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Influence of DL α-lipoic acid and vitamin-E against doxorubicin-induced biochemical and histological changes in the cardiac tissue of rats, 12/2005
Objective: The present study was undertaken to find out the preventive and curative role of lipoic acid (LA) and vitamin E (Vit. E) on doxorubicin... 
Antioxidants, cardiotoxicity, doxorubicin, free radicals
Journal
Circulation, ISSN 0009-7322, 06/2015, Volume 131, Issue 22, pp. 1946 - 1949
Journal Article
Journal Article
Journal of Cellular and Molecular Medicine: a journal of translational medicine, ISSN 1582-1838, 01/2017, Volume 21, Issue 12, pp. 3277 - 3287
textabstractThe use of doxorubicin (DOXO) as a chemotherapeutic drug has been hampered by cardiotoxicity leading to cardiomyopathy and heart failure. Folic... 
antioxidant enzyme | cardiotoxicity | nitric oxide | free radical | anthracycline | MORTALITY | MEDICINE, RESEARCH & EXPERIMENTAL | PRESSURE-OVERLOAD | MITOCHONDRIAL-FUNCTION | HEART-FAILURE | INJURY | CARDIAC DYSFUNCTION | PROTECTS | CELL BIOLOGY | INDUCED CARDIOTOXICITY | LEFT-VENTRICULAR DYSFUNCTION | CARDIOVASCULAR-DISEASE | Mitochondria - enzymology | Phosphorylation | Superoxide Dismutase - genetics | Antibiotics, Antineoplastic - toxicity | Folic Acid - pharmacology | Apoptosis - drug effects | Doxorubicin - antagonists & inhibitors | Cardiomyopathies - prevention & control | Male | Cardiomyopathies - enzymology | Cardiotoxicity - pathology | Myocytes, Cardiac - enzymology | Cardiomyopathies - mortality | Superoxides - metabolism | Superoxides - antagonists & inhibitors | Nitric Oxide Synthase Type III - metabolism | Cardiotoxicity - enzymology | Superoxide Dismutase - metabolism | Cardiotoxicity - prevention & control | Mice, Inbred C57BL | Gene Expression Regulation | Stroke Volume - drug effects | Antioxidants - pharmacology | Cardiotonic Agents - pharmacology | Mitochondria - drug effects | Mitochondria - pathology | Nitric Oxide Synthase Type III - genetics | Doxorubicin - toxicity | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Survival Analysis | Cardiotoxicity - mortality | Mice | Oxidative Stress - drug effects | Nitric Oxide - metabolism | Cardiomyopathies - chemically induced | Enzymes | Anthracyclines | Cardiomyopathy | Mortality | Superoxide | Folic acid | Endothelium | Antioxidants | Chemotherapy | Nitric oxide | Heart diseases | Cancer | Myocardial infarction | Heart | Oxidative stress | Superoxide dismutase | Doxorubicin | Atrophy | Mitochondria | Rodents | Supplementation | Pretreatment | Cerebral infarction | Stroke | Dietary supplements | Nitric-oxide synthase | Stroke volume | Acids | Fibrosis | Infarction | Apoptosis | Structure-function relationships | Index Medicus | Original
Journal Article
Circulation research, ISSN 0009-7330, 3/2018, Volume 122, Issue 7, pp. e62 - e63
Journal Article
Circulation research, ISSN 0009-7330, 3/2018, Volume 122, Issue 7, pp. e64 - e65
Journal Article
European Journal of Heart Failure, ISSN 1388-9842, 04/2014, Volume 16, Issue 4, pp. 358 - 366
Aims Doxorubicin is widely used against cancer; however, it can produce heart failure (HF). Among other hallmarks, oxidative stress is a major contributor to... 
Doxorubicin cardiotoxicity • Ranolazine • Heart failure • Oxidative stress • Na | Heart failure | Oxidative stress | CARDIAC & CARDIOVASCULAR SYSTEMS | MANAGEMENT | MYOCYTES | NITROSATIVE STRESS | ISOLATED RAT HEARTS | CARDIOTOXICITY | EUROPEAN-SOCIETY | Ranolazine | DIASTOLIC DYSFUNCTION | Doxorubicin cardiotoxicity | CALCIUM | Na+ | HEART-FAILURE ASSOCIATION | INHIBITOR | Antibiotics, Antineoplastic - toxicity | Reactive Oxygen Species - metabolism | Cardiotoxicity - diagnostic imaging | Ventricular Dysfunction, Left - chemically induced | Atrial Natriuretic Factor - genetics | Reverse Transcriptase Polymerase Chain Reaction - methods | Ultrasonography | Acetanilides - therapeutic use | Real-Time Polymerase Chain Reaction | Cardiotoxicity - prevention & control | Blotting, Western - methods | Natriuretic Peptide, Brain - genetics | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | RNA, Messenger - genetics | Oxidative Stress - genetics | Piperazines - therapeutic use | Enzyme Inhibitors - therapeutic use | Doxorubicin - toxicity | Sodium - blood | Ventricular Dysfunction, Left - prevention & control | Ventricular Dysfunction, Left - metabolism | Matrix Metalloproteinase 2 - genetics | Poly(ADP-ribose) Polymerases - metabolism | Animals | Connective Tissue Growth Factor - genetics | Mice | Oxidative Stress - drug effects | Index Medicus
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 04/2018, Volume 100, Issue 5, pp. 1298 - 1299
Journal Article
Journal Article
12/2009
Severe cardiac adverse effects are often related to intravenous phenytoin overdose. However, there is no reported cardiotoxicity resulting from oral overdose... 
overdose | oral phenytoin | Cardiotoxicity
Web Resource
Journal Article
Environmental Toxicology, ISSN 1520-4081, 03/2019, Volume 34, Issue 3, pp. 271 - 282
The risk of pesticides on the human health and environment has drawn increasing attention. Today, new tools are developed to reduce pesticide adverse effects.... 
cardiotoxicity | polysaccharide | cytotoxicity | oxidative stress | thiamethoxam | APOPTOSIS | LIPID-PEROXIDATION | ANTIOXIDANT ACTIVITIES | RATS | WATER RESOURCES | TOXICITY | TILMICOSIN-INDUCED CARDIOTOXICITY | ENVIRONMENTAL SCIENCES | GLUTATHIONE | FENUGREEK SEEDS | TOXICOLOGY | EXTRACT | Myocardial Infarction - genetics | Thiamethoxam - adverse effects | Rats, Wistar | Glutathione - metabolism | Antioxidants - metabolism | Humans | Male | Cardiotoxicity - etiology | Plant Extracts - administration & dosage | Trigonella - chemistry | Cardiotoxicity - drug therapy | Female | Polysaccharides - administration & dosage | Seeds - chemistry | Myocardial Infarction - etiology | Cardiotoxicity - genetics | Superoxide Dismutase - metabolism | DNA Damage - drug effects | Malondialdehyde - metabolism | Glutathione Peroxidase - metabolism | Rats | Thiobarbituric Acid Reactive Substances - metabolism | Myocardial Infarction - metabolism | Cholesterol - metabolism | Catalase - metabolism | Animals | Myocardial Infarction - drug therapy | Cardiotoxicity - metabolism | Oxidative Stress - drug effects | Antioxidants | Unsaturated fatty acids | Polysaccharides | Creatine kinase | Analysis | DNA damage | DNA | Genetic research | Aspartate | Heart attack | Heart | Myocardial infarction | Carbonyl compounds | Necroses | Lactate | Toxicity | Genotoxicity | Nicking | Insecticides | Lipids | Superoxide dismutase | Tissues | Creatine | Proteins | Risks | Catalase | Calcium-binding protein | Atherosclerosis | Fenugreek | Biocompatibility | Peroxidase | Oxidation | Damage | Deoxyribonucleic acid--DNA | Injuries | Glutathione | Carbonyls | Biodegradation | Glutathione peroxidase | Scavenging | Pesticides | Malondialdehyde | Cholesterol | L-Lactate dehydrogenase | Linoleic acid | Depletion | Arteriosclerosis | Levels | Infarction | In vivo methods and tests | Lactic acid | Viability | Apoptosis | Index Medicus | Life Sciences | Biodiversity
Journal Article
The Oncologist, ISSN 1083-7159, 04/2016, Volume 21, Issue 4, pp. 418 - 424
Journal Article