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1. Full Text CCAAT/enhancer binding protein beta (C/EBPβ) isoform balance as a regulator of epithelial-mesenchymal transition in mouse mammary epithelial cells
by Miura, Yuka and Hagiwara, Natsumi and Radisky, Derek C and Hirai, Yohei
Experimental cell research, ISSN 0014-4827, 09/2014, Volume 327, Issue 1, pp. 146 - 155
C/EBPβ | Mammary epithelia | Epimorphin | EMT | MET | Basement membrane | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Epithelial Cells - metabolism | Membrane Glycoproteins - metabolism | Gene Expression - genetics | Epithelial-Mesenchymal Transition - physiology | Epithelial-Mesenchymal Transition - genetics | Protein Isoforms - metabolism | Female | Collagen - genetics | Cell Line | CCAAT-Enhancer-Binding Protein-beta - genetics | Proteoglycans - metabolism | Basement Membrane - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Membrane Glycoproteins - genetics | Laminin - genetics | Collagen - metabolism | Animals | Transforming Growth Factor beta - genetics | Mammary Glands, Animal - metabolism | Mice | Laminin - metabolism | Transforming Growth Factor beta - metabolism | Drug Combinations | Proteoglycans - genetics | Protein Isoforms - genetics | Liver cancer | Gene expression | Intermediate filament proteins | Stem cells | Protein binding | Morphogenesis | Protein expression | Metastasis | Cellular biology | Binding sites | Cell adhesion & migration | Index Medicus | MORPHOGENESIS | TISSUE CULTURES | KERATIN | TETRACYCLINES | GENES | LIVER | METASTASES | CELL PROLIFERATION | MICE | 60 APPLIED LIFE SCIENCES | ADENOMAS
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2. Full Text CCAAT/enhancer-binding protein beta (C/EBP beta) knockdown reduces inflammation, ER stress, and apoptosis, and promotes autophagy in oxLDL-treated RAW264.7 macrophage cells
by Zahid, Md Khurshidul and Rogowski, Michael and Ponce, Christopher and Choudhury, Mahua and Moustaid-Moussa, Naima and Rahman, Shaikh M
Molecular and cellular biochemistry, ISSN 0300-8177, 01/2020, Volume 463, Issue 1-2, pp. 211 - 223
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Inflammation - pathology | CCAAT-Enhancer-Binding Protein-beta - genetics | Gene Expression Regulation | Lipoproteins, LDL - genetics | CCAAT-Enhancer-Binding Protein-beta - metabolism | Gene Knockdown Techniques | Inflammation - metabolism | Animals | Endoplasmic Reticulum Stress | Inflammation - genetics | RAW 264.7 Cells | Lipoproteins, LDL - metabolism | Mice | Foam Cells - pathology | Foam Cells - metabolism | Apoptosis | Type 2 diabetes | Proteins | Genes | Atherosclerosis | Inflammation | Macrophages | Gene expression | Cholesterol | Protein binding | TOR protein | Atherogenesis | Phosphorylation | Deregulation | Lipids | CCAAT/enhancer-binding protein | Kinases | Inactivation | Autophagy | Caspase-1 | Lipid metabolism | Efflux | NF-κB protein | Deactivation | Rapamycin | siRNA | Tumor necrosis factor-α | Metabolism | Stress | Arteriosclerosis | Endoplasmic reticulum | Phagocytosis | Index Medicus
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3. Full Text CCAAT/enhancer-binding protein β (C/EBPβ) expression regulates dietary-induced inflammation in macrophages and adipose tissue in mice
by Rahman, Shaikh M and Janssen, Rachel C and Choudhury, Mahua and Baquero, Karalee C and Aikens, Rebecca M and De La Houssaye, Becky A and Friedman, Jacob E
The Journal of biological chemistry, ISSN 0021-9258, 10/2012, Volume 287, Issue 41, pp. 34349 - 34360
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | CCAAT-Enhancer-Binding Protein-beta - biosynthesis | Inflammation - pathology | Inflammasomes - metabolism | Macrophage Activation - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Obesity - genetics | Transcription Factor RelA - genetics | Inflammation - metabolism | MAP Kinase Kinase 4 - metabolism | Mitochondria - genetics | Dietary Fats - pharmacology | Interleukin-10 - metabolism | Antigens, Differentiation - metabolism | Dietary Fats - adverse effects | Obesity - chemically induced | Macrophages - pathology | CCAAT-Enhancer-Binding Protein-beta - genetics | Mitochondria - metabolism | Mitochondria - pathology | 3T3-L1 Cells | Enzyme Activation - drug effects | Adipose Tissue, Brown - pathology | Inflammasomes - genetics | Mice, Knockout | Obesity - metabolism | Obesity - pathology | Gene Expression Regulation - drug effects | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Transcription Factor RelA - metabolism | Interleukin-10 - genetics | Inflammation - genetics | MAP Kinase Kinase 4 - genetics | Adipose Tissue, Brown - metabolism | Antigens, Differentiation - genetics | Mice | Enzyme Activation - genetics | Macrophage Activation - drug effects | Index Medicus | Obesity | Lipid Metabolism | Transcription Factors | Metabolism | EBP Transcription Factor | Cytokine
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4. Full Text Troxerutin counteracts domoic acid-induced memory deficits in mice by inhibiting CCAAT/enhancer binding protein β-mediated inflammatory response and oxidative stress
by Lu, Jun and Wu, Dong-Mei and Zheng, Yuan-Lin and Hu, Bin and Cheng, Wei and Zhang, Zi-Feng and Li, Meng-Qiu
The Journal of immunology (1950), ISSN 0022-1767, 04/2013, Volume 190, Issue 7, pp. 3466 - 3479
Life Sciences & Biomedicine | Immunology | Science & Technology | Reactive Oxygen Species - metabolism | Memory Disorders - genetics | Nitriles - pharmacology | Phosphoprotein Phosphatases - metabolism | Memory Disorders - metabolism | Male | Hippocampus - drug effects | Kainic Acid - adverse effects | Gene Knockdown Techniques | CDC2 Protein Kinase - metabolism | Inflammation - metabolism | Protein Kinase C - metabolism | Inflammation Mediators - metabolism | NADPH Oxidases - genetics | Phosphorylation - drug effects | Hydroxyethylrutoside - analogs & derivatives | Butadienes - pharmacology | Hydroxyethylrutoside - pharmacology | CCAAT-Enhancer-Binding Protein-beta - genetics | Mitochondria - metabolism | Mitochondria - drug effects | CCAAT-Enhancer-Binding Protein-beta - metabolism | Hippocampus - metabolism | Animals | Memory Disorders - drug therapy | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Hydroxyethylrutoside - administration & dosage | Mice | Oxidative Stress - drug effects | Genes, ras | Memory Disorders - chemically induced | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus | Abridged Index Medicus
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5. Full Text Promoter-distal RNA polymerase II binding discriminates active from inactive CCAAT/ enhancer-binding protein beta binding sites
by Savic, Daniel and Roberts, Brian S and Carleton, Julia B and Partridge, E. Christopher and White, Michæl A and Cohen, Barak A and Cooper, Gregory M and Gertz, Jason and Myers, Richard M
Genome research, ISSN 1088-9051, 12/2015, Volume 25, Issue 12, pp. 1791 - 1800
Biochemistry & Molecular Biology | Genetics & Heredity | Life Sciences & Biomedicine | Biotechnology & Applied Microbiology | Science & Technology | Promoter Regions, Genetic | Response Elements | Humans | Gene Expression Regulation | RNA Polymerase II - metabolism | Sequence Analysis, DNA | CCAAT-Enhancer-Binding Protein-beta - metabolism | Hep G2 Cells | Organ Specificity - genetics | Enhancer Elements, Genetic | K562 Cells | Protein Binding | Histones - metabolism | Binding Sites | Research | Binding proteins | RNA polymerases | Binding sites (Biochemistry) | Index Medicus
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6. Full Text CCAAT enhancer binding protein beta (C EBP )-2 transforms normal mammary epithelial cells and induces epithelial to mesenchymal transition in culture
by Bundy, L.M and Sealy, L
Oncogene, ISSN 0950-9232, 02/2003, Volume 22, Issue 6, pp. 869 - 883
Retroviral vector | Anchorage independence | Mammary gland | EMT | Invasion | C/EBP transcription factor | Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Cell physiology | Molecular and cellular biology | Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes | Prostaglandin-Endoperoxide Synthases - biosynthesis | Prostaglandin-Endoperoxide Synthases - genetics | Epithelium - pathology | Epithelium - metabolism | Isoenzymes - genetics | CCAAT-Enhancer-Binding Protein-beta - genetics | Cyclooxygenase 2 | Humans | Breast Neoplasms - etiology | Breast - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Membrane Proteins | Breast Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Female | Mesoderm - metabolism | Isoenzymes - biosynthesis | Breast - pathology | Cell Differentiation - physiology | Mesoderm - pathology | Index Medicus
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7. Full Text Nuclear factor erythroid-derived factor 2-related factor 2 regulates transcription of CCAAT/enhancer-binding protein β during adipogenesis
by Hou, Yongyong and Xue, Peng and Bai, Yushi and Liu, Dianxin and Woods, Courtney G and Yarborough, Kathy and Fu, Jingqi and Zhang, Qiang and Sun, Guifan and Collins, Sheila and Chan, Jefferson Y and Yamamoto, Masayuki and Andersen, Melvin E and Pi, Jingbo
Free radical biology & medicine, ISSN 0891-5849, 2012, Volume 52, Issue 2, pp. 462 - 472
C/EBPβ | Nrf2 | Adipogenesis | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Transcription, Genetic - drug effects | Reactive Oxygen Species - metabolism | Oxidative Stress | Cytoskeletal Proteins - genetics | Antioxidants - metabolism | Male | Gene Knockdown Techniques | Luciferases - genetics | Cell Nucleus - metabolism | Dexamethasone - pharmacology | Cytoskeletal Proteins - metabolism | Female | NF-E2-Related Factor 2 - genetics | Kelch-Like ECH-Associated Protein 1 | Genes, Reporter | Insulin - pharmacology | Response Elements | CCAAT-Enhancer-Binding Protein-beta - genetics | Mice, Inbred C57BL | Gene Expression Regulation | 3T3-L1 Cells | NF-E2-Related Factor 2 - deficiency | 1-Methyl-3-isobutylxanthine - pharmacology | CCAAT-Enhancer-Binding Protein-beta - metabolism | Mice, Knockout | Animals | NF-E2-Related Factor 2 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Mice | Primary Cell Culture | Adaptor Proteins, Signal Transducing - metabolism | Luciferases - biosynthesis | Index Medicus | C | EBPβ
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8. Full Text Mdm2 promotes myogenesis through the ubiquitination and degradation of CCAAT/Enhancer-binding protein β
by Fu, Dechen and Lala-Tabbert, Neena and Lee, Hwabin and Wiper-Bergeron, Nadine
The Journal of biological chemistry, ISSN 0021-9258, 04/2015, Volume 290, Issue 16, pp. 10200 - 10207
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | RNA, Small Interfering - genetics | Proto-Oncogene Proteins c-mdm2 - genetics | MyoD Protein - genetics | Humans | PAX7 Transcription Factor - genetics | Muscle, Skeletal - metabolism | PAX7 Transcription Factor - metabolism | Muscle, Skeletal - cytology | Myoblasts - metabolism | Ubiquitination | Gene Expression Regulation, Developmental | Proteolysis | Myoblasts - cytology | Cell Differentiation | Proto-Oncogene Proteins c-mdm2 - antagonists & inhibitors | Proto-Oncogene Proteins c-mdm2 - metabolism | Cell Line | Muscle, Skeletal - growth & development | Signal Transduction | CCAAT-Enhancer-Binding Protein-beta - genetics | Mice, Inbred C57BL | MyoD Protein - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Mice, Knockout | Animals | Muscle Development - genetics | Mice | Primary Cell Culture | Proteasome Endopeptidase Complex - metabolism | RNA, Small Interfering - metabolism | Index Medicus | ubiquitylation (ubiquitination) | EBP | CCAAT-enhancer-binding protein (C | muscle regeneration | mouse double minute 2 homolog (MDM2) | myogenesis | Cell Biology
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9. Full Text Theobromine suppresses adipogenesis through enhancement of CCAAT-enhancer-binding protein β degradation by adenosine receptor A1
by Mitani, Takakazu and Watanabe, Shun and Yoshioka, Yasukiyo and Katayama, Shigeru and Nakamura, Soichiro and Ashida, Hitoshi
Biochimica et biophysica acta. Molecular cell research, ISSN 0167-4889, 12/2017, Volume 1864, Issue 12, pp. 2438 - 2448
Sumoylation | Theobromine | Adenosine receptor | CCAAT/enhancer-binding protein | Adipogenesis | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Sumoylation - genetics | Receptors, Purinergic P1 - genetics | Signal Transduction | Adipogenesis - drug effects | 3T3-L1 Cells | CCAAT-Enhancer-Binding Protein-beta - metabolism | Cell Differentiation - genetics | Proteolysis - drug effects | Animals | Cysteine Endopeptidases - metabolism | Adipocytes - metabolism | Cysteine Endopeptidases - genetics | Theobromine - administration & dosage | Mice | Adipogenesis - genetics | Index Medicus
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