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Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2012, Volume 131, Issue 2, pp. 541 - 548.e9
Background Mast cells express receptors for complement anaphylatoxins C3a and C5a (ie, C3a receptor [C3aR] and C5a receptor [C5aR]), and C3a and C5a are... 
Allergy and Immunology | C3a | inflammation | IgE | anaphylaxis | Anaphylatoxin | complement | mast cells | C5a | HUMAN BASOPHILS | COMPLEMENT ACTIVATION | MOUSE | IMMUNOLOGY | EFFECTOR PHASE | ALLERGY | IN-VIVO | HMC-1 | ASTHMA | HYPERSENSITIVITY REACTIONS | C3A ANAPHYLATOXIN | Receptor, Anaphylatoxin C5a - genetics | Inflammation - pathology | Anaphylatoxins - genetics | Skin - metabolism | Complement C5a - metabolism | Complement C3a - genetics | Receptors, Complement - metabolism | Inflammation - metabolism | Immunoglobulin E - genetics | Receptors, Complement - biosynthesis | Mast Cells - metabolism | Receptors, Complement - genetics | Receptor, Anaphylatoxin C5a - immunology | Female | Complement C3a - metabolism | Skin - pathology | Skin - immunology | Mast Cells - immunology | Immunoglobulin E - metabolism | Mice, Inbred C57BL | Cells, Cultured | Anaphylatoxins - immunology | Inflammation - immunology | Animals | Receptors, Complement - immunology | Inflammation - genetics | Complement C5a - genetics | Mice | Receptor, Anaphylatoxin C5a - biosynthesis | Complement C3a - immunology | Complement C5a - immunology | Albumin | Medical colleges | Respiratory agents | Skin | Immunotherapy | Histamine | Antigens | Flow cytometry | Rodents | Experiments | Allergies | Methods | Index Medicus | Abridged Index Medicus | Complement receptors | Cell activation | Anaphylatoxins | Degranulation | Hypersensitivity (immediate) | Inflammation | Mast cells | Passive cutaneous anaphylaxis | complement component C5a | complement component C3a
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 10/2011, Volume 668, Issue 1-2, pp. 299 - 304
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 09/2011, Volume 112, Issue 9, pp. 2594 - 2605
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 351 - 15
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2017, Volume 199, Issue 3, pp. 1069 - 1085
Inflammatory processes play a key role in pathophysiology of many neurologic diseases/trauma, but the effect of immune cells and factors on... 
CELLULAR INFLAMMATORY RESPONSE | IN-VITRO | POLYMORPHONUCLEAR LEUKOCYTES | IMMUNE CELLS | ASTROCYTIC DIFFERENTIATION | COMPLEMENT CLEAVAGE PRODUCT | ADULT HIPPOCAMPAL NEUROGENESIS | STIMULATED HUMAN-MONOCYTES | STEM/PROGENITOR CELLS | SPINAL-CORD-INJURY | IMMUNOLOGY | Complement C1q - biosynthesis | Complement C1q - immunology | Humans | Complement C3a - genetics | Neural Stem Cells - immunology | Complement C3a - antagonists & inhibitors | Cell Differentiation - physiology | Neutrophils - metabolism | Astrocytes - drug effects | Complement C3a - biosynthesis | Cells, Cultured | Neural Stem Cells - drug effects | Neutrophils - immunology | Neural Stem Cells - physiology | Complement C1q - genetics | Immunity, Innate | Culture Media, Conditioned | Astrocytes - physiology | Animals | Complement C1q - antagonists & inhibitors | Spinal Cord Injuries - immunology | Mice | Spinal Cord Injuries - physiopathology | Complement C3a - immunology | Cell Movement | Spinal cord | Leukocyte migration | Central nervous system | Innate immunity | Leukocytes (neutrophilic) | Inflammation | Gliogenesis | Leukocytes | Spinal cord injury | Trauma | Immunity | Cell adhesion & migration | Neurological diseases | White blood cells | Immune systems | Proteins | Complement component C1q | Stem cells | Neural stem cells | Leukocytes (polymorphonuclear) | Immunity (humoral) | Conditioning | Cell migration | Index Medicus | Abridged Index Medicus | Innate Immunity and Inflammation
Journal Article
The EMBO Journal, ISSN 0261-4189, 03/2006, Volume 25, Issue 6, pp. 1364 - 1374
Through its involvement in inflammation, opsonization, and cytolysis, the complement protects against infectious agents. Although most of the complement... 
C3 | neurogenesis | mice | cerebral ischemia | complement system | Cerebral ischemia | Complement system | Mice | Neurogenesis | HEMATOPOIETIC STEM/PROGENITOR CELLS | DIRECTED MIGRATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUBVENTRICULAR ZONE | CELL BIOLOGY | NEURAL STEM-CELLS | C5A RECEPTOR | TARGETED DISRUPTION | LIVER-REGENERATION | CENTRAL-NERVOUS-SYSTEM | C3A RECEPTOR | FUNCTIONAL RECEPTOR | Receptor, Anaphylatoxin C5a - genetics | Central Nervous System - metabolism | Complement C3a - physiology | Complement Activation | Receptors, Complement - antagonists & inhibitors | Mice, Inbred C57BL | Male | Neurons - cytology | Complement C5a - physiology | Stem Cells - cytology | Stem Cells - metabolism | Complement C3a - genetics | Mice, Knockout | Receptors, Complement - metabolism | Animals | Central Nervous System - cytology | Receptors, Complement - genetics | Complement C5a - genetics | Female | Neurons - metabolism | Ischemia - pathology | Receptor, Anaphylatoxin C5a - metabolism | Proteins | Enzymes | Nervous system | Neurons | Rodents | Cells | Index Medicus | cytology | pathology | Complement | MEDICIN OCH HÄLSOVETENSKAP | Complement C5a | Stem Cells | Complement C3a | Receptors | genetics | Ischemia | MEDICAL AND HEALTH SCIENCES | physiology | Inbred C57BL | Knockout | antagonists & inhibitors | Central Nervous System | Receptor | Anaphylatoxin C5a | metabolism
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2011, Volume 187, Issue 1, pp. 172 - 180
Journal Article