X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (17662) 17662
Dissertation (186) 186
Book Chapter (157) 157
Conference Proceeding (99) 99
Publication (66) 66
Magazine Article (14) 14
Web Resource (10) 10
Book / eBook (9) 9
Government Document (5) 5
Journal / eJournal (5) 5
Book Review (2) 2
Trade Publication Article (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
science & technology (11781) 11781
life sciences & biomedicine (10783) 10783
animals (8004) 8004
calcium - metabolism (4690) 4690
humans (3256) 3256
biochemistry & molecular biology (3196) 3196
cell biology (3062) 3062
rats (3006) 3006
calcium (2974) 2974
ca2 (2822) 2822
male (2718) 2718
neurosciences (2148) 2148
mice (1993) 1993
neurosciences & neurology (1979) 1979
proteins (1734) 1734
pharmacology & pharmacy (1702) 1702
physiology (1664) 1664
cells, cultured (1444) 1444
ca (1299) 1299
phosphorylation (1290) 1290
biophysics (1288) 1288
female (1264) 1264
signal transduction (1229) 1229
kinases (1174) 1174
calmodulin (1070) 1070
in vitro techniques (1053) 1053
research article (940) 940
rodents (929) 929
gene expression (920) 920
channels (905) 905
ca2+/calmodulin-dependent protein kinase ii (879) 879
channel (876) 876
biology (871) 871
apoptosis (862) 862
rats, sprague-dawley (854) 854
science & technology - other topics (843) 843
calcium ions (831) 831
multidisciplinary sciences (822) 822
rats, wistar (801) 801
ca2+-transporting atpase (788) 788
calcium channels (757) 757
homeostasis (755) 755
mitochondria (735) 735
dose-response relationship, drug (734) 734
analysis (724) 724
patch-clamp techniques (723) 723
cardiovascular system & cardiology (720) 720
calcium-binding protein (718) 718
medicine (702) 702
signaling (700) 700
kinetics (695) 695
sarcoplasmic reticulum (693) 693
cell line (685) 685
endoplasmic reticulum (682) 682
calcium signaling (680) 680
neurons (680) 680
atpase (654) 654
calcium channels - metabolism (644) 644
calcium channel blockers - pharmacology (643) 643
calcium signalling (639) 639
cardiac & cardiovascular systems (625) 625
research (622) 622
physiological aspects (615) 615
ca2+ signaling (613) 613
intracellular ca (594) 594
heart (585) 585
physical sciences (571) 571
calcium - pharmacology (558) 558
science (558) 558
adenosine triphosphatase (555) 555
nervous system (554) 554
calcium signaling - drug effects (544) 544
biochemistry (533) 533
calcium signaling - physiology (529) 529
chemistry (527) 527
time factors (525) 525
neurons - metabolism (523) 523
cardiovascular system (513) 513
mice, inbred c57bl (510) 510
intracellular ca2 (501) 501
amino acid sequence (495) 495
electrophysiology (488) 488
protein binding (487) 487
calcium - physiology (481) 481
hippocampus (481) 481
enzyme inhibitors - pharmacology (471) 471
rabbits (470) 470
life sciences (454) 454
molecular sequence data (452) 452
mutation (440) 440
calcium influx (438) 438
neuroscience (428) 428
brain (426) 426
membrane potentials - drug effects (426) 426
smooth muscle (422) 422
protein kinase c (412) 412
myocardium - metabolism (409) 409
cardiomyocytes (408) 408
original (408) 408
guinea pigs (397) 397
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (17193) 17193
Japanese (1094) 1094
Chinese (567) 567
Portuguese (66) 66
Korean (59) 59
French (26) 26
German (18) 18
Russian (11) 11
Spanish (10) 10
Czech (5) 5
Norwegian (4) 4
Swedish (4) 4
Italian (3) 3
Croatian (2) 2
Persian (2) 2
Turkish (2) 2
Dutch (1) 1
Indonesian (1) 1
Lithuanian (1) 1
Polish (1) 1
Slovenian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Acta pharmaceutica Sinica. B, ISSN 2211-3835, 2017, Volume 7, Issue 1, pp. 3 - 17
The intracellular calcium ions(Ca2+) act as second messenger to regulate gene transcription,cell proliferation, migration and death. Accumulating evidences... 
Cell proliferation | entry | channels | Channel blockers | Cancer therapy | Migration | Store-operated Ca | Apoptosis | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Care and treatment | Cellular signal transduction | Research | Calcium ions | Cancer | Innovations | Genetic aspects | Gene expression | Health aspects | PM, plasma membrane | CaMKII, calmodulin-dependent protein kinase II | mAb, monoclonal antibody | NFAT, nuclear factor of activated T cells | CaM, calmodulin | sarcoplasmic reticulum | CBG, cannabigerol | Ca2+ channels | Ca2+ exchanger | RyR, ryanodine receptor | 20-GPPD, 20-O-β-D-glucopyranosyl-20(S)-protopanaxadiol | SPCA, secretory pathway Ca2+-ATPase | CYP3A4, cytochrome P450 3A4 | Store-operated Ca2+ entry | CPZ, capsazepine | SOCE, store-operated Ca2+ entry | CBD, cannabidiol | OSCC, oral squamous cell carcinoma cells | MLCK, myosin light-chain kinase | HCX, H | ER Ca2+-ATPase | Ca2+ exchangers | MCUR1, MCU uniporter regulator 1 | Review | TEA, tetraethylammonium | MICU (1, 2, 3), mitochondrial calcium uptake (type 1, type 2, type 3) | SR, endoplasmic | TG, thapsigargin | TRP (A, C, M, ML, N, P, V), transient receptor potential (ankyrin, canonical, melastatin, mucolipin, no mechanoreceptor potential C, polycystic, vanilloid) | IP3, inositol 1,4,5-trisphosphate | TPC2, two-pore channel 2 | ROS, reactive oxygen species | CRAC, Ca2+ release-activated Ca2+ channel | MCU, mitochondrial Ca2+ uniporter | PTP, permeability transition pore | NCX, Na+ | NSCLC, non-small cell lung cancer | CTL, cytotoxic T cells | PKC, protein kinase C | IP3R (1, 2, 3), IP3 receptor (type 1, type 2, type 3) | SERCA, SR | VGCC, voltage-gated Ca2+ channel | NF-κB, nuclear factor-κB | TRIM, 1-(2-(trifluoromethyl) phenyl) imidazole | PMCA, plasma membrane Ca2+-ATPase
Journal Article
FEBS letters, ISSN 0014-5793, 05/2010, Volume 584, Issue 10, pp. 2033 - 2042
Myogenic, or pressure‐induced, vasoconstriction is critical for local blood flow autoregulation. Underlying this vascular smooth muscle (VSM) response are... 
Calcium | MLC | Arteriole | Ca2+ spark | PLC | regulator of conductance for K | iberiotoxin | small conductance, Ca2+-activated K+ channel | Smooth muscle | CICR | RyR | voltage sensor domain | sarcoplasmic reticulum | STRess axis regulated EXon | Ca2+-induced Ca2+ release | myosin light chain | STOC | reactive oxygen species | BKCa | spontaneous transient outward current | ryanodine receptor | voltage-gated Ca2+ channel | vascular smooth muscle | endothelial cell | VSD | IP3 | Phospholipase C | VSM | intermediate conductance, Ca2+-activated K+ channel | inositol triphosphate | Myogenic | large conductance, Ca2+-activated K+ channel | membrane potential | phosphatidylinositol 4,5-bisphosphate | ROS | VGCC | RCK | STREX | PIP2 | IBTX | Large conductance, Ca | spark | channel | activated K | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Arterioles - physiology | Blood Pressure | Large-Conductance Calcium-Activated Potassium Channels - chemistry | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Animals | Arterioles - metabolism | Signal Transduction | Humans | Electrophysiological Phenomena | Arterioles - cytology | Index Medicus | myogenic | calcium | Ca2+ sparks | smooth muscle | large conductance | arterioles | Ca2+ - activated K+ channel
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2012, Volume 52, Issue 6, pp. 1299 - 1307
Abstract Hypertrophic cardiomyopathy (HCM) is frequently caused by mutations in MYBPC3 encoding cardiac myosin-binding protein C (cMyBP-C). The mechanisms... 
Cardiovascular | Diastolic dysfunction | Ca2+ sensitivity | Ca2+ transient | Mouse model | Hypertrophy | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cell Biology | Cardiomyopathy, Hypertrophic - genetics | Echocardiography | Diastole | Calcium - metabolism | Cardiomyopathy, Hypertrophic - metabolism | Mice, Transgenic | Gene Knock-In Techniques | Carrier Proteins - genetics | Animals | Heart Ventricles - physiopathology | Cardiomyopathy, Hypertrophic - physiopathology | Myocytes, Cardiac - metabolism | Heterozygote | Heart Ventricles - metabolism | Mice | Myofibrils - metabolism | Mutation | Gene Order | Index Medicus | cTnI, cardiac troponin I | Mybpc3, mouse cardiac myosin-binding protein C gene | PKA, cAMP-dependent protein kinase A | max F, maximal Ca2+-activated force | KI, homozygous Mybpc3-targeted knock-in mice | MYBPC3, human cardiac myosin-binding protein C gene | Het, heterozygous Mybpc3-targeted knock-in mice | PLB, phospholamban | KO, homozygous Mybpc3-targeted knock-out mice | Ca2+ exchanger | SERCA2, SR-Ca2+ ATPase | HCM, hypertrophic cardiomyopathy | NCX, Na+ | SL, sarcomere length | SR, sarcoplasmic reticulum | cMyBP-C, cardiac myosin-binding protein C | nH, Hill coefficient | pCa50, log of [Ca2+] required for 50% of maximal activation | LVH, left ventricular hypertrophy | CSQ, calsequestrin | Original
Journal Article
Pflügers Archiv, ISSN 1432-2013, 05/2009, Volume 458, Issue 5, pp. 869 - 876
Journal Article