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1. Full Text Fear of progression in patients 6 months after cancer rehabilitation—a validation study of the fear of progression questionnaire FoP-Q-12
by Hinz, Andreas and Mehnert, Anja and Ernst, Jochen and Herschbach, Peter and Schulte, Thomas
Supportive Care in Cancer, ISSN 0941-4355, 6/2015, Volume 23, Issue 6, pp. 1579 - 1587
Many cancer patients experience fear of progression (FoP). The purpose of this study was to test psychometric properties of the questionnaire FoP-Q-12, to... 
Cancer survivors | Pain Medicine | Nursing | Rehabilitation Medicine | Fear of recurrence | Medicine & Public Health | Oncology | Fear of progression | Rehabilitation | Nursing Management/Nursing Research | DIAGNOSIS | DEPRESSION SCALE | HOSPITAL ANXIETY | PREVALENCE | BREAST-CANCER | SUPPORTIVE CARE NEEDS | GENERAL-POPULATION | ONCOLOGY | HEALTH CARE SCIENCES & SERVICES | RECURRENCE | QUALITY-OF-LIFE | SURVIVORS | Reproducibility of Results | Neoplasms - rehabilitation | Humans | Middle Aged | Anxiety - psychology | Male | Psychometrics | Neoplasms - psychology | Disease Progression | Fear - psychology | Adult | Female | Surveys and Questionnaires | Aged | Anxiety - etiology | Surveys | Cancer patients | Analysis | Oncology, Experimental | Development and progression | Lymphomas | Anxiety | Research | Cancer | Fear & phobias | Survivor | Questionnaires | Index Medicus
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2. The chromosomal imbalance theory of cancer
: autocatalyzed progression of aneuploidy is carcinogenesis
by Rasnick, David William
2012, ISBN 9781578087372, cm.
Book
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3. Full Text Effect of Selumetinib vs Chemotherapy on Progression-Free Survival in Uveal Melanoma: A Randomized Clinical Trial
by Carvajal, Richard D and Sosman, Jeffrey A and Quevedo, Jorge Fernando and Milhem, Mohammed M and Joshua, Anthony M and Kudchadkar, Ragini R and Linette, Gerald P and Gajewski, Thomas F and Lutzky, Jose and Lawson, David H and Lao, Christopher D and Flynn, Patrick J and Albertini, Mark R and Sato, Takami and Lewis, Karl and Doyle, Austin and Ancell, Kristin and Panageas, Katherine S and Bluth, Mark and Hedvat, Cyrus and Erinjeri, Joseph and Ambrosini, Grazia and Marr, Brian and Abramson, David H and Dickson, Mark Andrew and Wolchok, Jedd D and Chapman, Paul B and Schwartz, Gary K
JAMA, ISSN 0098-7484, 06/2014, Volume 311, Issue 23, pp. 2397 - 2405
IMPORTANCE Uveal melanoma is characterized by mutations in GNAQ and GNA11, resulting in mitogen-activated protein kinase pathway activation. OBJECTIVE To... 
METASTATIC-DISEASE | LUNG-CANCER | MEDICINE, GENERAL & INTERNAL | MEK INHIBITOR | MUTANT GNAQ | SOLID TUMORS | CHOROIDAL MELANOMA | COLLABORATIVE OCULAR MELANOMA | GROWTH-FACTOR | MUTATIONS | CELL-DEATH | Dacarbazine - adverse effects | Dacarbazine - therapeutic use | Humans | Middle Aged | Male | Treatment Outcome | Disease Progression | Uveal Neoplasms - drug therapy | Antineoplastic Agents, Alkylating - therapeutic use | Melanoma - drug therapy | Dacarbazine - analogs & derivatives | Survival Analysis | Aged, 80 and over | Adult | Female | Aged | Antineoplastic Agents, Alkylating - adverse effects | Benzimidazoles - adverse effects | Benzimidazoles - therapeutic use | Chemotherapy | Care and treatment | Research | Health aspects | Melanoma | Cancer | Survival analysis | Prescription drugs | Oncology | Clinical outcomes | Index Medicus | Abridged Index Medicus
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4. Full Text Gefitinib plus chemotherapy versus placebo plus chemotherapy in EGFR -mutation-positive non-small-cell lung cancer after progression on first-line gefitinib (IMPRESS): a phase 3 randomised trial
by Soria, Jean-Charles, Prof|Wu, Yi-Long, Prof|Nakagawa, Kazuhiko, Prof|Kim, Sang-We, Prof|Yang, Jin-Ji, MD|Ahn, Myung-Ju, Prof|Wang, Jie, MD|Yang, James Chih-Hsin, Prof|Lu, You, Prof|Atagi, Shinji, MD|Ponce, Santiago, MD|Lee, Dae Ho, Prof|Liu, Yunpeng, Prof|Yoh, Kiyotaka, MD|Zhou, Jian-Ying, Prof|Shi, Xiaojin, MD|Webster, Alan, MSc|Jiang, Haiyi, MD|Mok, Tony S K, Prof
Lancet Oncology, The, ISSN 1470-2045, 2015, Volume 16, Issue 8, pp. 990 - 998
Summary Background Optimum management strategies for patients with advanced non-small-cell lung cancer (NSCLC) with acquired resistance to EGFR tyrosine-kinase... 
Hematology, Oncology and Palliative Medicine | TYROSINE KINASE INHIBITORS | MULTICENTER | ONCOLOGY | ADENOCARCINOMA | ACQUIRED-RESISTANCE | OPEN-LABEL | AFATINIB | ERLOTINIB | GEMCITABINE | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Prospective Studies | Guanine - analogs & derivatives | Lung Neoplasms - mortality | Humans | Middle Aged | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Lung Neoplasms - pathology | Male | Cisplatin - administration & dosage | Receptor, Epidermal Growth Factor - metabolism | Time Factors | Adult | Female | Quinazolines - administration & dosage | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Genetic Predisposition to Disease | Lung Neoplasms - enzymology | Drug Administration Schedule | Administration, Oral | Carcinoma, Non-Small-Cell Lung - genetics | Europe | Kaplan-Meier Estimate | Proportional Hazards Models | Treatment Outcome | Carcinoma, Non-Small-Cell Lung - mortality | Pemetrexed | Disease Progression | Disease-Free Survival | Protein Kinase Inhibitors - administration & dosage | Phenotype | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Asia | Glutamates - administration & dosage | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Aged | Biomarkers, Tumor - genetics | Carcinoma, Non-Small-Cell Lung - drug therapy | Carcinoma, Non-Small-Cell Lung - enzymology | Infusions, Intravenous | Mutation | Guanine - administration & dosage | Medical colleges | Chemotherapy | Strategic planning (Business) | Genetic aspects | Product development | Lung cancer, Non-small cell | Gefitinib | Cancer | Index Medicus
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5. Bone cancer
: progression and therapeutic approaches
by Heymann, Dominique
2010, 1st ed., ISBN 9780123748959, xv, 472 p., [10] p. of plates
The epidemiological and clinical importance of bone metastasis has long been recognized, but the past decade has seen an explosion in the fields of bone... 
Bone Neoplasms | Bones | Clinical & internal medicine | Treatment | Cancer
Book
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6. Full Text Progression-free survival/time to progression as a potential surrogate for overall survival in HR+, HER2- metastatic breast cancer
by Forsythe, Anna and Chandiwana, David and Barth, Janina and Thabane, Marroon and Baeck, Johan and Tremblay, Gabriel
Breast Cancer: Targets and Therapy, ISSN 1179-1314, 05/2018, Volume 10, pp. 69 - 78
Several recent randomized controlled trials (RCTs) in hormone receptor-positive (HR+), human epidermal growth factor receptor 2-negative (HER2-) metastatic... 
Time to progression | Breast cancer | Overall survival | Correlation analysis | Progression-free survival | Surrogate endpoint | Chemotherapy | Epidermal growth factor | Patient outcomes | Development and progression | Metastasis | Cancer | Drugs | Confidence intervals | Colorectal cancer | Clinical trials | FDA approval | Cancer therapies | Regulatory approval | Food | Pharmaceuticals | correlation analysis | overall survival | surrogate endpoint | time to progression | progression-free survival
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7. Full Text NICE Advice - Prolaris gene expression assay for assessing long-term risk of prostate cancer progression: (c) NICE (2016) Prolaris gene expression assay for assessing long-term risk of prostate cancer progression
BJU INTERNATIONAL, ISSN 1464-4096, 08/2018, Volume 122, Issue 2, pp. 173 - 180
UROLOGY & NEPHROLOGY | NICE Guidance | Genes | Genetic research | Development and progression | Genetic aspects | Gene expression | Health aspects | Prostate cancer | Cancer | Index Medicus
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8. Full Text Selumetinib Plus Docetaxel Compared With Docetaxel Alone and Progression-Free Survival in Patients With KRAS-Mutant Advanced Non–Small Cell Lung Cancer: The SELECT-1 Randomized Clinical Trial
by Jänne, Pasi A and van den Heuvel, Michel M and Barlesi, Fabrice and Cobo, Manuel and Mazieres, Julien and Crinò, Lucio and Orlov, Sergey and Blackhall, Fiona and Wolf, Juergen and Garrido, Pilar and Poltoratskiy, Artem and Mariani, Gabriella and Ghiorghiu, Dana and Kilgour, Elaine and Smith, Paul and Kohlmann, Alexander and Carlile, David J and Lawrence, David and Bowen, Karin and Vansteenkiste, Johan
JAMA, ISSN 0098-7484, 05/2017, Volume 317, Issue 18, pp. 1844 - 1853
IMPORTANCE: There are no specifically approved targeted therapies for the most common genomically defined subset of non–small cell lung cancer (NSCLC),... 
MEDICINE, GENERAL & INTERNAL | PHASE-II | MULTICENTER | MUTATIONS | AZD6244 ARRY-142886 | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Middle Aged | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Male | Treatment Outcome | Disease Progression | Disease-Free Survival | Taxoids - administration & dosage | Benzimidazoles - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Aged, 80 and over | Adult | Female | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Benzimidazoles - adverse effects | Taxoids - adverse effects | Drugs | Care and treatment | Cancer patients | Docetaxel | Dosage and administration | Diagnosis | Lung cancer, Non-small cell | Health aspects | Inhibitor drugs | Cell survival | Lung cancer | Clinical trials | Non-small cell lung carcinoma | MAP kinase | Patients | Survival | K-Ras protein | Side effects | Randomization | Protein kinase | Drug therapy | Cancer | Index Medicus | Abridged Index Medicus | Original Investigation | Research
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9. Full Text Fructose-1,6-bisphosphatase opposes renal carcinoma progression
by Li, B and Li, Bo and Qiu, B and Qiu, Bo and Lee, David S. M and Lee, David S M and Walton, Zandra E and Walton, Zandra E and Ochocki, Joshua D and Ochocki, Joshua D and Mathew, Lijoy K and Mathew, Lijoy K and Mancuso, Anthony and Mancuso, Anthony and Gade, Terence P F and Gade, Terence P. F and Keith, Brian and Keith, Brian and Nissim, Itzhak and Nissim, Itzhak and Simon, M Celeste and Simon, M. Celeste
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7517, pp. 251 - 255
Clear cell renal cell carcinoma (ccRCC), the most common form of kidney cancer(1), is characterized by elevated glycogen levels and fat deposition(2). These... 
INACTIVATION | METABOLISM | IDENTIFICATION | MULTIDISCIPLINARY SCIENCES | CELL CARCINOMA | DISEASE | Protein Structure, Tertiary | Cell Line | Kidney Neoplasms - genetics | Cell Proliferation | Epithelial Cells - metabolism | Humans | Carcinoma, Renal Cell - genetics | Models, Molecular | Disease Progression | Carcinoma, Renal Cell - physiopathology | Fructose-Bisphosphatase - metabolism | Animals | Basic Helix-Loop-Helix Transcription Factors - metabolism | Kidney Neoplasms - physiopathology | Swine | Kidney Neoplasms - enzymology | Cell Line, Tumor | Glycolysis | Carcinoma, Renal Cell - enzymology | Fructose-Bisphosphatase - genetics | Fructose-Bisphosphatase - chemistry | NADP - metabolism | Glucose metabolism | Epigenetic inheritance | Carcinoma | Patient outcomes | Development and progression | Genetic aspects | Research | Fructose | Cancer | Proteins | Enzymes | Cell growth | Dehydrogenases | Metabolites | Rodents | Kidney cancer | Hypoxia | Kinases | Metabolism | Tumors | Index Medicus
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10. Full Text TGFβ signalling: a complex web in cancer progression
by Miyazono, Kohei and Ikushima, Hiroaki and Medicinska och farmaceutiska vetenskapsområdet and Uppsala universitet and Medicinska och farmaceutiska vetenskapsområdet, centrumbildningar mm and Ludwiginstitutet för cancerforskning
Nature Reviews Cancer, ISSN 1474-175X, 06/2010, Volume 10, Issue 6, pp. 415 - 424
The distortion of growth factor signalling is the most important prerequisite in tumour progression. Transforming growth factor-beta (TGF beta) signalling... 
BREAST-CANCER | GROWTH-FACTOR-BETA | STEM-CELLS | RECEPTOR KINASE INHIBITOR | HORMONE-RELATED PROTEIN | PROMOTES TUMOR-GROWTH | ONCOLOGY | TRANSFORMING GROWTH-FACTOR-BETA-1 | TRANSCRIPTION FACTOR DEC1 | NON-HODGKINS-LYMPHOMA | EPITHELIAL-MESENCHYMAL TRANSITIONS | Medical and Health Sciences | Medicin och hälsovetenskap
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11. Full Text Sorafenib with ASC‐J9® synergistically suppresses the HCC progression via altering the pSTAT3‐CCL2/Bcl2 signals
by Xu, Junjie and Lin, Hui and Li, Gonghui and Sun, Yin and Shi, Liang and Ma, Wen‐Lung and Chen, Jiang and Cai, Xiujun and Chang, Chawnshang
International Journal of Cancer, ISSN 0020-7136, 02/2017, Volume 140, Issue 3, pp. 705 - 717
Sorafenib is currently used as a standard treatment to suppress the progression of hepatocellular carcinoma (HCC), especially in advanced stages. However,... 
synergism | ASC‐J9 | Sorafenib | ® | hepatocellular carcinoma | ASC-J9 | HEPATOCELLULAR-CARCINOMA CELLS | ANDROGEN RECEPTOR | ACTIVATION | TRANSCRIPTION 3 | STAT3 | ASC-J9((R)) | PROSTATE-CANCER METASTASIS | INHIBITION | ONCOLOGY | RESISTANCE | DEGRADATION | CURCUMIN ANALOGS | Niacinamide - analogs & derivatives | Curcumin - analogs & derivatives | Apoptosis - drug effects | Humans | Male | Proto-Oncogene Proteins c-bcl-2 - metabolism | Carcinoma, Hepatocellular - drug therapy | Liver Neoplasms - pathology | Chemokine CCL2 - metabolism | STAT3 Transcription Factor - metabolism | Curcumin - pharmacology | Liver Neoplasms - drug therapy | Disease Progression | Drug Synergism | Drug Therapy, Combination - methods | Animals | Signal Transduction - drug effects | Mice, Nude | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Mice | Phenylurea Compounds - pharmacology | Niacinamide - pharmacology | Carcinoma, Hepatocellular - metabolism | Antimitotic agents | Antineoplastic agents | Analysis | Liver cancer | Chemotherapy | Index Medicus | Cancer Therapy and Prevention
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