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Journal Article
Kidney International, ISSN 0085-2538, 08/2015, Volume 88, Issue 2, pp. 262 - 275
Tubulointerstitial hypoxia plays a critical role in the pathogenesis of kidney injury, and hypoxia-inducible factor (HIF)-1 is a master regulator of cellular... 
hypoxia | proximal tubule | inflammation | cell signaling | TRANSCRIPTION FACTORS | INDUCIBLE FACTOR | TUBULOINTERSTITIAL INJURY | 3 C/EBP ISOFORMS | INDUCTION | CANCER | PHASE | PATHWAY | GENE-EXPRESSION | UROLOGY & NEPHROLOGY | KIDNEY | Up-Regulation | Cell Hypoxia - physiology | Heme Oxygenase-1 - metabolism | Superoxide Dismutase - genetics | Kidney - pathology | Nephritis - genetics | Epithelial Cells - drug effects | Humans | Male | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Gene Knockdown Techniques | Hypoxia - metabolism | Heme Oxygenase-1 - genetics | Angiopoietin-like 4 Protein | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Inflammation Mediators - pharmacology | Microarray Analysis | Transcription, Genetic | CCAAT-Enhancer-Binding Protein-delta - metabolism | Reperfusion Injury - complications | Superoxide Dismutase - metabolism | Kidney Tubules, Proximal - cytology | Angiopoietins - metabolism | Gene Expression | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Hypoxia - etiology | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Renal Artery Obstruction - complications | Animals | Nephrectomy - adverse effects | CCAAT-Enhancer-Binding Protein-delta - genetics | Kidney Tubules, Proximal - metabolism | Cisplatin - adverse effects | Nephritis - metabolism | Mice | RNA, Small Interfering | Superoxide Dismutase-1 | Angiopoietins - genetics | Cell Hypoxia - genetics | Basic Research
Journal Article
JOURNAL OF BIOLOGICAL CHEMISTRY, ISSN 0021-9258, 06/2017, Volume 292, Issue 24, pp. 10275 - 10287
Journal Article
Redox Biology, ISSN 2213-2317, 06/2018, Volume 16, pp. 104 - 112
Excessive reactive oxygen species (ROS) can form an oxidative stress and an associated neuroinflammation. However, the contribution of astrocytes to ROS... 
Astrocyte | CEBPD | SOD1 and neuroinflammation | ROS | OXIDATIVE-STRESS | NADPH-OXIDASE | ACTIVATION | C/EBP-DELTA | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | ZINC | GENE | MICROGLIA | NEURONS | EXPRESSION
Journal Article
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 11/2015, Volume 14, Issue 11, pp. 2623 - 2633
Hepatocellular carcinoma (HCC) can arise from chronic inflammation due to viral infection, organ damage, drug toxicity, or alcohol abuse. Moreover, gene... 
APOPTOSIS | HEPATOCELLULAR-CARCINOMA | KINASE PATHWAY | ACTIVATION | C/EBP-DELTA | CEBPD | ONCOLOGY | SIGNALING PATHWAY | INFLAMMATION | GENE-EXPRESSION | GROWTH-FACTOR | Immunohistochemistry | Oligonucleotide Array Sequence Analysis | Apoptosis - drug effects | Humans | Apoptosis - genetics | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Carcinoma, Hepatocellular - drug therapy | Propane - analogs & derivatives | RNA Interference | Carcinoma, Hepatocellular - genetics | Tumor Burden - genetics | CCAAT-Enhancer-Binding Protein-delta - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Azacitidine - administration & dosage | Ketones - pharmacology | Propane - administration & dosage | Liver Neoplasms - genetics | Cells, Cultured | Liver Neoplasms - drug therapy | Azacitidine - analogs & derivatives | Gene Expression Profiling - methods | Ketones - administration & dosage | E2F1 Transcription Factor - metabolism | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Hep G2 Cells | Xenograft Model Antitumor Assays | Azacitidine - pharmacology | Animals | Tumor Burden - drug effects | CCAAT-Enhancer-Binding Protein-delta - antagonists & inhibitors | CCAAT-Enhancer-Binding Protein-delta - genetics | Liver Neoplasms - metabolism | Cell Line, Tumor | Mice, Inbred NOD | E2F1 Transcription Factor - genetics | Carcinoma, Hepatocellular - metabolism | Propane - pharmacology
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 11/2016, Volume 53, Issue 9, pp. 5912 - 5927
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e45378
Background: The up-regulation of CCAAT/enhancer binding protein delta (CEBPD) has frequently been observed in macrophages in age-associated disorders,... 
CELLS | ACTIVATION | C/EBP-DELTA | CYCLOOXYGENASE-2 | ANGIOGENESIS | COLORECTAL-CANCER | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DISEASE | INDUCTION | EXPRESSION | Arthritis, Experimental - genetics | Cell Adhesion Molecules - genetics | Chemokine CCL20 - metabolism | Cell Proliferation | Humans | Chemokine CXCL1 - genetics | Cell Movement - genetics | Cell Movement - physiology | Arthritis, Experimental - metabolism | Arthritis, Experimental - pathology | Arthritis, Rheumatoid - metabolism | Interleukin-23 Subunit p19 - metabolism | Chromatin Immunoprecipitation | Interleukin-23 Subunit p19 - genetics | Chemokine CCL20 - genetics | CCAAT-Enhancer-Binding Protein-delta - metabolism | Chemokine CXCL1 - metabolism | Cell Line | Cells, Cultured | Cell Adhesion Molecules - metabolism | Arthritis, Rheumatoid - pathology | Mice, Knockout | Arthritis, Rheumatoid - genetics | Animals | CCAAT-Enhancer-Binding Protein-delta - genetics | Cell Line, Tumor | Mice | Care and treatment | Research | Biological markers | Binding proteins | Health aspects | Rheumatoid arthritis | Cell proliferation | Immunohistochemistry | Transcription factors | Disease | Leukocyte migration | Pathogenesis | Arthritis | CCAAT/enhancer-binding protein | Macrophages | Inflammatory diseases | Proteins | Angiogenesis | Rodents | CCL20 protein | Tumor necrosis factor-TNF | Bioindicators | Age | Deoxyribonucleic acid--DNA | Cytokines | Inflammation | Gene expression | Synoviocytes | Endothelial cells | Medicine | Orthopedics | Collagen | Biomarkers | Cell migration | Deoxyribonucleic acid | DNA
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 04/2010, Volume 88, Issue 5, pp. 1113 - 1123
The transcription factor CCAAT/enhancer binding protein delta (C/EBP delta) regulates transcription of genes that play important roles in glial activation.... 
Lipopolysaccharide | Toll-like receptors | Glial activation | Transcription factors | Microglia | toll-like receptors | transcription factors | 3 C/EBP ISOFORMS | ACUTE-PHASE CONDITIONS | NEUROSCIENCES | INTERFERON-GAMMA | GENE-EXPRESSION | NITRIC-OXIDE | CYCLOOXYGENASE-2 EXPRESSION | CENTRAL-NERVOUS-SYSTEM | lipopolysaccharide | MOUSE MACROPHAGES | microglia | MOLECULAR-MECHANISMS | Microglia - metabolism | Coculture Techniques | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Gliosis - physiopathology | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Dose-Response Relationship, Drug | Microglia - immunology | Astrocytes - immunology | Cyclooxygenase 2 - genetics | Inflammation Mediators - pharmacology | Encephalitis - metabolism | Binding Sites - physiology | CCAAT-Enhancer-Binding Protein-delta - metabolism | Gliosis - immunology | Toll-Like Receptors - metabolism | RNA, Messenger - drug effects | Animals, Newborn | Astrocytes - drug effects | Encephalitis - genetics | Microglia - drug effects | Gliosis - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation - physiology | Encephalitis - immunology | Up-Regulation - genetics | CCAAT-Enhancer-Binding Protein-beta - metabolism | Toll-Like Receptors - agonists | Animals | Promoter Regions, Genetic - physiology | CCAAT-Enhancer-Binding Protein-delta - genetics | Lipopolysaccharides - pharmacology | Mice | Transcriptional Activation - physiology | Astrocytes - metabolism
Journal Article
by Wang, SM and Lee, YC and Ko, CY and Lai, MD and Lin, DY and Pao, PC and Chi, JY and Hsiao, YW and Liu, TL and Wang, JM
MOLECULAR NEUROBIOLOGY, ISSN 0893-7648, 02/2015, Volume 51, Issue 1, pp. 370 - 382
Reactive astrogliosis is a cellular manifestation of neuroinflammation and occurs in response to all forms and severities of the central nervous system (CNS)'s... 
ACTIVATION | C/EBP-DELTA | CEBPD | Astrocytes | Antiapoptosis | INDUCED APOPTOSIS | PHOSPHORYLATION | NEUROSCIENCES | BETA | REGULATOR | MICROGLIA | INFLAMMATION | Alzheimer's disease | EXPRESSION | ZNF179 | Insulin-Like Growth Factor Binding Protein 3 - genetics | Transcription, Genetic - drug effects | Interleukin-1beta - pharmacology | Alzheimer Disease - complications | Apoptosis - drug effects | Humans | Astrocytes - pathology | Molecular Sequence Data | Apoptosis - genetics | Male | DNA-Binding Proteins - deficiency | Alzheimer Disease - pathology | DNA-Binding Proteins - metabolism | Insulin-Like Growth Factor Binding Protein 3 - metabolism | CCAAT-Enhancer-Binding Protein-delta - deficiency | Kruppel-Like Transcription Factors - metabolism | Mitochondrial Proteins - metabolism | Base Sequence | Amyloid beta-Peptides - metabolism | CCAAT-Enhancer-Binding Protein-delta - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Astrocytes - drug effects | Plaque, Amyloid - pathology | Mice, Inbred C57BL | DNA-Binding Proteins - genetics | Down-Regulation - drug effects | Promyelocytic Leukemia Zinc Finger Protein | Plaque, Amyloid - complications | Animals | Cell Line, Tumor | Plaque, Amyloid - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Alzheimer Disease - genetics | Methyl Methanesulfonate - pharmacology | Astrocytes - metabolism | Yuan (China) | DNA binding proteins | Leukemia | Protein binding | Proteins | Alzheimers disease | Binding sites | Neurobiology
Journal Article
FASEB Journal, ISSN 0892-6638, 08/2013, Volume 27, Issue 8, pp. 2967 - 2976
Although uncontrolled inflammatory response plays a central role in the pathogenesis of acute lung injury (ALI), the precise molecular mechanisms underlying... 
C/EBP | LPS | SUPPRESSOR | KAPPA-B ACTIVATION | C/EBP-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | CYTOKINE | INDUCTION | MICE LACKING | IL-6 | CELL BIOLOGY | MURINE MODEL | MESSENGER-RNA | BIOLOGY | GENE-EXPRESSION | Bronchoalveolar Lavage Fluid - immunology | Acute Lung Injury - genetics | Lipopolysaccharides - immunology | Suppressor of Cytokine Signaling Proteins - deficiency | Signal Transduction - immunology | Myeloid Cells - immunology | Gene Expression - immunology | Acute Lung Injury - metabolism | CCAAT-Enhancer-Binding Protein-delta - metabolism | Lung - metabolism | Neutrophils - metabolism | Macrophages - immunology | Chemokines - immunology | Cytokines - immunology | Lung - pathology | Peroxidase - immunology | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Suppressor of Cytokine Signaling Proteins - immunology | Mice, Inbred C57BL | Cells, Cultured | Neutrophils - immunology | Suppressor of Cytokine Signaling Proteins - genetics | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Suppressor of Cytokine Signaling 3 Protein | Macrophages - metabolism | Animals | Myeloid Cells - metabolism | Acute Lung Injury - immunology | Chemokines - metabolism | Mice | Bronchoalveolar Lavage Fluid - chemistry | CCAAT-Enhancer-Binding Protein-delta - immunology | Lung - immunology | Peroxidase - metabolism | EBP | Research Communications
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2011, Volume 186, Issue 7, pp. 4467 - 4473
Subclinical levels of circulating endotoxin are associated with the pathogenesis of diverse human inflammatory diseases, by mildly inducing the expression of... 
TRANSREPRESSION | CELLS | INTEGRATION | MACROPHAGES | KINASE | TOLL-LIKE RECEPTORS | METABOLIC ENDOTOXEMIA | LIPOPOLYSACCHARIDE | KAPPA-B | IMMUNOLOGY | EXPRESSION | Cell Nucleus - immunology | Dose-Response Relationship, Immunologic | Interleukin-1 Receptor-Associated Kinases - genetics | Humans | Interleukin-1 Receptor-Associated Kinases - deficiency | I-kappa B Proteins - physiology | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - biosynthesis | Repressor Proteins - antagonists & inhibitors | Signal Transduction - immunology | Cell Nucleus - metabolism | Cell Nucleus - pathology | Bone Marrow Cells - immunology | Inflammation Mediators - metabolism | CCAAT-Enhancer-Binding Protein-delta - metabolism | Inflammation Mediators - physiology | Lipopolysaccharides - physiology | Macrophages - immunology | Repressor Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Lipopolysaccharides - toxicity | Macrophages - pathology | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Cells, Cultured | CCAAT-Enhancer-Binding Protein-delta - physiology | Signal Transduction - genetics | Mice, Knockout | Endotoxins - physiology | Macrophages - metabolism | Animals | Interleukin-1 Receptor-Associated Kinases - physiology | CCAAT-Enhancer-Binding Protein-delta - genetics | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | NF-kappa B - physiology | Proto-Oncogene Proteins - physiology | Mice | HeLa Cells | Bone Marrow Cells - metabolism | Endotoxins - toxicity | Receptors, Cytoplasmic and Nuclear - metabolism
Journal Article
15.