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1. Full Text Stromal cell-derived factor-1α confers protection against myocardial ischemia/reperfusion injury: Role of the cardiac stromal cell-derived factor-1α-CXCR4 axis
by Hu, Xiaofeng and Dai, Shujing and Wu, Wen-Jian and Tan, Wei and Zhu, Xiaoping and Mu, Jingyao and Guo, Yiru and Bolli, Roberto and Rokosh, Gregg
Circulation, ISSN 0009-7322, 08/2007, Volume 116, Issue 6, pp. 654 - 663
Background - Stromal cell-derived factor-1 alpha (SDF-1 alpha) binding to its cognate receptor, CXCR4, regulates a variety of cellular functions such as stem... 
Myocardial infarction | Reperfusion | Ischemia | CXCL12 chemokine | CXCR4 protein, mouse | SURVIVAL | STEM-CELLS | SIGNALING PATHWAYS | CARDIAC & CARDIOVASCULAR SYSTEMS | INDUCE | CHEMOKINE RECEPTOR CXCR4 | myocardial infarction | BONE-MARROW | FACTOR-I | G-CSF | ischemia | INFARCTION | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | reperfusion | Chemokine CXCL12 | Myocardial Ischemia - metabolism | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Myocardial Ischemia - prevention & control | Rats | Male | Hypoxia - metabolism | Myocardial Reperfusion Injury - metabolism | Paracrine Communication - physiology | Receptors, CXCR4 - physiology | Animals | Receptors, CXCR4 - biosynthesis | Hypoxia - pathology | Myocytes, Cardiac - physiology | Chemokines, CXC - physiology | Ischemic Preconditioning, Myocardial - methods | Mice | Stromal Cells - physiology | Cell Survival - physiology | Myocardial Reperfusion Injury - prevention & control
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2. Full Text Dietary Fiber Confers Protection against Flu by Shaping Ly6c− Patrolling Monocyte Hematopoiesis and CD8+ T Cell Metabolism
by Trompette, Aurélien and Gollwitzer, Eva S and Pattaroni, Céline and Lopez-Mejia, Isabel C and Riva, Erika and Pernot, Julie and Ubags, Niki and Fajas, Lluis and Nicod, Laurent P and Marsland, Benjamin J
Immunity, ISSN 1074-7613, 05/2018, Volume 48, Issue 5, pp. 992 - 1005.e8
Dietary fiber protects against chronic inflammatory diseases by dampening immune responses through short-chain fatty acids (SCFAs). Here we examined the effect... 
influenza | patrolling monocytes | CD8+ T cells | fiber | hematopoiesis | metabolism | SCFA | neutrophils | CD8 | T cells | LUNG | INFLUENZA-VIRUS INFECTION | DENDRITIC CELLS | CHAIN FATTY-ACIDS | MACROPHAGES | BONE-MARROW | GENE-EXPRESSION | GUT MICROBIOTA | BLOOD MONOCYTES | IMMUNOLOGY | ENERGY-METABOLISM | Immune response | Lymphocytes T | Adaptive immunity | Macrophages | Immunity | Fatty acids | Monocytes | Diet | Hematopoiesis | Lymphocytes | Influenza | Patrols | Bone marrow | Dietary fiber
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3. Full Text AKT2 Confers Protection Against Aortic Aneurysms and Dissections
by Shen, Ying H and Zhang, Lin and Ren, Pingping and Nguyen, Mary T and Zou, Sili and Wu, Darrell and Wang, Xing Li and Coselli, Joseph S and LeMaire, Scott A
Circulation Research, ISSN 0009-7330, 02/2013, Volume 112, Issue 4, pp. 618 - 632
RATIONALE:Aortic aneurysm and dissection (AAD) are major diseases of the adult aorta caused by progressive medial degeneration of the aortic wall. Although the... 
forkhead box protein O1 | tissue inhibitor of metalloproteinase-1 | AKT | matrix metalloproteinase-9 | aortic aneurysm and dissection | PHOSPHATIDYLINOSITOL 3-KINASE/AKT | APOPTOSIS | SIGNALING PATHWAYS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MICE LACKING | MATRIX METALLOPROTEINASES | INSULIN-RESISTANCE | IN-VIVO | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | MUSCLE-CELL-PROLIFERATION | HEMATOLOGY | Phosphorylation | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Apoptosis - drug effects | Aortic Aneurysm, Thoracic - prevention & control | Humans | Middle Aged | Proto-Oncogene Proteins c-akt - deficiency | Aortitis - enzymology | Aortitis - chemically induced | Case-Control Studies | Proto-Oncogene Proteins c-akt - genetics | Aneurysm, Dissecting - enzymology | Forkhead Transcription Factors - metabolism | Matrix Metalloproteinase 9 - genetics | Forkhead Transcription Factors - antagonists & inhibitors | Angiotensin II - toxicity | Aortitis - pathology | Myocytes, Smooth Muscle - drug effects | Aneurysm, Dissecting - prevention & control | Aneurysm, Dissecting - etiology | Aorta, Thoracic - pathology | Promoter Regions, Genetic | Angiotensin II - pharmacology | Cells, Cultured - drug effects | Matrix Metalloproteinase 9 - biosynthesis | Aortic Aneurysm, Thoracic - enzymology | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Aortitis - genetics | Enzyme Induction | Proto-Oncogene Proteins c-akt - physiology | Aortic Aneurysm, Thoracic - etiology | Cells, Cultured - enzymology | Mice, Knockout | Aorta, Thoracic - enzymology | Elastic Tissue - pathology | Tissue Inhibitor of Metalloproteinase-1 - genetics | Animals | Protein Binding | Aged | Forkhead Box Protein O1 | Mice | Protein Processing, Post-Translational | Aortic aneurysm and dissection | TIMP-1 | MMP-9 | FOXO1
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4. Full Text IL-18 production downstream of the Nlrp3 inflammasome confers protection against colorectal tumor formation
by Zaki, Mohammad Hasan and Vogel, Peter and Body-Malapel, Mathilde and Lamkanfi, Mohamed and Kanneganti, Thirumala-Devi
Journal of Immunology, ISSN 0022-1767, 10/2010, Volume 185, Issue 8, pp. 4912 - 4920
Colorectal cancer is a leading cause of cancer-related deaths worldwide. Chronic inflammation is recognized as a predisposing factor for the development of... 
TOLL-LIKE-RECEPTORS | MEDIATES IFN-GAMMA | INTERFERON-GAMMA | CROHNS-DISEASE | JAK-STAT | ULCERATIVE-COLITIS | CELL LINE | GAMMA-INDUCING FACTOR | KAPPA-B | IMMUNOLOGY | BOWEL-DISEASE | Interleukin-18 - immunology | Enzyme-Linked Immunosorbent Assay | NLR Family, Pyrin Domain-Containing 3 Protein | Mice, Inbred C57BL | Inflammation - immunology | Reverse Transcriptase Polymerase Chain Reaction | Cell Transformation, Neoplastic - metabolism | Blotting, Western | Mice, Knockout | Cell Transformation, Neoplastic - immunology | Inflammation - metabolism | Interleukin-18 - biosynthesis | Animals | Carrier Proteins - metabolism | Colorectal Neoplasms - immunology | Mice | Carrier Proteins - immunology | Colorectal Neoplasms - metabolism | Cytokines - biosynthesis | Life Sciences | Immunology
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5. Full Text Type III Interferons Produced by Human Placental Trophoblasts Confer Protection against Zika Virus Infection
by Bayer, Avraham and Lennemann, Nicholas J and Ouyang, Yingshi and Bramley, John C and Morosky, Stefanie and Marques, Ernesto Torres De Azeved and Cherry, Sara and Sadovsky, Yoel and Coyne, Carolyn B
Cell Host & Microbe, ISSN 1931-3128, 05/2016, Volume 19, Issue 5, pp. 705 - 712
During mammalian pregnancy, the placenta acts as a barrier between the maternal and fetal compartments. The recently observed association between Zika virus... 
IFNλ | placenta | Zika virus | type III interferon | virus | trophoblasts | CELLS | IN-VITRO | VIROLOGY | MICROBIOLOGY | DIFFERENTIATION | BRAIN | BRAZIL | PARASITOLOGY | LAMBDA | Interferons - biosynthesis | Pregnancy Complications, Infectious - virology | Humans | Fetal Diseases - prevention & control | Cercopithecus aethiops | Interferons - immunology | Microcephaly - virology | Interleukins - metabolism | Pregnancy Complications, Infectious - immunology | Interleukins - genetics | Microcephaly - immunology | Placenta - cytology | Microcephaly - prevention & control | Pregnancy Complications, Infectious - prevention & control | Female | Trophoblasts - cytology | Interleukins - pharmacology | Zika Virus Infection - prevention & control | Vero Cells | Zika Virus - immunology | Trophoblasts - metabolism | Placenta - immunology | Interferons - pharmacology | Cells, Cultured | Zika Virus - drug effects | Fetal Diseases - virology | Placenta - metabolism | Pregnancy | Trophoblasts - immunology | Animals | Fetal Diseases - immunology | Cell Line, Tumor | Zika Virus Infection - immunology | Virus diseases | Microbiology | Virus diseases in pregnancy | Interferon | Research institutes | Biological response modifiers | Health aspects | Virus | Trophoblasts | Placenta
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6. Full Text Sirt3 confers protection against neuronal ischemia by inducing autophagy: Involvement of the AMPK-mTOR pathway
by Dai, Shu-Hui and Chen, Tao and Li, Xia and Yue, Kang-Yi and Luo, Peng and Yang, Li-Kun and Zhu, Jie and Wang, Yu-Hai and Fei, Zhou and Jiang, Xiao-Fan
Free Radical Biology and Medicine, ISSN 0891-5849, 07/2017, Volume 108, pp. 345 - 353
Sirtuin3 (Sirt3) is a member of the silent information regulator 2 (Sir2) family of proteins located in mitochondria that influences almost every major aspect... 
Sirt3 | mTOR | AMPK | Ischemic stroke | Autophagy | OXIDATIVE STRESS | KINASE PATHWAY | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RAT CORTICAL-NEURONS | MECHANISMS | NEUROPROTECTION | CELL-DEATH | HYPOXIA-ISCHEMIA | ENDOCRINOLOGY & METABOLISM | 3-METHYLADENINE | INDUCED BRAIN-INJURY | Neurons - pathology | Up-Regulation | Sirtuin 3 - genetics | Sirtuin 3 - metabolism | Oxidative Stress | Signal Transduction | TOR Serine-Threonine Kinases - metabolism | Cells, Cultured | Brain Ischemia - metabolism | Rats | Rats, Sprague-Dawley | Hydrogen Peroxide - metabolism | Animals | Adenylate Kinase - metabolism | Membrane Potential, Mitochondrial | Beclin-1 - genetics | Glucose - metabolism | Neurons - metabolism | Beclin-1 - metabolism | Mitochondria - physiology | Proteins | Medical colleges | Neurosciences | Head injuries | Ischemia | Hydrogen | Neurons | Analysis | Electron microscopy
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7. Full Text Virus-like nanoparticle vaccine confers protection against Toxoplasma gondii
by Lee, Dong Hun and Lee, Su Hwa and Kim, Ah Ra and Quan, Fu Shi
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, p. e0161231
The inner membrane complex (IMC) of Toxoplasma gondii as a peripheral membrane system has unique and critical roles in parasite replication, motility and... 
IMMUNITY | CHALLENGE | RESPONSES | CELLS | REPLICATION | PARTICLES | MEMORY | MULTIDISCIPLINARY SCIENCES | MICE | INFECTION | INFLUENZA-VIRUS | Intestines - drug effects | Toxoplasma - immunology | Immunoglobulin A - biosynthesis | Vaccination | Protozoan Proteins - genetics | Toxoplasmosis, Animal - prevention & control | Intestines - immunology | CD4-CD8 Ratio | Immunoglobulin G - biosynthesis | Toxoplasmosis, Animal - parasitology | Feces - parasitology | Nanoparticles - therapeutic use | Intestines - parasitology | Female | Th1-Th2 Balance - drug effects | Brain - parasitology | Toxoplasmosis, Animal - mortality | Mice, Inbred C57BL | Nanoparticles - ultrastructure | Protozoan Vaccines - administration & dosage | Toxoplasma - pathogenicity | Antibodies, Protozoan - biosynthesis | Brain - drug effects | Vaccines, Virus-Like Particle - administration & dosage | Animals | Survival Analysis | Toxoplasmosis, Animal - immunology | Mice | Toxoplasma - drug effects | Brain - immunology | Protozoan Proteins - immunology | Toxoplasma | Immunoglobulins | Control | Viral vaccines | Physiological aspects | Causes of | Research | Health aspects | Brain | IgG antibody | CD8 antigen | Genes | Immunoglobulin G | Viruses | Infections | Lymphocytes T | Vaccines | Parasites | Immunoglobulin A | Nanoparticles | Lethal dose | Proteins | Replicating | Lymphocytes | Intestine | Rodents | Feces | Spleen | Protozoa | Cytokines | Cloning | CD4 antigen | Cysts | Microscopy | Influenza | Plasma cells | Budding
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8. Full Text Intermittent Fasting Confers Protection in CNS Autoimmunity by Altering the Gut Microbiota
by Cignarella, Francesca and Cantoni, Claudia and Ghezzi, Laura and Salter, Amber and Dorsett, Yair and Chen, Lei and Phillips, Daniel and Weinstock, George M and Fontana, Luigi and Cross, Anne H and Zhou, Yanjiao and Piccio, Laura
Cell Metabolism, ISSN 1550-4131, 06/2018, Volume 27, Issue 6, pp. 1222 - 1235.e6
Multiple sclerosis (MS) is more common in western countries with diet being a potential contributing factor. Here we show that intermittent fasting (IF)... 
intermittent fasting | experimental autoimmune encephalomyelitis | diet | multiple sclerosis | gut microbiota | OBESITY | MULTIPLE-SCLEROSIS | DIET | PROTEIN-COUPLED RECEPTOR | CHAIN FATTY-ACIDS | ENDOCRINOLOGY & METABOLISM | BODY-MASS INDEX | RISK | SCLEROSIS FUNCTIONAL COMPOSITE | SEGMENTED FILAMENTOUS BACTERIA | T-CELLS | CELL BIOLOGY | Autoimmunity | Medical colleges | Medical research | Multiple sclerosis | Fasting | Microbiota (Symbiotic organisms) | Analysis | Medicine, Experimental | T cells
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9. Full Text MICU1 Confers Protection from MCU-Dependent Manganese Toxicity
by Wettmarshausen, Jennifer and Goh, Valerie and Huang, Kai-Ting and Arduino, Daniela M and Tripathi, Utkarsh and Leimpek, Anja and Cheng, Yiming and Pittis, Alexandros A and Gabaldón, Toni and Mokranjac, Dejana and Hajnóczky, György and Perocchi, Fabiana
Cell Reports, ISSN 2211-1247, 11/2018, Volume 25, Issue 6, pp. 1425 - 1435.e7
The mitochondrial calcium uniporter is a highly selective ion channel composed of species- and tissue-specific subunits. However, the functional role of each... 
calcium | signaling | mitochondria | MICU1 | manganese | MCU | yeast | YEAST | EF-HAND | RAT-KIDNEY MITOCHONDRIA | CA2+ UPTAKE | ION | PROTEINS | IDENTIFICATION | MITOCHONDRIAL CALCIUM UNIPORTER | BINDING | LIVER MITOCHONDRIA | CELL BIOLOGY | Signaling | Mitochondria | Yeast | Calcium | Manganese
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10. Full Text Carbon Monoxide Confers Protection in Sepsis by Enhancing Beclin 1-Dependent Autophagy and Phagocytosis
by Lee, Seonmin and Lee, Seon-Jin and Coronata, Anna A and Fredenburgh, Laura E and Chung, Su Wol and Perrella, Mark A and Nakahira, Kiichi and Ryter, Stefan W and Choi, Augustine M.K
Antioxidants & Redox Signaling, ISSN 1523-0864, 01/2014, Volume 20, Issue 3, pp. 432 - 442
Aims: Sepsis, a systemic inflammatory response to infection, represents the leading cause of death in critically ill patients. However, the pathogenesis of... 
Forum Original Research Communications | PATHOGENESIS | APOPTOSIS | INHIBITION | HEME | GENE | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOCRINOLOGY & METABOLISM | NITRIC-OXIDE | RECEPTOR | EXPRESSION | DEFICIENCY | Beclin-1 | Wounds and Injuries - microbiology | Inflammation - pathology | Lung - microbiology | Phagocytosis - drug effects | Apoptosis - drug effects | Humans | Liver - microbiology | Carbon Monoxide - metabolism | Autophagy - drug effects | Apoptosis Regulatory Proteins - metabolism | Animals | Sepsis - pathology | Liver - drug effects | Inflammation - drug therapy | Sepsis - metabolism | Lung - drug effects | Apoptosis Regulatory Proteins - genetics | Wounds and Injuries - drug therapy | Cell Proliferation - drug effects | Mice | Punctures - adverse effects | Carbon Monoxide - administration & dosage | Physiological aspects | Autophagy (Cytology) | Sepsis | Carbon monoxide | Research | Phagocytosis
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11. Full Text Toll‐like receptor 9 inhibition confers protection from liver ischemia–reperfusion injury
by Bamboat, Zubin M and Balachandran, Vinod P and Ocuin, Lee M and Obaid, Hebroon and Plitas, George and DeMatteo, Ronald P
Hepatology, ISSN 0270-9139, 02/2010, Volume 51, Issue 2, pp. 621 - 632
Endogenous ligands such as high‐mobility group box 1 (HMGB1) and nucleic acids are released by dying cells and bind Toll‐like receptors (TLRs). Because TLR9... 
RAT-LIVER | CPG MOTIFS | SYSTEMIC AUTOIMMUNE-DISEASE | ACTIVATION | DENDRITIC CELLS | ENDOTHELIAL-CELLS | ENDOSOMAL TRANSLOCATION | MONOCLONAL-ANTIBODY | GASTROENTEROLOGY & HEPATOLOGY | HEPATIC ISCHEMIA/REPERFUSION INJURY | BACTERIAL-DNA | Reperfusion Injury - etiology | Liver - blood supply | Animals | Reperfusion Injury - prevention & control | Toll-Like Receptor 9 - antagonists & inhibitors | Mice, Inbred C57BL | Mice | Toll-Like Receptor 9 - physiology | HMGB1 Protein - physiology | HMGB1 Protein - antagonists & inhibitors | Bone marrow | Cytokines | Ischemia | Rodents | Hepatology | Immune system
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