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Tumor Biology, ISSN 1010-4283, 11/2015, Volume 36, Issue 11, pp. 8511 - 8517
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s13277-015-3592-y Prostate... 
CDKN1A | Prostate cancer (PC) | miR-208
Journal Article
Oncotarget, ISSN 1949-2553, 2018, Volume 9, Issue 21, pp. 15579 - 15590
MiR-106b is an oncomir and a potential target for anti-cancer therapy. We hypothesize that grape seed procyanidin extract (GSE) exerts antineoplastic effects... 
CDKN1A | Pharmacodynamics | Oncomir | Pharmacokinetics | P21
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 05/2017, Volume 18, Issue 5, p. 928
Our genomes are subject to potentially deleterious alterations resulting from endogenous sources (e.g., cellular metabolism, routine errors in DNA replication... 
CDKN1A | Premature senescence | Chemical genotoxic agents | DNAJB9 | p53 signaling | Mutational processes | Multinucleated giant cells | p21 | Apoptosis | CARCINOMA-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | apoptosis | GIANT CANCER-CELLS | KINASE C-DELTA | HUMAN BREAST-CANCER | CHEMISTRY, MULTIDISCIPLINARY | premature senescence | p21(WAF1) (CDKN1A) | STEM-LIKE CELLS | mutational processes | multinucleated giant cells | chemical genotoxic agents | TUMOR-SUPPRESSOR | IONIZING-RADIATION | DNA Damage - drug effects | Neoplasms - metabolism | HSP40 Heat-Shock Proteins - metabolism | Molecular Chaperones - metabolism | HSP40 Heat-Shock Proteins - genetics | Apoptosis - drug effects | Membrane Proteins - genetics | Humans | Molecular Chaperones - genetics | Tumor Suppressor Protein p53 - metabolism | Cellular Senescence - drug effects | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - chemistry | Tumor Suppressor Protein p53 - genetics | Antineoplastic Agents - toxicity | Neoplasms - drug therapy | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Membrane Proteins - metabolism | Neoplasms - pathology | Biotechnology | GTP-binding protein | Regulators | Senescence | Recombination | p53 Protein | DNA damage | Genotoxicity | Medical services | Homeostasis | Genomes | DNA repair | Alterations | Pathways | Cell cycle | Repair | Damage | Injuries | Deoxyribonucleic acid--DNA | Stresses | Retarding | Stability | Tumor cell lines | Metabolism | DNA biosynthesis | Progeny | Cyclin-dependent kinase inhibitor p21 | Chemotherapy | Stem cells | Diagnostic software | Tumor suppressor genes | Replication | Diagnostic systems | Solid tumors | Cancer | Tumors | Integrity
Journal Article
The Journal of Pathology, ISSN 0022-3417, 07/2012, Volume 227, Issue 3, pp. 275 - 285
MiR‐519d belongs to the chromosome 19 miRNA cluster (C19MC), the largest human miRNA cluster. One of its members, miR‐519d, is over‐expressed in hepatocellular... 
C19MC | methylation | AKT3 | CDKN1A/p21 | PTEN | TIMP2 | microRNA | HCC | Methylation | CDKN1A | INVOLVEMENT | DOXORUBICIN | PATHOLOGY | p21 | MTOR | ONCOLOGY | EXPRESSION | Transcription, Genetic - drug effects | Up-Regulation | Antibiotics, Antineoplastic - pharmacology | Humans | Middle Aged | Male | MicroRNAs - metabolism | Proto-Oncogene Proteins c-akt - genetics | Tissue Inhibitor of Metalloproteinase-2 - genetics | Tumor Suppressor Protein p53 - genetics | Tissue Inhibitor of Metalloproteinase-2 - metabolism | DNA Methylation | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Transfection | Chromatin Immunoprecipitation | RNA Interference | Carcinoma, Hepatocellular - genetics | Aged, 80 and over | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Female | Liver Neoplasms - pathology | Gene Expression Regulation, Neoplastic - drug effects | Liver Neoplasms - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Real-Time Polymerase Chain Reaction | PTEN Phosphohydrolase - genetics | Liver Neoplasms - genetics | Neoplasm Invasiveness | Tumor Suppressor Protein p53 - metabolism | PTEN Phosphohydrolase - metabolism | Carcinoma, Hepatocellular - enzymology | Hep G2 Cells | Cell Movement - drug effects | Signal Transduction - drug effects | Carcinoma, Hepatocellular - pathology | Aged | Cell Proliferation - drug effects | Cell Cycle - drug effects | Doxorubicin - pharmacology | Apoptosis
Journal Article
Journal of Hepatology, ISSN 0168-8278, 09/2019, Volume 71, Issue 3, pp. 553 - 562
Journal Article
Journal of Chinese Pharmaceutical Sciences, ISSN 1003-1057, 07/2017, Volume 26, Issue 7, pp. 488 - 495
Journal Article
The EMBO Journal, ISSN 0261-4189, 08/2017, Volume 36, Issue 15, pp. 2280 - 2295
Journal Article
Journal Article
Molecular cancer research : MCR, ISSN 1541-7786, 12/2016, Volume 14, Issue 12, pp. 1195 - 1203
Many drugs targeting the DNA damage response are being developed as anti-cancer therapies, either as single agents or in combination with ionising radiation or... 
CDKN1A | anticancer drugs | DNA damage | PARP inhibitors | olaparib
Journal Article
Cell Cycle, ISSN 1538-4101, 11/2009, Volume 8, Issue 22, pp. 3729 - 3741
Journal Article
Journal Article
Developmental Dynamics, ISSN 1058-8388, 03/2017, Volume 246, Issue 3, pp. 162 - 185
Background: Neural crest stem cells (NCSCs) are a population of adult multipotent stem cells. We are interested in studying whether oxygen tensions affect the... 
TPM1 (tropomyosin‐1) | hypoxia | stem cells | antioxidant | hyperoxia | S100A2 | neural crest | CDKN1A (p21CIP1/WAF1) | CXCR4 | HIF1α | oxygen | TP53 | ) | CDKN1A (p21 | TPM1 (tropomyosin-1) | CDKN1A (p21(CIP1/WAF1)) | OXIDATIVE STRESS | INDUCIBLE FACTOR | CHEMOKINE RECEPTOR CXCR4 | ANATOMY & MORPHOLOGY | BONE-MARROW | SELF-RENEWAL | OSTEOGENIC DIFFERENTIATION | DEVELOPMENTAL BIOLOGY | PROMOTES PROLIFERATION | TRANSCRIPTION FACTOR SOX10 | HIF1 alpha | ENHANCES PROLIFERATION | HEMATOPOIETIC STEM | Embryonic Stem Cells - metabolism | RNA, Small Interfering - genetics | Tropomyosin - genetics | Embryonic Stem Cells - cytology | Hyperoxia - physiopathology | Oxidative Stress - physiology | Cell Survival - genetics | Apoptosis - genetics | Tumor Suppressor Protein p53 - genetics | Hypoxia - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Hyperoxia - metabolism | Female | Receptors, CXCR4 - genetics | Cell Survival - physiology | Tropomyosin - metabolism | Cell Proliferation - genetics | Neural Crest - cytology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Cell Proliferation - physiology | Oxidative Stress - genetics | Tumor Suppressor Protein p53 - metabolism | Rats | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Blotting, Western | Receptors, CXCR4 - metabolism | Hair Follicle - cytology | Animals | Hair Follicle - metabolism | Fluorescent Antibody Technique | Hypoxia - physiopathology | Signal Transduction - physiology | Apoptosis - physiology
Journal Article