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Nature (London), ISSN 1476-4687, 2016, Volume 531, Issue 7593, pp. 253 - 257
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 1, p. e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment. Current understanding... 
MACROPHAGE INFILTRATION | PAIN | INDUCED MECHANICAL ALLODYNIA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | DORSAL-ROOT GANGLIA | SPINAL-CORD | NERVE INJURY | SENSORY NEURONS | SATELLITE GLIAL-CELLS | PACLITAXEL | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 6, p. e0128714
Introduction Understanding the factors that delineate the efficacy of T cell responses towards pathogens is crucial for our ability to develop potent therapies... 
ANTIGEN-SENSITIVITY | VIRUS | MULTIDISCIPLINARY SCIENCES | DISEASE | T-Lymphocyte Subsets - immunology | Leishmania major - immunology | CD8-Positive T-Lymphocytes - pathology | Humans | CD8-Positive T-Lymphocytes - parasitology | CD4-Positive T-Lymphocytes - parasitology | CD4-Positive T-Lymphocytes - pathology | Leishmaniasis, Cutaneous - immunology | CD4-Positive T-Lymphocytes - immunology | HIV Infections - immunology | HIV Infections - pathology | Clone Cells | CD4-Positive T-Lymphocytes - virology | Cytokines - immunology | Datasets as Topic | HIV Infections - virology | Leishmaniasis, Cutaneous - pathology | Immunophenotyping | Immunity, Innate | T-Lymphocyte Subsets - virology | HIV-1 - immunology | Models, Immunological | Algorithms | T-Lymphocyte Subsets - pathology | Leishmaniasis, Cutaneous - parasitology | T-Lymphocyte Subsets - parasitology | CD8-Positive T-Lymphocytes - virology | CD8-Positive T-Lymphocytes - immunology | Cytokines - biosynthesis | Cytotoxicity, Immunologic | Infection | T cells | Health aspects | Analysis | Evaluation | Pathogens | Antigens | Parameter estimation | Effectiveness | Cytokines | Control methods | CD8 antigen | Effector cells | Infections | Lymphocytes T | Molecular chains | CD4 antigen | Datasets | Infectious diseases | Lymphocytes | Correlation analysis | Human immunodeficiency virus--HIV | In vivo methods and tests | Combinatorial analysis | Quantitative analysis | Life Sciences | Human health and pathology | HIV | Human immunodeficiency virus
Journal Article
Journal Article
Nature (London), ISSN 1476-4687, 2015, Volume 523, Issue 7562, pp. 612 - 616
The clinical course of autoimmune and infectious disease varies greatly, even between individuals with the same condition. An understanding of the molecular... 
RESPONSES | INTERFERON | PREDICTS PROGNOSIS | ACUTE HEPATITIS-C | MULTIDISCIPLINARY SCIENCES | DISEASE | BIOLOGY | GENE-EXPRESSION | ACTIVITY INDEX | SIGNATURE | Inflammation - pathology | CD8-Positive T-Lymphocytes - pathology | Humans | Inflammatory Bowel Diseases - immunology | Transcriptome | Autoimmunity - genetics | Infection - virology | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - pathology | Autoimmune Diseases - genetics | CD4-Positive T-Lymphocytes - immunology | Autoimmunity - immunology | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Lupus Erythematosus, Systemic - immunology | CD8-Positive T-Lymphocytes - metabolism | Receptors, Antigen, T-Cell - immunology | Autoimmune Diseases - pathology | Receptors, Interleukin-7 - immunology | Receptors, Interleukin-7 - metabolism | Infection - immunology | Autoimmune Diseases - immunology | CD4-Positive T-Lymphocytes - metabolism | Inflammation - virology | Programmed Cell Death 1 Receptor - metabolism | Inflammation - immunology | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - genetics | Infection - genetics | Phenotype | Animals | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - immunology | Infection - pathology | Lupus Erythematosus, Systemic - genetics | Mice | Programmed Cell Death 1 Receptor - immunology | CD8-Positive T-Lymphocytes - immunology | Lupus Erythematosus, Systemic - pathology | CD2 Antigens - immunology | Viral antibodies | Antibodies | Interferon | T cells | Health aspects | Analysis | Hepatitis | Disease | Lymphocytes | T cell receptors | Infections | Genetic engineering | Regression analysis | Gene expression | Viral infections
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 4, p. e0154564
It has been established that mammalian target of Rapamycin (mTOR) inhibitors have anti-inflammatory effects in models of experimental colitis. However, the... 
B-CELLS | INTESTINAL INFLAMMATION | INFLAMMATORY-BOWEL-DISEASE | CROHNS-DISEASE | DENDRITIC CELLS | GM-CSF | MULTIDISCIPLINARY SCIENCES | IN-VIVO | KINASE INHIBITOR | ANTITUMOR-ACTIVITY | TH17 CELLS | T-Lymphocyte Subsets - immunology | Th1 Cells - pathology | CD8-Positive T-Lymphocytes - pathology | Colon - drug effects | Colitis - pathology | Colon - immunology | Male | CD4-Positive T-Lymphocytes - pathology | Th1 Cells - immunology | T-Lymphocytes, Regulatory - pathology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | TOR Serine-Threonine Kinases - antagonists & inhibitors | Th17 Cells - drug effects | T-Lymphocyte Subsets - drug effects | Inflammation Mediators - metabolism | Disease Models, Animal | Th17 Cells - pathology | Th1 Cells - drug effects | Colon - pathology | Mice, Inbred C57BL | Morpholines - pharmacology | Dextran Sulfate - toxicity | Colitis - etiology | T-Lymphocytes, Regulatory - drug effects | Animals | T-Lymphocyte Subsets - pathology | Cell Differentiation - drug effects | CD8-Positive T-Lymphocytes - drug effects | Th17 Cells - immunology | Cell Proliferation - drug effects | Mice | CD8-Positive T-Lymphocytes - immunology | Colitis - prevention & control | CD4-Positive T-Lymphocytes - drug effects | Dextran | Prevention | Dosage and administration | Interferon | Inflammation | Research | Colitis | T cells | CD8 antigen | Body weight | Body weight loss | Proteins | Cell growth | Surgery | Peripheral blood | Dextran sulfate | Bone marrow | Spleen | Cytokines | Lamina propria | Rapamycin | Gene expression | Studies | Inflammatory bowel disease | Hospitals | Sodium | Interleukin 10 | Cell proliferation | Animal models | Phosphorylation | Inflammatory bowel diseases | Leukemia | Interleukin | Helper cells | Lymphocytes T | Kinases | Sulfates | Autophagy | Lymph nodes | Interleukin 6 | Ethics | Granulocytes | Lymphocytes | Intestine | Rodents | Gastroenterology | Colon | Dendritic cells | Histology | CD4 antigen | Medicine | Sulfate | Laboratory animals
Journal Article