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2015, Topics in Medicinal Chemistry, ISBN 3319140604, Volume 14
Web Resource
American journal of respiratory cell and molecular biology, ISSN 1044-1549, 11/2015, Volume 53, Issue 5, pp. 676 - 688
Macrophages are dynamic cells that mature under the influence of signals from the local microenvironment into either classically (M1) or alternatively (M2)... 
classically activated macrophages (CAM/M1) | Phagocytosis/ endocytosis | phagocytosis/endocytosis | phenotypic stability | alternatively activated macrophages (AAM/M2) | reprogramming of polarization | PHAGOCYTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MONOCYTES | DIFFERENTIAL REGULATION | MURINE | CELL BIOLOGY | GM-CSF | RESPIRATORY SYSTEM | THP-1 CELLS | MANNOSE | RECEPTOR-ACTIVITY | POLARIZATION | EXPRESSION | Chemokines, CC - secretion | Gene Expression - drug effects | Monocytes - cytology | Humans | Monocytes - immunology | Chemokine CCL5 - secretion | Chemokine CCL17 - secretion | Chemokine CXCL10 - biosynthesis | Interleukin-4 - pharmacology | Chemokine CCL17 - biosynthesis | Gene Expression - immunology | Chemokines, CC - biosynthesis | Interleukin-8 - secretion | Cell Differentiation | Interleukin-1beta - biosynthesis | Macrophages - immunology | Endocytosis - immunology | Interleukin-1beta - secretion | Interleukin-8 - biosynthesis | Endocytosis - drug effects | Tumor Necrosis Factor-alpha - secretion | Immunophenotyping | Macrophages - cytology | Interleukin-13 - pharmacology | Monocytes - drug effects | Chemokine CCL5 - biosynthesis | Chemokine CXCL10 - secretion | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Primary Cell Culture | Macrophage Activation - drug effects | Tumor Necrosis Factor-alpha - biosynthesis | Interferon-gamma - pharmacology | Studies | Biology | Immunology | Blood & organ donations | Cytokines | Gene expression | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 11/2015, Volume 527, Issue 7577, pp. 249 - 253
Epigenetic silencing including histone modifications and DNA methylation is an important tumorigenic mechanism. However, its role in cancer immunopathology and... 
Polycomb Repressive Complex 2 - antagonists & inhibitors | CD8-Positive T-Lymphocytes - cytology | Immunotherapy - methods | Prognosis | Histones - chemistry | Chemokine CXCL9 - biosynthesis | Humans | Ovarian Neoplasms - pathology | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | Th1 Cells - immunology | DNA (Cytosine-5-)-Methyltransferases - metabolism | Th1 Cells - metabolism | Chemokine CXCL10 - biosynthesis | Chemokine CXCL10 - immunology | Female | Epigenesis, Genetic - drug effects | Lysine - metabolism | Tumor Cells, Cultured | Chemokines - immunology | Tumor Escape - immunology | Chemokines - biosynthesis | DNA (Cytosine-5-)-Methyltransferase 1 | Gene Silencing | Chemokine CXCL9 - immunology | Chemokine CXCL9 - genetics | Chemokines - genetics | Enhancer of Zeste Homolog 2 Protein | Ovarian Neoplasms - enzymology | Xenograft Model Antitumor Assays | B7-H1 Antigen - metabolism | Animals | Chemokine CXCL10 - genetics | Ovarian Neoplasms - therapy | Mice | Histones - metabolism | CD8-Positive T-Lymphocytes - immunology | Polycomb Repressive Complex 2 - metabolism | DNA Methylation - drug effects | Lymphocytes, Tumor-Infiltrating - immunology | Ovarian Neoplasms - immunology | Index Medicus | checkpoint | CXCL9 | histone modification | chemotherapy | epigenetics | Chemokine | CXCL10 | DNMT | T cell therapy | DNA methylation | PD-L1 | B7-H1 | cancer | trafficking | PD-1 | EZH2
Journal Article
The Journal of Immunology, ISSN 0022-1767, 06/2005, Volume 174, Issue 12, pp. 8183 - 8190
Journal Article
Diabetes Care, ISSN 0149-5992, 05/2010, Volume 33, Issue 5, pp. 1103 - 1108
OBJECTIVE - In view of the previously described anti-inflammatory effects of insulin, we investigated the potential suppressive effect of insulin on plasma... 
MIGRATION | INFLAMMATION | DISEASE | ENDOCRINOLOGY & METABOLISM | MONONUCLEAR-CELLS | ATHEROSCLEROSIS | SUBSETS | DEFICIENT MICE | KAPPA-B | EXPRESSION | ADIPOSE-TISSUE | Receptors, CCR2 - genetics | Interleukin-8 - genetics | Chemokines - blood | Obesity - immunology | Receptors, Chemokine - blood | Humans | Middle Aged | Chemokine CCL4 - genetics | Receptors, CCR5 - genetics | Insulin - blood | Obesity - blood | Inflammation - blood | Chemokine CCL5 - blood | Hypoglycemic Agents - blood | Chemokine CXCL12 - genetics | Chemokine CX3CL1 - blood | Hypoglycemic Agents - administration & dosage | Inflammation - drug therapy | Diabetes Mellitus, Type 2 - immunology | Gene Expression - immunology | Adult | Receptors, CXCR4 - genetics | Chemokine CCL4 - blood | Interleukin-8 - blood | Receptors, Chemokine - genetics | Immunosuppressive Agents - administration & dosage | Receptors, CCR5 - blood | Chemokine CCL11 - genetics | Chemokine CX3CL1 - genetics | Insulin - administration & dosage | Inflammation - immunology | Chemokines - genetics | Diabetes Mellitus, Type 2 - blood | Chemokine CCL5 - genetics | Chemokine CXCL12 - blood | Receptors, CCR2 - blood | Infusions, Intravenous | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | CX3C Chemokine Receptor 1 | Chemokine CCL11 - blood | Receptors, CXCR4 - blood | Physiological aspects | Dosage and administration | Insulin | Chemokine receptors | Chemokines | Methods | Diabetes therapy | Plasma | Pathogenesis | Rodents | Mortality | Atherosclerosis | Cardiovascular disease | Insulin resistance | Bone density | Diabetes | Blood | Index Medicus | Original Research
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2011, Volume 286, Issue 9, pp. 7214 - 7226
The Ca2+-binding protein of the EF-hand type, S100B, is abundantly expressed in and secreted by astrocytes, and release of S100B from damaged astrocytes occurs... 
CYTOPLASMIC DOMAIN | KAPPA-B ACTIVATION | IN-VITRO | NEURITE OUTGROWTH | INFLAMMATORY RESPONSES | BRAIN-INJURY | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | NITRIC-OXIDE | GROWTH-FACTOR | GLYCATION END-PRODUCTS | Microglia - metabolism | Chemokine CCL3 - immunology | Nerve Growth Factors - metabolism | S100 Proteins - immunology | Cell Movement - immunology | S100 Proteins - genetics | Chemokine CXCL12 - genetics | Microglia - immunology | Chemokine CCL3 - genetics | Calcium-Binding Proteins - immunology | Cattle | Encephalitis - metabolism | Chemokine CCL3 - metabolism | Chemokine CCL5 - metabolism | Receptors, Immunologic - immunology | Receptor for Advanced Glycation End Products | Chemokine CXCL12 - immunology | Chemokines - immunology | Calcium-Binding Proteins - metabolism | Microglia - cytology | Recombinant Proteins - metabolism | Cell Line | S100 Calcium Binding Protein beta Subunit | Mice, Inbred C57BL | Rats | Encephalitis - immunology | S100 Proteins - metabolism | Nerve Growth Factors - immunology | Recombinant Proteins - genetics | Chemokines - genetics | Animals | Carrier Proteins - metabolism | Chemokine CXCL12 - metabolism | Recombinant Proteins - immunology | Up-Regulation - immunology | Nerve Growth Factors - genetics | Chemokine CCL5 - genetics | Cytoskeleton - metabolism | Chemokines - metabolism | Mice | Receptors, Immunologic - genetics | Chemokine CCL5 - immunology | Receptors, Immunologic - metabolism | Calcium-Binding Proteins - genetics | Index Medicus | Immunology | Defensins | Signal Transduction | Neurodegeneration | Migration | Calcium-binding Proteins | Innate Immunity | RAGE | S100B | Chemokines | Microglia
Journal Article
Oral Diseases, ISSN 1354-523X, 11/2011, Volume 17, Issue 8, pp. 801 - 807
OBJECTIVE: Sjogren's syndrome is a chronic autoimmune disorder characterized by progressive lymphocytic infiltration within the salivary and lacrimal glands.... 
Innate immunity | Mouse | Sjögren's syndrome | Sialoadenitis | CELLS | Sjogren's syndrome | DOUBLE-STRANDED-RNA | SIALADENITIS | RECOGNITION | RIG-I | MECHANISMS | sialoadenitis | TOLL-LIKE RECEPTOR-3 | PATHOGENESIS | mouse | innate immunity | AUTOIMMUNE PANCREATITIS | DENTISTRY, ORAL SURGERY & MEDICINE | MICE | Monocyte Chemoattractant Proteins - drug effects | Chemokine CCL11 - analysis | Chemokine CXCL13 - analysis | Dendritic Cells - pathology | Sjogren's Syndrome - immunology | Killer Cells, Natural - pathology | CD4-Positive T-Lymphocytes - pathology | Chemokines, CC - analysis | Chemokine CXCL10 - analysis | Flow Cytometry | Chemokine CXCL10 - drug effects | Chemokine CCL4 - drug effects | Dendritic Cells - drug effects | Female | Chemokines, CXC - analysis | Chemokines, CXC - drug effects | Chemokine CCL4 - analysis | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Chemokines, CC - drug effects | Poly I-C - pharmacology | Sjogren's Syndrome - pathology | Immunity, Innate - drug effects | Submandibular Gland Diseases - pathology | Submandibular Gland Diseases - immunology | Toll-Like Receptor 3 - agonists | Chemokine CCL7 - analysis | Mice, Inbred NZB | Chemokine CCL3 - analysis | Chemokine CCL3 - drug effects | Sialadenitis - pathology | Immunity, Innate - immunology | Animals | Chemokine CCL7 - drug effects | Monocyte Chemoattractant Proteins - analysis | Chemokine CCL11 - drug effects | Killer Cells, Natural - drug effects | Mice | Chemokine CXCL13 - drug effects | CD4-Positive T-Lymphocytes - drug effects | Chemokine CCL2 - analysis | Chemokine CCL2 - drug effects | Sialadenitis - immunology | Immunohistochemistry | Dendritic cells | Analysis | Genes | Disease susceptibility | T cells | Gene expression | Dentistry
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 02/2004, Volume 94, Issue 2, pp. 253 - 261
ABSTRACT—Increasing evidence supports the involvement of inflammation in the early phases of atherogenesis. Recruitment of leukocytes within the vascular wall,... 
Atherogenesis | Leukocytes | Chemokine receptor antagonists | Chemokines | Vascular cells | atherogenesis | chemokine receptor antagonists | CARDIAC & CARDIOVASCULAR SYSTEMS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INDUCED LUNG INJURY | LESION FORMATION | DEFICIENT MICE | chemokines | KNOCKOUT MICE | vascular cells | IN-VIVO | leukocytes | MACROPHAGE INFLAMMATORY PROTEIN-2 | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | CC-CHEMOKINES | LYMPHOCYTES | Cholesterol, Dietary - toxicity | Apolipoproteins E - deficiency | Vasculitis - pathology | Male | Vasculitis - prevention & control | Chemokine CCL5 - analogs & derivatives | Chemotaxis, Leukocyte - drug effects | Receptors, Chemokine - biosynthesis | Receptors, LDL - deficiency | Chemokine CCL5 - metabolism | Receptors, Chemokine - antagonists & inhibitors | Receptors, CCR5 - physiology | Receptors, Chemokine - genetics | Receptors, Chemokine - drug effects | CCR5 Receptor Antagonists | Chemokines - biosynthesis | Aorta - drug effects | Mice, Inbred C57BL | RNA, Messenger - genetics | Chemokines - genetics | Mice, Knockout | Receptors, Chemokine - deficiency | Aorta - pathology | Gene Expression Regulation - drug effects | Arteriosclerosis - prevention & control | Animals | Chemokine CCL5 - pharmacology | Receptors, Chemokine - physiology | Endothelium, Vascular - metabolism | Apolipoproteins E - genetics | Protein Binding | Endothelium, Vascular - pathology | Mice | Receptors, CCR1 | Arteriosclerosis - pathology | Receptors, CCR2 | Index Medicus
Journal Article
Cellular Immunology, ISSN 0008-8749, 2011, Volume 270, Issue 2, pp. 164 - 171
► The chemokine receptors CCR1, CCR5, and CXCR2 are functionally expressed in primary rat microglia. ► The chemokines CCL2, CCL3, CCL5, and CXCL1 induce... 
Immunomodulation | CCL5 | CCL3 | CCL2 | CXCL1 | Chemokines | Microglia | MULTIPLE-SCLEROSIS LESIONS | ALZHEIMERS-DISEASE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | IMMUNOLOGY | CELL BIOLOGY | NITRIC-OXIDE | BETA-CHEMOKINES | CENTRAL-NERVOUS-SYSTEM | CHEMOKINE RECEPTORS | CCR5 | EXPRESSION | BRAIN | Microglia - metabolism | Chemokine CXCL1 - genetics | DNA Primers - genetics | RNA, Messenger - metabolism | Chemokine CXCL1 - pharmacology | Microglia - immunology | Chemokine CCL3 - genetics | Inflammation Mediators - pharmacology | Base Sequence | Inflammation Mediators - metabolism | Chemokine CCL3 - metabolism | Chemokine CCL2 - metabolism | Chemokine CCL5 - metabolism | Chemokine CXCL1 - metabolism | Recombinant Proteins - metabolism | Nitric Oxide - biosynthesis | Insulin-Like Growth Factor I - biosynthesis | Phagocytosis - drug effects | Microglia - drug effects | RNA, Messenger - genetics | Rats | Chemokine CCL2 - genetics | Chemokine CCL3 - pharmacology | Recombinant Proteins - genetics | Chemotaxis - drug effects | Recombinant Proteins - pharmacology | Rats, Sprague-Dawley | Animals | Chemokine CCL5 - pharmacology | Chemokine CCL2 - pharmacology | Interleukin-10 - genetics | Receptors, Interleukin-8B - metabolism | Chemokine CCL5 - genetics | Receptors, CCR1 - metabolism | Interleukin-10 - biosynthesis | In Vitro Techniques | Index Medicus
Journal Article
Immunological Reviews, ISSN 0105-2896, 05/2019, Volume 289, Issue 1, pp. 5 - 8
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, pp. e0172821 - e0172821
The IFN-gamma-inducible chemokines CXCL9, CXCL10, and CXCL11 play a key role in many inflammatory conditions, particularly those mediated by T cells.... 
LICHEN-PLANUS | ATTRACTANT CHEMOKINE | INTERFERON-GAMMA | CXC-CHEMOKINE | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | CELL ALPHA CHEMOATTRACTANT | I-TAC | CONTACT HYPERSENSITIVITY | CD8(+) T-CELLS | RECEPTOR CXCR3 | Chemokine CXCL11 - genetics | Lichen Planus, Oral - metabolism | Humans | RNA, Messenger - metabolism | Lichen Planus, Oral - genetics | Lichen Planus, Oral - immunology | Lichen Planus, Oral - pathology | T-Lymphocytes - drug effects | Mouth Mucosa - pathology | Receptors, CXCR3 - metabolism | RNA, Messenger - genetics | Chemokines, CXC - genetics | Organ Specificity | Chemokine CXCL9 - genetics | Keratinocytes - immunology | Gene Expression Regulation - drug effects | Cell Movement - drug effects | Keratinocytes - drug effects | T-Lymphocytes - cytology | Chemokine CXCL10 - genetics | Keratinocytes - metabolism | Chemokine CXCL9 - metabolism | Chemokines, CXC - metabolism | Cell Line, Tumor | Chemokine CXCL11 - metabolism | Chemokine CXCL10 - metabolism | Interferon-gamma - pharmacology | Keratinocytes | Genetic aspects | Research | Lichen planus | Chemokines | Flow cytometry | Cell culture | Disease | Pathogenesis | CD45RA antigen | Mucosa | Fluorescence | Lymphocytes T | Family medical history | Tissues | Immunological diseases | Immunology | Lymphocytes | Rodents | Fibroblasts | Enzyme-linked immunosorbent assay | Cytokines | Inflammation | Chemotaxis | Patients | CD4 antigen | Oral hygiene | γ-Interferon | CXCL10 protein | Ligands | CXCL11 protein | Interferon | Cancer | Index Medicus
Journal Article
Cytokine, ISSN 1043-4666, 12/2011, Volume 56, Issue 3, pp. 573 - 580
► Adiponectin induces the CC chemokines CCL2, -3, -4 and -5 in human monocytes. ► Surface abundance of CCR2 and CCR5 is reduced while CCR1 is not affected. ►... 
CC-chemokines | Obesity | CC-chemokine receptors | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | IMMUNOLOGY | CELL BIOLOGY | INHIBITION | INSULIN-RESISTANCE | DISEASE | CHEMOATTRACTANT PROTEIN-1 | MICE | CHEMOKINE RECEPTORS | HEPATIC STEATOSIS | EXPRESSION | ADIPOSE-TISSUE | Chemokine CCL3 - secretion | Chemokine CCL4 - secretion | Chemokines, CC - blood | Chemokines, CC - secretion | Chemokine CCL2 - secretion | Monocytes - cytology | Humans | Middle Aged | Chemokine CCL4 - genetics | Body Weight - drug effects | Male | Monocytes - immunology | RNA, Messenger - metabolism | Chemokine CCL5 - secretion | Chemokine CCL5 - blood | Receptors, CCR5 - metabolism | Chemokine CCL3 - genetics | Diabetes Mellitus, Type 2 - immunology | Chemokine CCL3 - blood | Adult | Cell Membrane - metabolism | Chemokine CCL4 - blood | Cell Membrane - drug effects | Monocytes - secretion | Adiponectin - pharmacology | Subcellular Fractions - immunology | Subcellular Fractions - drug effects | RNA, Messenger - genetics | Cells, Cultured | Overweight - immunology | Chemokine CCL2 - genetics | Chemokine CCL2 - blood | Monocytes - drug effects | Up-Regulation - drug effects | Diabetes Mellitus, Type 2 - blood | Receptors, CCR2 - metabolism | Overweight - blood | Chemokine CCL5 - genetics | Chemokines, CC - genetics | Aged | Type 2 diabetes | Index Medicus
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 02/2013, Volume 304, Issue 3, pp. 162 - 169