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1. Full Text SIRT1 regulates adiponectin gene expression through Foxo1-C/enhancer- binding protein α transcriptional complex
by Qiao, Liping and Shao, Jianhua
The Journal of biological chemistry, ISSN 0021-9258, 12/2006, Volume 281, Issue 52, pp. 39915 - 39924
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Adiponectin - biosynthesis | Humans | Molecular Sequence Data | Adipocytes - cytology | CCAAT-Enhancer-Binding Protein-alpha - deficiency | Male | Forkhead Transcription Factors - physiology | Sirtuins - deficiency | Cell Differentiation - genetics | CCAAT-Enhancer-Binding Protein-alpha - physiology | Forkhead Transcription Factors - metabolism | Sirtuin 1 | Adiponectin - genetics | Base Sequence | Up-Regulation - physiology | Sirtuins - genetics | Cell Differentiation - physiology | Forkhead Transcription Factors - deficiency | Cell Line | Promoter Regions, Genetic | Mice, Inbred C57BL | Sirtuins - physiology | Up-Regulation - genetics | Binding Sites - genetics | 3T3-L1 Cells | Forkhead Transcription Factors - genetics | Transcription, Genetic - physiology | Animals | Adipocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice, Obese | Forkhead Box Protein O1 | Mice | Index Medicus
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2. Full Text CCAAT/enhancer-binding protein α negatively regulates IFN-γ expression in T cells
by Tanaka, Shinya and Tanaka, Kentaro and Magnusson, Fay and Chung, Yeonseok and Martinez, Gustavo J and Wang, Yi-Hong and Nurieva, Roza I and Kurosaki, Tomohiro and Dong, Chen
The Journal of immunology (1950), ISSN 0022-1767, 12/2014, Volume 193, Issue 12, pp. 6152 - 6160
Life Sciences & Biomedicine | Immunology | Science & Technology | T-Lymphocyte Subsets - immunology | CCAAT-Enhancer-Binding Protein-alpha - metabolism | T-Lymphocyte Subsets - cytology | CCAAT-Enhancer-Binding Protein-alpha - deficiency | Gene Expression Profiling | Interferon-gamma - metabolism | Cell Differentiation - genetics | Immunity, Humoral | Immunoglobulin G - immunology | T-Lymphocytes, Helper-Inducer - immunology | T-Lymphocytes, Helper-Inducer - cytology | Transcription, Genetic | Interferon-gamma - genetics | Immunoglobulin Class Switching | B-Lymphocytes - metabolism | T-Lymphocytes, Helper-Inducer - metabolism | Gene Expression Regulation | T-Box Domain Proteins - metabolism | Mice, Knockout | Cell Differentiation - immunology | Animals | B-Lymphocytes - immunology | T-Lymphocyte Subsets - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice | Index Medicus | Abridged Index Medicus
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3. Full Text Wnt signaling stimulates osteoblastogenesis of mesenchymal precursors by suppressing CCAAT/enhancer-binding protein α and peroxisome proliferator-activated receptor γ
by Kang, Sona and Bennett, Christina N and Gerin, Isabelle and Rapp, Lauren A and Hankenson, Kurt D and MacDougald, Ormond A
The Journal of biological chemistry, ISSN 0021-9258, 05/2007, Volume 282, Issue 19, pp. 14515 - 14524
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Homeodomain Proteins - metabolism | Bone Marrow Cells - physiology | Adipocytes - cytology | PPAR gamma - metabolism | Wnt Proteins - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Embryo, Mammalian - metabolism | Adipogenesis - physiology | Mesenchymal Stromal Cells - cytology | Wnt Proteins - genetics | Mesenchymal Stromal Cells - physiology | Fibroblasts - metabolism | PPAR gamma - genetics | Signal Transduction | Bone Marrow Cells - cytology | Cells, Cultured | Transcription Factors - genetics | beta Catenin - metabolism | Homeodomain Proteins - genetics | beta Catenin - genetics | Transcription Factors - metabolism | Animals | Embryo, Mammalian - cytology | Adipocytes - metabolism | Sp7 Transcription Factor | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice | Stromal Cells - physiology | Core Binding Factor Alpha 1 Subunit - genetics | Osteogenesis | Stromal Cells - cytology | Index Medicus
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4. Full Text Dominant-negative mutations of CEBPA , encoding CCAAT/enhancer binding protein-α (C/EBPα), in acute myeloid leukemia
by Pabst, Thomas and Radomska, Hanna S and Tenen, Daniel G and Hiddemann, Wolfgang and Schnittger, Susanne and Narravula, Sailaja and Zhang, Pu and Mueller, Beatrice U and Behre, Gerhard
Nature genetics, ISSN 1061-4036, 03/2001, Volume 27, Issue 3, pp. 263 - 270
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Sequence Deletion | DNA, Neoplasm - metabolism | Leukemia, Myeloid, Acute - pathology | Humans | Leukemia, Myeloid, Acute - metabolism | Transcriptional Activation | DNA Primers - genetics | CCAAT-Enhancer-Binding Protein-alpha - chemistry | Cell Differentiation - genetics | CCAAT-Enhancer-Binding Protein-alpha - biosynthesis | Cell Nucleus - metabolism | Genes, Dominant | Base Sequence | CCAAT-Enhancer-Binding Protein-alpha - genetics | Granulocytes - pathology | Heterozygote | Mutation | In Vitro Techniques | Leukemia, Myeloid, Acute - genetics | Neutrophils - pathology | CCAAT/enhancer binding protein-^a | CEBPA gene | Index Medicus
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5. Full Text Epigenetic modification of CCAAT/enhancer binding protein α expression in acute myeloid leukemia
by Hackanson, Björn and Bennett, Kristi L and Brena, Romulo M and Jiang, Jinmai and Claus, Rainer and Chen, Shih-Shih and Blagitko-Dorfs, Nadya and Maharry, Katie and Whitman, Susan P and Schmittgen, Thomas D and Lübbert, Michael and Marcucci, Guido and Bloomfield, Clara D and Plass, Christoph
Cancer research (Chicago, Ill.), ISSN 0008-5472, 05/2008, Volume 68, Issue 9, pp. 3142 - 3151
Life Sciences & Biomedicine | Oncology | Science & Technology | Promoter Regions, Genetic | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Epigenesis, Genetic | Humans | Hematopoiesis - genetics | Gene Expression Regulation, Leukemic | RNA, Messenger - metabolism | DNA Methylation | RNA Interference | U937 Cells | K562 Cells | CCAAT-Enhancer-Binding Protein-alpha - genetics | HL-60 Cells | Cytogenetic Analysis | MicroRNAs - physiology | Tumor Cells, Cultured | Leukemia, Myeloid, Acute - classification | Cluster Analysis | Leukemia, Myeloid, Acute - genetics | Index Medicus
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6. Upregulation of long non-coding RNA urothelial carcinoma associated 1 by CCAAT/enhancer binding protein alpha contributes to bladder cancer cell growth and reduced apoptosis
by Xue, Mei and Li, Xu and Wu, Wenjing and Zhang, Shuwan and Wu, Shouzhen and Li, Zhengkun and Chen, Wei
Oncology reports, ISSN 1021-335X, 05/2014, Volume 31, Issue 5, pp. 1993 - 2000
Life Sciences & Biomedicine | Oncology | Science & Technology | Promoter Regions, Genetic | Up-Regulation | Cell Proliferation - genetics | Protein Binding - genetics | Transcriptional Activation - genetics | Humans | Cell Survival - genetics | Apoptosis - genetics | RNA, Long Noncoding - genetics | Binding Sites - genetics | Point Mutation | RNA Interference | RNA, Long Noncoding - biosynthesis | Urinary Bladder Neoplasms - pathology | Biomarkers | CCAAT-Enhancer-Binding Protein-alpha - genetics | Cell Line, Tumor | Transcription, Genetic | Biomarkers, Tumor - genetics | Electrophoretic Mobility Shift Assay | RNA, Small Interfering
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7. Full Text Functional interaction of CCAAT/enhancer-binding-protein-α basic region mutants with E2F transcription factors and DNA
by Kowenz-Leutz, Elisabeth and Schuetz, Anja and Liu, Qingbin and Knoblich, Maria and Heinemann, Udo and Leutz, Achim
Biochimica et biophysica acta. Gene regulatory mechanisms, ISSN 1874-9399, 07/2016, Volume 1859, Issue 7, pp. 841 - 847
Cell proliferation | E2F transcription factor | CCAAT-enhancer-binding protein (C/EBP) | Differentiation | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | CCAAT-Enhancer-Binding Proteins - metabolism | Amino Acid Sequence | Humans | Cells, Cultured | Mutant Proteins - genetics | Models, Molecular | Molecular Sequence Data | Mutant Proteins - metabolism | DNA - metabolism | E2F Transcription Factors - metabolism | Protein Structure, Secondary - genetics | Sequence Homology, Amino Acid | Animals | Protein Interaction Domains and Motifs - genetics | Mutant Proteins - chemistry | HEK293 Cells | CCAAT-Enhancer-Binding Proteins - chemistry | Mice | CCAAT-Enhancer-Binding Proteins - genetics | Protein Binding - physiology
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8. Full Text CCAAT/Enhancer-Binding Protein α Is Epigenetically Silenced by Histone Deacetylation in Endometriosis and Promotes the Pathogenesis of Endometriosis
by Kawano, Yukie and Nasu, Kaei and Hijiya, Naoki and Tsukamoto, Yoshiyuki and Amada, Kohei and Abe, Wakana and Kai, Kentaro and Moriyama, Masatsugu and Narahara, Hisashi
The journal of clinical endocrinology and metabolism, ISSN 0021-972X, 09/2013, Volume 98, Issue 9, pp. E1474 - E1482
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Ovarian Diseases - metabolism | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Endometriosis - genetics | Apoptosis - drug effects | Epigenesis, Genetic | Humans | PPAR gamma - metabolism | Valproic Acid - pharmacology | Caspases - metabolism | Period Circadian Proteins - genetics | Stromal Cells - drug effects | Adult | Female | Acetylation | bcl-2-Associated X Protein - genetics | Apoptosis Inducing Factor - metabolism | PPAR gamma - genetics | Ovarian Diseases - genetics | Caspases - genetics | Stromal Cells - metabolism | Gene Silencing | bcl-2-Associated X Protein - metabolism | Apoptosis Inducing Factor - genetics | Histones - genetics | Period Circadian Proteins - metabolism | Endometriosis - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Apoptosis - physiology | Histones - metabolism | Index Medicus | Abridged Index Medicus
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9. Full Text CCAAT/enhancer-binding protein alpha (CEBPA) gene haploinsufficiency does not alter hematopoiesis or induce leukemia in lck-CALM/AF10 transgenic mice
by Lange, A.P and Almeida, L.Y and Silva, C. L. Araújo and Scheucher, P.S and Chahud, F and Krause, A and Bohlander, S.K and Rego, E.M
Brazilian journal of medical and biological research, ISSN 0100-879X, 2019, Volume 52, Issue 6, pp. e8424 - e8424
CEBPA | CALM/AF10 | Leukemia | Transgenic mice | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Prognosis | Genetic aspects | Genetic engineering | T cells | Gene expression | Hematopoietic stem cells | Cancer | Protein binding | Stem cell transplantation | Lymphocytes T | CCAAT/enhancer-binding protein | CEBPA gene | Macrophages | T-cell receptor | Mac1 protein | Rodents | Bone marrow | Fusion protein | Phenotypes | Acute lymphatic leukemia | Myeloid leukemia | T cell receptors | Lymphatic leukemia | c-Kit protein | Progenitor cells | Hemopoiesis | Haploinsufficiency | B220 antigen | Lck protein | Leukemogenesis | Leukocytes (granulocytic) | Acute myeloid leukemia | Index Medicus
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10. Full Text Hypoxia down-regulates CCAAT/enhancer binding protein-α expression in breast cancer cells
by Seifeddine, Ramzi and Dreiem, Anne and Blanc, Etienne and Fulchignoni-Lataud, Marie-Claude and Belda, Marie-Aude Le Frère and Lecuru, Fabrice and Mayi, Thérèse Hervèe and Mazure, Nathalie and Favaudon, Vincent and Massaad, Charbel and Barouki, Robert and Massaad-Massade, Liliane
Cancer research (Chicago, Ill.), ISSN 0008-5472, 04/2008, Volume 68, Issue 7, pp. 2158 - 2165
Life Sciences & Biomedicine | Oncology | Science & Technology | Gynecology. Andrology. Obstetrics | Mammary gland diseases | Pharmacology. Drug treatments | Biological and medical sciences | Medical sciences | Antineoplastic agents | Tumors | Immunohistochemistry | RNA, Small Interfering - genetics | Transcription, Genetic - drug effects | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Humans | Molecular Sequence Data | Cell Growth Processes - physiology | Promoter Regions, Genetic - drug effects | Breast Neoplasms - metabolism | RNA, Messenger - biosynthesis | CCAAT-Enhancer-Binding Protein-alpha - biosynthesis | Transfection | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Base Sequence | Electrophoretic Mobility Shift Assay | Deferoxamine - pharmacology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Down-Regulation | RNA, Messenger - genetics | Reverse Transcriptase Polymerase Chain Reaction | Subcellular Fractions - metabolism | Blotting, Western | Breast Neoplasms - genetics | Breast Neoplasms - pathology | CCAAT-Enhancer-Binding Protein-alpha - genetics | Cell Cycle - physiology | Cell Line, Tumor | Cell Hypoxia - genetics | Index Medicus | Promoter Regions (Genetics) | Biochemistry, Molecular Biology | Breast Neoplasms | Hypoxia-Inducible Factor 1, alpha Subunit | Cell Growth Processes | Deferoxamine | Cell Hypoxia | Life Sciences | CCAAT-Enhancer-Binding Protein-alpha | Cell Cycle | Transcription, Genetic | RNA, Small Interfering | Subcellular Fractions | RNA, Messenger
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11. Full Text The CCAAT Enhancer-binding Protein α (C/EBPα) Requires a SWI/SNF Complex for Proliferation Arrest
by Müller, Christine and Calkhoven, Cornelis F and Sha, Xiaojing and Leutz, Achim
The Journal of biological chemistry, ISSN 0021-9258, 02/2004, Volume 279, Issue 8, pp. 7353 - 7358
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Protein Structure, Tertiary | Cell Line | NIH 3T3 Cells | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Rats | Plasmids - metabolism | Blotting, Western | Animals | Transfection | Cell Cycle | RNA Interference | Retroviridae - genetics | Time Factors | CCAAT-Enhancer-Binding Protein-alpha - physiology | Cell Division | Protein Isoforms | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice | Mice, Inbred BALB C | Models, Genetic | Genetic Vectors | Genes, Reporter | Chromatin - chemistry | Fibroblasts - metabolism | C/EBP^a protein | SWI/SNF complex | Index Medicus
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12. Full Text Yin Yang-1 suppresses differentiation of hepatocellular carcinoma cells through the downregulation of CCAAT/enhancer-binding protein alpha
by Zhang, Shengjie and Jiang, Tingting and Feng, Lifeng and Sun, Jie and Lu, Haiqi and Wang, Qinchuan and Pan, Min and Huang, Dongsheng and Wang, Xian and Wang, Linbo and Jin, Hongchuan
Journal of molecular medicine (Berlin, Germany), ISSN 0946-2716, 9/2012, Volume 90, Issue 9, pp. 1069 - 1077
Human Genetics | Biomedicine | Oncogene | Internal Medicine | Molecular Medicine | Carcinogenesis | Cancer | Genetics & Heredity | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Gastroenterology. Liver. Pancreas. Abdomen | Biological and medical sciences | General aspects | Medical sciences | Liver. Biliary tract. Portal circulation. Exocrine pancreas | Tumors | CCAAT-Enhancer-Binding Proteins - metabolism | Cell Line | Liver Neoplasms - genetics | Down-Regulation | Humans | Gene Expression Regulation, Neoplastic | YY1 Transcription Factor - genetics | YY1 Transcription Factor - metabolism | DNA Methylation | Carcinoma, Hepatocellular - genetics | Gene Deletion | Liver Neoplasms - metabolism | Cell Line, Tumor | Cell Differentiation | Liver - cytology | CCAAT-Enhancer-Binding Proteins - genetics | Carcinoma, Hepatocellular - metabolism | Index Medicus
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