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Cell calcium, ISSN 0143-4160, 1980
Journal
2007, Subcellular biochemistry, ISBN 9781402061905, Volume 45., xii, 566, [26] p. of plates
This topic is a new entry in the area of cellular calcium signaling: yet, it now spans the entire area, with discoveries that cover both genetic and acquired... 
Calcium | Pathophysiology | Physiological effect | Human Genetics | Biochemistry, general | Molecular Medicine | Biomedicine | Neurobiology
Book
Circulation Research, ISSN 0009-7330, 02/2007, Volume 100, Issue 3, pp. 342 - 353
A large body of evidence has accrued indicating that voltage-gated Ca channel subtypes, including L-, T-, N-, and P/Q-type, are present within renal vascular... 
Renal microcirculation | Mibefradil | channels | Afferent arteriole | channel blockers | Efferent arteriole | Renal disease | Voltage-dependent Ca | Efonidipine | Ca2+ channel blockers | N-TYPE | CARDIAC & CARDIOVASCULAR SYSTEMS | EFFERENT ARTERIOLES | efonidipine | SPONTANEOUSLY HYPERTENSIVE-RATS | RHO-KINASE INHIBITOR | renal microcirculation | ANGIOTENSIN-II | T-TYPE | renal disease | mibefradil | DEPENDENT CALCIUM-CHANNELS | CARDIAC L-TYPE | RENAL MICROVASCULAR CONSTRICTION | efferent arteriole | voltage-dependent Ca2+ channels | PERIPHERAL VASCULAR DISEASE | afferent arteriole | HEMATOLOGY | IN-VIVO VISUALIZATION | Arterioles - physiology | Kidney - physiology | Protein Subunits | Antihypertensive Agents - pharmacology | Kidney - blood supply | Humans | Calcium Channel Blockers - therapeutic use | Hypertension - drug therapy | Antihypertensive Agents - classification | Calcium Channels - physiology | Calcium Channels, T-Type - chemistry | Neurotransmitter Agents - secretion | Calcium Channels - drug effects | Calcium Channels, N-Type - drug effects | Cardiovascular Diseases - physiopathology | Kidney - drug effects | Calcium Channels - classification | Rats | Calcium Channels, L-Type - physiology | Antihypertensive Agents - therapeutic use | Arterioles - drug effects | Disease Progression | Antihypertensive Agents - adverse effects | Mice, Knockout | Calcium Signaling - drug effects | Diabetes Mellitus - physiopathology | Models, Biological | Calcium Channels - chemistry | Calcium Channels, T-Type - drug effects | Mice | Vasodilation - drug effects | Hydronephrosis - physiopathology | Calcium Channel Blockers - adverse effects | Calcium Signaling - physiology | Cardiovascular Diseases - drug therapy | Renal Circulation - physiology | Microcirculation - drug effects | Microcirculation - physiology | Renal Circulation - drug effects | Blood Pressure - drug effects | Calcium Channels, L-Type - chemistry | Kidney Diseases - metabolism | Calcium Channels, T-Type - physiology | Renin - secretion | Aldosterone - physiology | Kidney Diseases - drug therapy | Renin-Angiotensin System - physiology | Calcium Channel Blockers - pharmacology | Hypertension - physiopathology | Animals | Calcium Channels, L-Type - drug effects | Calcium Channels, N-Type - physiology | Calcium Channels, N-Type - chemistry
Journal Article
1994, ISBN 0125641419, Volume 40., xiv, 368
Book
Journal Article
2004, Novartis Foundation symposium, ISBN 0470862548, Volume 258, 288
Chair's Introduction (J. Putney). Molecular genetics of Drosophila TRP channels (C. Montell). Mammalian TRPC channnel subunit assembly (W. Schilling and M.... 
TRP channels | Life Sciences | Biochemistry | SCIENCE | Congresses
eBook
2002, 1st ed., Advances in cell aging and gerontology, ISBN 9780444511355, Volume 10., viii, 202
Book
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 4, p. e18037
Background: The ability to establish human induced pluripotent stem cells (hiPSCs) by reprogramming of adult fibroblasts and to coax their differentiation into... 
HEART | DEFINED FACTORS | CONTRACTION | MULTIDISCIPLINARY SCIENCES | CARDIAC MYOCYTES | MECHANISMS | SARCOPLASMIC-RETICULUM | INDUCTION | HUMAN SOMATIC-CELLS | RELEASE CHANNEL | EXPRESSION | Cell Line | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Myocytes, Cardiac - cytology | Calcium - metabolism | Humans | Gene Expression Regulation | Ryanodine Receptor Calcium Release Channel - metabolism | Myocytes, Cardiac - enzymology | Animals | Calcium Channels, L-Type - genetics | Biological Transport | Ryanodine Receptor Calcium Release Channel - genetics | Inositol 1,4,5-Trisphosphate Receptors - metabolism | Intracellular Space - metabolism | Myocytes, Cardiac - metabolism | Cell Differentiation | Mice | Calcium Channels, L-Type - metabolism | Induced Pluripotent Stem Cells - cytology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Inositol 1,4,5-Trisphosphate - metabolism | Sarcolemma - metabolism | Inositol 1,4,5-Trisphosphate Receptors - genetics | Proteins | Stem cells | Heart cells | Research | Adenosine triphosphatase | Nifedipine | Caffeine | Calcium | Laboratories | Immunocytochemistry | Ryanodine receptors | Confocal | Channels | Calcium influx | Ca2+-transporting ATPase | Thapsigargin | Sarcoplasmic reticulum | Rodents | Fibroblasts | Physiology | Sequestering | Inositol 1,4,5-trisphosphate | Heart failure | Calcium channels | Calcium (intracellular) | Cloning | Calcium imaging | Cardiomyocytes | Biophysics | Gene expression | Polymerase chain reaction | Medicine | Studies | Hypotheses | Human performance | Calcium channels (L-type) | Pluripotency | Calcium (extracellular) | Calcium (reticular) | Handling | Ca super(2+)-transporting ATPase | cardiomyocytes | Lasers | Differentiation | thapsigargin
Journal Article
PLoS biology, ISSN 1545-7885, 2011, Volume 9, Issue 3, p. e1001025
Store-operated Ca2+ entry (SOCE) has been associated with two types of channels: CRAC channels that require Orai1 and STIM1 and SOC channels that involve... 
B-LYMPHOCYTES | OPERATED CALCIUM-CHANNELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOLOGY | PORE SUBUNIT | SMOOTH-MUSCLE-CELLS | ACINAR-CELLS | STORE DEPLETION | ACTIVATES CRAC CHANNELS | GLAND FLUID SECRETION | I-CRAC | ENDOTHELIAL PERMEABILITY | Calcium Signaling - physiology | Humans | Neoplasm Proteins - physiology | TRPC Cation Channels - analysis | Calcium - chemistry | Membrane Proteins - analysis | Gene Knockdown Techniques | Cell Membrane - chemistry | Calcium Channels - physiology | Membrane Proteins - physiology | Cell Membrane - metabolism | Neoplasm Proteins - genetics | TRPC Cation Channels - genetics | Calcium Channels - genetics | Stromal Interaction Molecule 1 | Cell Line | Membrane Proteins - genetics | ORAI1 Protein | Cytosol - chemistry | Calcium Channels - analysis | Patch-Clamp Techniques | Animals | Models, Biological | TRPC Cation Channels - metabolism | Cytosol - metabolism | Neoplasm Proteins - analysis | Mice | Mice, Inbred BALB C | Proteins | Plasma | Calcium | Cells | Recruitment | Channel pores | Calcium channels | Channel gating | Ion currents | STIM1 protein | NF-AT protein | Orai1 protein | transient receptor potential proteins | Calcium influx | Potassium channels (calcium-gated) | Calcium signalling | Signal transduction | Plasma membranes | Ion channels | Endoplasmic reticulum | thapsigargin | Calcium (extracellular) | Calcium (reticular)
Journal Article
Journal Article