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Journal Article
Neuron, ISSN 0896-6273, 01/2015, Volume 85, Issue 2, pp. 296 - 302
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2013, Volume 110, Issue 13, pp. 5193 - 5198
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2012, Volume 18, Issue 7, pp. 1087 - 1094
Temporal lobe epilepsy is a common, chronic neurological disorder characterized by recurrent spontaneous seizures. MicroRNAs (miRNAs) are small, noncoding RNAs... 
MEDICINE, RESEARCH & EXPERIMENTAL | TEMPORAL-LOBE EPILEPSY | SYNAPTIC REORGANIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | STATUS-EPILEPTICUS | BRAIN-DAMAGE | KAINIC ACID | CELL-DEATH | CELL BIOLOGY | DENDRITIC SPINES | IN-VIVO | ELECTROGRAPHIC SEIZURES | LONG-TERM POTENTIATION | Neurons - pathology | Recurrence | Gene Silencing - drug effects | Seizures - genetics | Seizures - prevention & control | Humans | Middle Aged | Male | MicroRNAs - metabolism | Status Epilepticus - genetics | Neuroprotective Agents - metabolism | CA3 Region, Hippocampal - drug effects | Dendritic Spines - drug effects | Epilepsy, Temporal Lobe - genetics | CA3 Region, Hippocampal - metabolism | Adult | Neurons - metabolism | Pyramidal Cells - drug effects | Cell Death - drug effects | Kainic Acid - toxicity | Neurons - drug effects | Epilepsy, Temporal Lobe - pathology | Real-Time Polymerase Chain Reaction | Mice, Inbred C57BL | Status Epilepticus - pathology | Up-Regulation - genetics | Up-Regulation - drug effects | Animals | Dendritic Spines - pathology | Pyramidal Cells - pathology | Mice | MicroRNAs - genetics | CA3 Region, Hippocampal - pathology | Physiological aspects | Genetic aspects | MicroRNA | Research | Neurons | Epilepsy | Ribonucleic acid--RNA | Convulsions & seizures | Hippocampal sclerosis | Epileptogenesis | Temporal lobe epilepsy | Dicer | Synaptogenesis
Journal Article
Hippocampus, ISSN 1050-9631, 08/2012, Volume 22, Issue 8, pp. 1691 - 1702
Iron is a necessary substrate for neuronal function throughout the lifespan, but particularly during development. Early life iron deficiency (ID) in humans... 
transgenic tet‐OFF mouse | CA1 | iron deficiency | BDNF | parvalbumin | Transgenic tet-OFF mouse | Iron deficiency | Parvalbumin | PERINATAL IRON | POSTNATAL ONTOGENESIS | NEUROSCIENCES | CA1 PYRAMIDAL NEURONS | DEFICIENCY ANEMIA | TRANSFERRIN RECEPTOR | VISUAL-CORTEX | DEVELOPMENTAL-CHANGES | transgenic tet-OFF mouse | PERINEURONAL NETS | EXTRACELLULAR-MATRIX | RECOGNITION MEMORY | Pyramidal Cells - metabolism | CA1 Region, Hippocampal - cytology | Age Factors | Humans | Extracellular Matrix - metabolism | Parvalbumins - metabolism | Time Factors | Iron - deficiency | CA3 Region, Hippocampal - metabolism | CA1 Region, Hippocampal - metabolism | Female | Brain-Derived Neurotrophic Factor - metabolism | Receptors, Transferrin - genetics | Iron - pharmacology | Hippocampus - growth & development | CHO Cells | Animals, Newborn | Dendrites - metabolism | Cricetinae | Hippocampus - embryology | CA3 Region, Hippocampal - cytology | Mice, Inbred C57BL | Mice, Transgenic | CA1 Region, Hippocampal - embryology | Hippocampus - cytology | Maze Learning - drug effects | Receptors, Transferrin - metabolism | Prenatal Exposure Delayed Effects - metabolism | Iron, Dietary - metabolism | Pregnancy | Animals | Interneurons - metabolism | Memory Disorders - drug therapy | Analysis of Variance | CA3 Region, Hippocampal - embryology | Nerve Net - metabolism | Pyramidal Cells - embryology | Mice | Memory - physiology | Medical colleges | Neurosciences | Transferrin | Anemia | Neurons | Animal behavior | Analysis | Transgenic tet-OFF Mouse | Iron Deficiency
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 03/2013, Volume 19, Issue 3, pp. 351 - 357
Previous attempts to identify neuroprotective targets by studying the ischemic cascade and devising ways to suppress it have failed to translate to efficacious... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | BRAIN-INJURY | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | HEAT-SHOCK PROTEIN | RAT MODEL | DELAYED NEURONAL DEATH | CELL BIOLOGY | HIPPOCAMPUS | RAPAMYCIN PROTECTS | CA1 | TRANSIENT FOREBRAIN ISCHEMIA | Rats, Wistar | Multiprotein Complexes | Male | Hypoxia-Ischemia, Brain - prevention & control | Autophagy - drug effects | Neuroprotective Agents - metabolism | Mechanistic Target of Rapamycin Complex 1 | RNA Interference | Tumor Suppressor Proteins - genetics | Prosencephalon - blood supply | CA3 Region, Hippocampal - metabolism | CA1 Region, Hippocampal - metabolism | Hypoxia-Ischemia, Brain - metabolism | Tumor Suppressor Proteins - metabolism | Adenine - analogs & derivatives | CA1 Region, Hippocampal - physiology | CA3 Region, Hippocampal - physiology | Cells, Cultured | Ischemic Preconditioning | Prosencephalon - injuries | Rats | Adenine - pharmacology | Sirolimus - pharmacology | Animals | Proteins - metabolism | Hypoxia | RNA, Small Interfering | TOR Serine-Threonine Kinases | Proteins - antagonists & inhibitors | Stroke (Disease) | Complications and side effects | Autophagy (Cytology) | Care and treatment | Physiological aspects | Genetic aspects | Research | Pharmacology | Ischemia | Gene expression | Neurons | Autophagy | global cerebral ischemia | endogenous neuroprotection | autophagy | CA3 | proteomics | hippocampus | hamartin | preconditioning
Journal Article
Neuropharmacology, ISSN 0028-3908, 04/2014, Volume 79, pp. 307 - 313
Journal Article