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Nature Medicine, ISSN 1078-8956, 01/2007, Volume 13, Issue 1, pp. 54 - 61
Anthracyclin-treated tumor cells are particularly effective in eliciting an anticancer immune response, whereas other DNA-damaging agents such as etoposide and... 
MEDICINE, RESEARCH & EXPERIMENTAL | CLEARANCE | DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR | CHAPERONE | CELL BIOLOGY | DYING CELLS | APOPTOTIC CELLS | SURFACE CALRETICULIN | ENDOPLASMIC-RETICULUM | ENCODING CALRETICULIN | ANTIGEN | Apoptosis - drug effects | Colonic Neoplasms - drug therapy | Dendritic Cells - immunology | Electrophoresis, Gel, Two-Dimensional | Immunoblotting | Antineoplastic Agents - therapeutic use | Protein Transport - drug effects | Calreticulin - genetics | Colonic Neoplasms - metabolism | Cell Cycle Proteins - antagonists & inhibitors | RNA Interference | Anthracyclines - pharmacology | Neoplasms, Experimental - immunology | Calreticulin - metabolism | Antigens, Differentiation - metabolism | Antineoplastic Agents - pharmacology | Cell Membrane - metabolism | Protein Phosphatase 1 | Cell Membrane - drug effects | Mitomycin - therapeutic use | Cell Cycle Proteins - metabolism | Phagocytosis - immunology | Etoposide - pharmacology | Etoposide - therapeutic use | Recombinant Proteins - pharmacology | Mitomycin - pharmacology | Animals | Apoptosis - immunology | Mice, Nude | Colonic Neoplasms - pathology | Cell Line, Tumor | Cell Membrane - immunology | Mice | Mice, Inbred BALB C | Anthracyclines - therapeutic use | Calreticulin - immunology | Neoplasms, Experimental - metabolism | Neoplasms, Experimental - drug therapy | Chemotherapy | Immunology | DNA damage | Rodents | Cancer | Apoptosis | Tumors | Antigens, Differentiation | Cell Membrane | Anthracyclines | Dendritic Cells | Recombinant Proteins | Life Sciences | Calreticulin | Neoplasms, Experimental | Mitomycin | Etoposide | Antineoplastic Agents | Protein Transport | Phagocytosis | Cell Cycle Proteins | Colonic Neoplasms
Journal Article
The EMBO Journal, ISSN 0261-4189, 03/2009, Volume 28, Issue 5, pp. 578 - 590
Journal Article
The EMBO Journal, ISSN 0261-4189, 03/2012, Volume 31, Issue 5, pp. 1062 - 1079
Journal Article
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2016, Volume 95, Issue 1, pp. 120 - 130
Journal Article
Nature Biotechnology, ISSN 1087-0156, 02/2007, Volume 25, Issue 2, pp. 192 - 193
Journal Article
Molecular Oncology, ISSN 1574-7891, 04/2015, Volume 9, Issue 4, pp. 834 - 849
Peritoneal dissemination is a major clinical obstacle in gastrointestinal cancer therapy, and it accounts for the majority of cancer-related mortality.... 
Calreticulin | Carcinogenesis | Epithelial-to-mesenchymal transition | Immunogenic cell death | Endoplasmic reticulum stress | ANGIOGENESIS | CANCER-CELL DEATH | OVEREXPRESSION | THERAPY | ONCOLOGY | PATHWAY | APOPTOTIC CELLS | HEALTH | TUMOR-GROWTH | EXPRESSION | CONTRIBUTES | Calpain - metabolism | Humans | Middle Aged | Methylnitronitrosoguanidine | Epithelial-Mesenchymal Transition - drug effects | Male | Stomach Neoplasms - pathology | PPAR gamma - metabolism | Calreticulin - genetics | Lignans - pharmacology | Promoter Regions, Genetic - genetics | Gene Knockdown Techniques | Biphenyl Compounds - pharmacology | Protein Binding - drug effects | Biomarkers, Tumor - metabolism | Calreticulin - metabolism | Adult | Female | Cell Death - drug effects | Phagocytosis - drug effects | Endoplasmic Reticulum Stress - drug effects | Neoplasm Invasiveness | Mice, Inbred C57BL | Down-Regulation - drug effects | Stomach Neoplasms - immunology | Up-Regulation - drug effects | Macrophages - metabolism | Animals | Cell Line, Tumor | Macrophages - drug effects | Mice, Inbred BALB C | Immunohistochemistry | Medical colleges | Care and treatment | RNA | Genes | Calpain | Stress (Physiology) | Metastasis | Transforming growth factors | Cells | Cell death | Gastrointestinal diseases | Stem cells | Genetic engineering | Health aspects | Cancer | Phosphorylation | Mesenchyme | Stomach cancer | Lymph nodes | Metastases | Cell adhesion & migration | Conflicts of interest | Tumorigenesis | Growth factors | Gastric cancer | Lymphatic system | RNA-mediated interference | siRNA | Tumor cell lines | Gene expression | Bladder cancer | Gene silencing | Immunogenicity | Medical prognosis | Cell lines | Endoplasmic reticulum | Prostate cancer | Cell migration | Tumors | Apoptosis | Peritoneum | Pharmaceuticals
Journal Article