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Journal of Neurochemistry, ISSN 0022-3042, 09/2018, Volume 146, Issue 5, pp. 526 - 539
The chemokine CCL5 prevents neuronal cell death mediated both by amyloid β, as well as the human immunodeficiency virus viral proteins gp120 and Tat. Because... 
CXCR4 | AKT | CCR5 | signal transduction | CRISPR‐Cas9 | ERK | CRISPR-Cas9 | NEURONAL APOPTOSIS | ALZHEIMERS-DISEASE | DESENSITIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GP120 | NEUROSCIENCES | PEPTIDE | CELL-DEATH | INTERNALIZATION | MOUSE MODEL | Receptors, G-Protein-Coupled - metabolism | Embryo, Mammalian | Humans | Protein Transport - drug effects | Cerebral Cortex - cytology | CRISPR-Associated Protein 9 - genetics | CRISPR-Associated Protein 9 - metabolism | Antineoplastic Agents - pharmacology | Chemokine CCL5 - metabolism | Neurons - metabolism | Neurons - drug effects | Neuroblastoma - pathology | Pertussis Toxin - pharmacology | Tretinoin - pharmacology | T-Lymphocytes | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mutagenesis - genetics | Gene Expression Regulation - physiology | Rats | Protein Transport - genetics | Animals | Chemokine CCL5 - pharmacology | Signal Transduction - drug effects | Chemokine CCL5 - genetics | Signal Transduction - physiology | Receptors, G-Protein-Coupled - genetics | Synthesis | Viral proteins | Glycogen | Cell death | Cellular signal transduction | Protein kinases | Protein binding | Neuroprotection | CC chemokine receptors | Flow cytometry | Phosphorylation | Viruses | AKT protein | Antagonists | Kinases | Neuroblastoma cells | Proteins | CCR1 protein | Time dependence | Receptors | Human immunodeficiency virus--HIV | CRISPR | CCR5 protein | Extracellular signal-regulated kinase | Glycogen synthase kinase 3 | Pharmacology | Tat protein | Signaling | Materials information | Internalization | Chemokines | Glycoprotein gp120
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2018, Volume 77, p. 1681
Journal Article
Cellular Signalling, ISSN 0898-6568, 01/2018, Volume 42, pp. 88 - 96
Cell migration towards a chemotactic stimulus relies on the re-arrangement of the cytoskeleton, which is triggered by activation of small G proteins RhoA, Rac1... 
Chemokine receptor | Antagonist | Rac1 | Chemotaxis | Ligand bias | ACTIVATION | METASTASIS | CANCER | CELL BIOLOGY | INDUCED CELL-MIGRATION | G-BETA-GAMMA | INTERNALIZATION | CCR5 | AXIS | ACTS | Humans | Receptors, CCR5 - genetics | cdc42 GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - genetics | Pyrones - pharmacology | Quinolines - pharmacology | DNA-Binding Proteins - metabolism | Receptors, CCR5 - metabolism | Peptides - chemical synthesis | MCF-7 Cells | Receptors, CXCR4 - genetics | Aminoquinolines - pharmacology | Amino Acid Sequence | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Jurkat Cells | Gene Expression Regulation | THP-1 Cells | Chemokine CCL3 - pharmacology | Transcription Factors - antagonists & inhibitors | Chemotaxis - drug effects | Cytoskeleton - ultrastructure | Pyrimidines - pharmacology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Peptides - pharmacology | Receptors, CXCR4 - metabolism | Transcription Factors - metabolism | Receptors, CCR1 - genetics | Chemokine CXCL12 - pharmacology | cdc42 GTP-Binding Protein - genetics | Receptors, CCR1 - metabolism | rac1 GTP-Binding Protein - antagonists & inhibitors | Cytoskeleton - metabolism | Cytoskeleton - drug effects | rac1 GTP-Binding Protein - metabolism | rac1 GTP-Binding Protein - genetics | Phosphates | G proteins | T cells | Muscle proteins | Membrane proteins
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 02/2017, Volume 23, Issue 3, pp. 833 - 844
Purpose: We have reported loss of SMAD4 promotes expression of CCL15 from colorectal cancer to recruit CCR1(+) myeloid cells through the CCL15-CCR1 axis, which... 
COLON-CANCER | HEPATOCELLULAR-CARCINOMA | PROGNOSTIC-FACTORS | ONCOLOGY | RESECTION | IMMATURE MYELOID CELLS | CHEMOKINE | SUPPRESSOR-CELLS | LIVER METASTASIS | EXPRESSION | PULMONARY METASTASES | Receptors, CCR1 - physiology | Prognosis | Humans | Lung Neoplasms - metabolism | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Macrophage Inflammatory Proteins - physiology | Neutrophil Infiltration | Heterografts | Lung Neoplasms - secondary | Chemokines, CC - biosynthesis | Female | Neoplasm Proteins - genetics | Colorectal Neoplasms - metabolism | Genes, Reporter | Neoplasm Proteins - biosynthesis | Smad4 Protein - physiology | Proportional Hazards Models | Mice, SCID | Macrophage Inflammatory Proteins - genetics | Disease-Free Survival | Lung Neoplasms - immunology | Animals | Chemokines, CC - physiology | Mice, Nude | Chemokines, CC - genetics | Cell Line, Tumor | Myeloid Cells - metabolism | Mice | Neoplasm Proteins - deficiency | Smad4 Protein - deficiency | Colorectal Neoplasms - pathology | Cell Movement | Macrophage Inflammatory Proteins - biosynthesis | CC chemokine receptors | Myeloid cells | Liver | Lung cancer | Colorectal carcinoma | Colorectal cancer | Leukocytes (neutrophilic) | Metastasis | Multivariate analysis | Patients | Smad4 protein | Recruitment | Metastases | CCR1 protein | Experimental design | Rodents | Medical prognosis | Biomarkers | Cancer
Journal Article
Hematological Oncology, ISSN 0278-0232, 09/2017, Volume 35, Issue 3, pp. 310 - 316
Journal Article
Oncogene, ISSN 0950-9232, 2017, Volume 36, Issue 26, pp. 3651 - 3660
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 03/2010, Volume 207, Issue 3, pp. 623 - 635
Journal Article
Journal Article
Translational Research, ISSN 1931-5244, 2015, Volume 165, Issue 4, pp. 512 - 530
Renal fibrosis is the hallmark of virtually all progressive kidney diseases and strongly correlates with the deterioration of kidney function. The... 
Internal Medicine | activating transcription factor 2 | inhibitor of kappa B | activator protein-1 | unilateral ureteral obstruction | AP-1 | CTGF | ANG II | TGF-β-inducible gene | TβRII | angiotensin-converting enzyme | bone morphogenetic protein receptor | Monocyte chemoattractant protein-1 | idiopathic pulmonary fibrosis | PPARγ | ARB | plasminogen activator inhibitor-1 | TAK1-binding protein 1 | receptor-regulated Smads | angiotensin II receptor blocker | MAPK kinase kinase kinase | stz | CCL2-CCR2 | uterine sensitization-associated gene-1 | ESRD | chronic kidney disease | TSP 1 | nuclear factor kappa B | acute tubular necrosis | mitogen-activated protein kinase | Peroxisome proliferator-activated receptor γ | TrKA | A Study of Cardiovascular Events in Diabetes | TAK1 | USAG-1 | ASCEND | transforming growth factor β1 | von Willebrand factor | βig-h3 | FSGS | MKKK | heparan sulfate proteoglycan | CCR1 | ACE | HSPG | BMP | MKK | bone morphogenetic protein | ECM | endothelin | thrombospondin type 1 | tyrosine receptor kinase A | ETA | focal segmental glomerular sclerosis | ATN | phosphodiesterase | TAB1 | ATF 2 | extracellular matrix | Kielin/chordin-like protein | JNK | RAAS | small interfering RNA | LRP | lipoprotein receptor-related protein | NF-κB | renin-angiotensin-aldosterone system | BMPR | PAI-1 | LC3 | TGF-β type I receptor | c-Jun N-terminal kinase | TβRI | IGF-1 | end-stage renal disease | MAPK | EMT | UUO | epithelial-to-mesenchymal transition | connective tissue growth factor | streptozotocin | Nox | ROS | mitogen-activated protein kinase kinase | insulin-like growth factor 1 | nicotinamide adenine dinucleotide phosphate oxidase | MCP-1 | tumor necrosis factor α | estimated glomerular filtration rate | vWF | CC motif chemokine ligand 2-CC receptor type 2 | TGF-β type II receptor | IκB | microtubule-associated protein 1 light chain 3 | PDE | TGF-β-activated kinase 1 | reactive oxygen species | CC receptor type 1 | Angiotensin II | CKD | endothelin A receptor | eGFR | VEGF | siRNA | TNF-α | R-Smads | vascular endothelial growth factor | KCP | TGF-β1 | IPF | MEDICINE, RESEARCH & EXPERIMENTAL | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | ANGIOTENSIN-ALDOSTERONE SYSTEM | DIABETIC-NEPHROPATHY | MATRIX GENE-EXPRESSION | RENAL FIBROSIS | MEDICINE, GENERAL & INTERNAL | TISSUE GROWTH-FACTOR | TRANSFORMING GROWTH-FACTOR-BETA-1 | BONE MORPHOGENETIC PROTEIN-7 | TGF-BETA ANTIBODY | MEDICAL LABORATORY TECHNOLOGY | IDIOPATHIC PULMONARY-FIBROSIS | Fibrosis - drug therapy | Signal Transduction | Humans | Transforming Growth Factor beta1 - metabolism | Drug Design | Transforming Growth Factor beta1 - genetics | Fibrosis - pathology | Renal Insufficiency, Chronic - drug therapy | Renal Insufficiency, Chronic - pathology | Oxidases | Phosphates | Niacinamide | Medical colleges | Development and progression | Transforming growth factors | Homeopathy | Materia medica and therapeutics | Purines | Chronic kidney failure | Nitric oxide | Therapeutics | Angiotensin | Bone morphogenetic proteins | Health aspects | bone morphogenic protein-7 | autophagy | transforming growth factor-β | NADPH oxidase | tumor necrosis factor-α | Kidney fibrosis | endothelin-1 | C-C motif chemokine | chemokine receptor
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2017, Volume 199, Issue 4, pp. 1516 - 1525
Tyrosine kinase inhibitors (TKIs) are used in the clinical management of hematological neoplasms. Moreover, in solid tumors such as stage 4 neuroblastomas... 
MESYLATE | IN-VITRO | THERAPY | EFFICACY | SAFETY | BCR-ABL | TYROSINE KINASE INHIBITOR | C-KIT | RECEPTOR | LIGAND | IMMUNOLOGY | Humans | Programmed Cell Death 1 Ligand 2 Protein - metabolism | Monocytes - immunology | Neuroblastoma - immunology | Killer Cells, Natural - immunology | Receptors, CXCR4 - genetics | Antineoplastic Agents - pharmacology | Receptors, CCR1 - immunology | Monocytes - physiology | Macrophages - immunology | Cytokines - immunology | Macrophages - physiology | Receptors, CXCR3 - metabolism | Imatinib Mesylate - pharmacology | Programmed Cell Death 1 Ligand 2 Protein - genetics | Cytokines - secretion | Pyrimidines - pharmacology | B7-H1 Antigen - genetics | Killer Cells, Natural - physiology | Receptors, CXCR4 - metabolism | Monocytes - drug effects | B7-H1 Antigen - metabolism | Receptors, CCR1 - genetics | Cell Differentiation - drug effects | Lymphocyte Activation - drug effects | Receptors, CXCR3 - genetics | Receptors, CCR1 - metabolism | Macrophages - drug effects | Killer Cells, Natural - drug effects | Lymphocyte receptors | CC chemokine receptors | Parkinson's disease | Transcription | Leukemia | Cytotoxicity | Macrophages | B7 antigen | Neoplasms | CCR1 protein | Receptors | Immunology | Natural killer cells | Protein-tyrosine kinase | Movement disorders | Immune system | Tyrosine | Imatinib | Immune response | Immunostimulation | Hematology | Neurodegenerative diseases | Cytokines | Myeloid leukemia | Chronic myeloid leukemia | CXCR3 protein | Patients | CXCR4 protein | Monocytes | Immunosuppression | PD-L1 protein | Antitumor activity | Histocompatibility antigen HLA | Solid tumors | Tumors
Journal Article