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Separation and Purification Technology, ISSN 1383-5866, 03/2015, Volume 142, pp. 155 - 161
Journal Article
FEBS Letters, ISSN 0014-5793, 06/2014, Volume 588, Issue 12, pp. 2095 - 2100
•CCl4-induced elevation of serum ALP and TBIL was reduced in TLR2−/− mice.•CCl4-induced liver collagen deposition was attenuated in TLR2−/− mice.•CCl4-induced... 
NF-κB | Liver fibrosis | Hepatic stellate cell | Toll like receptor 2 | Carbon tetrachloride | mitogen-activated protein kinases | extracellular signal-regulated kinase | Col1A1 | total bilirubin | ALP | extracellular matrix proteins | JNK | nuclear factor kappa B | Toll like receptor | platelet-derived growth factor | TNF | α-SMA | HSC | alkaline phosphatase | hepatic stellate cell | TLR | interleukin | hematoxylin and eosin | c-Jun N-terminal kinase | H&E | carbon tetrachloride | MAPK | smooth muscle alpha-actin | ECM | PAMP | TGF | type I procollagen α1 chain | pathogen-associated molecular patterns | tumor necrosis factor | damage-associated molecular patterns | TBIL | PDGF | CCl4 | LTA | lipoteichoic acid | wild-type | transforming growth factor | ERK | DAMP | APOPTOSIS | ACTIVATION | CONTRIBUTE | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | CELL BIOLOGY | NF-kappa B | HEPATIC STELLATE CELLS | INHIBITION | BIOPHYSICS | INFLAMMATION | SENESCENCE | MODULATION | Toll-Like Receptor 2 - genetics | Carbon Tetrachloride - pharmacology | Hepatic Stellate Cells - metabolism | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | Liver Cirrhosis - chemically induced | Toll-Like Receptor 2 - deficiency | Phosphorylation - genetics | MAP Kinase Signaling System - genetics | Liver Cirrhosis - metabolism | Phosphorylation - drug effects | Cytokines - genetics | Liver Cirrhosis - genetics | Hepatic Stellate Cells - drug effects | Mice, Inbred C57BL | RNA, Messenger - genetics | Down-Regulation - drug effects | Gene Knockout Techniques | Down-Regulation - genetics | Collagen - metabolism | Animals | MAP Kinase Signaling System - drug effects | Liver Cirrhosis - pathology | Mice | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Journal Article
Journal Article
Journal Article
Toxicology, ISSN 0300-483X, 2010, Volume 273, Issue 1, pp. 45 - 52
Journal Article
Mediators of Inflammation, ISSN 0962-9351, 2014, Volume 2014, pp. 696383 - 15
This study investigated the anti-inflammatory effects of DEC on the CCl4-induced hepatotoxicity in C57BL/6 mice. Chronic inflammation was induced by i.p.... 
GROWTH-FACTOR-BETA | OXIDATIVE STRESS | ACTIVATION | HEPATIC STELLATE CELLS | TGF-BETA | NITRIC-OXIDE SYNTHASE | ENDOTHELIAL-CELLS | GENE-EXPRESSION | INTERLEUKIN-10 | IMMUNOLOGY | NF-KAPPA-B | CELL BIOLOGY | Inflammation - pathology | Gene Expression - drug effects | Male | RNA, Messenger - metabolism | Inflammation - metabolism | Cyclooxygenase 2 - genetics | Inflammation - drug therapy | Liver Cirrhosis - metabolism | Chemical and Drug Induced Liver Injury - pathology | Disease Models, Animal | Liver Cirrhosis - drug therapy | Cytokines - metabolism | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | RNA, Messenger - genetics | Diethylcarbamazine - pharmacology | Carbon Tetrachloride - toxicity | Collagen - metabolism | Animals | Nitric Oxide Synthase Type II - genetics | Chemical and Drug Induced Liver Injury - metabolism | Chemical and Drug Induced Liver Injury - drug therapy | Cyclooxygenase 2 - metabolism | Liver Cirrhosis - pathology | Mice | Nitric Oxide - metabolism | Prevention | Liver diseases | Drug interactions | Carbon tetrachloride | Fibrosis | Physiological aspects | Diethylcarbamazine | Dosage and administration | Inflammation | Identification and classification | Health aspects | Oxidative stress | Enzymes | Free radicals | Cytokines | Laboratories | Alcoholism | Mortality | Smooth muscle | Metabolism | Carbon | Metabolites | Rodents | Nitric oxide | Collagen | Tumor necrosis factor-TNF | Liver cirrhosis | Growth factors | Injuries
Journal Article
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 10/2017, Volume 95, Issue 10, pp. 1077 - 1089
Alcoholic liver disease (ALD) is a leading cause of liver cirrhosis, liver cancer, and related mortality. The endocannabinoid system contributes to the... 
Human Genetics | Therapy | Cannabinoids | Biomedicine | Liver | Methylhonokiol | Internal Medicine | Alcohol | Molecular Medicine | MEDICINE, RESEARCH & EXPERIMENTAL | BETA-SECRETASE ACTIVITY | OXIDATIVE STRESS | P21-MEDIATED SUPPRESSION | ALZHEIMERS-DISEASE | MEMORY IMPAIRMENT | ENDOCANNABINOID SYSTEM | MOUSE MODEL | GENETICS & HEREDITY | MAGNOLIA-OFFICINALIS | KAPPA-B ACTIVITY | GAMMA AGONIST | Liver Diseases, Alcoholic - pathology | Chemical and Drug Induced Liver Injury - prevention & control | Liver - pathology | Liver Cirrhosis - prevention & control | Lignans - chemistry | Carbon Tetrachloride | Mice, Inbred C57BL | Liver Diseases, Alcoholic - prevention & control | Male | Protective Agents - chemistry | Protective Agents - therapeutic use | Biphenyl Compounds - therapeutic use | Lignans - therapeutic use | Magnolia - chemistry | Animals | Liver - drug effects | Endocannabinoids - analysis | Liver Cirrhosis - pathology | Biphenyl Compounds - chemistry | Chemical and Drug Induced Liver Injury - pathology | Prevention | Liver diseases | Causes of | Genetic aspects | Research | Health aspects | Alcohols | Matrix metalloproteinases | Bcl-2 protein | Carbon tetrachloride | Diacylglycerol | mRNA | Activation | Damage prevention | Liver cancer | Endocannabinoid system | Diacylglycerol O-acyltransferase | Rodents | Animal tissues | Biphenyl | Cannabinoid CB2 receptors | Lesions | Stellate cells | Damage assessment | Mass spectroscopy | JNK protein | Liquid chromatography | Histology | Cannabinoid CB1 receptors | Gene expression | CCL4 protein | Fatty acids | Fatty-acid synthase | Mcl-1 protein | Anandamide | Cirrhosis | Fibrosis | Interleukin 10 | Alcoholic beverages | Mice | Mass spectrometry | Apoptosis
Journal Article
Journal Article