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PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e72278
Background: Opioidergic SLP (sustained ligand-activated preconditioning) induced by 3-5 days of opioid receptor (OR) agonism induces persistent protection... 
NATRIURETIC-PEPTIDE | ISCHEMIA/REPERFUSION INJURY | INFARCTION | OPIOID RECEPTOR | MULTIDISCIPLINARY SCIENCES | CARDIOPROTECTIVE PHENOTYPE | MICE | MECHANISMS | ISCHEMIA-REPERFUSION | MOUSE HEARTS | MYOSIN HEAVY-CHAIN | Myocardial Ischemia - genetics | Animals | Polymerase Chain Reaction | Ischemic Preconditioning | Myocardial Ischemia - physiopathology | Ligands | Male | Transcription, Genetic | Gene Expression Profiling | Mice | Mice, Inbred BALB C | Heart | Genes | HLA histocompatibility antigens | Heat shock proteins | Genetic transcription | Proteins | Histocompatibility antigens | DNA microarrays | Ischemia | Analysis | Genetic research | Genetic aspects | Natriuretic peptides | Heart attacks | Peptides | Morphine | Immunity | Interrogation | Egr-3 protein | Cytokines | Contractility | GSTM1 protein | Gene expression | CD83 antigen | Gene silencing | Injury prevention | Myocardium | Preconditioning | Monocyte chemoattractant protein 1 | Health sciences | Oxidative stress | CD86 antigen | Carbon tetrachloride | Kinases | Interleukin 6 | Reperfusion | Fos protein | Rodents | Interleukin 1 | Drb1 protein | Egr-2 protein | Heart diseases | Hypertension | Opioid receptors | CCL4 protein | Muscle contraction | Signaling | Protein expression | Infarction | Histocompatibility antigen HLA | Diabetes | Stress proteins | Chemokines | Cellular stress response | Apoptosis | Questioning
Journal Article
Fundamental & Clinical Pharmacology, ISSN 0767-3981, 04/2010, Volume 24, Issue 2, pp. 189 - 198
Four pre‐medication drugs are used to relieve pain, allay anxiety, reduce secretion and enhance hypnosis, were evaluated for their effects on ischemia... 
ischemia reperfusion | ischemic pre‐conditioning | pre‐medication | anesthetics | Anesthetics | Ischemic pre-conditioning | Ischemia reperfusion | Pre-medication | PROTECTION | CARDIOPROTECTIVE PHENOTYPE | pre-medication | ischemic pre-conditioning | SEVOFLURANE | ISCHEMIA-REPERFUSION INJURY | INFARCTION | RECEPTOR STIMULATION | OPIOID RECEPTOR | VOLATILE ANESTHETICS | PHARMACOLOGY & PHARMACY | RAT-HEART | HISTAMINE | Adjuvants, Anesthesia - administration & dosage | Anti-Anxiety Agents - administration & dosage | Adjuvants, Anesthesia - pharmacology | Kidney - blood supply | Kidney - pathology | Rats, Wistar | Analgesics, Opioid - pharmacology | Dose-Response Relationship, Drug | Ischemic Preconditioning - methods | Histamine H1 Antagonists - pharmacology | Alprazolam - administration & dosage | Histamine H1 Antagonists - administration & dosage | Female | Promethazine - administration & dosage | Alprazolam - pharmacology | Premedication - methods | Kidney - drug effects | Morphine - pharmacology | Anti-Anxiety Agents - pharmacology | Rats | Atropine - pharmacology | Animals | Reperfusion Injury - prevention & control | Analgesics, Opioid - administration & dosage | Reperfusion Injury - physiopathology | Atropine - administration & dosage | Morphine - administration & dosage | Promethazine - pharmacology | Alprazolam | Morphine | Care and treatment | Pain
Journal Article
Journal Article
American Journal of Drug Discovery and Development, ISSN 2150-427X, 2011, Volume 1, Issue 4, pp. 209 - 219
Journal Article
Pharmacology and Therapeutics, ISSN 0163-7258, 06/2018, Volume 186, pp. 73 - 87
Acute myocardial infarction (AMI) and the heart failure that often follows, are major causes of death and disability worldwide. As such, new therapies are... 
Monocytes | Acute myocardial ischemia and reperfusion injury | Dendritic cells | Cytokines | Lymphocytes | Innate immunity | Inflammation | Acute myocardial infarction | Macrophages | Chemokines | ISCHEMIA/REPERFUSION INJURY | MANNOSE-BINDING LECTIN | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | ANTI-C5 COMPLEMENT ANTIBODY | ISCHEMIA-REPERFUSION INJURY | TOLL-LIKE RECEPTOR-2 | PERCUTANEOUS CORONARY INTERVENTION | CARDIOPROTECTIVE THERAPIES CAESAR | REGULATORY T-CELLS | MOBILITY GROUP BOX-1 | PHARMACOLOGY & PHARMACY | Humans | Myocardial Infarction - immunology | Heart Failure - prevention & control | Animals | Myocardial Infarction - drug therapy | Heart Failure - immunology | Inflammation Mediators - metabolism | Anti-Inflammatory Agents - therapeutic use | Anti-Inflammatory Agents - administration & dosage | Ventricular Remodeling - immunology | Ventricular Remodeling - drug effects | Disease Models, Animal | Transluminal angioplasty | Heart | Fibronectins | Chromosomal proteins | C-reactive protein | Heat shock proteins | Antigen presenting cells | Transforming growth factors | T cells | Biological response modifiers | Heart attack | Prevention | Oligomers | Interleukins | Cardiac patients | Nitric oxide | Bone morphogenetic proteins | Interferon | Health aspects | CCR5, Chemokine receptor 5 | NO, Nitric oxide | EDA, Extra domain A | PMN, Polymorphonuclear leukocytes | AMI, Acute myocardial infarction | MMPs, Matrix metalloproteinases | MCP-1, Monocyte chemoattractant protein-1 | ICAM-1 | Tregs, Regulatory T cells | BAFF, B-cell activating factor | FN-EDA, Fibronectin-end domain A | IFN-γ, Interferon-γ | STEMI, ST segment elevation myocardial infarction | AGEs, Advanced glycation end-products | RAGE, Receptor for advanced glycation end-products | ACS, Acute coronary syndrome | IRF5, Interferon regulatory factor 5 | LTB4, Leukotriene B4 | NLRP3, Nucleotide-binding oligomerization domain-like receptor family of cytosolic proteins | MyD, Myeloid differentiation primary response gene | TLRs, Toll-like receptors | CCL5, Chemokine ligand 5 | CCR2, Chemokine receptor 2 | hs-CRP, High-sensitivity C-reactive protein | DAMPs, Damage-associated molecular patterns | HMGB1, High mobility group box 1 | IHD, Ischemic heart disease | CINC-1, CXCL1, GRO α, KC, Cytokine-induced neutrophil chemoattractant 1 | IL-1, Interleukin-1 | MIP-2α, CXCL2, GRO β, Macrophage inflammatory protein-2α | PS, Phosphatidylserine | CCR9, Chemokine receptor 5 | TNFα, Tumor necrosis factor-alpha | IL-8, CXCL8, Interleukin-8 | MI, Myocardial infarction | PPCI, Primary percutaneous coronary intervention | HSPs, Heat shock proteins | LV, Left ventricular | CR1, Complement receptor 1 | eRNA, Extracellular ribonucleic acids | CCL2, Chemokine ligand 2 | NF-κB, Nuclear factor kappa-light-chain-enhancer of activated B cells | TGF-β, Transforming growth factor-β | ROS, Reactive oxygen species | ICAM-2, Intercellular adhesion molecule | ECM, Extracellular matrix | C1-INH, C1-inhibitor | NLRs, NOD-like receptors | PRRs, Pattern recognition receptors
Journal Article
Experimental and Therapeutic Medicine, ISSN 1792-0981, 08/2016, Volume 12, Issue 2, pp. 1147 - 1152
The aim of the present study was to investigate the cardioprotective effect of tanshinone IIA and the underlying molecular mechanisms. An in vitro model of... 
Cardioprotective effectß | Akt kinase | Tanshinone IIA | B cell lymphoma-2 | MiR-133 | MEDICINE, RESEARCH & EXPERIMENTAL | MECHANISM | INDUCED APOPTOSIS | MUSCLE-SPECIFIC MICRORNA | REPERFUSION INJURY | SULFONATE | tanshinone IIA | DANSHEN | CARDIOMYOCYTES | PATHWAY | miR-133 | CARDIAC-HYPERTROPHY | cardioprotective effect | EXPRESSION
Journal Article
Basic Research in Cardiology, ISSN 0300-8428, 9/2018, Volume 113, Issue 5, pp. 1 - 73
Journal Article