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1. Full Text Contradictory intrahepatic immune responses activated in high-load hepatitis C virus livers compared with low-load livers
by Ishibashi, Mariko and Ishibashi, Mariko and Yamaguchi, Hiromi and Yamaguchi, Hiromi and Hirotani, Yukari and Hirotani, Yukari and Sakurada, Akihisa and Sakurada, Akihisa and Endo, Toshihide and Endo, Toshihide and Sugitani, Masahiko and Sugitani, Masahiko and Takayama, Tadatoshi and Takayama, Tadatoshi and Makishima, Makoto and Makishima, Makoto and Esumi, Mariko and Esumi, Mariko
Archives of virology, ISSN 0304-8608, 4/2018, Volume 163, Issue 4, pp. 855 - 865
Medical Microbiology | Infectious Diseases | Biomedicine | Virology | Life Sciences & Biomedicine | Science & Technology | HLA-DQ alpha-Chains - immunology | Liver - virology | Dendritic Cells - immunology | Humans | Hepatitis C, Chronic - virology | Hepacivirus - pathogenicity | Antigens, CD - genetics | CD11c Antigen - immunology | CD40 Ligand - immunology | CD4-Positive T-Lymphocytes - immunology | Liver - immunology | CD11c Antigen - genetics | Carcinoma, Hepatocellular - immunology | Liver Neoplasms - virology | Antigens, CD20 - immunology | Liver Neoplasms - genetics | Signal Transduction | Immune Tolerance | Liver Neoplasms - immunology | Carcinoma, Hepatocellular - virology | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Viral Load - immunology | Antigens, CD20 - genetics | CD40 Ligand - genetics | Hepatocytes - virology | Hepacivirus - growth & development | CD8-Positive T-Lymphocytes - immunology | Forkhead Transcription Factors - immunology | Antigens, CD - immunology | Hepacivirus - immunology | Transcriptome - immunology | Hepatitis C, Chronic - complications | HLA-DQ alpha-Chains - genetics | Kupffer Cells - virology | Liver Neoplasms - etiology | Carcinoma, Hepatocellular - genetics | Carcinoma, Hepatocellular - etiology | CD4-Positive T-Lymphocytes - virology | Dendritic Cells - virology | Antigens, Differentiation, Myelomonocytic - immunology | Adaptive Immunity | Gene Expression Regulation | Forkhead Transcription Factors - genetics | Hepatocytes - immunology | Viral Load - genetics | Antigens, Differentiation, Myelomonocytic - genetics | CD8-Positive T-Lymphocytes - virology | Hepatitis C, Chronic - immunology | Hepatitis C, Chronic - genetics | Kupffer Cells - immunology | Medical colleges | Histocompatibility antigens | Immune response | Microbiology | RNA | Cell death | Liver | HLA histocompatibility antigens | B cells | Comparative analysis | Hepatitis C virus | T cells | DQA1 protein | CD8 antigen | Helper cells | Kupffer cells | Lymphocytes T | Macrophages | Hepatitis | Foxp3 protein | CD154 antigen | CD11c antigen | Antigens | Load | CD3 antigen | T cell receptors | Adaptive immunity | Immunological tolerance | CD4 antigen | Hepatocytes | Lymphocytes B | Antigen-presenting cells | PD-L1 protein | CD20 antigen | Histocompatibility antigen HLA | Hepatitis C | Apoptosis | Index Medicus
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2. Full Text Thymic B Cells Are Licensed to Present Self Antigens for Central T Cell Tolerance Induction
by Yamano, Tomoyoshi and Nedjic, Jelena and Hinterberger, Maria and Steinert, Madlen and Koser, Sandra and Pinto, Sheena and Gerdes, Norbert and Lutgens, Esther and Ishimaru, Naozumi and Busslinger, Meinrad and Brors, Benedikt and Kyewski, Bruno and Klein, Ludger
Immunity (Cambridge, Mass.), ISSN 1074-7613, 06/2015, Volume 42, Issue 6, pp. 1048 - 1061
Life Sciences & Biomedicine | Immunology | Science & Technology | B7-1 Antigen - genetics | Signal Transduction | Humans | Cells, Cultured | B-Lymphocytes - physiology | Antigen Presentation - genetics | Central Tolerance - genetics | B7-1 Antigen - metabolism | Clonal Selection, Antigen-Mediated - genetics | Transcription Factors - genetics | Mice, Knockout | CD4-Positive T-Lymphocytes - immunology | Transcription Factors - metabolism | Cell Differentiation - genetics | Animals | CD40 Antigens - metabolism | Autoantigens - immunology | Histocompatibility Antigens Class II - metabolism | Thymus Gland - immunology | CD40 Antigens - genetics | Mice | Mice, Inbred BALB C | Histocompatibility Antigens Class II - genetics | Immunoglobulin Class Switching - genetics | Autoimmunity | Antigens | B cells | Dendritic cells | T cells | Spleen | Flow cytometry | Lymphocytes | Bone marrow | T cell receptors | Gene expression | Index Medicus
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3. Full Text A TLR9-dependent checkpoint governs B cell responses to DNA-containing antigens
by Sindhava, Vishal J and Oropallo, Michael A and Moody, Krishna and Naradikian, Martin and Higdon, Lauren E and Zhou, Lin and Myles, Arpita and Green, Nathaniel and Nündel, Kerstin and Stohl, William and Schmidt, Amanda M and Cao, Wei and Dorta-Estremera, Stephanie and Kambayashi, Taku and Marshak-Rothstein, Ann and Cancro, Michael P
The Journal of clinical investigation, ISSN 0021-9738, 05/2017, Volume 127, Issue 5, pp. 1651 - 1663
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Toll-Like Receptor 9 - genetics | Male | T-Box Domain Proteins - immunology | G1 Phase Cell Cycle Checkpoints - immunology | MAP Kinase Signaling System - immunology | Toll-Like Receptor 9 - immunology | Cell Differentiation - genetics | Interleukins - genetics | MAP Kinase Signaling System - genetics | Interleukins - immunology | B-Cell Activating Factor - immunology | Female | CD40 Antigens - genetics | Interferon-gamma - genetics | Cell Line | DNA - immunology | p38 Mitogen-Activated Protein Kinases - genetics | p38 Mitogen-Activated Protein Kinases - immunology | T-Box Domain Proteins - genetics | G1 Phase Cell Cycle Checkpoints - genetics | Receptors, Antigen, B-Cell - immunology | Mice, Knockout | Cell Differentiation - immunology | Animals | B-Lymphocytes - immunology | B-Cell Activating Factor - genetics | Interferon-gamma - immunology | Antigens - immunology | Receptors, Antigen, B-Cell - genetics | Mice | CD40 Antigens - immunology | Antigens | DNA | Cytokine receptors | TLR9 | Cellular signal transduction | Research | B cells | CD27 antigen | CD40 antigen | Laboratories | B-cell receptor | Kinases | Genotype & phenotype | Mitochondria | Cell activation | Lymphocytes | Cell cycle | Interleukin 21 | Toll-like receptors | Deoxyribonucleic acid--DNA | Immunization | Cell survival | Cytokines | Cell division | T cell receptors | Survival | TLR9 protein | Immune response (humoral) | Medicine | Lymphocytes B | BLyS protein | Ligands | Interferon | Regulation | Autoimmune diseases | Apoptosis | Index Medicus | Abridged Index Medicus
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4. Full Text CD19 and BAFF‐R can signal to promote B‐cell survival in the absence of Syk
by Hobeika, Elias and Levit‐Zerdoun, Ella and Anastasopoulou, Vasiliki and Pohlmeyer, Roland and Altmeier, Simon and Alsadeq, Ameera and Dobenecker, Marc‐Werner and Pelanda, Roberta and Reth, Michael
The EMBO journal, ISSN 0261-4189, 04/2015, Volume 34, Issue 7, pp. 925 - 939
BAFF receptor | mb1‐CreERT2 | Syk | B‐cell antigen receptor | CD19 | mb1-CreER T 2 | B-cell antigen receptor | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Antigens, CD - immunology | CD40 Antigens - antagonists & inhibitors | Cell Survival - genetics | Intracellular Signaling Peptides and Proteins - immunology | Antigens, CD19 - genetics | Antigens, CD - genetics | Phosphatidylinositol 3-Kinases - immunology | Protein-Tyrosine Kinases - immunology | Signal Transduction - immunology | Protein-Tyrosine Kinases - genetics | ADP-ribosyl Cyclase 1 - immunology | CD40 Antigens - genetics | Interleukin-4 - genetics | Membrane Glycoproteins - immunology | B-Cell Activation Factor Receptor - genetics | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Antigens, CD19 - immunology | B-Cell Activation Factor Receptor - immunology | B-Lymphocytes - cytology | Interleukin-4 - antagonists & inhibitors | ADP-ribosyl Cyclase 1 - genetics | Signal Transduction - genetics | Cell Survival - immunology | Receptors, Antigen, B-Cell - immunology | Membrane Glycoproteins - genetics | Mice, Knockout | Antibodies - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Interleukin-4 - immunology | Animals | B-Lymphocytes - immunology | Lipopolysaccharides - pharmacology | Receptors, Antigen, B-Cell - genetics | Mice | Oligodeoxyribonucleotides - pharmacology | CD40 Antigens - immunology | Lymphocyte receptors | T cell receptors | Kinases | Immune system | Index Medicus | mb1-CreERT2
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5. Full Text Peptide-Conjugated Nanoparticles Reduce Positive Co-stimulatory Expression and T Cell Activity to Induce Tolerance
by Kuo, Robert and Saito, Eiji and Miller, Stephen D and Shea, Lonnie D
Molecular therapy, ISSN 1525-0016, 07/2017, Volume 25, Issue 7, pp. 1676 - 1685
tolerance induction mechanism | antigen-specific tolerance | immune tolerance | PLGA | PLG nanoparticles | B7-2 Antigen - genetics | Myelin Proteolipid Protein - immunology | B7-1 Antigen - genetics | Spleen - immunology | Nanoparticles - chemistry | Ovalbumin - immunology | Encephalomyelitis, Autoimmune, Experimental - immunology | Delayed-Action Preparations - chemistry | Spleen - drug effects | Polyglactin 910 - metabolism | T-Lymphocytes, Regulatory - pathology | Delayed-Action Preparations - administration & dosage | Myelin Proteolipid Protein - pharmacology | T-Lymphocytes, Regulatory - immunology | Antigen-Presenting Cells - pathology | Immunoconjugates - pharmacology | Female | CD40 Antigens - genetics | Encephalomyelitis, Autoimmune, Experimental - genetics | Polyglactin 910 - chemistry | B7-2 Antigen - immunology | Myelin Proteolipid Protein - chemistry | Spleen - pathology | Ovalbumin - chemistry | Immune Tolerance - drug effects | Gene Expression | Encephalomyelitis, Autoimmune, Experimental - pathology | B7-1 Antigen - immunology | Immunoconjugates - metabolism | Antigen-Presenting Cells - drug effects | Mice, Inbred C57BL | Ovalbumin - pharmacology | Antigen-Presenting Cells - immunology | Immunoconjugates - chemistry | Antigens - chemistry | Particle Size | Encephalomyelitis, Autoimmune, Experimental - therapy | T-Lymphocytes, Regulatory - drug effects | Animals | Antigens - immunology | Mice | Nanoparticles - administration & dosage | Primary Cell Culture | Antigens - pharmacology | CD40 Antigens - immunology | Cell proliferation | Multiple sclerosis | Transcription factors | CD40 antigen | Disease | Peptides | CD86 antigen | Lymphocytes T | Nanoparticles | Myelin proteolipid protein | Lymphocytes | Localization | Immune system | Antigen presentation | Cytokines | Dendritic cells | Myelin | CD80 antigen | Inflammation | T cell receptors | Immunological tolerance | Studies | Major histocompatibility complex | Antigen-presenting cells | Internalization | PD-L1 protein | Proteolipid protein | Poly(lactide-co-glycolide) | Autoimmune diseases | Apoptosis | Index Medicus | Original
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6. Full Text The etiology of autoimmune thyroid disease: A story of genes and environment
by Tomer, Yaron and Huber, Amanda
Journal of autoimmunity, ISSN 0896-8411, 2009, Volume 32, Issue 3, pp. 231 - 239
Allergy and Immunology | Environment | Graves' Disease | Genes | Hashimoto's thyroiditis | Life Sciences & Biomedicine | Immunology | Science & Technology | Antigens, CD - immunology | Receptors, Thyrotropin - genetics | Thyroiditis, Autoimmune - immunology | Thyroglobulin - metabolism | Humans | Autoimmunity - genetics | Antigens, CD - genetics | Antigens, CD - metabolism | HLA-DR Antigens - genetics | Thyroglobulin - immunology | Thyroiditis, Autoimmune - etiology | CD40 Antigens - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - immunology | Autoimmunity - immunology | Thyroiditis, Autoimmune - metabolism | CD40 Antigens - genetics | Genetic Predisposition to Disease | Receptors, Thyrotropin - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - metabolism | Receptors, Thyrotropin - immunology | Thyroiditis, Autoimmune - genetics | CTLA-4 Antigen | Thyroglobulin - genetics | HLA-DR Antigens - immunology | CD40 Antigens - immunology | Autoimmunity | Development and progression | Thyroid diseases | Index Medicus
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7. Full Text Regulated Expansion and Survival of Chimeric Antigen Receptor-Modified T Cells Using Small Molecule-Dependent Inducible MyD88/CD40
by Foster, Aaron E and Mahendravada, Aruna and Shinners, Nicholas P and Chang, Wei-Chun and Crisostomo, Jeannette and Lu, An and Khalil, Mariam and Morschl, Eva and Shaw, Joanne L and Saha, Sunandan and Duong, MyLinh T and Collinson-Pautz, Matthew R and Torres, David L and Rodriguez, Tania and Pentcheva-Hoang, Tsvetelina and Bayle, J. Henri and Slawin, Kevin M and Spencer, David M
Molecular therapy, ISSN 1525-0016, 09/2017, Volume 25, Issue 9, pp. 2176 - 2188
CD40 | inducible | costimulation | T cell | chimeric antigen receptor | dimerizer | MyD88 | Genetics & Heredity | Life Sciences & Biomedicine | Biotechnology & Applied Microbiology | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Cell Proliferation | Humans | Gene Expression Profiling | Leukemia - metabolism | Recombinant Fusion Proteins | CD28 Antigens - metabolism | Lymphocyte Activation - immunology | CD40 Antigens - metabolism | CD28 Antigens - genetics | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | CD40 Antigens - genetics | Toll-Like Receptors - metabolism | Leukemia - genetics | Disease Models, Animal | Immunotherapy, Adoptive - methods | Receptors, Antigen, T-Cell - metabolism | Signal Transduction | Cell Survival | Leukemia - therapy | Xenograft Model Antitumor Assays | Animals | Leukemia - immunology | Lymphocyte Activation - drug effects | T-Lymphocytes - immunology | Mice | Receptors, Antigen, T-Cell - genetics | Cluster Analysis | Cell proliferation | Antigens | Graft-versus-host reaction | CD40 antigen | Cell survival | Cytokines | CD8 antigen | Cytotoxicity | T cell receptors | Lymphocytes T | Gene expression | Kinases | Adoptive transfer | CD4 antigen | Chimeric antigen receptors | Cell growth | Cell activation | Lymphocytes | Toll-like receptors | MyD88 protein | Tumor necrosis factor-TNF | Ligands | Software | Tumors | Index Medicus | Original
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