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2004, ISBN 3527309802
Web Resource
2004, ISBN 6610519692
Web Resource
2015, First edition., Methods in cell biology, ISBN 0128024496, Volume 129.
"This new volume of Methods in Cell Biology looks at methods for analyzing centrosomes and centrioles. Chapters cover such topics as methods to analyze... 
Centrosomes | Cell division | Centrioles | Centrosome
Web Resource
2015, First edition., Methods in cell biology, ISBN 0128024496, Volume 129.
"This new volume of Methods in Cell Biology looks at methods for analyzing centrosomes and centrioles. Chapters cover such topics as methods to analyze... 
Centrosomes | Cell division | Centrioles | Centrosome
Web Resource
Cell Reports, ISSN 2211-1247, 09/2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Index Medicus | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
Journal Article
Anticancer Research, ISSN 0250-7005, 06/2018, Volume 38, Issue 6, pp. 3393 - 3400
Background/Aim: Owing to the frequent observation of centrosome amplification in human cancers, cancer cells have a unique mechanism to suppress detrimental... 
Centrosome amplification | Radiosensitizer | Breast cancer | Radiotherapy | Centrosome clustering | centrosome amplification | IN-VITRO | AMPLIFICATION | INSTABILITY | ONCOLOGY | radiotherapy | breast cancer | radiosensitizer | INDUCTION | CIP2A | INHIBITOR | Humans | Spindle Apparatus - radiation effects | Cell Survival - genetics | Centrosome - drug effects | Centrosome - radiation effects | Breast Neoplasms - metabolism | Spindle Apparatus - metabolism | MCF-7 Cells | RNA Interference | Kinesin - genetics | Female | Griseofulvin - pharmacology | Fibroblasts - metabolism | Cell Division - genetics | Cell Line | Cell Survival - drug effects | Radiation-Sensitizing Agents - pharmacology | Cell Division - radiation effects | Cell Survival - radiation effects | Cell Division - drug effects | Centrosome - metabolism | Kinesin - metabolism | Phenanthrenes - pharmacology | Breast Neoplasms - genetics | Fibroblasts - radiation effects | Breast Neoplasms - pathology | Fibroblasts - drug effects | Cell Line, Tumor | Spindle Apparatus - drug effects | Biotechnology | Radiation effects | Radiosensitivity | Centrosomes | siRNA | Radiation therapy | Tumor cell lines | Clustering | Amplification | Ionizing radiation | Cell lines | Fibroblasts | Breast | Irradiation | Sensitivity enhancement | Division | Inhibition | Immunofluorescence | Spindles | Viability | Radiation damage | Cancer | Tumors | Index Medicus
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 6/2004, Volume 165, Issue 5, pp. 709 - 721
Humans with mutations in either DCX or LIS1 display nearly identical neuronal migration defects, known as lissencephaly. To define subcellular mechanisms, we... 
Neuroscience | Neurons | Microtubules | Centrosomes | Nuclear membrane | Antibodies | Retroviridae | Genetic mutation | Cells | Animal migration behavior | Nucleus | Doublecortin | Migration | Centrosome | Lis1 | CEREBELLAR GRANULE NEURONS | CYTOPLASMIC DYNEIN | doublecortin | VARIABLE PHENOTYPES | ASPERGILLUS-NIDULANS | Lis 1 | nucleus | centrosome | MICROTUBULE-ASSOCIATED PROTEIN | CORTICAL DEVELOPMENT | MISSENSE MUTATIONS | CEREBRAL-CORTEX | CELL BIOLOGY | DOUBLE CORTEX SYNDROME | migration | SUBCORTICAL BAND HETEROTOPIA | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Gene Expression Regulation, Developmental - genetics | Neurons - cytology | Brain - abnormalities | Cell Movement - genetics | Brain - metabolism | Centrosome - ultrastructure | Cell Differentiation - genetics | Microtubules - metabolism | Cell Nucleus - metabolism | Nervous System Malformations - metabolism | Microtubules - ultrastructure | Nervous System Malformations - physiopathology | 1-Alkyl-2-acetylglycerophosphocholine Esterase | Neurons - metabolism | Neuropeptides - genetics | Nervous System Malformations - genetics | Dyneins - metabolism | Brain - cytology | Cells, Cultured | Neuropeptides - metabolism | Up-Regulation - genetics | Cell Nucleus - ultrastructure | Mutation - genetics | Centrosome - metabolism | Macromolecular Substances | Animals | Microtubules - genetics | Mice | Dyneins - genetics | Research | Mutation | Cellular biology | Cell adhesion & migration | Index Medicus | migration; Lis1; doublecortin; centrosome; nucleus
Journal Article
Cell, ISSN 0092-8674, 06/2017, Volume 169, Issue 6, pp. 1066 - 1077.e10
Journal Article
11/2013
Robust bipolar spindle formation and faithful transmission of genetic material are vital to the maintenance of genome integrity and cellular homeostasis.... 
0379 | Mitosis | Mitotic Spindle | 0307 | Centrosome
Dissertation