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PLoS Genetics, ISSN 1553-7390, 02/2018, Volume 14, Issue 2, p. e1007212
The lack of a mutant phenotype in homozygous mutant individuals' due to compensatory gene expression triggered upstream of protein function has been identified... 
HEART | SKELETAL-MUSCLE | ROBUSTNESS | DUPLICATE GENES | GENETICS & HEREDITY | ALPHA-CARDIAC ACTIN | ABSENCE | MICE | MYOPATHY | EXPRESSION | Physiological aspects | Muscle diseases | Genetic aspects | Gene mutations | Actin | Muscle proteins | Gene expression | Analysis | Phenotypes | Transcription | Funding | Zebrafish | Antisense | Genomes | Muscular dystrophy | Proteins | Musculoskeletal system | Vertebrates | Nemaline myopathy | Compensation | Muscle function | Mutation | Myopathy
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 08/2017, Volume 490, Issue 2, pp. 324 - 329
Myocardium of mammals contains a wide range of isoforms of proteins that provides contractile function of the heart. These are two isoforms of ventricular and... 
In vitro motility assay | Cardiac myosin | Actin | Tropomyosin | Calcium regulation | BETA-TROPOMYOSIN | MOUSE HEART | BIOCHEMISTRY & MOLECULAR BIOLOGY | RABBIT SKELETAL-MUSCLE | MYOFILAMENT CALCIUM SENSITIVITY | IN-VITRO | BIOPHYSICS | In vitro motility assay | STRIATED-MUSCLE TROPOMYOSIN | LIGHT-CHAIN ISOFORMS | EXPRESSION | BINDING
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