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Free radical biology & medicine, ISSN 0891-5849, 2011, Volume 50, Issue 7, pp. 777 - 793
.... Here, we review the main redox-regulated pathways that are involved in cardiac myocyte excitation... 
Heart failure | Reactive oxygen species | Mitochondria | Free radicals | NADPH oxidase | Cardiac myocyte | Redox signaling | Hypertrophy | HYPOXIA-INDUCIBLE FACTOR | NOX2-CONTAINING NADPH OXIDASE | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR-KAPPA-B | MITOCHONDRIAL PERMEABILITY TRANSITION | EMBRYONIC STEM-CELLS | RECEPTOR-STIMULATED HYPERTROPHY | ENDOCRINOLOGY & METABOLISM | INDUCED CARDIOMYOCYTE HYPERTROPHY | XANTHINE OXIDOREDUCTASE INHIBITION | RAT VENTRICULAR MYOCYTES | Excitation Contraction Coupling - physiology | Myocardial Ischemia - metabolism | Rabbits | Cell Proliferation | Reactive Oxygen Species - metabolism | Heart Diseases - metabolism | Oxidation-Reduction | Oxidative Stress | Heart Diseases - physiopathology | Humans | NADPH Oxidases - metabolism | Stress, Physiological | Rats | Mitochondria - metabolism | Hypertrophy - metabolism | Oxygen - metabolism | Hypertrophy - physiopathology | Animals | Myocytes, Cardiac - physiology | Myocardial Ischemia - physiopathology | Thioredoxins - metabolism | Mice | Cell Differentiation - physiology | ARC, apoptosis repressor with caspase recruitment domain | PKG, protein kinase G | AIF, apoptosis-inducing factor | Review | MAO-A, monoamine oxidase-A | MI, myocardial infarction | TNFα, tumor necrosis factor-α | ES, embryonic stem | GPCR, G-protein-coupled receptor | RyR, ryanodine receptor | PHD, prolyl hydroxylase dioxygenase | CTGF, connective tissue growth factor | mtDNA, mitochondrial DNA | ROS, reactive oxygen species | ETC, electron transport chain | HDAC, histone deacetylase | EB, embryoid body | Ca exchanger | G6PDH, glucose-6-phosphate dehydrogenase | PKC, protein kinase C | SR, sarcoplasmic reticulum | SERCA, sarcoplasmic reticulum calcium ATPase | VEGF, vascular endothelial growth factor | Hif, hypoxia-inducible factor | NOS, nitric oxide synthase | NCX, Na | MPTP, mitochondrial permeability transition pore | ECC, excitation–contraction coupling | CamKII, calmodulin kinase II | ER, endoplasmic reticulum | MMP, matrix metalloproteinase | PKA, protein kinase A | Trx1, thioredoxin1
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