRERI | GLOBULAR HEAD | TUMOR-NECROSIS-FACTOR | ZOOLOGY | COMPLEMENT COMPONENT C1Q | SUBCOMPONENT C1Q | Receptors, Pattern Recognition - immunology | Actinomycetales Infections - genetics | Complement C1q - immunology | Hemocytes - immunology | Humans | Gram-Negative Bacterial Infections - immunology | Molecular Sequence Data | Phylogeny | Complement C1q - metabolism | Receptors, Pattern Recognition - metabolism | Pectinidae - genetics | Micrococcus luteus - immunology | Actinomycetales Infections - immunology | Mycoses - metabolism | Hepatopancreas - metabolism | Agglutination | Hemocytes - microbiology | Pichia - pathogenicity | Complement C1q - chemistry | Amino Acid Sequence | Micrococcus luteus - pathogenicity | Pectinidae - microbiology | Receptors, Pattern Recognition - chemistry | Gram-Negative Bacterial Infections - genetics | Mycoses - genetics | Protein Structure, Tertiary - genetics | Complement C1q - genetics | Listonella - pathogenicity | Immunity, Innate | Gram-Negative Bacterial Infections - metabolism | Hemocytes - metabolism | Protein Folding | Animals | Hepatopancreas - microbiology | Receptors, Pattern Recognition - genetics | Listonella - immunology | Mycoses - immunology | Actinomycetales Infections - metabolism | Hepatopancreas - immunology | Pichia - immunology | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 09/2013, Volume 14, Issue 9, pp. 917 - 926
Journal Article
Arkhiv Patologii, ISSN 0004-1955, 01/2018, Volume 80, Issue 1, pp. 46 - 51
Journal Article
Cell, ISSN 0092-8674, 05/2016, Volume 165, Issue 4, pp. 921 - 935
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 03/2017, Volume 312, Issue 3, pp. F516 - F532
We have examined the pathogenic role of increased complement expression and activation during kidney fibrosis. Here, we show that PDGFR beta-positive pericytes... 
Pericytes | C1q | SYSTEM | C1Q | REPAIR | PHYSIOLOGY | PATHWAY | INJURY | UROLOGY & NEPHROLOGY | CKD PROGRESSION | RENAL ISCHEMIA-REPERFUSION | DISTINCT ROLES | CHRONIC KIDNEY-DISEASE | EXPRESSION | Wnt3A Protein - metabolism | Ureteral Obstruction - complications | Leukocyte Common Antigens - metabolism | Complement Activation | Complement C1q - immunology | Male | Complement C1q - metabolism | Renal Insufficiency, Chronic - metabolism | Complement C3 - deficiency | Time Factors | Pericytes - pathology | Inflammation Mediators - metabolism | Renal Insufficiency, Chronic - genetics | Kidney Tubules - pathology | Kidney Tubules - metabolism | Complement C3 - genetics | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Wnt Signaling Pathway | Disease Models, Animal | Receptor, Platelet-Derived Growth Factor beta - metabolism | Folic Acid | Kidney Tubules - immunology | Renal Insufficiency, Chronic - immunology | Cytokines - metabolism | Macrophages - pathology | Complement C3 - metabolism | Mice, Inbred C57BL | Pericytes - metabolism | Cell Communication | Genotype | Complement C1q - genetics | Disease Progression | Complement C3 - immunology | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Macrophages - metabolism | Pericytes - immunology | Phenotype | Animals | Fibrosis | Complement C1q - deficiency | Physiological aspects | Cell interaction | Immune response | Observations | Health aspects | Index Medicus | pericytes
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2017, Volume 199, Issue 3, pp. 1069 - 1085
Inflammatory processes play a key role in pathophysiology of many neurologic diseases/trauma, but the effect of immune cells and factors on... 
CELLULAR INFLAMMATORY RESPONSE | IN-VITRO | POLYMORPHONUCLEAR LEUKOCYTES | IMMUNE CELLS | ASTROCYTIC DIFFERENTIATION | COMPLEMENT CLEAVAGE PRODUCT | ADULT HIPPOCAMPAL NEUROGENESIS | STIMULATED HUMAN-MONOCYTES | STEM/PROGENITOR CELLS | SPINAL-CORD-INJURY | IMMUNOLOGY | Complement C1q - biosynthesis | Complement C1q - immunology | Humans | Complement C3a - genetics | Neural Stem Cells - immunology | Complement C3a - antagonists & inhibitors | Cell Differentiation - physiology | Neutrophils - metabolism | Astrocytes - drug effects | Complement C3a - biosynthesis | Cells, Cultured | Neural Stem Cells - drug effects | Neutrophils - immunology | Neural Stem Cells - physiology | Complement C1q - genetics | Immunity, Innate | Culture Media, Conditioned | Astrocytes - physiology | Animals | Complement C1q - antagonists & inhibitors | Spinal Cord Injuries - immunology | Mice | Spinal Cord Injuries - physiopathology | Complement C3a - immunology | Cell Movement | Spinal cord | Leukocyte migration | Central nervous system | Innate immunity | Leukocytes (neutrophilic) | Inflammation | Gliogenesis | Leukocytes | Spinal cord injury | Trauma | Immunity | Cell adhesion & migration | Neurological diseases | White blood cells | Immune systems | Proteins | Complement component C1q | Stem cells | Neural stem cells | Leukocytes (polymorphonuclear) | Immunity (humoral) | Conditioning | Cell migration | Index Medicus | Abridged Index Medicus | Innate Immunity and Inflammation
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2019, Volume 1901, pp. 183 - 189
Anti-C1q autoantibodies may be found in many conditions, most commonly in systemic lupus erythematosus (SLE) and hypocomplementemic urticarial vasculitis... 
Western blot | Hypocomplementemic urticarial vasculitis syndrome | Systemic lupus erythematosus | Anti-C1q autoantibodies | C1q protein chains B and C | Protein Binding | Autoantibodies - analysis | Blotting, Western - methods | Complement C1q - isolation & purification | Complement C1q - immunology | Humans
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