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2017, Second edition., Advances in experimental medicine and biology, ISBN 9783319577319, Volume 993., xxix, 645 pages
This book thoroughly explores robotics in neurorehabilitative therapy, describing state-of-the-art applications of robotic devices for locomotion -- posture... 
Calcium channels | Cellular signal transduction | Intracellular calcium | Disease | Calcium Signaling | Cardiology | Nervous system | Robotics in medicine
Book
2004, Methods in molecular biology, ISBN 9781592596713, Volume 250., xi, 332
Mitogen-activated protein kinase (MAPK) signaling cascades initiate highly significant cellular processes and are involved in a large number of diseases,... 
Transduction du signal cellulaire | Protéine kinase | Methodology | Cellular signal transduction | Research | Recherche | Protein kinases | Méthodologie | Biochemistry, general | Biochemistry | Life Sciences
Book
Cell Metabolism, ISSN 1550-4131, 06/2016, Volume 23, Issue 6, pp. 1013 - 1021
Cellular senescence is a complex stress response that permanently arrests the proliferation of cells at risk for oncogenic transformation. However, senescent... 
INCREASED GLYCOLYSIS | CANCER-CELLS | PREMATURE SENESCENCE | DNA-DAMAGE | ENDOCRINOLOGY & METABOLISM | EXTENDS LIFE-SPAN | TUMOR-SUPPRESSOR | AUTOPHAGY | ONCOGENE-INDUCED SENESCENCE | REGULATES SENESCENCE | P53 | CELL BIOLOGY | Animals | Signal Transduction | Cellular Senescence | Humans | Mitochondria - metabolism | Autophagy | NAD - metabolism | Aging - metabolism | Prevention | Physiological aspects | Cancer
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 1/2012, Volume 347, Issue 1, pp. 11 - 20
Transforming growth factor-beta (TGFβ) is a key regulator of cell fate during embryogenesis and has also emerged as a potent driver of the... 
Human Genetics | Smads | Biomedicine | TAK1 | TRAF6 | Proteomics | TGFβ | Molecular Medicine | Non-Smads | DISTINCT MODES | TGF-BETA | PROTEIN | BETA-ACTIVATED KINASE-1 | PHOSPHATIDYLINOSITOL 3-KINASE | P38 MAPK | ERK MAP KINASE | MESENCHYMAL TRANSITION | CELL BIOLOGY | CROSS-TALK | II RECEPTOR | TGF beta | GTP Phosphohydrolases - metabolism | Receptors, Transforming Growth Factor beta - metabolism | Epithelial-Mesenchymal Transition - physiology | TNF Receptor-Associated Factor 6 - metabolism | Signal Transduction - physiology | MAP Kinase Kinase Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Integrins - metabolism | Smad Proteins - metabolism | Transforming Growth Factor beta - metabolism | Ubiquitin | Tumor necrosis factor | Ligases | Development and progression | Bone morphogenetic proteins | G proteins | Transforming growth factors | Mitogens | Tumors | Proteins | Phosphorylation | Cellular biology | Kinases | Indexing in process | Medical Biotechnology | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinsk bioteknologi | Medical Biotechnology (with a focus on Cell Biology (including Stem Cell Biology), Molecular Biology, Microbiology, Biochemistry or Biopharmacy) | Medicinsk bioteknologi (med inriktning mot cellbiologi (inklusive stamcellsbiologi), molekylärbiologi, mikrobiologi, biokemi eller biofarmaci)
Journal Article
Journal of Internal Medicine, ISSN 0954-6820, 03/2014, Volume 275, Issue 3, pp. 296 - 303
Recent trials of anti‐amyloid agents have not produced convincing improvements in clinical outcome in Alzheimer's disease; however, the reason for these poor... 
tau | amyloid | cell biology | genetics | Alzheimer's disease | Cell biology | Genetics | Tau | Amyloid | THERAPEUTICS | COMMON VARIANTS | COMPLEMENT ACTIVATION | DEPOSITION | ANTIBODY | BETA-PEPTIDE | MECHANISMS | AMYLOID PRECURSOR PROTEIN | APOLIPOPROTEIN-E | MEDICINE, GENERAL & INTERNAL | A-BETA | Brain - metabolism | Amyloid - genetics | Amyloid beta-Peptides - antagonists & inhibitors | Genetic Predisposition to Disease | Immunotherapy - methods | Amyloid - metabolism | Early Medical Intervention | Humans | Alzheimer Disease - metabolism | Alzheimer Disease - genetics | Genetic research | Amyloidosis | Cells | Brain | Neurosciences | cholesterol transport | Alzheimer disease | cholesterol synthesis | immunoglobulin G1 antibody | genetic association | bapineuzumab | follow up | solanezumab | phase 3 clinical trial (topic) | cellular distribution | phase 1 clinical trial (topic) | innate immunity | amyloid precursor protein | presenilin | Immunotherapy | Amyloid beta-Peptides | priority journal | Notch3 receptor | Neurovetenskaper | clusterin | cerebrospinal fluid level | clinical pathway | signal transduction | avagacestat | cholesterol metabolism | randomized controlled trial (topic) | risk factor | article | genetic variability | phase 2 clinical trial (topic) | pathogenesis | semagacestat | genetic analysis | complement classical pathway | apolipoprotein E | gamma secretase inhibitor | nonhuman | drug clearance
Journal Article
2005, ISBN 9781588293541, x, 214
Cutting-edge articles review our current understanding of lipid microdomain signaling mechanisms and their physiological and pathological importance. The book... 
Membrane Microdomains | Membrane lipids | pathology | Signal Transduction | physiology | Lipids | Cellular signal transduction | metabolism | Synapses | Apoptosis | Biology, life sciences | Life Sciences | Cell Biology
Book
1999, Modern genetics, ISBN 905702392X, Volume 5., xiii, 317
Apoptosis, or programmed cell death, is a necessary process by which a cell may die without adversely affecting its environment. It plays a crucial role in... 
Cell death | Apoptosis | Cellular signal transduction
Book