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Journal of Translational Medicine, ISSN 1479-5876, 10/2015, Volume 13, Issue 1, pp. 330 - 330
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 09/2012, Volume 169, Issue 3, pp. 302 - 310
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2009, Volume 29, Issue 7, pp. 2089 - 2102
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 02/2017, Volume 16, Issue 2, pp. 312 - 322
Hepatocellular carcinoma, a deadly disease, commonly arises in the setting of chronic inflammation. C-C motif chemokine ligand 2 (CCL2/MCP1), a chemokine that... 
MACROPHAGE INFILTRATION | INHIBITION | NK CELLS | ONCOLOGY | IN-VIVO | GENES | CCL2 | CHEMOKINE | LIVER-DISEASE | BLOCKADE | CARCINOMA | Immunohistochemistry | Receptors, CCR2 - genetics | Humans | Antibodies, Blocking - pharmacology | Carcinoma, Hepatocellular - drug therapy | Flow Cytometry | Cell Transformation, Neoplastic - genetics | Killer Cells, Natural - immunology | Antineoplastic Agents - pharmacology | Chemokine CCL2 - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Disease Models, Animal | Tumor Microenvironment - drug effects | Gene Expression | Carcinoma, Hepatocellular - diagnosis | Antibodies, Monoclonal - pharmacology | Liver Neoplasms - drug therapy | Antibodies, Neutralizing - pharmacology | Chemokine CCL2 - genetics | Cell Transformation, Neoplastic - metabolism | Mice, Knockout | Tumor Microenvironment - immunology | Xenograft Model Antitumor Assays | Receptors, CCR2 - metabolism | Animals | Liver Neoplasms - diagnosis | Signal Transduction - drug effects | Tumor Burden - drug effects | Biopsy | Liver Neoplasms - metabolism | Biomarkers | Cell Line, Tumor | Killer Cells, Natural - drug effects | Mice | MicroRNAs - genetics | Killer Cells, Natural - metabolism | Chemokine CCL2 - antagonists & inhibitors | Carcinoma, Hepatocellular - metabolism | CC chemokine receptors | Toxicity | Liver | c-Myc protein | Cytotoxicity | Hepatocellular carcinoma | Myc protein | Interleukin 6 | Hepatitis | Liver cancer | CCR2 protein | Immunotherapy | Rodents | Tumorigenesis | Inhibition | Carbon-carbon composites | NF-κB protein | Myeloid cells | Liver diseases | Stat3 protein | Inflammation | Tumor necrosis factor-α | Immune systems | Neutralizing | γ-Interferon | Chemokines | Monocyte chemoattractant protein 1 | Cancer | Index Medicus | microRNA-122 (miR-122) | CD11bhighGr1+ cells | CCL2 neutralizing antibody | Natural killer (NK) cells | Hepatocellular carcinoma (HCC)
Journal Article
Nature, ISSN 0028-0836, 11/2014, Volume 515, Issue 7525, pp. 130 - 133
Journal Article
EMBO reports, ISSN 1469-221X, 12/2016, Volume 17, Issue 12, pp. 1738 - 1752
Sickness behavior defines the endocrine, autonomic, behavioral, and metabolic responses associated with infection. While inflammatory responses were suggested... 
weight loss | CCR2 signaling pathway | melanin‐concentrating hormone | CCL2 chemokine | neuroinflammation | melanin-concentrating hormone | MESSENGER-RNA EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ARCUATE NUCLEUS | CELL BIOLOGY | ENERGY-BALANCE | BODY-WEIGHT | INDUCED SICKNESS BEHAVIOR | INDUCED ANOREXIA | REGULATE FEEDING-BEHAVIOR | RAT-BRAIN | CHEMOKINE RECEPTORS | Chemokine CCL2 - immunology | Lipopolysaccharides - immunology | Melanins - metabolism | Inflammation - metabolism | Pituitary Hormones - metabolism | Hypothalamic Hormones - immunology | Chemokine CCL2 - metabolism | Neurons - metabolism | Cytokines - genetics | Cytokines - immunology | Signal Transduction | Melanins - immunology | Neurons - immunology | Chemokine CCL2 - genetics | Melanins - genetics | Chemokine CCL2 - deficiency | Receptors, CCR2 - metabolism | Animals | Hypothalamus - metabolism | Hypothalamic Hormones - metabolism | Weight Loss | Pituitary Hormones - genetics | Hypothalamic Hormones - genetics | Mice | Pituitary Hormones - immunology | Illness Behavior | Cytokines - biosynthesis | Inflammation | Behavior | Metabolism | Neurons | Index Medicus | Life Sciences | Neurons and Cognition | Biochemistry, Molecular Biology | Neurobiology | Biomolecules | Cellular Biology | Cell Behavior | Neuroscience | Immunology | Scientific Reports | Scientific Report
Journal Article
JOURNAL OF CLINICAL INVESTIGATION, ISSN 0021-9738, 03/2013, Volume 123, Issue 3, pp. 1371 - 1381
The BRAF mutant, BRAF(V600E), is expressed in nearly half of melanomas, and oral BRAF inhibitors induce substantial tumor regression in patients with... 
MEDICINE, RESEARCH & EXPERIMENTAL | METASTATIC MELANOMA | THERAPY | RESISTANCE | TUMOR-ASSOCIATED MACROPHAGES | ANTIBODY | MICE | CANCER-IMMUNOTHERAPY | ANTITUMOR IMMUNITY | ESTABLISHED TUMORS | BRAF(V600E) | Chemokine CCL2 - immunology | Humans | Drug Resistance, Neoplasm | Male | Antineoplastic Agents - administration & dosage | Mutation, Missense | Molecular Targeted Therapy | Indoles - administration & dosage | T-Lymphocytes, Regulatory - immunology | Immunotherapy | Indoles - pharmacology | Antineoplastic Agents - pharmacology | Chemokine CCL2 - metabolism | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Tumor Necrosis Factor Receptor Superfamily, Member 9 - immunology | Chemokine CCL2 - genetics | Melanoma - pathology | Down-Regulation - drug effects | Sulfonamides - pharmacology | Drug Synergism | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Xenograft Model Antitumor Assays | Receptors, CCR2 - metabolism | Animals | Antibodies, Monoclonal - administration & dosage | Melanoma - immunology | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Cell Line, Tumor | Mice | CD8-Positive T-Lymphocytes - immunology | Immunity, Cellular | Sulfonamides - administration & dosage | Care and treatment | Melanoma | Physiological aspects | Genetic aspects | Research | Chemokine receptors | Index Medicus | Abridged Index Medicus
Journal Article
Neuroscience Bulletin, ISSN 1673-7067, 2/2018, Volume 34, Issue 1, pp. 13 - 21
Mounting evidence supports an important role of chemokines, produced by spinal cord astrocytes, in promoting central sensitization and chronic pain. In... 
Neurology | Neuron–glial interaction | Neurosciences | Pain Medicine | Biomedicine | Human Physiology | Monocyte chemoattractant protein 1 (MCP-1) | Anesthesiology | Anatomy | Chemokines | C–C motif chemokine ligand 2 (CCL2) | ACTIVATION | LAMINA II | RECEPTOR | C-C motif chemokine ligand 2 (CCL2) | NERVE INJURY | NEUROSCIENCES | SENSORY NEURONS | DORSAL-HORN | NEUROPATHIC PAIN | CORD ASTROCYTES | Neuron-glial interaction | MICE | CONTRIBUTES | Hyperalgesia - chemically induced | Myelitis - chemically induced | Freund's Adjuvant - toxicity | Pain Management | Male | Spiro Compounds - therapeutic use | Myelitis - drug therapy | Long-Term Potentiation - drug effects | Excitatory Amino Acid Agents - pharmacology | Long-Term Potentiation - physiology | Female | Chemokine CCL2 - metabolism | Spinal Cord - cytology | Neurons - drug effects | Somatostatin - metabolism | Vesicular Glutamate Transport Protein 2 - genetics | Somatostatin - genetics | Hyperalgesia - metabolism | Mice, Inbred C57BL | Vesicular Glutamate Transport Protein 2 - metabolism | Hyperalgesia - prevention & control | Chemokine CCL2 - genetics | Mice, Transgenic | Excitatory Amino Acid Agonists - pharmacology | Benzoxazines - therapeutic use | Animals | Vesicular Inhibitory Amino Acid Transport Proteins - metabolism | Chemokine CCL2 - pharmacology | Vesicular Inhibitory Amino Acid Transport Proteins - genetics | Benzoxazines - pharmacology | Luminescent Proteins - genetics | Myelitis - metabolism | Mice | Chemokine CCL2 - antagonists & inhibitors | Spiro Compounds - pharmacology | Luminescent Proteins - metabolism | Methyl aspartate | Medical colleges | Neurons | Physiological aspects | Glutamate | Chronic pain | Neurophysiology | Index Medicus
Journal Article
European Journal of Pain, ISSN 1090-3801, 2008, Volume 13, Issue 3, pp. 263 - 272
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 03/2009, Volume 179, Issue 5, pp. 414 - 425
Journal Article