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Neoplasia, ISSN 1476-5586, 08/2019, Volume 21, Issue 8, pp. 740 - 751
Myxoid liposarcoma is a malignant lipogenic tumor that develops in deep soft tissues. While local control rates are good, current chemotherapy options remain... 
TRANSFORMATION | REARRANGEMENT | DOMAIN | ONCOLOGY | ONCOGENIC FUSION | EWS-FLI1 | BAF COMPLEXES | GENES | CHOP | CANCER | RNA-BINDING PROTEIN
Journal Article
by Liu, XY and Sun, HL and Yu, M and Liu, J and Yang, BN and Wu, YR and Wang, JW
JOURNAL OF CELLULAR PHYSIOLOGY, ISSN 0021-9541, 07/2019, Volume 234, Issue 7, pp. 11602 - 11609
DDIT3 is of great importance in endoplasmic reticulum stress and is involved in many inflammatory diseases and mineralization processes. The cementum protects... 
APOPTOSIS | PHYSIOLOGY | signal transduction | CELLS IN-VITRO | ER STRESS | IDH1 | CEMENTUM | SUFFICIENT | CELL BIOLOGY | DNA damage-inducible transcript 3 | GENE | gene regulation | MUTATION | nuclear factor-B (NF-kappa B) | cementogenesis | CHOP | DIFFERENTIATION | isocitrate dehydrogenase 1 (IDH1) | RNA
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 10/2017, Volume 23, Issue 20, pp. 6227 - 6239
Purpose: Myxoid liposarcoma is an aggressive disease with particular propensity to develop hematogenic metastases. Over 90% of myxoid liposarcoma are... 
SYNOVIAL SARCOMA | ONCOLOGY | PATHWAY | CELL LIPOSARCOMA | IN-VIVO | GROWTH | RECEPTOR | CHOP | REFRACTORY EWING SARCOMA | SOFT-TISSUE SARCOMA | HUMAN CANCER | Liposarcoma, Myxoid - metabolism | Receptor, IGF Type 1 - metabolism | Oncogene Proteins, Fusion - metabolism | Phosphorylation | Humans | Middle Aged | Liposarcoma, Myxoid - pathology | Male | Phosphatidylinositol 3-Kinases - metabolism | Molecular Targeted Therapy | Gene Knockdown Techniques | Young Adult | Adult | Female | Antineoplastic Agents - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Cell Survival - drug effects | Gene Expression | Tumor Burden | Receptor, IGF Type 1 - genetics | Xenograft Model Antitumor Assays | Animals | Liposarcoma, Myxoid - drug therapy | Signal Transduction - drug effects | Oncogene Proteins, Fusion - genetics | Biomarkers | Cell Line, Tumor | Aged | Mice | Apoptosis | Biotechnology | Transcription | Pathogenesis | Fibrosarcoma | AKT protein | Insulin-like growth factors | Kinases | Inactivation | Metastases | Signal transduction | FUS protein | Insulin-like growth factor II | Fusion protein | Translocation | Deactivation | RNA-mediated interference | Tumor cell lines | Gene expression | 1-Phosphatidylinositol 3-kinase | Signaling | Chorioallantoic membrane | Experimental design | Cell lines | Effectors | Liposarcoma | In vivo methods and tests | Aberration | Cancer | in-vivo | synovial sarcoma | soft-tissue sarcoma | chop | Oncology | refractory ewing sarcoma | cell liposarcoma | receptor | human cancer | pathway | growth | Cancer and Oncology | Cancer och onkologi
Journal Article
Molecular Neurodegeneration, ISSN 1750-1326, 10/2017, Volume 12, Issue 1, pp. 71 - 13
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 06/2019, Volume 38, Issue 23, pp. 4560 - 4573
Journal Article
Oncotarget, ISSN 1949-2553, 2015, Volume 6, Issue 14, pp. 12637 - 12653
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e33208
DDIT3, also known as GADD153 or CHOP, encodes a basic leucine zipper transcription factor of the dimer forming C/EBP family. DDIT3 is known as a key regulator... 
TRANSCRIPTION FACTOR CHOP | IN-VITRO | MYXOID LIPOSARCOMA | INDUCED APOPTOSIS | BIOLOGY | BINDING-PROTEIN | GENE-EXPRESSION | PANCREATIC BETA-CELLS | ENDOPLASMIC-RETICULUM STRESS | AMINO-ACID LIMITATION | C/EBP-HOMOLOGOUS PROTEIN | Transcription Factor CHOP - genetics | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Humans | Cytoplasm - metabolism | Gene Expression Profiling | Green Fluorescent Proteins - genetics | Fibrosarcoma - metabolism | Promoter Regions, Genetic - genetics | Liposarcoma - metabolism | Cell Nucleus - metabolism | Flow Cytometry | Biomarkers, Tumor - metabolism | Real-Time Polymerase Chain Reaction | Fibroblasts - metabolism | Recombinant Proteins - metabolism | Antineoplastic Agents, Hormonal - pharmacology | Liposarcoma - drug therapy | RNA, Messenger - genetics | Cells, Cultured | Liposarcoma - genetics | Recombinant Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion | Blotting, Western | Fibrosarcoma - genetics | Cell Cycle | Cell Nucleus - genetics | Fibroblasts - drug effects | Tamoxifen - pharmacology | Biomarkers, Tumor - genetics | Fibroblasts - cytology | Cell Nucleus - drug effects | Transcription Factor CHOP - metabolism | Cytoplasm - drug effects | Fibrosarcoma - drug therapy | Cell Movement | Genomes | Genetic transcription | DNA binding proteins | Tamoxifen | Genes | Genomics | Cell culture | Transcription factors | Target recognition | Amino acids | Biochemistry | CCAAT/enhancer-binding protein | Leucine | Kinases | Experiments | Proteins | Cell growth | Annotations | Cell cycle | Fibroblasts | Physiology | Localization | Stress response | Deoxyribonucleic acid--DNA | Translocation | Stresses | Cell survival | Cloning | Forming | Gene expression | Stress | Nuclear transport | Leucine zipper proteins | Pathology | DNA microarrays | Ligands | Prostate | Cell migration | CHOP protein | Cellular stress response | Apoptosis | Cancer | cytology | Green Fluorescent Proteins | RNA | Hormonal | Genetic | Fibrosarcoma | Recombinant Proteins | Blotting | Tumor Markers | Western | genetics | Transcription Factor CHOP | pharmacology | Cultured | drug therapy | Promoter Regions | Antineoplastic Agents | drug effects | Messenger | Biological | Cells | Liposarcoma | metabolism | Cancer and Oncology | Cell Nucleus | Cytoplasm | Cancer och onkologi | Deoxyribonucleic acid | DNA
Journal Article
Experimental & molecular medicine, ISSN 1226-3613, 09/2017, Volume 49, Issue 9, pp. e372 - e372
Cadmium (Cd), a major component of cigarette smoke, disrupts the normal functions of airway cells and can lead to the development of various pulmonary diseases... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | UNFOLDED PROTEIN RESPONSE | C/EBP-BETA | ENDOPLASMIC-RETICULUM-STRESS | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | INVOLVEMENT | GENE-EXPRESSION | TOXICITY | COPD | Inflammation - pathology | Transcription Factor CHOP - genetics | Epithelial Cells - metabolism | Respiratory Mucosa - drug effects | Epithelial Cells - drug effects | Humans | Endoplasmic Reticulum - metabolism | Gene Expression Profiling | Gene Knockdown Techniques | Cadmium - adverse effects | Respiratory Mucosa - pathology | Inflammation - metabolism | Endoplasmic Reticulum - drug effects | Inflammation Mediators - metabolism | Homeostasis - drug effects | CCAAT-Enhancer-Binding Proteins - metabolism | Cell Line | Cell Survival - drug effects | Cytokines - metabolism | Endoplasmic Reticulum Stress - drug effects | Inflammation - etiology | Gene Expression Regulation - drug effects | Signal Transduction - drug effects | Respiratory Mucosa - metabolism | Transcription Factor CHOP - metabolism | Cluster Analysis | Oxidative stress | Cadmium | Epithelial cells | Lung diseases | siRNA | Inflammation | CCAAT/enhancer-binding protein | Respiratory tract | Signal transduction | Molecular modelling | Transcription activation | Obstructive lung disease | Cell cycle | Chronic obstructive pulmonary disease | Transduction | Cigarette smoke | Apoptosis | Original
Journal Article
International Journal of Cancer, ISSN 0020-7136, 07/2005, Volume 115, Issue 4, pp. 556 - 560
Journal Article