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Clinica Chimica Acta, ISSN 0009-8981, 11/2019, Volume 498, pp. 68 - 75
Platelet antibodies can lead to clinical diseases such as platelet transfusion refractoriness (PTR), fetal/neonatal alloimmune thrombocytopenia (FNAIT), etc.... 
CD36 deficiency | Fetal/neonatal alloimmune thrombocytopenia | Platelet transfusion refractoriness | Platelet alloantibody | Viral antibodies | Antigens | Transfusion | Blood platelets | Pregnant women | Analysis | Antibodies | Blood donors
Journal Article
Zhongguo shi yan xue ye xue za zhi, ISSN 1009-2137, 06/2016, Volume 24, Issue 3, p. 934
CD36 is a transmembrane glycoprotein, a multi-ligand receptor, possesses various biological functions. CD36 deficiency may stimulate the body to produce... 
Pregnancy | CD36 Antigens - deficiency | Blood Transfusion | Platelet Transfusion | Autoantibodies - blood | Blood Platelet Disorders | Humans | Blood Platelets | Female | Genetic Diseases, Inborn | Histocompatibility
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 01/2013, Volume 112, Issue 1, pp. 103 - 112
RATIONALE:A prothrombotic state and increased platelet reactivity are common in pathophysiological conditions associated with oxidative stress and infections.... 
carboxy(alkylpyrrole) protein adducts | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | HUMAN PLASMA | MACROPHAGES | DEPENDENT PROTEIN-KINASE | MACULAR DEGENERATION | Toll-like receptor 9 | platelets | LOW-DENSITY LIPOPROTEINS | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | OXIDIZED PHOSPHOLIPIDS | CD36 | HEMATOLOGY | oxidative stress | B SCAVENGER RECEPTORS | Toll-Like Receptor 9 - blood | CD36 Antigens - deficiency | Phosphorylation | Blood Platelets - immunology | CD36 Antigens - genetics | Toll-Like Receptor 2 - genetics | Oxidative Stress | Humans | Toll-Like Receptor 9 - genetics | Toll-Like Receptor 2 - deficiency | Transfection | src-Family Kinases - blood | Time Factors | Toll-Like Receptor 6 - genetics | Phosphatidylinositol 3-Kinase - blood | Thrombosis - blood | Genes, Reporter | Disease Models, Animal | Cell Line | Scavenger Receptors, Class B - deficiency | Signal Transduction | Mice, Inbred C57BL | Toll-Like Receptor 9 - deficiency | Myeloid Differentiation Factor 88 - genetics | Thrombosis - immunology | Scavenger Receptors, Class B - genetics | Immunity, Innate | Toll-Like Receptor 6 - deficiency | Mice, Knockout | Platelet Aggregation | Animals | Myeloid Differentiation Factor 88 - deficiency | Blood Platelets - metabolism | Platelet Activation | Proto-Oncogene Proteins c-akt - blood | Ligands | Thrombosis - genetics | Interleukin-1 Receptor-Associated Kinases - blood | Mice | Serum Albumin - metabolism
Journal Article
Circulation Research, ISSN 0009-7330, 09/2013, Volume 113, Issue 8, pp. 1004 - 1012
RATIONALE:Efficient clearance of apoptotic cells (efferocytosis) is a prerequisite for inflammation resolution and tissue repair. After myocardial infarction,... 
Myocardial infarction | Efferocytosis | Inflammation | Macrophages | Phagocytosis | phagocytosis | CLEARANCE | CELL ACCUMULATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHOSPHATIDYLSERINE RECEPTOR | efferocytosis | myocardial infarction | HEART-FAILURE | PLAQUE NECROSIS | ACCELERATES ATHEROSCLEROSIS | MEDIATED PHAGOCYTOSIS | MICE LACKING | macrophages | inflammation | ACUTE MYOCARDIAL-INFARCTION | PERIPHERAL VASCULAR DISEASE | MER | HEMATOLOGY | Inflammation - pathology | Myocardial Infarction - genetics | CD36 Antigens - deficiency | CD36 Antigens - genetics | Ventricular Function, Left | Myocytes, Cardiac - immunology | Coculture Techniques | Male | Wound Healing | Myocardial Infarction - immunology | Recovery of Function | Myocytes, Cardiac - enzymology | Ventricular Remodeling | Time Factors | Myocardial Infarction - pathology | c-Mer Tyrosine Kinase | Bone Marrow Transplantation | Antigens, Ly - metabolism | Female | Myocardial Infarction - physiopathology | Macrophages - immunology | Disease Models, Animal | Myocardial Infarction - enzymology | Myocardial Contraction | Proto-Oncogene Proteins - metabolism | Receptor Protein-Tyrosine Kinases - deficiency | Macrophages - pathology | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Proto-Oncogene Proteins - genetics | Inflammation - immunology | Proto-Oncogene Proteins - deficiency | Receptor Protein-Tyrosine Kinases - metabolism | Transplantation Chimera | Macrophages - enzymology | Mice, Knockout | Myocytes, Cardiac - pathology | Animals | Receptor Protein-Tyrosine Kinases - genetics | Inflammation - genetics | Mice | Inflammation - enzymology | Apoptosis | Index Medicus
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 10/2014, Volume 115, Issue 9, pp. 770 - 780
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2015, Volume 35, Issue 8, pp. 1787 - 1797
OBJECTIVE—To examine infiltration of blood foamy monocytes, containing intracellular lipid droplets, into early atherosclerotic lesions and its contribution to... 
atherosclerosis | monocytes | inflammation | diet, high-fat | lipoproteins | RECRUITMENT | FAMILIAL HYPERCHOLESTEROLEMIA | STERILE INFLAMMATION | MACROPHAGES | PREDICT CARDIOVASCULAR EVENTS | SUBSETS | DEFICIENT MICE | high-fat | DISEASE | PERIPHERAL VASCULAR DISEASE | diet | BLOOD MONOCYTES | ACCUMULATION | HEMATOLOGY | Hypercholesterolemia - immunology | CD36 Antigens - deficiency | CD36 Antigens - genetics | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Male | Monocytes - metabolism | Monocytes - immunology | Aorta - metabolism | Hypercholesterolemia - drug therapy | Aortic Diseases - metabolism | CD11c Antigen - genetics | Time Factors | Toll-Like Receptor 4 - deficiency | Diet, High-Fat | Receptors, LDL - deficiency | Aortic Diseases - prevention & control | Aortic Diseases - pathology | Disease Models, Animal | Cell Line | Receptors, LDL - genetics | Atherosclerosis - pathology | Atherosclerosis - immunology | Cholesterol, VLDL - metabolism | Aortic Diseases - immunology | Aorta - drug effects | Mice, Inbred C57BL | Toll-Like Receptor 4 - genetics | Aorta - immunology | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Monocytes - drug effects | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Hypercholesterolemia - genetics | Cholesterol, Dietary | Hypercholesterolemia - metabolism | Atherosclerosis - prevention & control | Atherosclerosis
Journal Article
PLoS ONE, ISSN 1932-6203, 2012, Volume 7, Issue 3, pp. e34378 - 7 p.
Non-alcoholic steatohepatitis (NASH) is characterized by steatosis and inflammation, which can further progress into fibrosis and cirrhosis. Recently, we... 
LDL | SCAVENGER RECEPTOR CD36 | MULTIDISCIPLINARY SCIENCES | DISEASE | ATHEROSCLEROSIS | ACCUMULATION | PROTECTS | Inflammation - pathology | Kupffer Cells - pathology | CD36 Antigens - deficiency | Liver - pathology | CD36 Antigens - genetics | Fatty Liver - pathology | Lipids - chemistry | Lysosomes - metabolism | CD36 Antigens - metabolism | Bone Marrow Transplantation | Non-alcoholic Fatty Liver Disease | Receptors, LDL - deficiency | Female | Scavenger Receptors, Class A - genetics | Kupffer Cells - metabolism | Receptors, LDL - genetics | Fatty Liver - metabolism | Liver - metabolism | Scavenger Receptors, Class A - deficiency | Receptors, LDL - metabolism | Lipid Metabolism | Cholesterol - metabolism | Mice, Knockout | Animals | Mice | Scavenger Receptors, Class A - metabolism | Lipids | Inflammation | Macrophages | Liver cirrhosis | Cholesterol | Syngeneic grafts | Trafficking | Liver | Kupffer cells | Bone marrow transplantation | Acidification | Homeostasis | Inflammatory response | Biology | Autophagy | Accumulation | Liver cancer | Signal transduction | Receptors | Toxicology | Clonal deletion | Rodents | Atherosclerosis | Deletion | Bone marrow | Scavenger receptors | Lipoprotein (low density) receptors | Liver diseases | CD36 antigen | Steatosis | Nutrition research | Pathology | Cirrhosis | Lipoproteins | Microscopy | Internalization | Fibrosis | Intracellular | Laboratory animals
Journal Article
Circulation Research, ISSN 0009-7330, 07/2015, Volume 117, Issue 4, pp. 321 - 332
RATIONALE:Oxidative stress is an important contributing factor in several human pathologies ranging from atherosclerosis to cancer progression; however, the... 
Antigens | Wound healing | CD36 | Angiogenesis inducing agents | Macrophages | Toll-like receptor 2 | wound healing | CARDIAC & CARDIOVASCULAR SYSTEMS | GENOMIC BIOMARKERS | LOW-DENSITY-LIPOPROTEIN | antigens, CD36 | MACULAR DEGENERATION | ENDOGENOUS LIGANDS | VASCULAR-DISEASE | angiogenesis inducing agents | macrophages | ACCESSORY MOLECULES | PERIPHERAL VASCULAR DISEASE | OXIDIZED PHOSPHOLIPIDS | STRESS | HEMATOLOGY | MACROPHAGE SUBSETS | toll-like receptor 2 | CD36 Antigens - deficiency | CD36 Antigens - genetics | Toll-Like Receptor 2 - genetics | Apolipoproteins E - deficiency | Oxidative Stress | Atherosclerosis - genetics | Humans | Melanoma, Experimental - metabolism | Wound Healing | Toll-Like Receptor 2 - deficiency | Transfection | CD36 Antigens - metabolism | RNA Interference | Time Factors | HEK293 Cells | Antigens, Differentiation - metabolism | Lipoproteins, LDL - metabolism | Disease Models, Animal | Pyrroles - metabolism | Signal Transduction | Mice, Inbred C57BL | Macrophages, Peritoneal - immunology | Melanoma, Experimental - pathology | Toll-Like Receptor 2 - metabolism | Tumor Burden | Atherosclerosis - metabolism | Mice, Knockout | Phenotype | Animals | Apolipoproteins E - genetics | Macrophages, Peritoneal - metabolism | Neovascularization, Physiologic | Oxidized lipids | angiogenesis | macrophage | CEP | TLR2
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 08/2017, Volume 27, Issue 4, pp. 21 - 214
Aims: Cluster of differentiation 36 (CD36) is involved in the development of nonalcoholic steatohepatitis (NASH). Excess CD36 facilitates liver cells taking... 
Original Research Communications | MCP-1 | CD36 | NASH | ROS | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | OXIDIZED LDL | PALMITATE | INHIBITION | SCAVENGER RECEPTOR CD36 | INSULIN-RESISTANCE | LIVER | ENDOCRINOLOGY & METABOLISM | KUPFFER CELLS | MICE | STEATOHEPATITIS | Up-Regulation | CD36 Antigens - deficiency | CD36 Antigens - genetics | Reactive Oxygen Species - metabolism | Coculture Techniques | Humans | Hepatocytes - metabolism | Hepatocytes - cytology | Cell Nucleus - metabolism | Disease Models, Animal | Promoter Regions, Genetic | Non-alcoholic Fatty Liver Disease - genetics | Signal Transduction | Cells, Cultured | THP-1 Cells | Chemokine CCL2 - genetics | Non-alcoholic Fatty Liver Disease - metabolism | Macrophages - cytology | Gene Knockout Techniques | Hep G2 Cells | Macrophages - metabolism | Animals | Mice | Histone Deacetylase 2 - metabolism | Histones - metabolism | Cell Movement | Histones | Monocytes | Inflammation | Research | Gene expression | Macrophages | Histone deacetylase | Reactive oxygen species | Hydrogen peroxide | Leukocyte migration | Liver | Innovations | Inflammatory response | Reduction | Fatty liver | Clonal deletion | Rodents | Transcription activation | Clusters | Deletion | HDAC2 protein | Liver diseases | CD36 antigen | Fatty acids | Promoters | Steatosis | Hepatocytes | Fibrosis | Infiltration | Differentiation | Monocyte chemoattractant protein 1 | Cell migration
Journal Article
Circulation Research, ISSN 0009-7330, 09/2017, Volume 121, Issue 8, pp. 951 - 962
RATIONALE:Platelet hyperreactivity, which is common in many pathological conditions, is associated with increased atherothrombotic risk. The mechanisms leading... 
Innate immune system | Platelets | Hyperlipidemia | Thrombosis | TLR2 | OXIDATION | hyperlipidemia | ACTIVATION | COMPLEX | CARDIAC & CARDIOVASCULAR SYSTEMS | HYPERCHOLESTEROLEMIA | platelets | innate immune system | thrombosis | PATTERN-RECOGNITION | SCAVENGER RECEPTOR CD36 | DISEASE | TOLL-LIKE RECEPTORS | PERIPHERAL VASCULAR DISEASE | OXIDIZED PHOSPHOLIPIDS | HEMATOLOGY | LIPOPROTEINS | CD36 Antigens - deficiency | Hyperlipidemias - genetics | Blood Platelets - immunology | CD36 Antigens - genetics | Hyperlipidemias - blood | Toll-Like Receptor 2 - genetics | Apolipoproteins E - deficiency | Humans | Male | Toll-Like Receptor 2 - deficiency | Transfection | Toll-Like Receptor 6 - genetics | HEK293 Cells | Female | Phospholipids - blood | Thrombosis - blood | Disease Models, Animal | Hyperlipidemias - immunology | Toll-Like Receptor 6 - metabolism | Genetic Predisposition to Disease | Hyperlipidemias - metabolism | Oxidation-Reduction | Signal Transduction | Mice, Inbred C57BL | Myeloid Differentiation Factor 88 - genetics | Thrombosis - immunology | Toll-Like Receptor 2 - metabolism | Immunity, Innate | Toll-Like Receptor 6 - deficiency | Mice, Knockout | Phenotype | Thrombosis - metabolism | Animals | Myeloid Differentiation Factor 88 - deficiency | Blood Platelets - metabolism | Platelet Activation | Apolipoproteins E - genetics | Thrombosis - genetics | Hyperreactivity | Dyslipidemia | Lipid peroxidation | Phospholipids | Clonal deletion | Apolipoprotein E | Rodents | Interleukin 1 | Toll-like receptors | Protein-tyrosine kinase | Immune system | Spleen | TRAF6 protein | Cytokines | TIRAP protein | Pattern recognition | CD36 antigen | Phospholipase C | Phospholipase | Tumor necrosis factor | TLR2 protein | MyD88 protein | Syk protein | Ligands | Lipids and Cholesterol | Oxidative Stress | signal transduction | lipids and lipoproteins | singaling pathways | Cell Signaling
Journal Article
Journal of Lipid Research, ISSN 0022-2275, 05/2009, Volume 50, Issue 5, pp. 999 - 1111
Journal Article