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1. Full Text C3 glomerulopathy: consensus report
by Pickering, Matthew C and D'Agati, Vivette D and Nester, Carla M and Smith, Richard J and Haas, Mark and Appel, Gerald B and Alpers, Charles E and Bajema, Ingeborg M and Bedrosian, Camille and Braun, Michael and Doyle, Mittie and Fakhouri, Fadi and Fervenza, Fernando C and Fogo, Agnes B and Frémeaux-Bacchi, Véronique and Gale, Daniel P and Goicoechea de Jorge, Elena and Griffin, Gene and Harris, Claire L and Michael Holers, V and Johnson, Sally and Lavin, Peter J and Medjeral-Thomas, Nicholas and Paul Morgan, B and Nast, Cynthia C and Noel, Laure-Hélène and Keith Peters, D and Rodríguez de Córdoba, Santiago and Servais, Aude and Sethi, Sanjeev and Song, Wen-Chao and Tamburini, Paul and Thurman, Joshua M and Zavros, Michael and Terence Cook, H
Kidney International, ISSN 0085-2538, 12/2013, Volume 84, Issue 6, pp. 1079 - 1089
C3 glomerulopathy is a recently introduced pathological entity whose original definition was glomerular pathology characterized by C3 accumulation with absent... 
immunology and pathology | complement | glomerulonephritis | clinical nephrology | clinical immunology | ECULIZUMAB | ACTIVATION | MECHANISM | CLASSIFICATION | COMPLEMENT FACTOR-H | PROVIDES INSIGHTS | MUTATION | DYSREGULATION | UROLOGY & NEPHROLOGY | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | DENSE-DEPOSIT DISEASE | Complement C3 - analysis | Predictive Value of Tests | Glomerulonephritis - therapy | Prognosis | Humans | Cooperative Behavior | Glomerulonephritis - diagnosis | Glomerulonephritis - immunology | International Cooperation | Kidney Glomerulus - pathology | Biopsy | Biomedical Research | Kidney Glomerulus - immunology | Meeting Report
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2. Full Text C3 Glomerulopathy
by Riedl, Magdalena and Thorner, Paul and Licht, Christoph
Pediatric Nephrology, ISSN 0931-041X, 1/2017, Volume 32, Issue 1, pp. 43 - 57
Recent advances in our understanding of the disease pathology of membranoproliferative glomerulonephritis has resulted in its re-classification as complement... 
Pediatrics | Nephrology | Medicine & Public Health | Membranoproliferative glomerulonephritis | Dense deposit disease | Eculizumab | Complement | C3 Glomerulopathy | C3 Nephritic factor | Urology | HEMOLYTIC-UREMIC SYNDROME | MESANGIOCAPILLARY GLOMERULONEPHRITIS | GLOMERULONEPHRITIS TYPE-II | ALTERNATE-DAY PREDNISONE | COMPLEMENT FACTOR-H | PLASMA-EXCHANGE | NEPHRITIC FACTOR | RECURRENT MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | UROLOGY & NEPHROLOGY | PEDIATRICS | MYCOPHENOLATE-MOFETIL | DENSE-DEPOSIT DISEASE | Glomerulonephritis, Membranoproliferative - pathology | Animals | Complement C3 | Humans | Complement C3 Nephritic Factor | Treatment Outcome | Glomerulonephritis, Membranoproliferative - diagnosis | Glomerulonephritis, Membranoproliferative - therapy | Disease Models, Animal | Glomerulonephritis | Development and progression | Care and treatment | Genetic aspects | Diagnosis | Research
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3. Full Text C3 glomerulopathy-associated CFHR1 mutation alters FHR oligomerization and complement regulation
by Tortajada, Agustín and Yébenes, Hugo and Abarrategui-Garrido, Cynthia and Anter, Jaouad and García-Fernández, Jesús M and Martínez-Barricarte, Rubén and Alba-Domínguez, María and Malik, Talat H and Bedoya, Rafael and Pérez, Rocío Cabrera and Trascasa, Margarita López and Pickering, Matthew C and Harris, Claire L and Sánchez-Corral, Pilar and Llorca, Oscar and De Córdoba, Santiago Rodríguez
Journal of Clinical Investigation, ISSN 0021-9738, 06/2013, Volume 123, Issue 6, pp. 2434 - 2446
C3 glomerulopathies (C3G) are a group of severe renal diseases with distinct patterns of glomerular inflammation and C3 deposition caused by complement... 
ECULIZUMAB | GLOMERULONEPHRITIS | MEDICINE, RESEARCH & EXPERIMENTAL | PROVIDES INSIGHTS | HEMOLYTIC-UREMIC SYNDROME | MPGN-II | MACULAR DEGENERATION | HEPARIN | H-RELATED PROTEINS | DENSE-DEPOSIT DISEASE | REVEALS | Gene Duplication | Hemolysis | Immobilized Proteins - chemistry | Kidney - pathology | Complement C3 - metabolism | Humans | Middle Aged | Protein Multimerization | Complement C3b Inactivator Proteins - genetics | Complement C3b Inactivator Proteins - chemistry | Male | Sequence Analysis, DNA | Kidney Diseases - genetics | Complement System Proteins - metabolism | Complement C3b Inactivator Proteins - metabolism | Complement C3 - chemistry | Pedigree | Immobilized Proteins - metabolism | Protein Structure, Quaternary | Complement System Proteins - isolation & purification | Protein Binding | Female | Mutagenesis, Insertional | Child | Glomerulonephritis | Complement factor H | Gene mutations | Physiological aspects | Causes of | Genetic aspects | Research | Proteins | Competition | Plasma | Carbohydrates | Mutation | Crystal structure
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4. C3 glomerulopathy
by Pickering, Matthew
2017, The complement system
Glomerulonephritis | Kidney Diseases | Kidneys | pathology | Complement C3 | therapy | eculizumab | Complement Factor H | analysis | Diseases | Antibodies, Monoclonal, Humanized | Immunology | Complement (Immunology)
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5. Full Text Update on C3 glomerulopathy
by Barbour, Thomas D and Ruseva, Marieta M and Pickering, Matthew C
Nephrology Dialysis Transplantation, ISSN 0931-0509, 05/2016, Volume 31, Issue 5, pp. 717 - 725
C3 glomerulopathy refers to a disease process in which abnormal control of complement activation, degradation or deposition results in predominant C3 fragment... 
complement | factor H | C3 glomerulopathy | dense deposit | ECULIZUMAB | GLOMERULONEPHRITIS | RENAL-TRANSPLANTATION | COMPLEMENT REGULATION | TRANSPLANTATION | THERAPY | CFHR1 | MUTATION | UROLOGY & NEPHROLOGY | PATIENT | DENSE-DEPOSIT DISEASE | Kidney Diseases - physiopathology | Animals | Complement Factor H - metabolism | Complement Activation | Complement C3 - metabolism | Humans | Kidney Glomerulus - metabolism | Kidney Glomerulus - pathology | Cutting-Edge Renal Science
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6. Full Text C3 glomerulopathy: a new classification
by Pickering, Matthew C and Cook, H. Terence and Noël, Laure-Hélène and Frémeaux-Bacchi, Véronique and Fakhouri, Fadi
Nature Reviews Nephrology, ISSN 1759-5061, 08/2010, Volume 6, Issue 8, pp. 494 - 499
Several distinct pathological patterns of glomerular inflammation are associated with abnormal regulation of the complement system, specifically, with... 
COMPLEMENT FACTOR-H | HEMOLYTIC-UREMIC SYNDROME | MESANGIOCAPILLARY GLOMERULONEPHRITIS | UROLOGY & NEPHROLOGY | GLOMERULONEPHRITIS TYPE-III | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | VARIANT | DENSE-DEPOSIT DISEASE | CHILDREN | Complement C3 - metabolism | Humans | Risk Factors | Kidney Glomerulus - metabolism | Glomerulonephritis - classification | Glomerulonephritis - etiology | Kidney Glomerulus - pathology | Glomerulonephritis | Immunoglobulins | Research | Health aspects | Risk factors
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7. Full Text Complement inhibition in C3 glomerulopathy
by Nester, Carla M and Smith, Richard J.H
Seminars in Immunology, ISSN 1044-5323, 2016, Volume 28, Issue 3, pp. 241 - 249
Highlights • Dysregulation of the alternative pathway of complement plays a critical role in C3 glomerulopathy. • Ultra-rare variants in complement genes are... 
Allergy and Immunology | Eculizumab | Complement | Membranoproliferative glomerulonephritis | Dense deposit disease | C3 glomerulopathy | C3 glomerulonephritis | ACTIVATION | IMMUNOLOGY | FACTOR-H DEFICIENCY | NEPHRITIC FACTOR | GENE | DYSREGULATION | MONOCLONAL GAMMOPATHY | MUTATIONS | DENSE-DEPOSIT DISEASE | Biomarkers - metabolism | Immunotherapy - methods | Kidney - pathology | Gene Frequency | Humans | Glomerulonephritis, Membranous - genetics | Genotype | Complement C3 - immunology | Polymorphism, Genetic | Kidney - metabolism | Animals | Glomerulonephritis, Membranous - immunology | Complement Inactivating Agents - therapeutic use | Complement Pathway, Alternative | Complement C3 - genetics | Glomerulonephritis, Membranous - therapy | Medical colleges
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8. Full Text Recent insights into C3 glomerulopathy
by Barbour, Thomas D and Pickering, Matthew C and Terence Cook, H
Nephrology Dialysis Transplantation, ISSN 0931-0509, 07/2013, Volume 28, Issue 7, pp. 1685 - 1693
'C3 glomerulopathy' is a recent disease classification comprising several rare types of glomerulonephritis (GN), including dense deposit disease (DDD), C3... 
Glomerulonephritis | Eculizumab | Complement | Dense deposit | Kidney | HEMOLYTIC-UREMIC SYNDROME | GLOMERULONEPHRITIS TYPE-II | eculizumab | RENAL-TRANSPLANTATION | dense deposit | TRANSPLANTATION | kidney | COMPLEMENT FACTOR-H | FACTOR B | NEPHRITIC FACTOR | ALTERNATIVE PATHWAY | UROLOGY & NEPHROLOGY | MONOCLONAL GAMMOPATHY | complement | glomerulonephritis | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | DENSE-DEPOSIT DISEASE | Animals | Complement C3 - metabolism | Kidney Diseases - pathology | Humans | Kidney Glomerulus - metabolism | Kidney Glomerulus - pathology | Kidney Diseases - metabolism | Cutting-Edge Renal Science
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9. Full Text The role of complement in C3 glomerulopathy
by Zipfel, Peter F and Skerka, Christine and Chen, Qian and Wiech, Thorsten and Goodship, Tim and Johnson, Sally and Fremeaux-Bacchi, Veronique and Nester, Clara and Córdoba, Santiago Rodríguez de and Noris, Marina and Pickering, Matthew and Smith, Richard
Molecular Immunology, ISSN 0161-5890, 09/2015, Volume 67, Issue 1, pp. 21 - 30
C3 glomerulopathy describes a spectrum of disorders with glomerular pathology associated with C3 cleavage product deposition and with defective complement... 
MPGN membranoproliferative | C3 nephritic factor | Dense deposit diseases | Complement C3 glomerulopathy | HEMOLYTIC-UREMIC SYNDROME | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLOMERULONEPHRITIS TYPE-II | MACULAR DEGENERATION | IMMUNOLOGY | FACTOR-H-AUTOANTIBODIES | DENSE DEPOSIT DISEASE | NEPHRITIC FACTOR | ALTERNATIVE PATHWAY | BINDING-AFFINITY | NEISSERIA-GONORRHOEAE | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | Complement C3 Nephritic Factor - genetics | Complement Activation | Humans | Complement C3b Inactivator Proteins - genetics | Complement C3b Inactivator Proteins - chemistry | Complement C3 - chemistry | Protein Aggregation, Pathological - pathology | Protein Aggregation, Pathological - diagnosis | B-Lymphocytes - pathology | Protein Aggregation, Pathological - immunology | T-Lymphocytes - pathology | Complement C3 - genetics | Protein Aggregation, Pathological - genetics | Adaptive Immunity | Glomerulonephritis, Membranoproliferative - pathology | Gene Expression | Complement C3 Nephritic Factor - chemistry | Biomarkers - blood | Kidney Glomerulus - pathology | Glomerulonephritis, Membranoproliferative - genetics | Glomerulonephritis, Membranoproliferative - immunology | B-Lymphocytes - immunology | T-Lymphocytes - immunology | Glomerulonephritis, Membranoproliferative - diagnosis | Kidney Glomerulus - immunology | Kidney Glomerulus - chemistry | Medical colleges
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10. Full Text C3 glomerulopathy associated with monoclonal Ig is a distinct subtype
by Ravindran, Aishwarya and Fervenza, Fernando C and Smith, Richard J.H and Sethi, Sanjeev
Kidney International, ISSN 0085-2538, 07/2018, Volume 94, Issue 1, pp. 178 - 186
Monoclonal immunoglobulins (MIg) may play a causal role in C3 glomerulopathy (C3G) by impairing regulation of the alternative pathway of complement.... 
dense deposit disease | monoclonal Ig | C3 glomerulopathy | alternative pathway of complement | C3 glomerulonephritis | MULTIPLE-MYELOMA | CRITERIA | ABNORMALITIES | COMPLEMENT ALTERNATIVE PATHWAY | RISK | GAMMOPATHY | PROLIFERATIVE GLOMERULONEPHRITIS | UNDETERMINED SIGNIFICANCE | UROLOGY & NEPHROLOGY | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | Complement C3 - analysis | Age Factors | Follow-Up Studies | Immunoglobulin G - blood | Humans | Middle Aged | Child, Preschool | Male | Glomerulonephritis, Membranoproliferative - blood | Young Adult | Paraproteinemias - pathology | Immunoglobulin G - immunology | Aged, 80 and over | Adult | Female | Child | Antibodies, Monoclonal - immunology | Paraproteinemias - immunology | Glomerulonephritis, Membranoproliferative - pathology | Kidney Glomerulus - pathology | Complement C3 - immunology | Glomerulonephritis, Membranoproliferative - immunology | Biopsy | Complement Pathway, Alternative - immunology | Adolescent | Aged | Paraproteinemias - blood | Kidney Glomerulus - immunology
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11. Full Text Treatment options for C3 glomerulopathy
by Nester, Carla M and Smith, Richard J
Current Opinion in Nephrology and Hypertension, ISSN 1062-4821, 03/2013, Volume 22, Issue 2, pp. 231 - 237
PURPOSE OF REVIEWThe purpose of this review is to discuss emerging nomenclature, review the salient clinicopathological features and describe the therapeutic... 
dense deposit disease | membranoproliferative glomerulonephritis | C3 glomerulopathy | Alternate pathway of complement | C3 glomerulonephritis | ECULIZUMAB | ACTIVATION | HEMOLYTIC-UREMIC SYNDROME | ABNORMALITIES | DEFICIENT MICE | PROTEIN 5 | NEPHROPATHY | COMPLEMENT FACTOR-H | alternate pathway of complement | UROLOGY & NEPHROLOGY | PERIPHERAL VASCULAR DISEASE | DENSE-DEPOSIT DISEASE | Kidney - drug effects | Kidney - pathology | Complement C3 - metabolism | Humans | Immunosuppressive Agents - therapeutic use | Kidney - immunology | Treatment Outcome | Glomerulonephritis, Membranoproliferative - drug therapy | Young Adult | Glomerulonephritis, Membranoproliferative - immunology | Animals | Complement Pathway, Alternative - drug effects | Adolescent | Adult | Female | Immunosuppressive Agents - adverse effects | Glomerulonephritis, Membranoproliferative - diagnosis | Complement Activation - drug effects | Dense Deposit Disease | C3 Glomerulonephritis | C3 Glomerulopathy
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12. Full Text Membranoproliferative glomerulonephritis and C3 glomerulopathy: resolving the confusion
by Sethi, Sanjeev and Nester, Carla M and Smith, Richard J.H
Kidney International, ISSN 0085-2538, 03/2012, Volume 81, Issue 5, pp. 434 - 441
Membranoproliferative glomerulonephritis (MPGN) denotes a general pattern of glomerular injury that is easily recognized by light microscopy. With additional... 
pathology | glomerular disease | complement | immunology | membranoproliferative glomerulonephritis (MPGN) | DENSE DEPOSIT DISEASE | PROTEIN | ALTERNATIVE PATHWAY | MUTATION | UROLOGY & NEPHROLOGY | FACTOR-H | membrano-proliferative glomerulonephritis (MPGN) | REVEALS | Glomerulonephritis, Membranoproliferative - drug therapy | Glomerulonephritis, Membranoproliferative - pathology | Complement C3 - antagonists & inhibitors | Glomerulonephritis, Membranoproliferative - classification | Complement C3 - metabolism | Glomerulonephritis - pathology | Humans | Glomerulonephritis - immunology | Glomerulonephritis - classification | Microscopy, Electron | Antimitotic Agents - therapeutic use | Microscopy, Fluorescence
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13. Full Text Treating C3 glomerulopathy with eculizumab
by Welte, Thomas and Arnold, Frederic and Kappes, Julia and Seidl, Maximilian and Häffner, Karsten and Bergmann, Carsten and Walz, Gerd and Neumann-Haefelin, Elke
BMC Nephrology, ISSN 1471-2369, 01/2018, Volume 19, Issue 1, pp. 7 - 10
Background: C3 glomerulopathy (C3G) is a rare, but severe glomerular disease with grim prognosis. The complex pathogenesis is just unfolding, and involves... 
Eculizumab | Complement | Dense deposit disease | C3 glomerulopathy | C3 glomerulonephritis | HEMOLYTIC-UREMIC SYNDROME | COMPLEMENT INHIBITOR ECULIZUMAB | RENAL-TRANSPLANTATION | MPGN | THERAPY | ALTERNATIVE PATHWAY | UROLOGY & NEPHROLOGY | PATIENT | DYSFUNCTION | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | DENSE-DEPOSIT DISEASE | Antibodies, Monoclonal, Humanized - therapeutic use | Complement C3 - metabolism | Glomerulonephritis - blood | Humans | Middle Aged | Male | Treatment Outcome | Glomerulonephritis - drug therapy | Young Adult | Adolescent | Adult | Female | Retrospective Studies | Glomerulonephritis | Diagnosis | Research | Drug therapy
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