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Free Radical Biology and Medicine, ISSN 0891-5849, 11/2015, Volume 88, Issue Pt B, pp. 417 - 426
In light of the emerging interplay between redox and metabolic signaling pathways we investigated the potential cross talk between nuclear factor E2-related... 
Xanthohumol | Nrf2 | ER stress | AMPK | HO-1 | PERK | LKB1 | MEF | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | MECHANISMS | ENERGY SENSOR | CELL DEFENSE PATHWAY | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | NRF2-MEDIATED INDUCTION | HEME OXYGENASE-1 | STRESS | AMP-Activated Protein Kinases - metabolism | Heme Oxygenase-1 - metabolism | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Oxidative Stress - physiology | Propiophenones - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Cross-Talk - physiology | Blotting, Western | Animals | Flow Cytometry | Fibroblasts - drug effects | NF-E2-Related Factor 2 - metabolism | Signal Transduction - physiology | Flavonoids - pharmacology | Membrane Proteins - metabolism | Mice | Real-Time Polymerase Chain Reaction | Unfolded Protein Response - physiology | Fibroblasts - metabolism | RNA | Glycogen | Synthesis | ACC, acetyl-CoA carboxylase | CHO, Chinese hamster ovary | GSH, glutathione (reduced) | OCR, oxygen consumption rate | HO-1, heme oxygenase 1 | CaMKK, calcium calmodulin-dependent kinase kinase | Maf, small musculoaponeurotic fibrosarcoma | PERK, protein kinase RNA-like endoplasmic reticulum kinase | Xn, xanthohumol | Hrd1, synoviolin | TAK, transforming growth factor β-activated kinase | UPR, unfolded protein response | Hrd1 (HMG-CoA reductase degradation)-ubiquitin ligase | MEF, mouse embryonic fibroblasts | βTrcP1, β-transducin-repeat containing protein 1 | mTOR, mammalian target of rapamycin | ROS, reactive oxygen species | LKB1, liver kinase B1 | Keap1, Kelch-like ECH-associated protein | GSSG, oxidized glutathione | ARE, antioxidant response element | WT, wild type | GSK3β, glycogen synthase kinase 3β | Nrf2, nuclear factor E2-related factor 2 | AMPK, AMP-activated kinase | DCF, dichlorofluorescein | NADPH, nicotinamide adenine dinucleotide phosphate | DMSO, dimethyl sulfoxide | ER, endoplasmic reticulum | FCS, fetal calf serum
Journal Article
Nature, ISSN 0028-0836, 11/2012, Volume 491, Issue 7423, pp. 269 - 273
Myocardial cell death is initiated by excessive mitochondrial Ca2+ entry causing Ca2+ overload, mitochondrial permeability transition pore (mPTP) opening and... 
OXIDATION | HYPERTROPHY | APOPTOSIS | INNER MEMBRANE | II INHIBITION PROTECTS | MULTIDISCIPLINARY SCIENCES | CALCIUM UNIPORTER | IN-VIVO | CALMODULIN KINASE | FAILURE | KINASE-II | Mitochondria, Heart - metabolism | Apoptosis - drug effects | Calcium - metabolism | Cyclosporine - pharmacology | Mitochondria, Heart - pathology | Membrane Potential, Mitochondrial - drug effects | Heart Failure - prevention & control | Myocardium - metabolism | Membrane Potential, Mitochondrial - physiology | Female | Stress, Physiological - drug effects | Reperfusion Injury - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Heart - physiopathology | Mitochondrial Membrane Transport Proteins - metabolism | Calcium - pharmacology | Reperfusion Injury - pathology | Reperfusion Injury - enzymology | Mice, Inbred C57BL | Mitochondria, Heart - enzymology | Mice, Transgenic | Myocardium - pathology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Serine - metabolism | Heart Failure - drug therapy | Myocardium - enzymology | Animals | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - chemistry | Myocardial Infarction - drug therapy | Reperfusion Injury - prevention & control | Heart - drug effects | Mice | Myocardial Infarction - prevention & control | Heart failure | Cell death | Heart cells | Research | Mitochondrial myopathies | Properties | Observations | Protein kinases | Calmodulin | Proteins | Heart | Mitochondria | Heart attacks | Rodents | Homeostasis | Apoptosis
Journal Article
The New Phytologist, ISSN 0028-646X, 8/2008, Volume 179, Issue 3, pp. 675 - 686
Guard cell movements are regulated by environmental cues including, for example, elevations in extracellular Ca²⁺ concentration. Here, the subcellular... 
Proteins | Calcium | Chloroplasts | Fluorescence | Guard cells | Thylakoids | Gene expression regulation | Plants | Perceptual localization | Plant cells | stomatal movement | calcium | guard cells | chloroplast | signal transduction | Ca2+‐sensing receptor (CAS) | Signal transduction | sensing receptor (CAS) | Stomatal movement | Ca | Chloroplast | Ca2+-sensing receptor (CAS) | RED-LIGHT RESPONSE | SENSITIVITY | FREE CALCIUM | BIOENERGETIC PATHWAYS | PLANT SCIENCES | CA2 | GUARD-CELLS | THYLAKOID MEMBRANE PROTEOME | ARABIDOPSIS | OSCILLATIONS | Arabidopsis - physiology | Cell Fractionation | Calcium - metabolism | Recombinant Fusion Proteins - analysis | Cytoplasm - metabolism | Thylakoids - chemistry | Plant Stomata - metabolism | Receptors, Calcium-Sensing - analysis | Sequence Analysis, Protein | Thylakoids - ultrastructure | Conserved Sequence | Arabidopsis Proteins - analysis | Thylakoids - physiology | Amino Acid Sequence | Arabidopsis Proteins - genetics | Arabidopsis Proteins - physiology | Receptors, Calcium-Sensing - physiology | Green Fluorescent Proteins - analysis | Calcium-Binding Proteins - analysis | Arabidopsis - ultrastructure | Signal Transduction - genetics | Arabidopsis - metabolism | Arabidopsis Proteins - chemistry | Tobacco - genetics | Calcium-Binding Proteins - physiology | Receptors, Calcium-Sensing - genetics | Tobacco - ultrastructure | Plant Stomata - physiology | Calcium-Binding Proteins - genetics
Journal Article
Cell Metabolism, ISSN 1550-4131, 09/2016, Volume 24, Issue 3, pp. 389 - 401
Journal Article
Journal Article
The FASEB Journal, ISSN 0892-6638, 08/2014, Volume 28, Issue 8, pp. 3301 - 3312
Journal Article
Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7285, pp. 104 - 107
Injury causes a systemic inflammatory response syndrome (SIRS) that is clinically much like sepsis. Microbial pathogen-associated molecular patterns (PAMPs)... 
SHOCK | SIRS | OPERATED CALCIUM INFLUX | HUMAN NEUTROPHILS | PEPTIDES | MODELS | MOBILIZATION | CHEMOTAXIS | MULTIDISCIPLINARY SCIENCES | RECEPTORS | TRAUMA | Liver - pathology | Phosphorylation | Humans | Male | Mitochondria - immunology | Liver - injuries | N-Formylmethionine Leucyl-Phenylalanine - metabolism | Liver - immunology | Systemic Inflammatory Response Syndrome - blood | Muscle, Skeletal - immunology | Sepsis - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Neutrophils - metabolism | Calcium Signaling | Femur - injuries | Sepsis - immunology | CpG Islands - immunology | Neutrophils - enzymology | Wounds and Injuries - pathology | Fractures, Bone - immunology | Wounds and Injuries - blood | Cells, Cultured | Neutrophils - immunology | Wounds and Injuries - complications | Rats | Wounds and Injuries - immunology | Acute Lung Injury - pathology | DNA, Mitochondrial - blood | Rats, Sprague-Dawley | DNA, Mitochondrial - immunology | Immunity, Innate - immunology | Systemic Inflammatory Response Syndrome - pathology | Animals | Mitochondria - secretion | Acute Lung Injury - immunology | Fractures, Bone - pathology | Systemic Inflammatory Response Syndrome - complications | Muscle, Skeletal - pathology | Toll-Like Receptor 9 - metabolism | Sepsis - microbiology | Mitogen-Activated Protein Kinases - metabolism | N-Formylmethionine Leucyl-Phenylalanine - immunology | Infection | Peptides | Physiological aspects | Mitochondrial DNA | Inflammation | Research | Risk factors | Fractures | Bacterial proteins | Rheumatoid arthritis | Trauma | Deoxyribonucleic acid--DNA
Journal Article