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Molecular therapy, ISSN 1525-0016, 2017, Volume 25, Issue 7, pp. 1676 - 1685
.... Antigen-specific tolerance was induced using poly(lactide-co-glycolide) nanoparticles conjugated with disease-relevant antigen to treat a model of multiple sclerosis... 
tolerance induction mechanism | antigen-specific tolerance | immune tolerance | PLGA | PLG nanoparticles | MEDICINE, RESEARCH & EXPERIMENTAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | DENDRITIC CELLS | ANTIGENS | IL-10 | IMMUNOLOGICAL-TOLERANCE | DELIVERY | RESPONSES | MULTIPLE-SCLEROSIS | PATHWAY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | CROSS-PRESENTATION | B7-2 Antigen - genetics | Myelin Proteolipid Protein - immunology | B7-1 Antigen - genetics | Spleen - immunology | Nanoparticles - chemistry | Ovalbumin - immunology | Encephalomyelitis, Autoimmune, Experimental - immunology | Delayed-Action Preparations - chemistry | Spleen - drug effects | Polyglactin 910 - metabolism | T-Lymphocytes, Regulatory - pathology | Delayed-Action Preparations - administration & dosage | Myelin Proteolipid Protein - pharmacology | T-Lymphocytes, Regulatory - immunology | Antigen-Presenting Cells - pathology | Immunoconjugates - pharmacology | Female | CD40 Antigens - genetics | Encephalomyelitis, Autoimmune, Experimental - genetics | Polyglactin 910 - chemistry | B7-2 Antigen - immunology | Myelin Proteolipid Protein - chemistry | Spleen - pathology | Ovalbumin - chemistry | Immune Tolerance - drug effects | Gene Expression | Encephalomyelitis, Autoimmune, Experimental - pathology | B7-1 Antigen - immunology | Immunoconjugates - metabolism | Antigen-Presenting Cells - drug effects | Mice, Inbred C57BL | Ovalbumin - pharmacology | Antigen-Presenting Cells - immunology | Immunoconjugates - chemistry | Antigens - chemistry | Particle Size | Encephalomyelitis, Autoimmune, Experimental - therapy | T-Lymphocytes, Regulatory - drug effects | Animals | Antigens - immunology | Mice | Nanoparticles - administration & dosage | Primary Cell Culture | Antigens - pharmacology | CD40 Antigens - immunology | Cell proliferation | Multiple sclerosis | Transcription factors | CD40 antigen | Disease | Peptides | CD86 antigen | Lymphocytes T | Nanoparticles | Myelin proteolipid protein | Lymphocytes | Localization | Immune system | Antigen presentation | Cytokines | Dendritic cells | Myelin | CD80 antigen | Inflammation | T cell receptors | Immunological tolerance | Studies | Major histocompatibility complex | Antigen-presenting cells | Internalization | PD-L1 protein | Proteolipid protein | Poly(lactide-co-glycolide) | Autoimmune diseases | Apoptosis | Original
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2002, Volume 196, Issue 12, pp. 1627 - 1638
Journal Article
Clinical Immunology, ISSN 1521-6616, 2015, Volume 160, Issue 1, pp. 103 - 123
Journal Article
Journal Article
Vaccine, ISSN 0264-410X, 2011, Volume 30, Issue 9, pp. 1624 - 1635
Journal Article
The Journal of Immunology, ISSN 0022-1767, 12/2003, Volume 171, Issue 12, pp. 6431 - 6441
An important prerequisite in using regulatory T cells for immunotherapy is their ex vivo expansion without loss of suppressor function. Human anergic... 
BETA-CHAIN | IN-VITRO | MEMORY | TOLERANCE | INTERLEUKIN-15 | PROLIFERATION | GROWTH-FACTOR | INDUCTION | IMMUNOLOGY | EX-VIVO | VERSUS-HOST-DISEASE | Interleukin-15 - physiology | T-Lymphocyte Subsets - immunology | Antigens, CD - immunology | Intracellular Fluid - immunology | T-Lymphocyte Subsets - cytology | Coculture Techniques | Humans | Interphase - immunology | Antibodies, Blocking - pharmacology | Interleukin-10 - physiology | CD4-Positive T-Lymphocytes - immunology | B7-2 Antigen | Lymphocyte Activation - immunology | Cell Division - immunology | Intracellular Fluid - chemistry | Histocompatibility Antigens Class II - physiology | Membrane Glycoproteins - immunology | Bystander Effect - immunology | Immunologic Memory - immunology | Receptors, Antigen, T-Cell - metabolism | Receptors, Interleukin-2 - biosynthesis | CD4-Positive T-Lymphocytes - cytology | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Transforming Growth Factor beta - physiology | Immunophenotyping | Immunity, Innate | Proto-Oncogene Proteins c-bcl-2 - biosynthesis | Receptors, Antigen, T-Cell - physiology | Antigens, Differentiation - physiology | CTLA-4 Antigen | Interleukin-15 - pharmacology | Epitopes, T-Lymphocyte - physiology | Up-Regulation - immunology | T-Lymphocyte Subsets - metabolism | Staining and Labeling | Clonal Anergy - immunology | Interleukin-2 - pharmacology | CD40 Antigens - immunology
Journal Article
Journal Article
Molecular therapy, ISSN 1525-0016, 2017, Volume 25, Issue 9, pp. 2176 - 2188
Anti-tumor efficacy of T cells engineered to express chimeric antigen receptors (CARs) is dependent on their specificity, survival... 
CD40 | inducible | costimulation | T cell | chimeric antigen receptor | dimerizer | MyD88 | MEDICINE, RESEARCH & EXPERIMENTAL | EFFECTOR FUNCTIONS | TOLL-LIKE RECEPTOR | ANTITUMOR-ACTIVITY | CUTTING EDGE | THERAPY | MEMORY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | CPG-OLIGODEOXYNUCLEOTIDES | LYMPHODEPLETION | COSTIMULATION | EXPRESSION | Cell Proliferation | Humans | Gene Expression Profiling | Leukemia - metabolism | Recombinant Fusion Proteins | CD28 Antigens - metabolism | Lymphocyte Activation - immunology | CD40 Antigens - metabolism | CD28 Antigens - genetics | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | CD40 Antigens - genetics | Toll-Like Receptors - metabolism | Leukemia - genetics | Disease Models, Animal | Immunotherapy, Adoptive - methods | Receptors, Antigen, T-Cell - metabolism | Signal Transduction | Cell Survival | Leukemia - therapy | Xenograft Model Antitumor Assays | Animals | Leukemia - immunology | Lymphocyte Activation - drug effects | T-Lymphocytes - immunology | Mice | Receptors, Antigen, T-Cell - genetics | Cluster Analysis | Cell proliferation | Antigens | Graft-versus-host reaction | CD40 antigen | Cell survival | Cytokines | CD8 antigen | Cytotoxicity | T cell receptors | Lymphocytes T | Gene expression | Kinases | Adoptive transfer | CD4 antigen | Cell growth | Cell activation | Lymphocytes | Toll-like receptors | MyD88 protein | Tumor necrosis factor-TNF | Ligands | Software | Tumors | Original
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 2015, Volume 64, Issue 1, pp. 158 - 171
Journal Article