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Circulation Research, ISSN 0009-7330, 11/2004, Volume 95, Issue 10, pp. 957 - 970
Journal Article
JOURNAL OF CLINICAL INVESTIGATION, ISSN 0021-9738, 05/2003, Volume 111, Issue 10, pp. 1497 - 1504
Heart failure is a common, lethal condition whose pathogenesis is poorly understood. Recent studies have identified low levels of myocyte apoptosis (80-250... 
MYOCARDIUM | MEDICINE, RESEARCH & EXPERIMENTAL | INDUCED-PROXIMITY MODEL | CONTRACTILITY | MICE DEFICIENT | FAILING HUMAN HEART | RECEPTORS | STRESS | CASPASE ACTIVATION | INTERLEUKIN-1-BETA CONVERTING-ENZYME
Journal Article
Circulation, ISSN 0009-7322, 08/2004, Volume 110, Issue 6, pp. 705 - 712
Background-The endoplasmic reticulum (ER) is recognized as an organelle that participates in folding secretory and membrane proteins. The ER responds to stress... 
Heart failure | Endoplasmic reticulum | Apoptosis | Hypertrophy | heart failure | CELLS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | hypertrophy | apoptosis | COORDINATION | BETA | UNFOLDED PROTEIN RESPONSE | DISEASE | DEGRADATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | endoplasmic reticulum | Tetrazoles - pharmacology | Humans | Heart Failure - physiopathology | Rats, Inbred WKY | Male | Natriuretic Peptide, Brain - physiology | Angiotensin II Type 1 Receptor Blockers - pharmacology | RNA, Messenger - biosynthesis | Stress, Physiological - chemically induced | Ligation | Endoplasmic Reticulum - drug effects | Cells, Cultured - pathology | Heart Failure - etiology | Thapsigargin - toxicity | Disease Models, Animal | Molecular Chaperones - biosynthesis | Olmesartan Medoxomil | Angiotensin II - pharmacology | Cells, Cultured - drug effects | Tunicamycin - toxicity | Signal Transduction | Mice, Inbred C57BL | RNA, Messenger - genetics | Aortic Valve Stenosis - physiopathology | Endoplasmic Reticulum - physiology | Molecular Chaperones - genetics | Cardiomegaly - physiopathology | Rats | Heart Failure - genetics | Stress, Physiological - physiopathology | Imidazoles - pharmacology | Cardiomegaly - complications | Disease Progression | Gene Expression Regulation - drug effects | Myocytes, Cardiac - pathology | Aortic Valve Stenosis - complications | Animals | Myocytes, Cardiac - drug effects | Mice | Cardiomegaly - genetics | Angiotensin II - physiology
Journal Article
Journal of Cardiovascular Pharmacology, ISSN 0160-2446, 2017, Volume 70, Issue 2, pp. 94 - 101
Prolonged or excessive β-adrenergic activation leads to cardiac myocyte loss and heart dysfunction; however, the underlying cellular mechanisms are still... 
20-HETE | cardiac myocytes | apoptosis | cytochrome P450 ω-hydroxylase | Animals, Newborn | Animals | Cytochrome P-450 CYP4A - physiology | Myocytes, Cardiac - drug effects | Rats, Wistar | Myocytes, Cardiac - physiology | Cells, Cultured | Adrenergic beta-Agonists - pharmacology | Rats | Receptors, Adrenergic, beta - physiology
Journal Article
Journal Article