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cardiac-specific overexpression (155) 155
animals (119) 119
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Circulation, ISSN 0009-7322, 11/2011, Volume 124, Issue 19, pp. 2106 - 2116
Background-Transgenic mice with cardiac-restricted overexpression of tumor necrosis factor (MHCsTNF mice) develop progressive myocardial fibrosis, diastolic... 
fibroblasts | fibrosis | tumor necrosis factor | mast cells | transforming growth factor beta | CARDIAC & CARDIOVASCULAR SYSTEMS | STIMULATION | CARDIAC-SPECIFIC OVEREXPRESSION | ISCHEMIA | FAILURE | DENSITY | GROWTH-FACTOR-BETA | DISEASE | MYOFIBROBLASTS | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | EXPRESSION | Cell Communication - immunology | Myocardium - immunology | Tumor Necrosis Factor-alpha - genetics | Signal Transduction - immunology | Gene Expression - immunology | Tumor Necrosis Factor-alpha - immunology | Endomyocardial Fibrosis - pathology | Endomyocardial Fibrosis - immunology | Myocarditis - immunology | Mast Cells - immunology | Pteridines - pharmacology | Mice, Transgenic | Myocardium - pathology | Fibroblasts - pathology | Phenotype | Animals | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Signal Transduction - drug effects | Mast Cells - pathology | Fibroblasts - immunology | Endomyocardial Fibrosis - physiopathology | Mice | Myocarditis - pathology | Primary Cell Culture | Myocarditis - physiopathology | Transforming Growth Factor beta - metabolism | Cell interaction | Tumor necrosis factor | Fibrosis | Fibroblasts | Physiological aspects | Development and progression | Mast cells | Research | Transforming growth factor-beta (TGF-β) | Tumor necrosis factor (TNF) | cardiac fibroblasts | myocardial fibrosis
Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, ISSN 0363-6135, 03/2018, Volume 314, Issue 3, pp. H643 - H658
Transgenic (TG) mice overexpressing beta(2)-adrenergic receptors (beta(2)-ARs) demonstrate enhanced myocardial function, which manifests in increased basal... 
CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | PROTEIN | CARDIAC-SPECIFIC OVEREXPRESSION | HEART-FAILURE | RECEPTOR | CARDIOMYOCYTES | calcium transient | CA2+ CURRENT | transgenic mice | CONTRACTION | beta-adrenergic receptors | PERIPHERAL VASCULAR DISEASE | zinterol | cardiac action potential | COMPARTMENTATION
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2009, Volume 47, Issue 3, pp. 382 - 390
Abstract The occurrence of cardiovascular dysfunction in sepsis is associated with a significantly increased mortality rate of 70% to 90% compared with 20% in... 
Cardiovascular | Myocardial function | NF-κB | Sepsis | Small heat shock protein | Apoptosis | ISCHEMIA/REPERFUSION INJURY | CARDIAC & CARDIOVASCULAR SYSTEMS | PHOSPHORYLATION | CARDIAC-SPECIFIC EXPRESSION | STRESS-RESPONSE | RATS | EXERCISE | HEAT-SHOCK-PROTEIN | CELL BIOLOGY | NF-kappa B | INFLAMMATION | MICE | SEVERE SEPSIS | small heat shock protein | myocardial function | sepsis
Journal Article
Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, ISSN 0363-6135, 03/2009, Volume 296, Issue 3, pp. H704 - H718
Rivard K, Trepanier-Boulay V, Rindt H, Fiset C. Electrical remodeling in a transgenic mouse model of alpha(1B)-adrenergic receptor overexpression. Am J Physiol... 
CHANNEL EXPRESSION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ALPHA-SUBUNIT | CARDIAC-SPECIFIC OVEREXPRESSION | HYPERTROPHIC FACTORS | HEART-FAILURE | OUTWARD K+ CURRENT | K+ currents | ANGIOTENSIN-II | ACTION-POTENTIAL PROLONGATION | mouse ventricle | arrhythmias | PERIPHERAL VASCULAR DISEASE | RAT VENTRICULAR MYOCYTES | TARGETED DELETION
Journal Article
Journal Article
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES, ISSN 1422-0067, 08/2019, Volume 20, Issue 15, p. 3673
Journal Article
by Tan, Y and Chen, SN and Zhong, JK and Ren, J and Dong, ML
CURRENT PHARMACEUTICAL DESIGN, ISSN 1381-6128, 2019, Volume 25, Issue 18, pp. 2060 - 2070
Background: Sepsis and septic shock are known to prompt multiple organ failure including cardiac contractile dysfunction, which is typically referred to as... 
heart failure | OXIDATIVE STRESS | ACTIVATION | CANINE MODEL | CARDIAC-SPECIFIC OVEREXPRESSION | NITRIC-OXIDE SYNTHASE | CONTRACTILE DYSFUNCTION | heart | SHOCK | Mitochondria | contraction | INDUCED MYOCARDIAL DYSFUNCTION | PHARMACOLOGY & PHARMACY | intervention | sepsis
Journal Article
Journal Article
CELLULAR SIGNALLING, ISSN 0898-6568, 10/2014, Volume 26, Issue 10, pp. 2147 - 2160
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2007, Volume 104, Issue 45, pp. 17867 - 17872
Journal Article