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Journal of Neurophysiology, ISSN 0022-3077, 12/2016, Volume 116, Issue 6, pp. 2950 - 2960
Journal Article
Nature, ISSN 0028-0836, 06/2007, Volume 447, Issue 7148, pp. 1081 - 1086
Why dopamine-containing neurons of the brain's substantia nigra pars compacta die in Parkinson's disease has been an enduring mystery. Our studies suggest that... 
PATHOGENESIS | SUBSTANTIA-NIGRA NEURONS | STRIATAL LEVEL | CHOLINERGIC INTERNEURONS | MULTIDISCIPLINARY SCIENCES | CA2+ CHANNELS | CALCIUM-ANTAGONISTS | MECHANISMS | MITOCHONDRIAL-DNA DELETIONS | MEDIUM SPINY NEURONS | MIDBRAIN DOPAMINERGIC-NEURONS | Neurons - pathology | Antiparkinson Agents - pharmacology | Substantia Nigra - pathology | Calcium - metabolism | Male | Neurons - cytology | Electric Conductivity | Parkinson Disease - drug therapy | Substantia Nigra - metabolism | Substantia Nigra - cytology | Gene Deletion | Rotenone - pharmacology | Aging | Neurons - metabolism | Parkinson Disease - metabolism | Neurons - drug effects | Dopamine - metabolism | Disease Models, Animal | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Dendrites - metabolism | Parkinson Disease - pathology | Calcium - pharmacology | Mice, Inbred C57BL | Mitochondria - drug effects | Disease Progression | Parkinson Disease - prevention & control | Animals | Calcium Channels, L-Type - genetics | Calcium Channels, L-Type - deficiency | Mice | Calcium Channels, L-Type - metabolism | Models, Neurological | Parkinson's disease | Brain stem | Rejuvenation | Physiological aspects | Development and progression | Research | Dopaminergic mechanisms | Medical research | Models | Brain research | Neurons | Rodents | Parkinsons disease | Biomedical materials | Progressions | In vivo testing | In vivo tests | Parkinson disease | Strategy | Channels
Journal Article