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Neuropharmacology, ISSN 0028-3908, 10/2012, Volume 63, Issue 5, pp. 905 - 915
Mu-opioid and CB1-cannabinoid agonists produce analgesia; however, adverse effects limit use of drugs in both classes. Additive or synergistic effects... 
Analgesia | Morphine | Marijuana | G-protein | Adenylyl cyclase | CB1 receptors | AGONIST MORPHINE | ACTIVATION | PROTEIN-KINASE | CB1 CANNABINOID RECEPTOR | SYNERGISTIC INTERACTIONS | NEUROSCIENCES | INHIBITION | KAPPA | FULL AGONIST | ADENYLYL-CYCLASE | PHARMACOLOGY & PHARMACY | RAT-BRAIN | Pyrazoles - therapeutic use | Receptor, Cannabinoid, CB2 - agonists | Humans | Analgesics, Opioid - pharmacology | Receptors, Opioid, mu - agonists | Piperidines - pharmacology | Cannabinoid Receptor Agonists - therapeutic use | Recombinant Proteins - antagonists & inhibitors | Piperidines - metabolism | Receptors, Opioid, mu - metabolism | Morpholines - metabolism | Analgesics, Non-Narcotic - metabolism | Drug Inverse Agonism | Mice | Analgesics, Opioid - metabolism | Kinetics | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Analgesics, Opioid - antagonists & inhibitors | Cannabinoid Receptor Agonists - metabolism | Cricetulus | Analgesics, Non-Narcotic - pharmacology | Cannabinoid Receptor Agonists - pharmacology | Receptor, Cannabinoid, CB2 - genetics | Analgesics, Opioid - adverse effects | Narcotic Antagonists - metabolism | Receptor, Cannabinoid, CB1 - agonists | Analgesics, Non-Narcotic - adverse effects | Cannabinoid Receptor Agonists - adverse effects | Morpholines - adverse effects | CHO Cells | Pyrazoles - adverse effects | Pyrazoles - pharmacology | Binding, Competitive | Recombinant Proteins - metabolism | Cricetinae | Morpholines - pharmacology | Narcotic Antagonists - adverse effects | Recombinant Proteins - agonists | Analgesics, Non-Narcotic - therapeutic use | Pyrazoles - metabolism | Mice, Inbred Strains | Receptors, Opioid, mu - antagonists & inhibitors | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Narcotic Antagonists - therapeutic use | Piperidines - therapeutic use | Narcotic Antagonists - pharmacology | Piperidines - adverse effects | Receptors, Opioid, mu - genetics | Index Medicus | Drugs | Pain perception | Opioid receptors | Cyclic AMP | Opioid receptors (type mu) | Antagonists | Cannabinoid CB1 receptors | Guanine nucleotide-binding protein | Antagonism | Cell activation | Side effects | Inverse agonists | CB1 Receptors | Adenylyl Cyclase
Journal Article
Neuropharmacology, ISSN 0028-3908, 08/2006, Volume 51, Issue 2, pp. 358 - 366
Journal Article
Neuropharmacology, ISSN 0028-3908, 09/2012, Volume 63, Issue 4, pp. 653 - 666
Cannabinoids have been shown to exert neuroprotective effects in a plethora of neurodegenerative conditions. Over the past decade, some studies demonstrate... 
Neuroinflammation | CB2 | CB1 | WIN55212-2 | PPAR-γ | Beta-amyloid | CB | NITRIC-OXIDE SYNTHASE | ALZHEIMERS-DISEASE | NEUROSCIENCES | PPAR-gamma | CELL-DEATH | CASPASE-3 ACTIVATION | CORTICAL-NEURONS | SIGNALING PATHWAY | DNA FRAGMENTATION | IN-VIVO | PHARMACOLOGY & PHARMACY | INFLAMMATORY RESPONSE | NF-KAPPA-B | Receptor, Cannabinoid, CB2 - agonists | Neuroglia - pathology | Injections, Intraventricular | Male | PPAR gamma - metabolism | Alzheimer Disease - pathology | Cannabinoid Receptor Agonists - therapeutic use | Neuroglia - immunology | Nootropic Agents - antagonists & inhibitors | Cannabinoid Receptor Antagonists - pharmacology | Neurons - metabolism | Alzheimer Disease - immunology | Morpholines - therapeutic use | Receptors, Cannabinoid - chemistry | Disease Models, Animal | Benzoxazines - administration & dosage | Nerve Tissue Proteins - agonists | Alzheimer Disease - drug therapy | Rats | Hippocampus - pathology | Random Allocation | PPAR gamma - antagonists & inhibitors | Signal Transduction - drug effects | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Naphthalenes - therapeutic use | Neurons - pathology | Rats, Wistar | Hippocampus - drug effects | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Cannabinoid Receptor Agonists - chemistry | Neuroglia - drug effects | Receptors, Cannabinoid - metabolism | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | Receptor, Cannabinoid, CB1 - agonists | Morpholines - antagonists & inhibitors | Benzoxazines - antagonists & inhibitors | Neurons - drug effects | Nerve Tissue Proteins - antagonists & inhibitors | Hippocampus - immunology | Morpholines - administration & dosage | Neurons - immunology | Nootropic Agents - therapeutic use | Cannabinoid Receptor Agonists - administration & dosage | Benzoxazines - therapeutic use | Nerve Tissue Proteins - metabolism | Hippocampus - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Nootropic Agents - administration & dosage | Naphthalenes - administration & dosage | Alzheimer Disease - metabolism | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | PPAR gamma - agonists | Neuroglia - metabolism | Naphthalenes - antagonists & inhibitors
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 01/2012, Volume 37, Issue 2, pp. 478 - 486
The midbrain dorsal periaqueductal gray (dPAG) has an important role in orchestrating anxiety-and panic-related responses. Given the cellular and behavioral... 
vanilloid | anxiety | anandamide | cannabinoid | periaqueductal gray | panic | LOCALIZATION | GRAY | PSYCHIATRY | BEHAVIOR | PERIAQUEDUCTAL GREY | ENDOCANNABINOID SYSTEM | NEUROSCIENCES | ACID AMIDE HYDROLASE | LONG-TERM DEPRESSION | PHARMACOLOGY & PHARMACY | BRAIN | Rats, Wistar | Capsaicin - pharmacology | Cinnamates - administration & dosage | Microinjections - methods | Periaqueductal Gray - drug effects | Male | Piperidines - antagonists & inhibitors | Receptor, Cannabinoid, CB1 - physiology | Capsaicin - administration & dosage | TRPV Cation Channels - metabolism | Drug Interactions | Receptor, Cannabinoid, CB1 - agonists | Piperidines - pharmacology | Arachidonic Acids - administration & dosage | TRPV Cation Channels - antagonists & inhibitors | Electric Stimulation - methods | Pyrazoles - pharmacology | Panic - drug effects | Piperidines - administration & dosage | TRPV Cation Channels - physiology | Rats | Capsaicin - analogs & derivatives | Cinnamates - pharmacology | Arachidonic Acids - pharmacology | Receptor, Cannabinoid, CB1 - metabolism | Panic - physiology | Pyrazoles - administration & dosage | Animals | Anilides - administration & dosage | Anilides - pharmacology | Periaqueductal Gray - physiology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Pyrazoles - antagonists & inhibitors | animal models | psychopharmacology | mood | stress disorders | cannabinoids | Original
Journal Article
Pharmacological Reviews, ISSN 0031-6997, 09/2011, Volume 63, Issue 3, pp. 461 - 470
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 03/2007, Volume 150, Issue 5, pp. 613 - 623
Background and purpose: A nonpsychoactive constituent of the cannabis plant, cannabidiol has been demonstrated to have low affinity for both cannabinoid CB1... 
cannabidiol | CB2 receptor | CP55940 | O‐2654 | GTPγS assay | CB1 receptor | mouse brain | inverse agonism | neutral antagonism | O-2654 | GTP gamma S assay | INHIBITION | LIGANDS | SELECTIVE INVERSE AGONIST | MOUSE VAS-DEFERENS | PERIPHERAL CANNABINOID RECEPTOR | PHARMACOLOGY & PHARMACY | BINDING | BRAIN | Cricetulus | Cyclohexanols | Receptor, Cannabinoid, CB2 - agonists | Cannabinoid Receptor Antagonists | Cyclohexanes - pharmacology | Humans | Bornanes - pharmacology | Anti-Inflammatory Agents - metabolism | Brain - metabolism | Dose-Response Relationship, Drug | Receptors, Cannabinoid - metabolism | Receptor, Cannabinoid, CB1 - agonists | Transfection | Piperidines - pharmacology | Morpholines - antagonists & inhibitors | Phenols - antagonists & inhibitors | Cyclohexanes - antagonists & inhibitors | Benzoxazines - antagonists & inhibitors | Cell Membrane - metabolism | Cell Membrane - drug effects | Receptors, Cannabinoid - genetics | CHO Cells | Phenols - pharmacology | Pyrazoles - pharmacology | Cricetinae | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Morpholines - pharmacology | Cannabidiol - pharmacology | Mice, Knockout | Naphthalenes - pharmacology | Brain - drug effects | Cannabinoid Receptor Agonists | Guanosine 5'-O-(3-Thiotriphosphate) - metabolism | Animals | Benzoxazines - pharmacology | Protein Binding | Mice | Cannabidiol - metabolism | In Vitro Techniques | Naphthalenes - antagonists & inhibitors | Research Papers
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 07/2013, Volume 346, Issue 1, pp. 54 - 66
The G protein-coupled receptor 55 (GPR55) is a lysophosphatidylinositol (LPI) receptor that is also responsive to certain cannabinoids. Although GPR55 has been... 
IN-VITRO | ACTIVATION | CANNABINOID RECEPTOR | ASSAY | L-ALPHA-LYSOPHOSPHATIDYLINOSITOL | LIGANDS | GPR55 | PHARMACOLOGY & PHARMACY | PROLIFERATION | IDENTIFICATION | CANCER | Azabicyclo Compounds - pharmacology | Endothelium, Vascular - cytology | Lysophospholipids - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | NFATC Transcription Factors - agonists | Endothelium, Vascular - drug effects | NF-kappa B - metabolism | Protein Transport - drug effects | Sulfones - antagonists & inhibitors | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Sulfones - pharmacology | Recombinant Fusion Proteins - metabolism | HEK293 Cells | Platelet Aggregation - ethics | Cannabinoid Receptor Antagonists - pharmacology | Piperazines - antagonists & inhibitors | Platelet Aggregation - drug effects | Blood Platelets - drug effects | Wound Healing - drug effects | Gene Expression | NF-kappa B - antagonists & inhibitors | Lysophospholipids - antagonists & inhibitors | NFATC Transcription Factors - metabolism | Cells, Cultured | Piperazines - pharmacology | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Receptor, Cannabinoid, CB1 - genetics | Blood Platelets - metabolism | Calcium Signaling - drug effects | Endothelium, Vascular - metabolism | Receptors, G-Protein-Coupled - antagonists & inhibitors | Benzoates - pharmacology | Ligands | Receptors, G-Protein-Coupled - genetics | NFATC Transcription Factors - antagonists & inhibitors | Receptors, G-Protein-Coupled - chemistry
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 04/2012, Volume 83, Issue 7, pp. 952 - 961
Journal Article