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Biochimica et biophysica acta. Molecular and cell biology of lipids, ISSN 1388-1981, 2015, Volume 1851, Issue 6, pp. 844 - 856
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 7, p. e0180154
Ion channels regulate a variety of physiological processes and represent an important class of drug target. Among the many methods of studying ion channel... 
TARGET | DYE | KV1.3 | NA(V)1.7 | MODULATORS | MULTIDISCIPLINARY SCIENCES | EXTREME PAIN DISORDER | PATCH-CLAMP | MUTATIONS | Cricetulus | High-Throughput Screening Assays - instrumentation | NAV1.3 Voltage-Gated Sodium Channel - genetics | NAV1.1 Voltage-Gated Sodium Channel - genetics | NAV1.5 Voltage-Gated Sodium Channel - metabolism | NAV1.1 Voltage-Gated Sodium Channel - metabolism | NAV1.4 Voltage-Gated Sodium Channel - metabolism | Kv1.3 Potassium Channel - genetics | NAV1.2 Voltage-Gated Sodium Channel - metabolism | NAV1.3 Voltage-Gated Sodium Channel - metabolism | Patch-Clamp Techniques - methods | Kv1.3 Potassium Channel - deficiency | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Sodium Channels - metabolism | NAV1.7 Voltage-Gated Sodium Channel - genetics | NAV1.5 Voltage-Gated Sodium Channel - genetics | Drug Evaluation, Preclinical | Transgenes | Potassium Channel Blockers - pharmacology | CHO Cells | High-Throughput Screening Assays - methods | Gene Expression | NAV1.4 Voltage-Gated Sodium Channel - genetics | NAV1.2 Voltage-Gated Sodium Channel - genetics | Rats | Sodium Channel Blockers - pharmacology | Membrane Potentials - physiology | Animals | T-Lymphocytes - cytology | NAV1.6 Voltage-Gated Sodium Channel - metabolism | NAV1.7 Voltage-Gated Sodium Channel - metabolism | NAV1.6 Voltage-Gated Sodium Channel - genetics | Sodium Channels - genetics | Primary Cell Culture | Patch-Clamp Techniques - instrumentation | Ion channels | Information management | Drug discovery | T cells | Modules | Electrophysiology | Patching | Lymphocytes T | Boundaries | Assaying | Channels | Step voltage | Immunology | Pain | Lymphocytes | Sodium channels (voltage-gated) | Physiology | Potassium channels (voltage-gated) | Gold | Platforms | Data points | Automation | Cloning | Pharmacology | Sodium | Antigen-presenting cells | Ligands | Mutation | Recording | Potassium
Journal Article
Current Medicinal Chemistry, ISSN 0929-8673, 04/2013, Volume 20, Issue 10, pp. 1241 - 1285
Ion channel targeted drugs have always been related with either the central nervous system (CNS), the peripheral nervous system, or the cardiovascular system.... 
ASIC | TRP | Central nervous system | P2X | KCNQ | NMDA | CNS | Ion channels | CHEMISTRY, MEDICINAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | GATED SODIUM-CHANNELS | SUBTYPE-SELECTIVE AGONIST | D-ASPARTATE RECEPTOR | ion channels | ACID-SENSING ION-CHANNEL-1 | central nervous system | GLYCINE ANTAGONIST GV150526 | TARANTULA TOXIN PSALMOTOXIN-1 | TRAUMATIC BRAIN-INJURY | ISCHEMIC CELL-DEATH | PHARMACOLOGY & PHARMACY | PERIPHERAL NEUROPATHIC PAIN | TRANSGENIC MOUSE MODEL | Ligand-Gated Ion Channels - antagonists & inhibitors | Transient Receptor Potential Channels - antagonists & inhibitors | Calcium Channels - metabolism | Humans | Calcium Channel Blockers - therapeutic use | Potassium Channel Blockers - therapeutic use | Sodium Channel Blockers - pharmacology | Calcium Channel Blockers - pharmacology | Ion Channels - antagonists & inhibitors | Potassium Channels - metabolism | Animals | Ion Channels - metabolism | Sodium Channels - chemistry | Calcium Channels - chemistry | Central Nervous System Diseases - drug therapy | Sodium Channels - metabolism | Calcium Channel Blockers - chemistry | Sodium Channel Blockers - therapeutic use | Potassium Channels - chemistry | Sodium Channel Blockers - chemistry | Transient Receptor Potential Channels - metabolism | Ligand-Gated Ion Channels - metabolism | Potassium Channel Blockers - chemistry | Potassium Channel Blockers - pharmacology
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 2004, Volume 24, Issue 26, pp. 5922 - 5930
We used a model system in which dopaminergic (DA) neurons from embryonic rat mesencephalon undergo spontaneous and selective degeneration as they develop in... 
Excitability | GDNF | Sodium | Channel | Trophic | Dopaminergic | TYROSINE-HYDROXYLASE | POTASSIUM CHANNELS | NEUROTROPHIC FACTOR | channel | trophic | NEUROSCIENCES | sodium | CYCLIC-AMP | excitability | CALCIUM-CHANNELS | SIGNALING PATHWAY | MEMBRANE-PROPERTIES | CENTRAL-NERVOUS-SYSTEM | dopaminergic | SMALL-CONDUCTANCE | PARKINSONS-DISEASE | Mesencephalon - cytology | Sodium-Potassium-Exchanging ATPase - physiology | Rats, Wistar | Apoptosis - drug effects | gamma-Aminobutyric Acid - metabolism | Cells, Cultured - cytology | Neurons - cytology | Sodium Channels - physiology | Apamin - pharmacology | Nerve Growth Factors - pharmacology | Neuroprotective Agents - pharmacology | Glial Cell Line-Derived Neurotrophic Factor | Ion Transport - drug effects | Neurons - metabolism | Aspirin - pharmacology | Tetrodotoxin - pharmacology | Neurons - drug effects | Dopamine - metabolism | Ouabain - pharmacology | Potassium Channel Blockers - pharmacology | Sodium - pharmacology | Cells, Cultured - drug effects | Nerve Tissue Proteins - physiology | Nifedipine - pharmacology | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Biological Transport, Active - drug effects | Rats | Sodium Channel Blockers - pharmacology | Sodium Channels - drug effects | Calcium Channel Blockers - pharmacology | Nerve Tissue Proteins - drug effects | Veratridine - pharmacology | Scorpion Venoms - pharmacology | Animals | Calcium Channels, T-Type - drug effects | Serotonin - metabolism | Calcium Channel Agonists - pharmacology | Cells, Cultured - metabolism | Potassium Channels, Calcium-Activated - antagonists & inhibitors | Ion Channel Gating | Sodium - physiology | Tetrodotoxin | Neuroprotective Agents | Veratridine | Mesencephalon | Neurons and Cognition | Ouabain | Calcium Channel Agonists | Calcium Channels, T-Type | Potassium Channels, Calcium-Activated | Life Sciences | Biological Transport, Active | Sodium-Potassium-Exchanging ATPase | Ion Transport | Potassium Channel Blockers | gamma-Aminobutyric Acid | Aspirin | Apamin | Dopamine | Serotonin | Neurons | Sodium Channels | Cells, Cultured | Nerve Tissue Proteins | Nifedipine | Scorpion Venoms | Calcium Channel Blockers | Nerve Growth Factors | Sodium Channel Blockers | Apoptosis | Plasticity | Development | Repair
Journal Article
Circulation. Arrhythmia and electrophysiology, ISSN 1941-3084, 2017, Volume 10, Issue 4, pp. e003560 - e003560
BACKGROUND—The widely used macrolide antibiotic azithromycin increases risk of cardiovascular and sudden cardiac death, although the underlying mechanisms are... 
calcium channel | sodium channels | mice | potassium channels | pharmacology | TORSADES-DE-POINTES | SODIUM | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | MACROLIDE | TRAFFICKING | ARRHYTHMIA | QT-INTERVAL PROLONGATION | IN-VIVO | CHANNELS | MUTATIONS | Arrhythmias, Cardiac - chemically induced | KCNQ1 Potassium Channel - genetics | Cricetulus | Humans | KCNQ1 Potassium Channel - metabolism | Azithromycin - toxicity | NAV1.5 Voltage-Gated Sodium Channel - metabolism | Arrhythmias, Cardiac - physiopathology | Action Potentials | Dose-Response Relationship, Drug | Young Adult | Heart Rate - drug effects | Transfection | Potassium Channels, Voltage-Gated - metabolism | Time Factors | NAV1.5 Voltage-Gated Sodium Channel - drug effects | HEK293 Cells | KCNQ1 Potassium Channel - antagonists & inhibitors | Female | Potassium Channels, Inwardly Rectifying - antagonists & inhibitors | Potassium Channel Blockers - toxicity | Anti-Bacterial Agents - toxicity | NAV1.5 Voltage-Gated Sodium Channel - genetics | CHO Cells | Potassium Channels, Voltage-Gated - genetics | Sodium Channel Blockers - toxicity | Arrhythmias, Cardiac - metabolism | Rabbits | Mice, Inbred C57BL | Potassium Channels, Inwardly Rectifying - genetics | Potassium Channels, Voltage-Gated - antagonists & inhibitors | Telemetry | Calcium Channel Blockers - toxicity | Animals | Calcium Channels, L-Type - genetics | Myocytes, Cardiac - drug effects | Calcium Channels, L-Type - drug effects | Myocytes, Cardiac - metabolism | Calcium Channels, L-Type - metabolism | Potassium Channels, Inwardly Rectifying - metabolism | Electrocardiography, Ambulatory | Ion Channels | HL-1 cells | Membrane Transport | Electrophysiology | azithromycin | proarrhythmia | Translational Studies | mouse | Arrhythmias | Basic Science Research
Journal Article
Circulation Research, ISSN 0009-7330, 02/2007, Volume 100, Issue 3, pp. 342 - 353
A large body of evidence has accrued indicating that voltage-gated Ca channel subtypes, including L-, T-, N-, and P/Q-type, are present within renal vascular... 
Renal microcirculation | Mibefradil | channels | Afferent arteriole | channel blockers | Efferent arteriole | Renal disease | Voltage-dependent Ca | Efonidipine | Ca2+ channel blockers | N-TYPE | CARDIAC & CARDIOVASCULAR SYSTEMS | EFFERENT ARTERIOLES | efonidipine | SPONTANEOUSLY HYPERTENSIVE-RATS | RHO-KINASE INHIBITOR | renal microcirculation | ANGIOTENSIN-II | T-TYPE | renal disease | mibefradil | DEPENDENT CALCIUM-CHANNELS | CARDIAC L-TYPE | RENAL MICROVASCULAR CONSTRICTION | efferent arteriole | voltage-dependent Ca2+ channels | PERIPHERAL VASCULAR DISEASE | afferent arteriole | HEMATOLOGY | IN-VIVO VISUALIZATION | Arterioles - physiology | Kidney - physiology | Protein Subunits | Antihypertensive Agents - pharmacology | Kidney - blood supply | Humans | Calcium Channel Blockers - therapeutic use | Hypertension - drug therapy | Antihypertensive Agents - classification | Calcium Channels - physiology | Calcium Channels, T-Type - chemistry | Neurotransmitter Agents - secretion | Calcium Channels - drug effects | Calcium Channels, N-Type - drug effects | Cardiovascular Diseases - physiopathology | Kidney - drug effects | Calcium Channels - classification | Rats | Calcium Channels, L-Type - physiology | Antihypertensive Agents - therapeutic use | Arterioles - drug effects | Disease Progression | Antihypertensive Agents - adverse effects | Mice, Knockout | Calcium Signaling - drug effects | Diabetes Mellitus - physiopathology | Models, Biological | Calcium Channels - chemistry | Calcium Channels, T-Type - drug effects | Mice | Vasodilation - drug effects | Hydronephrosis - physiopathology | Calcium Channel Blockers - adverse effects | Calcium Signaling - physiology | Cardiovascular Diseases - drug therapy | Renal Circulation - physiology | Microcirculation - drug effects | Microcirculation - physiology | Renal Circulation - drug effects | Blood Pressure - drug effects | Calcium Channels, L-Type - chemistry | Kidney Diseases - metabolism | Calcium Channels, T-Type - physiology | Renin - secretion | Aldosterone - physiology | Kidney Diseases - drug therapy | Renin-Angiotensin System - physiology | Calcium Channel Blockers - pharmacology | Hypertension - physiopathology | Animals | Calcium Channels, L-Type - drug effects | Calcium Channels, N-Type - physiology | Calcium Channels, N-Type - chemistry
Journal Article