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Chemistry - A European Journal, ISSN 0947-6539, 11/2011, Volume 17, Issue 45, pp. 12573 - 12577
The oxidant directs: A mild, practical, efficient, and regioselective Ru-catalyzed isoquinolone synthesis with a broad substrate scope was reported (see... 
ruthenium | CiH activation | homogeneous catalysis | directing group | alkynes | DIRECT ARYLATION | BOND FORMATION | INTERNAL ALKYNES | CHEMISTRY, MULTIDISCIPLINARY | CARBON-HYDROGEN | FUNCTIONALIZATION | C-H activation | BENZAMIDES | OXIDATIVE COUPLING/CYCLIZATION | COUPLING REACTIONS | METAL | BENZOIC-ACIDS | Synthesis | Oxidants | Oxidizing agents | Activation | Oxidation | Bonding | Substrates
Journal Article
Nature Immunology, ISSN 1529-2908, 05/2010, Volume 11, Issue 5, pp. 411 - 418
Sensors of pathogens, such as Toll-like receptors (TLRs), detect microbes to activate transcriptional programs that orchestrate adaptive responses to specific... 
UNFOLDED PROTEIN RESPONSE | PATHWAY | ER STRESS | HOST-DEFENSE | ENDOPLASMIC-RETICULUM STRESS | FRANCISELLA-TULARENSIS INFECTION | IMMUNOLOGY | LIVE VACCINE STRAIN | INFLAMMATORY RESPONSE | NEGATIVE REGULATOR | SYSTEMS BIOLOGY | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 02/2011, Volume 116, Issue 3, pp. 438 - 448
P>Death-inducing ligands tumor necrosis factor alpha (TNF alpha) and Fas ligand (FasL) do not kill cultured astrocytes; instead they induce a variety of... 
NF‐κB | astrocytes | chemokines | heat shock factor‐1 | heat shock proteins | NF-κB | heat shock factor-1 | DEATH RECEPTOR | DENDRITIC CELLS | HSP70 PROTECTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | LIGAND-INDUCED APOPTOSIS | BINDING ACTIVITY | DOWN-REGULATION | HUMAN FETAL ASTROCYTES | MEDIATED APOPTOSIS | NEUROSCIENCES | NF-kappa B | GENE-EXPRESSION | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Chemokines - biosynthesis | Fas Ligand Protein - metabolism | Transcriptional Activation - genetics | Cells, Cultured | Cell Survival - genetics | Chemokines - physiology | Heat-Shock Proteins - biosynthesis | Apoptosis - genetics | fas Receptor - metabolism | Mice, Inbred C3H | Heat Shock Transcription Factors | DNA-Binding Proteins - metabolism | Fas Ligand Protein - physiology | Transcription Factors - metabolism | Heat-Shock Proteins - genetics | Astrocytes - physiology | Animals | Tumor Necrosis Factor-alpha - physiology | Mice | Enzyme Activation - genetics | Heat-Shock Proteins - physiology | Astrocytes - metabolism | Tumor necrosis factor | DNA | Heat shock proteins | Protein biosynthesis | Necrosis | Tumors | Protein binding | Cell culture | Cell survival | Fas antigen | Astrocytes | Extracellular signal-regulated kinase | CCL3 protein | Data processing | Signal transduction | NF- Kappa B protein | FasL protein | CXC chemokines | CXCL10 protein | Hsp72 protein | Quercetin | Interleukin 8 | Monocyte chemoattractant protein 1 | Tumor necrosis factor- alpha
Journal Article
Science, ISSN 0036-8075, 11/2002, Volume 298, Issue 5595, pp. 1025 - 1029
Journal Article
Journal Article
Journal Article
Biochemical Journal, ISSN 0264-6021, 02/2015, Volume 466, Issue 1, pp. 1 - 11
Growth hormone (GH) and structurally related cytokines regulate a great number of physiological and pathological processes. They do this by coupling their... 
Srk family kinases | Class I cytokine receptors | Janus kinase 2 (JAK2) | Cytokine receptor signalling | Growth hormone | Growth hormone receptor | STIMULATING FACTOR-RECEPTOR | PROLACTIN RECEPTOR | growth hormone receptor | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | LYN TYROSINE KINASE | TRANSMEMBRANE DOMAIN | COMMON BETA-SUBUNIT | cytokine receptor signalling | SRC-FAMILY KINASES | SIGNAL-TRANSDUCTION | ERYTHROPOIETIN RECEPTOR | growth hormone | class I cytokine receptors | THROMBOPOIETIN RECEPTOR | Receptors, Somatotropin - metabolism | Receptor Cross-Talk | Cytokines - metabolism | Signal Transduction | Growth Hormone - metabolism | Humans | Protein Multimerization | Gene Expression Regulation | Janus Kinase 2 - genetics | Models, Molecular | Receptors, Somatotropin - genetics | Growth Hormone - genetics | Animals | Janus Kinase 2 - metabolism | Protein Binding | Protein Interaction Domains and Motifs | Enzyme Activation | Receptors, Cytokine - metabolism | Cytokines - genetics | Receptors, Cytokine - genetics | CT-1, cardiotropin-1 | TPO, thrombopoietin | JM, juxtamembrane | OSM, oncostatin-M | Review | GH, growth hormone | FNIII, fibronectin III-like | ECD, extracellular domain | JAK, Janus kinase | MAb, monoclonal antibody | CRH, cytokine receptor homology | EPO, erythropoietin | CNTF, ciliary neurotropic factor | GM-CSF, granulocyte-macrophage colony-stimulating factor | PK, pseudokinase | TMD, transmembrane domain
Journal Article
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 12/2012, Volume 209, Issue 13, pp. 2395 - 2408
Journal Article