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ccaat-enhancer-binding protein-beta - genetics (148) 148
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1. Full Text Wnt signaling stimulates osteoblastogenesis of mesenchymal precursors by suppressing CCAAT/enhancer-binding protein α and peroxisome proliferator-activated receptor γ
by Kang, Sona and Bennett, Christina N and Gerin, Isabelle and Rapp, Lauren A and Hankenson, Kurt D and MacDougald, Ormond A
Journal of Biological Chemistry, ISSN 0021-9258, 05/2007, Volume 282, Issue 19, pp. 14515 - 14524
Mesenchymal precursor cells have the potential to differentiate into several cell types, including adipocytes and osteoblasts. Activation of Wnt/beta-catenin... 
GLYCOGEN-SYNTHASE KINASE-3-BETA | IN-VITRO | HIGH-BONE-MASS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | PROMOTES OSTEOGENESIS | PPAR-GAMMA | BETA-CATENIN | TRANSCRIPTION FACTOR | BROWN ADIPOCYTE DIFFERENTIATION | 3T3-L1 CELLS | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Homeodomain Proteins - metabolism | Bone Marrow Cells - physiology | Adipocytes - cytology | PPAR gamma - metabolism | Wnt Proteins - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Embryo, Mammalian - metabolism | Adipogenesis - physiology | Mesenchymal Stromal Cells - cytology | Wnt Proteins - genetics | Mesenchymal Stromal Cells - physiology | Fibroblasts - metabolism | PPAR gamma - genetics | Signal Transduction | Bone Marrow Cells - cytology | Cells, Cultured | Transcription Factors - genetics | beta Catenin - metabolism | Homeodomain Proteins - genetics | beta Catenin - genetics | Transcription Factors - metabolism | Animals | Embryo, Mammalian - cytology | Adipocytes - metabolism | Sp7 Transcription Factor | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice | Stromal Cells - physiology | Core Binding Factor Alpha 1 Subunit - genetics | Osteogenesis | Stromal Cells - cytology | Index Medicus
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2. Full Text SIRT1 regulates adiponectin gene expression through Foxo1-C/enhancer- binding protein α transcriptional complex
by Qiao, Liping and Shao, Jianhua
Journal of Biological Chemistry, ISSN 0021-9258, 12/2006, Volume 281, Issue 52, pp. 39915 - 39924
Adiponectin is an adipose-derived hormone that plays an important role in maintaining energy homeostasis. Adiponectin gene expression is diminished in both... 
INSULIN | METABOLISM | FATTY-ACID OXIDATION | PHOSPHORYLATION | ACETYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | BINDING-PROTEIN | FACTOR FOXO1 | ADIPOCYTE DIFFERENTIATION | PROMOTER | ADIPOSE-TISSUE | Adiponectin - biosynthesis | Humans | Molecular Sequence Data | Adipocytes - cytology | CCAAT-Enhancer-Binding Protein-alpha - deficiency | Male | Forkhead Transcription Factors - physiology | Sirtuins - deficiency | Cell Differentiation - genetics | CCAAT-Enhancer-Binding Protein-alpha - physiology | Forkhead Transcription Factors - metabolism | Sirtuin 1 | Adiponectin - genetics | Base Sequence | Up-Regulation - physiology | Sirtuins - genetics | Cell Differentiation - physiology | Forkhead Transcription Factors - deficiency | Cell Line | Promoter Regions, Genetic | Mice, Inbred C57BL | Sirtuins - physiology | Up-Regulation - genetics | Binding Sites - genetics | 3T3-L1 Cells | Forkhead Transcription Factors - genetics | Transcription, Genetic - physiology | Animals | Adipocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice, Obese | Forkhead Box Protein O1 | Mice | Index Medicus
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3. Full Text Epigenetic modification of CCAAT/enhancer binding protein α expression in acute myeloid leukemia
by Hackanson, Björn and Bennett, Kristi L and Brena, Romulo M and Jiang, Jinmai and Claus, Rainer and Chen, Shih-Shih and Blagitko-Dorfs, Nadya and Maharry, Katie and Whitman, Susan P and Schmittgen, Thomas D and Lübbert, Michael and Marcucci, Guido and Bloomfield, Clara D and Plass, Christoph
Cancer Research, ISSN 0008-5472, 05/2008, Volume 68, Issue 9, pp. 3142 - 3151
Functional loss of CCAAT/enhancer binding protein alpha (C/ EBP alpha), a master regulatory transcription factor in the hematopoietic system, can result in a... 
DOWN-MODULATION | C/EBP-ALPHA | MICRORNA GENE | HYPERMETHYLATION | ONCOLOGY | GENE-EXPRESSION | TUMOR-SUPPRESSOR | VALPROIC ACID | HUMAN CANCER-CELLS | TRANSCRIPTION FACTOR | DNA-METHYLATION | Promoter Regions, Genetic | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Epigenesis, Genetic | Humans | Hematopoiesis - genetics | Gene Expression Regulation, Leukemic | RNA, Messenger - metabolism | DNA Methylation | RNA Interference | U937 Cells | K562 Cells | CCAAT-Enhancer-Binding Protein-alpha - genetics | HL-60 Cells | Cytogenetic Analysis | MicroRNAs - physiology | Tumor Cells, Cultured | Leukemia, Myeloid, Acute - classification | Cluster Analysis | Leukemia, Myeloid, Acute - genetics | Index Medicus
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4. Full Text Mutations and Treatment Outcome in Cytogenetically Normal Acute Myeloid Leukemia
by Schlenk, Richard F and Döhner, Konstanze and Döhner, Hartmut and Krauter, Jürgen and Fröhling, Stefan and Corbacioglu, Andrea and Bullinger, Lars and Habdank, Marianne and Späth, Daniela and Morgan, Michael and Benner, Axel and Schlegelberger, Brigitte and Heil, Gerhard and Ganser, Arnold and German Austrian Acute Myeloid and German-Austrian Acute Myeloid Leukemia Study Group
The New England Journal of Medicine, ISSN 0028-4793, 05/2008, Volume 358, Issue 18, pp. 1909 - 1918
Almost half of patients with acute myeloid leukemia (AML) do not have detectable cytogenetic abnormalities. This study of more than 870 such patients... 
INTERNAL TANDEM DUPLICATION | STUDY-GROUP ULM | MEDICINE, GENERAL & INTERNAL | NORMAL KARYOTYPE | FAVORABLE PROGNOSTIC-SIGNIFICANCE | KINASE DOMAIN MUTATIONS | ACUTE MYELOGENOUS LEUKEMIA | YOUNGER ADULTS | NUCLEOPHOSMIN NPM1 | CEBPA MUTATIONS | GROUP-B | Humans | Middle Aged | Kaplan-Meier Estimate | Genotype | Male | Treatment Outcome | Genetic Markers | Remission Induction | Leukemia, Myeloid, Acute - mortality | Stem Cell Transplantation | fms-Like Tyrosine Kinase 3 - genetics | Myeloid-Lymphoid Leukemia Protein - genetics | Adolescent | CCAAT-Enhancer-Binding Protein-alpha - genetics | Adult | Female | Oncogene Proteins, Viral - genetics | Mutation | Nuclear Proteins - genetics | Leukemia, Myeloid, Acute - therapy | Leukemia, Myeloid, Acute - genetics | Care and treatment | Genetic aspects | Nonlymphoid leukemia | Gene mutations | Myelocytic leukemia | Health aspects | Bone marrow | Consolidation | Transplants & implants | Kinases | Gene expression | Leukemia | Index Medicus | Abridged Index Medicus
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5. Full Text Upregulation of long non-coding RNA urothelial carcinoma associated 1 by CCAAT/enhancer binding protein α contributes to bladder cancer cell growth and reduced apoptosis
by XUE, MEI and LI, XU and WU, WENJING and ZHANG, SHUWAN and WU, SHOUZHEN and LI, ZHENGKUN and CHEN, WEI
Oncology Reports, ISSN 1021-335X, 5/2014, Volume 31, Issue 5, pp. 1993 - 2000
Long non-coding RNA urothelial carcinoma associated 1 (lncRNA-UCA1) is upregulated in bladder cancer and plays a pivotal role in bladder cancer progression and... 
transcription factor | urothelial carcinoma associated 1 | long non-coding RNA | CCAAT/enhancer binding protein α | bladder cancer | CREB | DATABASE | ONCOLOGY | HULC | GENES | UCA1 | CCAAT/enhancer binding protein alpha | Promoter Regions, Genetic | Up-Regulation | Cell Proliferation - genetics | Protein Binding - genetics | Transcriptional Activation - genetics | Humans | Cell Survival - genetics | Apoptosis - genetics | RNA, Long Noncoding - genetics | Binding Sites - genetics | Point Mutation | RNA Interference | RNA, Long Noncoding - biosynthesis | Urinary Bladder Neoplasms - pathology | Biomarkers | CCAAT-Enhancer-Binding Protein-alpha - genetics | Cell Line, Tumor | Transcription, Genetic | Biomarkers, Tumor - genetics | Electrophoretic Mobility Shift Assay | RNA, Small Interfering | Transcription factors | Genes | Metastasis | Drug resistance | Bladder cancer | Studies | Proteins | Cell growth | Mutagenesis | Cell cycle | Software | Binding sites | Deoxyribonucleic acid--DNA | Apoptosis | Index Medicus | enhancer binding protein α | CCAAT
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6. Full Text MicroRNA expression profiling in relation to the genetic heterogeneity of acute myeloid leukemia
by Jongen-Lavrencic, Mojca and Sun, S.M and Dijkstra, M.K and Valk, Peter and Löwenberg, Bob
Blood, ISSN 0006-4971, 05/2008, Volume 111, Issue 10, pp. 5078 - 5085
textabstractAcute myeloid leukemia (AML) is a highly diverse disease characterized by various cytogenetic and molecular abnormalities. MicroRNAs are small... 
REAL-TIME PCR | COLONY-STIMULATING FACTOR | HUMAN LUNG CANCERS | MESSENGER-RNA | FAVORABLE PROGNOSTIC-SIGNIFICANCE | HEMATOPOIETIC STEM-CELLS | TUMOR-SUPPRESSOR GENE | NORMAL CYTOGENETICS | YOUNGER ADULTS | NUCLEOPHOSMIN NPMC(+) AML | HEMATOLOGY | Blast Crisis - pathology | Humans | Gene Expression Profiling - methods | Gene Expression Regulation, Leukemic | Genetic Heterogeneity | Mutant Proteins | fms-Like Tyrosine Kinase 3 - genetics | Leukemia, Myeloid, Acute - diagnosis | CCAAT-Enhancer-Binding Protein-alpha - genetics | MicroRNAs - genetics | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | Leukemia, Myeloid, Acute - classification | Leukemia, Myeloid, Acute - genetics | Index Medicus | Abridged Index Medicus
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7. Full Text C/EBPα poises B cells for rapid reprogramming into induced pluripotent stem cells
by Di Stefano, Bruno and Sardina, Jose Luis and Van Oevelen, Chris and Collombet, Samuel and Kallin, Eric M and Vicent, Guillermo P and Lu, Jun and Thieffry, Denis and Beato, Miguel and Graf, Thomas
Nature, ISSN 0028-0836, 2014, Volume 506, Issue 7487, pp. 235 - 239
CCAAT/enhancer binding protein-alpha (C/EBP alpha) induces transdifferentiation of B cells into macrophages at high efficiencies and enhances reprogramming... 
NETWORK | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | TET2 | TRANSDIFFERENTIATION | REQUIREMENT | REVEALS | Chromatin - metabolism | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Epithelial-Mesenchymal Transition - genetics | SOXB1 Transcription Factors - metabolism | DNA-Binding Proteins - metabolism | Octamer Transcription Factor-3 - genetics | DNA Methylation | SOXB1 Transcription Factors - genetics | Cytosine - metabolism | Kruppel-Like Transcription Factors - metabolism | Cell Transdifferentiation | Induced Pluripotent Stem Cells - cytology | Cellular Reprogramming - genetics | B-Lymphocytes - metabolism | Induced Pluripotent Stem Cells - metabolism | Proto-Oncogene Proteins - metabolism | B-Lymphocytes - cytology | Cells, Cultured | Proto-Oncogene Proteins - genetics | Up-Regulation - genetics | DNA-Binding Proteins - genetics | Proto-Oncogene Proteins c-myc - metabolism | Deoxyribonuclease I - metabolism | Animals | Octamer Transcription Factor-3 - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice | Proto-Oncogene Proteins c-myc - genetics | Chromatin - genetics | Kruppel-Like Transcription Factors - genetics | Index Medicus
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8. Full Text CCAAT/enhancer binding protein α (C/EBPα)-induced transdifferentiation of pre-B cells into macrophages involves no overt retrodifferentiation
by Alessandro Di Tullio and Thien Phong Vu Manh and Alexis Schubert and Robert Månsson and Thomas Graf and Stem Cell Center and Lund University and Stamcellscentrum (SCC) and Lunds universitet
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2011, Volume 108, Issue 41, pp. 17016 - 17021
Earlier work has shown that pre-B cells can be converted into macrophages by the transcription factor CCAAT/enhancer binding protein α at very high... 
T lymphocytes | B lymphocytes | Epithelial cells | Genes | Stromal cells | Stem cells | Cell lines | Macrophages | Progenitor cells | Hematopoietic stem cells | Cell reprogramming | Hematopoietic differentiation lineage commitment | Cell fate decision | PROGENITOR CELLS | STEM-CELLS | DEFINED FACTORS | MULTIDISCIPLINARY SCIENCES | NEUTROPHILS | PLURIPOTENCY | FIBROBLASTS | cell reprogramming | C/EBP-ALPHA | cell fate decision | IN-VIVO | DIFFERENTIATION | hematopoietic differentiation lineage commitment | HEMATOPOIETIC STEM | Precursor Cells, B-Lymphoid - metabolism | RNA, Messenger - genetics | Cells, Cultured | Transcriptome | Cell Transdifferentiation - genetics | Macrophages - cytology | RNA, Messenger - metabolism | fms-Like Tyrosine Kinase 3 - genetics | Macrophages - metabolism | Animals | Cell Transdifferentiation - physiology | T-Lymphocytes - cytology | T-Lymphocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - physiology | Precursor Cells, B-Lymphoid - cytology | Erythrocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Erythrocytes - cytology | Proto-Oncogene Proteins c-kit - genetics | Mice | Megakaryocytes - cytology | Megakaryocytes - metabolism | Cell Lineage - genetics | Genes, cdc | Index Medicus | Biological Sciences | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | hematopoietic differentiation | Medicinska och farmaceutiska grundvetenskaper | Cell and Molecular Biology | lineage commitment | Cell- och molekylärbiologi
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9. Full Text Extensive chromatin remodelling and establishment of transcription factor 'hotspots' during early adipogenesis
by Sung, Myong-Hee and Loft, Anne and Baek, Songjoon and Mandrup, Susanne and Nielsen, Ronni and Hager, Gordon L and Siersbæk, Rasmus and John, Sam
The EMBO Journal, ISSN 0261-4189, 04/2011, Volume 30, Issue 8, pp. 1459 - 1472
Adipogenesis is tightly controlled by a complex network of transcription factors acting at different stages of differentiation. Peroxisome... 
C/EBPβ | adipocyte differentiation | DNase I hypersensitivity | chromatin remodelling | ChIP‐seq | ChIP-seq | C/EBP beta | C/EBP-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | BINDING PROTEIN-BETA | HUMAN GENOME | GLUCOCORTICOID-RECEPTOR | IN-VIVO | ACTIVATED-RECEPTOR-GAMMA | PPAR-GAMMA | STIMULATES ADIPOGENESIS | ADIPOSE-TISSUE | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Adipocytes - cytology | Adipogenesis - physiology | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Chromatin Immunoprecipitation | Tumor Suppressor Proteins - genetics | Transcription, Genetic | Cell Differentiation | Retinoid X Receptors - genetics | Retinoid X Receptors - metabolism | Tumor Suppressor Proteins - metabolism | CCAAT-Enhancer-Binding Protein-beta - genetics | RNA, Messenger - genetics | Chromatin Assembly and Disassembly | Transcription Factors - genetics | 3T3-L1 Cells | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | CCAAT-Enhancer-Binding Protein-beta - metabolism | Transcription Factors - metabolism | Algorithms | Animals | Adipocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice | Chromatin | Genes | Adipocytes | Index Medicus | EBPβ
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10. Full Text Dominant-negative mutations of CEBPA , encoding CCAAT/enhancer binding protein-α (C/EBPα), in acute myeloid leukemia
by Pabst, Thomas and Radomska, Hanna S and Tenen, Daniel G and Hiddemann, Wolfgang and Schnittger, Susanne and Narravula, Sailaja and Zhang, Pu and Mueller, Beatrice U and Behre, Gerhard
Nature Genetics, ISSN 1061-4036, 03/2001, Volume 27, Issue 3, pp. 263 - 270
The transcription factor C/EBP alpha (for CCAAT/enhancer binding protein-alpha; encoded by the gene CEBPA) is crucial for the differentiation of granulocytes.... 
ACTIVATION | MESSENGER-RNA | GENE | TRANSCRIPTION | GENETICS & HEREDITY | CELL-PROLIFERATION | DEFICIENT MICE | DIFFERENTIATION | TRANSLATION | EXPRESSION | EPSILON | Sequence Deletion | DNA, Neoplasm - metabolism | Leukemia, Myeloid, Acute - pathology | Humans | Leukemia, Myeloid, Acute - metabolism | Transcriptional Activation | DNA Primers - genetics | CCAAT-Enhancer-Binding Protein-alpha - chemistry | Cell Differentiation - genetics | CCAAT-Enhancer-Binding Protein-alpha - biosynthesis | Cell Nucleus - metabolism | Genes, Dominant | Base Sequence | CCAAT-Enhancer-Binding Protein-alpha - genetics | Granulocytes - pathology | Heterozygote | Mutation | In Vitro Techniques | Leukemia, Myeloid, Acute - genetics | Neutrophils - pathology | CCAAT/enhancer binding protein-^a | CEBPA gene | Index Medicus
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11. Full Text Prognostic impact, concurrent genetic mutations, and gene expression features of AML with CEBPA mutations in a cohort of 1182 cytogenetically normal AML patients: Further evidence for CEBPA double mutant AML as a distinctive disease entity
by Taskesen, Erdogan and Bullinger, Lars and Corbacioglu, Andrea and Sanders, Mathijs A and Erpelinck, Claudia A. J and Wouters, Bas J and Van Der Poel-van De Luytgaarde, Sonja C and Damm, Frederik and Krauter, Jürgen and Ganser, Arnold and Schlenk, Richard F and Löwenberg, Bob and Delwel, Ruud and Döhner, Hartmut and Valk, Peter J. M and Döhner, Konstanze
Blood, ISSN 0006-4971, 02/2011, Volume 117, Issue 8, pp. 2469 - 2475
We evaluated concurrent gene mutations, clinical outcome, and gene expression signatures of CCAAT/enhancer binding protein alpha (CEBPA) double mutations... 
BINDING-PROTEIN-ALPHA | TRIAL | C/EBP-ALPHA | NORMAL KARYOTYPE | COLONY-STIMULATING FACTOR | FAVORABLE PROGNOSIS | CONSOLIDATION THERAPY | YOUNGER ADULTS | ACUTE MYELOID-LEUKEMIA | HEMATOLOGY | MYELODYSPLASTIC SYNDROMES | Molecular Diagnostic Techniques - methods | Prognosis | Humans | Middle Aged | Gene Expression Profiling - methods | Treatment Outcome | Young Adult | Mutation - physiology | Leukemia, Myeloid, Acute - diagnosis | Sensitivity and Specificity | Adolescent | Classification | Survival Analysis | CCAAT-Enhancer-Binding Protein-alpha - genetics | Adult | Molecular Diagnostic Techniques - standards | Leukemia, Myeloid, Acute - classification | Cohort Studies | Leukemia, Myeloid, Acute - genetics | Index Medicus | Abridged Index Medicus
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12. Full Text TRIB2 Acts Downstream of Wnt/TCF in Liver Cancer Cells to Regulate YAP and C/EBPα Function
by Wang, Jiayi and Park, Joo-Seop and Wei, Yingying and Rajurkar, Mihir and Cotton, Jennifer L and Fan, Qishi and Lewis, Brian C and Ji, Hongkai and Mao, Junhao
Molecular Cell, ISSN 1097-2765, 07/2013, Volume 51, Issue 2, pp. 211 - 225
Dysregulation of Wnt signaling is closely associated with human liver tumorigenesis. However, liver cancer-specific Wnt transcriptional programs and downstream... 
BETA-CATENIN GENE | TRANSCRIPTION FACTORS | ORGAN SIZE CONTROL | WNT/BETA-CATENIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | HIPPO PATHWAY | PHENOTYPE | PROLIFERATION | HEPATOCELLULAR CARCINOMAS | MYC DELETION | CELL BIOLOGY | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Cell Proliferation | Immunoprecipitation | Oligonucleotide Array Sequence Analysis | Humans | Gene Expression Profiling | Intracellular Signaling Peptides and Proteins - metabolism | Wnt Proteins - metabolism | Immunoenzyme Techniques | Wnt Proteins - genetics | Carcinoma, Hepatocellular - genetics | Biomarkers, Tumor - metabolism | Liver Neoplasms - pathology | Cell Differentiation | Nuclear Proteins - genetics | T Cell Transcription Factor 1 - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Real-Time Polymerase Chain Reaction | Liver Neoplasms - genetics | RNA, Messenger - genetics | Nuclear Proteins - metabolism | Transcription Factors - genetics | Calcium-Calmodulin-Dependent Protein Kinases | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | Blotting, Western | beta Catenin - genetics | T Cell Transcription Factor 1 - genetics | Transcription Factors - metabolism | Carcinoma, Hepatocellular - pathology | Fluorescent Antibody Technique | Liver Neoplasms - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Biomarkers, Tumor - genetics | Carcinoma, Hepatocellular - metabolism | Index Medicus
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13. Full Text Cell-cycle regulator E2F1 and microRNA-223 comprise an autoregulatory negative feedback loop in acute myeloid leukemia
by Pulikkan, John A and Dengler, Viola and Peramangalam, Philomina S and Peer Zada, Abdul A and Müller-Tidow, Carsten and Bohlander, Stefan K and Tenen, Daniel G and Behre, Gerhard
Blood, ISSN 0006-4971, 03/2010, Volume 115, Issue 9, pp. 1768 - 1778
Transcription factor CCAAT enhancer binding protein alpha (C/EBP alpha) is essential for granulopoiesis and its function is deregulated in leukemia. Inhibition... 
TRANSCRIPTION FACTORS | C/EBP-ALPHA | DIRECT REPRESSION | PLASMINOGEN-ACTIVATOR | GENE | ENHANCER-BINDING-PROTEIN | IN-VIVO | C-MYC | DIFFERENTIATION | HEMATOLOGY | EXPRESSION | Leukopoiesis - genetics | Up-Regulation | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | MicroRNAs - metabolism | DNA Primers - genetics | Base Sequence | Granulocytes - pathology | Granulocytes - metabolism | Cell Line | Leukopoiesis - physiology | Leukemia, Myeloid, Acute - pathology | Down-Regulation | E2F1 Transcription Factor - metabolism | Mice, Knockout | Feedback, Physiological | Animals | Cell Cycle | Models, Biological | K562 Cells | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice | MicroRNAs - genetics | Mutation | E2F1 Transcription Factor - genetics | Leukemia, Myeloid, Acute - genetics | Index Medicus | Abridged Index Medicus | Myeloid Neoplasia
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