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PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6, pp. e0157613 - e0157613
Journal Article
Current Molecular Medicine, ISSN 1566-5240, 06/2007, Volume 7, Issue 4, pp. 427 - 445
The Notch signaling pathway is involved in a wide variety of highly conserved developmental processes in mammals. Importantly, mutations of the Notch protein... 
Delta-like protein | Notch gene | O-Fucose modification | Coagulation factor | Endoplasmic reticulum | Epidermal Growth Factor | MEDICINE, RESEARCH & EXPERIMENTAL | DROSOPHILA WING DEVELOPMENT | FACTOR-LIKE MODULES | ALAGILLE-SYNDROME | ACUTE LYMPHOBLASTIC-LEUKEMIA | UDP-D-XYLOSE | AUTOSOMAL-DOMINANT ARTERIOPATHY | O-LINKED FUCOSE | NULL FEMALE MICE | CELL LINE HEPG2 | FACTOR-LIKE REPEATS | Alagille Syndrome - enzymology | CADASIL - pathology | Receptors, Notch - metabolism | Fucose - metabolism | Humans | N-Acetylglucosaminyltransferases - genetics | Receptors, Notch - genetics | Alagille Syndrome - pathology | Endoplasmic Reticulum - pathology | Intercellular Signaling Peptides and Proteins - metabolism | Alagille Syndrome - genetics | Serrate-Jagged Proteins | CADASIL - therapy | Membrane Proteins - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Endoplasmic Reticulum - enzymology | CADASIL - enzymology | Fucosyltransferases - genetics | Multiple Sclerosis - enzymology | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Intracellular Signaling Peptides and Proteins | Fucose - genetics | Multiple Sclerosis - genetics | Glycosylation | Protein Structure, Tertiary - genetics | Signal Transduction - genetics | Alagille Syndrome - therapy | Multiple Sclerosis - therapy | CADASIL - genetics | N-Acetylglucosaminyltransferases - metabolism | Protein Modification, Translational | Fucosyltransferases - metabolism | Leukemia, T-Cell - enzymology | Leukemia, T-Cell - genetics | Multiple Sclerosis - pathology | Leukemia, T-Cell - pathology | Leukemia, T-Cell - therapy | Calcium-Binding Proteins - genetics
Journal Article
REVISTA DE NEUROLOGIA, ISSN 0210-0010, 09/2008, Volume 47, Issue 6, pp. 299 - 300
Journal Article
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2015, Volume 73, pp. 189 - 203
Abstract Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL), a Notch3 dominant mutation-induced cerebral... 
Neurology | Angiogenesis | CADASIL | CAA | SCF | Bone marrow-derived cell | Vascular smooth muscle cell | G-CSF | Neurogenesis | Apoptosis | SUBCORTICAL INFARCTS | BONE-MARROW | NEUROSCIENCES | AMYLOID ANGIOPATHY | ENDOTHELIAL-CELLS | NEURONAL CELLS | AUTOSOMAL-DOMINANT ARTERIOPATHY | NOTCH3 MUTATIONS | CEREBRAL-BLOOD-FLOW | ISCHEMIC-STROKE | TRANSGENIC MICE | Neovascularization, Physiologic - drug effects | Muscle, Smooth, Vascular - metabolism | Stem Cell Factor - therapeutic use | Caspase 3 - metabolism | Male | Receptors, Notch - genetics | Neurogenesis - genetics | Time Factors | Cognition Disorders - etiology | Bone Marrow Transplantation | CADASIL - complications | CADASIL - surgery | Neurogenesis - drug effects | Cell Death - drug effects | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Neovascularization, Physiologic - genetics | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Cognition Disorders - drug therapy | Granulocyte Colony-Stimulating Factor - therapeutic use | Maze Learning - drug effects | Mutation - genetics | CADASIL - genetics | Animals | CADASIL - drug therapy | Receptor, Notch3 | Mice | Stroke (Disease) | Granulocyte colony-stimulating factor | Analysis | Stem cells | Macrophage colony stimulating factor | Health aspects | Dementia | Blood circulation disorders | Cell death | Genetic engineering | Index Medicus
Journal Article
Journal Article